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1. Nerve endings
: the discovery of the synapse
by Rapport, Richard L
2005, 1st ed., ISBN 0393060195, 240
Book
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2. Full Text 3,4‐Methylenedioxypyrovalerone prevents while methylone enhances methamphetamine‐induced damage to dopamine nerve endings: β‐ketoamphetamine modulation of neurotoxicity by the dopamine transporter
by Anneken, John H and Angoa‐Pérez, Mariana and Kuhn, Donald M
Journal of Neurochemistry, ISSN 0022-3042, 04/2015, Volume 133, Issue 2, pp. 211 - 222
Methylone, 3,4‐methylenedioxypyrovalerone (MDPV), and mephedrone are psychoactive ingredients of ‘bath salts’ and their abuse represents a growing public... 
dopamine transporter | neurotoxicity | neurotoxic amphetamines | dopamine nerve ending | β‐ketoamphetamines | Neurotoxic amphetamines | Dopamine transporter | Neurotoxicity | β-ketoamphetamines | Dopamine nerve ending | LOCOMOTOR STIMULANT | CONDITIONED PLACE PREFERENCE | PHARMACOLOGICAL CHARACTERIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROSCIENCES | BATH SALTS | NUCLEUS-ACCUMBENS | ketoamphetamines | INTRACRANIAL SELF-STIMULATION | MEPHEDRONE 4-METHYLMETHCATHINONE | METHYLENEDIOXYPYROVALERONE MDPV | SYNTHETIC CATHINONES | AMPHETAMINE | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Dopamine Plasma Membrane Transport Proteins - metabolism | Tyrosine 3-Monooxygenase - metabolism | Body Temperature - drug effects | Methamphetamine - toxicity | Mice, Inbred C57BL | Glial Fibrillary Acidic Protein - metabolism | Central Nervous System Stimulants - toxicity | Corpus Striatum - cytology | Pyrrolidines - pharmacology | Dose-Response Relationship, Drug | Neurotoxins - pharmacology | Animals | Nerve Endings - drug effects | Psychotropic Drugs - pharmacology | Corpus Striatum - drug effects | Female | Mice | Methamphetamine - analogs & derivatives | Benzodioxoles - pharmacology | Dopamine - metabolism | Index Medicus
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3. Iconography of sensory nerve endings
by Ábrahám, A. (Ambrus)
1981, ISBN 9630523167, 396
Neural receptors
Book
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4. Full Text Mephedrone, an abused psychoactive component of ‘bath salts’ and methamphetamine congener, does not cause neurotoxicity to dopamine nerve endings of the striatum
by Angoa‐Pérez, Mariana and Kane, Michael J and Francescutti, Dina M and Sykes, Katherine E and Shah, Mrudang M and Mohammed, Abiy M and Thomas, David M and Kuhn, Donald M
Journal of Neurochemistry, ISSN 0022-3042, 03/2012, Volume 120, Issue 6, pp. 1097 - 1107
J. Neurochem. (2012) 120, 1097–1107. Mephedrone (4‐methylmethcathinone) is a β‐ketoamphetamine with close structural analogy to substituted amphetamines and... 
mephedrone | neurotoxicity | methamphetamine | dopamine | β‐ketoamphetamines | microglial activation | Mephedrone | Neurotoxicity | β-ketoamphetamines | Dopamine | Microglial activation | Methamphetamine | SUBSTITUTED AMPHETAMINES | HYPERTHERMIA | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | METHCATHINONE | NEUROSCIENCES | CATHA-EDULIS | NUCLEUS-ACCUMBENS | ss-ketoamphetamines | GLUTAMATE | PHARMACOLOGY | 3,4-METHYLENEDIOXYMETHAMPHETAMINE MDMA | Methamphetamine - pharmacology | Dopamine Plasma Membrane Transport Proteins - metabolism | Tyrosine 3-Monooxygenase - metabolism | Central Nervous System Stimulants - pharmacology | Body Temperature - drug effects | Mice, Inbred C57BL | Corpus Striatum - cytology | Dopaminergic Neurons - cytology | Neuroglia - drug effects | Animals | Nerve Endings - drug effects | Corpus Striatum - drug effects | Behavior, Animal - drug effects | Female | Mice | Methamphetamine - analogs & derivatives | Dopamine - metabolism | Tyrosine | Neurosciences | Bath products | Phenols | Hydroxylases | Intermediate filament proteins | Public health | Neurochemistry | Psychotropic drugs | Brain | Stimulants | Neurons | Toxicity | Nerve endings | Drug abuse | Salts | Congeners | Glial fibrillary acidic protein | Tyrosine 3-monooxygenase | Amphetamine | Neostriatum | Dopamine transporter | Hyperthermia
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5. Full Text Virus-specific CD8+ T cells accumulate near sensory nerve endings in genital skin during subclinical HSV-2 reactivation
by Zhu, Jia and Koelle, David M and Cao, Jianhong and Vazquez, Julio and Meei, Li Huang and Hladik, Florian and Wald, Anna and Corey, Lawrence
Journal of Experimental Medicine, ISSN 0022-1007, 03/2007, Volume 204, Issue 3, pp. 595 - 603
Cytotoxic CD8(+) T cells play a critical role in controlling herpes simplex virus (HSV) infection and reactivation. However, little is known about the... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | HERPES-SIMPLEX-VIRUS | ACYCLOVIR | HIV | LATENCY | IMMUNOLOGY | PROTEINS | TYPE-2 | LESIONS | AUTOFLUORESCENCE | LYMPHOCYTES | Genitalia, Male - innervation | CD8-Positive T-Lymphocytes - pathology | Nerve Endings - immunology | Humans | Middle Aged | Male | Skin Diseases, Viral - virology | Genitalia, Female - innervation | Skin Diseases, Viral - immunology | Cell Movement - immunology | Herpesvirus 2, Human - immunology | Herpes Genitalis - pathology | Nerve Endings - virology | CD8-Positive T-Lymphocytes - virology | Herpes Genitalis - virology | Skin Diseases, Viral - pathology | Adult | Female | CD8-Positive T-Lymphocytes - immunology | Herpes Genitalis - immunology | Virus Activation - immunology | Nerve Endings - pathology
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6. Full Text Sertraline inhibits pre‐synaptic Na+ channel‐mediated responses in hippocampus‐isolated nerve endings
by Aldana, Blanca I and Sitges, María
Journal of Neurochemistry, ISSN 0022-3042, 04/2012, Volume 121, Issue 2, pp. 197 - 205
J. Neurochem. (2012) 121, 197–205. In the present study, a possible sertraline action on cerebral pre‐synaptic Na+ channels was investigated. For this purpose,... 
GABA | veratridine | Glu | sodium channels | neurotransmitter release | 5‐HT | Sodium channels | Neurotransmitter release | Veratridine | 5-HT | Calcium - pharmacology | Sertraline - pharmacology | Presynaptic Terminals - drug effects | Rats, Wistar | gamma-Aminobutyric Acid - metabolism | Neurotransmitter Agents - metabolism | Rats | Male | Hydroxyindoleacetic Acid - metabolism | Sodium Channels - drug effects | Hippocampus - drug effects | Sodium - metabolism | Hippocampus - cytology | Veratridine - pharmacology | Animals | Nerve Endings - drug effects | Serotonin - metabolism | Glutamic Acid - metabolism | Sodium Channel Blockers | Tetrodotoxin - pharmacology | In Vitro Techniques | Neurochemistry | Brain | Neurotransmitters | Neurons | Antidepressants | Amino acids
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7. Full Text Spinal afferent nerve endings in visceral organs: Recent advances
by Spencer, Nick J and Zagorodnyuk, Vladimir and Brookes, Simon J and Hibberd, Tim
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 2016, Volume 311, Issue 6, pp. G1056 - G1063
Spinal afferent neurons play a major role in detection and transduction of painful stimuli from internal (visceral) organs. Recent technical advances have made... 
Nociception | Spinal afferent | Dorsal root ganglia | Pain | Visceral afferent | pain | PHYSIOLOGY | RECTAL MECHANORECEPTORS | dorsal root ganglia | GASTROINTESTINAL-TRACT | nociception | SMALL-INTESTINE | ALPHA-CGRP | GUINEA-PIG ESOPHAGUS | TRANSDUCTION SITES | spinal afferent | INTRAGANGLIONIC LAMINAR ENDINGS | SENSORY NEURONS | SMOOTH-MUSCLE | visceral afferent | GASTROENTEROLOGY & HEPATOLOGY | COLORECTAL DISTENSION | Animals | Visceral Afferents - metabolism | Nerve Endings - physiology | Intestines - innervation | Ganglia, Spinal - physiology | Mice | Nerve Endings - metabolism | Visceral Afferents - physiology | Ganglia, Spinal - metabolism | Nerve endings | Physiological aspects | Afferent pathways | Spine | Viscera
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8. Full Text Vinpocetine and α‐tocopherol prevent the increase in DA and oxidative stress induced by 3‐NPA in striatum isolated nerve endings
by Herrera‐Mundo, Nieves and Sitges, María
Journal of Neurochemistry, ISSN 0022-3042, 01/2013, Volume 124, Issue 2, pp. 233 - 240
Vinpocetine is a neuroprotective drug that exerts beneficial effects on neurological symptoms and cerebrovascular disease. 3‐nitropropionic acid (3‐NPA) is a... 
3‐nitropropionic acid | MAO | VMA | dopamine–quinone adducts | synaptosomes | vinpocetine | 3-nitropropionic acid | dopamine-quinone adducts | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARBAMAZEPINE | RELEASE | NEUROSCIENCES | INHIBITION | DOPAMINE | CHAIN ACTIVITY | BRAIN | PARKINSONS-DISEASE | Nerve Endings - chemistry | Propionates - pharmacology | Oxidative Stress - physiology | Dopamine - biosynthesis | Rats | Antioxidants - pharmacology | Anticonvulsants - pharmacology | Corpus Striatum - chemistry | Neural Inhibition - physiology | Animals | Neuroprotective Agents - pharmacology | Vinca Alkaloids - pharmacology | Nerve Endings - drug effects | Corpus Striatum - drug effects | Nitro Compounds - pharmacology | Oxidative Stress - drug effects | alpha-Tocopherol - pharmacology | Neural Inhibition - drug effects | Dopamine - metabolism | Drugs | Nerve endings | Brain | Enzymes | Oxidative stress | Reactive oxygen species | Dopamine | Free radicals | Fluorescence | Lipid peroxidation | Neuroprotective agents | Amine oxidase (flavin-containing) | Antioxidants | Vitamin E | 3-Nitropropionic acid | Neostriatum | Synaptosomes | Electron transport
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9. Full Text Degeneration of proprioceptive sensory nerve endings in mice harboring amyotrophic lateral sclerosis–causing mutations
by Vaughan, Sydney K and Kemp, Zachary and Hatzipetros, Theo and Vieira, Fernando and Valdez, Gregorio
Journal of Comparative Neurology, ISSN 0021-9967, 12/2015, Volume 523, Issue 17, pp. 2477 - 2494
ABSTRACT Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease that primarily targets the motor system. Although much is known about the effects... 
SOD1g93A | Amyotrophic Lateral Sclerosis (ALS) | TDP43A315T | Proprioceptive sensory neuron | Neuromuscular junction (NMJ) | Motor neuron | VGLUT1 | VAChT | TDP43 | SOD1 | LIFE-SPAN | ALS | SEMAPHORIN 3A | NEUROSCIENCES | SYNAPTOTAGMIN-I | ZOOLOGY | MOTOR-NEURON DEGENERATION | DISTAL AXONOPATHY | MOUSE MODEL | SCHWANN-CELLS | SOD1(g93A) | TDP43(A315T) | SUPEROXIDE-DISMUTASE | DISEASE PROGRESSION | Superoxide Dismutase - genetics | Spinal Cord - metabolism | Age Factors | Humans | Nerve Degeneration - genetics | Parvalbumins - metabolism | Muscle, Skeletal - metabolism | Synapses - pathology | Sympathetic Nervous System - metabolism | Motor Neurons - pathology | Nerve Degeneration - metabolism | Synapses - metabolism | Parvalbumins - genetics | Sensory Receptor Cells - metabolism | Sympathetic Nervous System - pathology | Disease Models, Animal | Sensory Receptor Cells - pathology | Cholera Toxin - metabolism | Gene Expression Regulation - genetics | Mice, Transgenic | DNA-Binding Proteins - genetics | Mutation - genetics | Nerve Degeneration - pathology | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Animals | Mice | Muscle, Skeletal - pathology | Physiological aspects | Amyotrophic lateral sclerosis | Nervous system diseases | Genetic aspects | Cholera toxin
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10. Full Text Morphology of P2X3‐immunoreactive nerve endings in the rat tracheal mucosa
by Yamamoto, Yoshio and Nakamuta, Nobuaki
Journal of Comparative Neurology, ISSN 0021-9967, 02/2018, Volume 526, Issue 3, pp. 550 - 566
Nerve endings with immunoreactivity for the P2X3 purinoreceptor (P2X3) in the rat tracheal mucosa were examined by immunohistochemistry of whole‐mount... 
RRID: AB_2301751 | trachea | RRID: AB_2299063 | RRID: AB_1587626 | purinoreceptor | sensory nerve endings | respiration | RRID: AB_2290729 | RRID: AB_11212910 | RRID: AB_2187832 | RRID: AB_2157930 | RRID: AB_322417 | RAPIDLY ADAPTING RECEPTORS | RESPIRATORY-TRACT | NEUROEPITHELIAL BODIES | NEUROSCIENCES | SENSORY NEURONS | AUDITORY BRAIN-STEM | ZOOLOGY | PULMONARY RECEPTORS | IRRITANT RECEPTORS | P2X3 RECEPTOR | CAROTID-BODY | ATP RELEASE | Immunohistochemistry | Nerve endings | Presynapse | Epithelial cells | Mucosa | Lamina propria | Confocal | Epithelium | Laser microscopy | Calcitonin gene-related peptide | Axons | Receptors | Rodents | Nodose ganglion | SNAP-25 protein | Terminals | Trachea
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11. Full Text Tear fluid hyperosmolality increases nerve impulse activity of cold thermoreceptor endings of the cornea
by Parra, Andres and Gonzalez-Gonzalez, Omar and Gallar, Juana and Belmonte, Carlos
Pain, ISSN 0304-3959, 2014, Volume 155, Issue 8, pp. 1481 - 1491
Dry eye disease (DED) is a multifactorial disorder affecting the composition and volume of tears. DED causes ocular surface dryness, cooling, and... 
Polymodal nociceptors | Cornea | Cold thermoreceptors | Dry eye | Tear osmolality | Hyperosmotic stress | OCULAR SURFACE | GUINEA-PIG CORNEA | DRY-EYE DISEASE | FILM LIPID LAYER | NEUROSCIENCES | SENSORY RECEPTORS | CLINICAL NEUROLOGY | EVAPORATION | IN-VITRO | CATS CORNEA | ION-CHANNEL | NEURONS | ANESTHESIOLOGY | Cold Temperature | Dry Eye Syndromes - physiopathology | Mice, Inbred C57BL | Male | Mice, Inbred ICR | Action Potentials - physiology | Thermoreceptors - physiology | Animals | Osmolar Concentration | Nerve Endings - physiology | Cornea - innervation | Cornea - physiopathology | Mice | Tears - physiology
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