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Development (Cambridge), ISSN 0950-1991, 11/2016, Volume 143, Issue 21, pp. 4038 - 4052
Microcephaly and medulloblastoma may both result from mutations that compromise genomic stability. We report that ATR, which is mutated in the microcephalic... 
Mouse | DNA damage | Medulloblastoma | Chromosomal instability | Microcephaly | Neural progenitor | ATR | INDUCED APOPTOSIS | PROTEIN-KINASE | DNA-DAMAGE | DEVELOPMENTAL BIOLOGY | CELL-DEATH | NERVOUS-SYSTEM DEVELOPMENT | BREAST-CANCER | GENOME INTEGRITY | MITOTIC ARREST | MICE | ATAXIA-TELANGIECTASIA | Apoptosis - drug effects | Cerebellum - drug effects | Apoptosis - genetics | Male | Developmental Disabilities - genetics | Cerebellum - abnormalities | Neurogenesis - genetics | Developmental Disabilities - pathology | Cell Transformation, Neoplastic - genetics | Gene Deletion | Medulloblastoma - pathology | Neurons - physiology | Ataxia Telangiectasia Mutated Proteins - antagonists & inhibitors | Female | Neurogenesis - drug effects | Cerebellar Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Medulloblastoma - genetics | Nervous System Malformations - genetics | Neurons - drug effects | Animals, Newborn | Nervous System Malformations - pathology | Cerebellum - metabolism | Mice, Inbred C57BL | Gene Expression Regulation, Developmental - drug effects | Chromosomal Instability - drug effects | Cerebellar Neoplasms - genetics | Ataxia Telangiectasia Mutated Proteins - physiology | Cerebellum - pathology | Mice, Knockout | Isoxazoles - pharmacology | Chromosomal Instability - genetics | Animals | Cerebellum - growth & development | Mice | Cell Transformation, Neoplastic - drug effects | Ataxia Telangiectasia Mutated Proteins - genetics | Pyrazines - pharmacology | Index Medicus | 205
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2002, Volume 22, Issue 19, pp. 8563 - 8573
Many ligand/receptor families are known to contribute to axonal growth and targeting. Thus far, there have been no reports implicating Wnts and Frizzleds in... 
Forebrain | Axonal growth | Fiber tracts | Wnt signaling | Frizzled | Mouse brain development | WNT SIGNALING PATHWAY | NERVOUS-SYSTEM | TISSUE POLARITY | mouse brain development | INT-1 PROTOONCOGENE | NEUROSCIENCES | DROSOPHILA | CELL POLARITY | TARGETED DISRUPTION | axonal growth | frizzled | fiber tracts | TRANSMEMBRANE DOMAINS | forebrain | NEURAL CREST | EXPRESSION | Abnormalities, Multiple - pathology | Central Nervous System - metabolism | Prosencephalon - abnormalities | Central Nervous System - abnormalities | Central Nervous System - pathology | Neural Pathways - pathology | Cell Movement - physiology | Nerve Fibers - physiology | Cerebral Ventricles - embryology | Prosencephalon - metabolism | Nervous System Malformations - genetics | Abnormalities, Multiple - genetics | Genes, Reporter | Prosencephalon - pathology | Frizzled Receptors | In Situ Nick-End Labeling | Nerve Fibers - pathology | Gene Targeting | Nervous System Malformations - pathology | Mice, Inbred C57BL | Receptors, G-Protein-Coupled | Receptors, Cell Surface - metabolism | Mice, Transgenic | Cell Division - physiology | Respiratory Insufficiency - pathology | Phenotype | Animals | Neural Pathways - abnormalities | Respiratory Insufficiency - genetics | Axons - pathology | Receptors, Cell Surface - deficiency | Mice | Cerebral Ventricles - cytology | Apoptosis | Lac Operon | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
PLoS Biology, ISSN 1544-9173, 06/2018, Volume 16, Issue 6, pp. e2004880 - e2004880
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2010, Volume 41, Issue 2, pp. 469 - 480
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 10/2011, Volume 89, Issue 10, pp. 1575 - 1585
Maternal infection during pregnancy is an environmental risk factor for the development of severe brain disorders in offspring, including schizophrenia and... 
maternal immune challenge | corticogenesis | schizophrenia | polyriboinosinic‐polyribocytidylic acid (Poly I:C) | autism | Schizophrenia | Autism | Corticogenesis | Maternal immune challenge | Polyriboinosinic-polyribocytidylic acid (Poly I:C) | MIGRATION | NEOCORTICAL NEURONS | ACTIVATION | NEURONAL DIFFERENTIATION | polyriboinosinic-polyribocytidylic acid (Poly I:C) | NEUROSCIENCES | INTERMEDIATE PROGENITOR CELLS | VISUAL-CORTEX | PRECURSOR CELLS | EXPRESSION | BRAIN | Nervous System Malformations - immunology | Nervous System Malformations - pathology | Cerebral Cortex - pathology | Neurogenesis - immunology | Cerebral Cortex - virology | Male | Autoimmune Diseases of the Nervous System - pathology | Poly I-C - toxicity | Nervous System Malformations - virology | Pregnancy | Autoimmune Diseases of the Nervous System - immunology | Animals | Prenatal Exposure Delayed Effects - immunology | Prenatal Exposure Delayed Effects - virology | Cerebral Cortex - immunology | Female | Animals, Outbred Strains | Mice | Prenatal Exposure Delayed Effects - pathology | Autoimmune Diseases of the Nervous System - chemically induced | Brain | Transcription factors | Mental disorders | Synaptogenesis | Cognitive ability | Cortex | Nervous system | Poly (I:C) | Gene expression | Neurodevelopmental disorders | Intrauterine exposure | Neurogenesis | Risk factors | Infection | Pax6 protein | Progeny | Neural stem cells | Prenatal experience | Immune system | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2003, Volume 23, Issue 12, pp. 5123 - 5130
The chemotactic factors directing interneuron migration during cerebrocortical development are essentially unknown. Here we identify the CXC chemokine receptor... 
Stromal cell-derived factor-1 | Cerebral cortex | Reelin | Immunocytochemistry | Development | CXCR4 | Cajal-Retzius cell | CXC chemokine | In situ hybridization | Hippocampus | Interneuron migration | NEURONAL MIGRATION | development | hippocampus | ADULT RATS | MESSENGER-RNA EXPRESSION | CHEMOKINE RECEPTOR CXCR4 | immunocytochemistry | DEVELOPING CEREBRAL-CORTEX | NEUROSCIENCES | interneuron migration | cerebral cortex | CORTICAL NEUROGENESIS | GANGLIONIC EMINENCE | CELL-DERIVED FACTOR-1 | reelin | stromal cell-derived factor-1 | DENTATE GYRUS | CAJAL-RETZIUS CELLS | in situ hybridization | Immunohistochemistry | Extracellular Matrix Proteins - biosynthesis | Rats, Wistar | Cell Count | Stem Cells - cytology | Stem Cells - metabolism | Cell Movement - physiology | Neocortex - metabolism | RNA, Messenger - biosynthesis | In Situ Hybridization | Gene Expression Regulation, Developmental | Receptors, CXCR4 - deficiency | Receptors, CXCR4 - genetics | Interneurons - cytology | Choristoma - genetics | Serine Endopeptidases | Nervous System Malformations - genetics | Chemokine CXCL12 | Nervous System Malformations - pathology | Mice, Inbred C57BL | Rats | Nerve Tissue Proteins | Chemokines, CXC - genetics | Chemokines, CXC - deficiency | Mice, Knockout | Cell Movement - drug effects | Receptors, CXCR4 - physiology | Animals | Interneurons - metabolism | Cell Adhesion Molecules, Neuronal - biosynthesis | Neocortex - abnormalities | Chemokines, CXC - physiology | Neocortex - cytology | Signal Transduction - physiology | Mice | Choristoma - pathology | Index Medicus
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 07/2012, Volume 90, Issue 7, pp. 1403 - 1412
Journal Article