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PloS one, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e96239
...-Oncology, Departments of Neurology, Microbiology, and Biochemistry and Molecular Genetics, University of Virginia Health System, Charlottesville, Virginia, United... 
GLIOMA-CELLS | CANCER-CELLS | GROWTH-FACTOR RECEPTOR | NERVOUS-SYSTEM | IN-VITRO | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | AKT PATHWAY | SPHEROID CULTURES | PRECURSOR CELLS | BRAIN-TUMORS | Neoplastic Stem Cells - cytology | Caspase 9 - genetics | Caspase 9 - metabolism | Humans | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | Glycoproteins - metabolism | Peptides - genetics | 3' Untranslated Regions - genetics | Cell Survival - genetics | MicroRNAs - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Flow Cytometry | Peptides - metabolism | Bcl-2-Like Protein 11 | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Caspase 3 - genetics | Apoptosis Regulatory Proteins - genetics | Glioblastoma - metabolism | Membrane Proteins - metabolism | Cell Survival - physiology | Glycoproteins - genetics | Proto-Oncogene Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | Proto-Oncogene Proteins - genetics | AC133 Antigen | Apoptosis Regulatory Proteins - metabolism | Cell Line, Tumor | MicroRNAs - genetics | DNA microarrays | MicroRNA | Analysis | Luciferase | Stem cells | Glioblastoma multiforme | Apoptosis | Brain | Flow cytometry | Brain tumors | Brain cancer | Glioblastoma | Fluorescence | Oncology | Biochemistry | Neurosurgery | Caspase-3 | Cancer therapies | Genetics | miRNA | Cell survival | Caspase | siRNA | Ribonucleic acid--RNA | Survival | Neurology | Brain research | Mutagenesis | 3' Untranslated regions | Cell number | MicroRNAs | Caspase-9 | Neural stem cells | Annexin V | BIM protein | Cancer | Tumors | RNA | Ribonucleic acid
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2005, Volume 8, Issue 6, pp. 797 - 804
Journal Article
Oncogene, ISSN 1476-5594, 2018, Volume 37, Issue 31, pp. 4313 - 4333
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 340 - 347
Neurofibromatosis type 1 (NF1) patients develop benign neurofibromas and malignant peripheral nerve sheath tumors (MPNST). These incurable peripheral nerve... 
MEDICINE, RESEARCH & EXPERIMENTAL | SIGNALING PATHWAYS | PLEXIFORM NEUROFIBROMAS | GENOMIC ANALYSES | THERAPEUTIC TARGET | NF1 | SCHWANN-CELLS | HYPERACTIVE RAS | CANCER | NERVE SHEATH TUMORS | PREVIOUSLY TREATED PATIENTS | Neoplasm Transplantation | Mitogen-Activated Protein Kinase Kinases - genetics | Diphenylamine - pharmacology | Humans | Child, Preschool | Male | Transplantation, Heterologous | Extracellular Signal-Regulated MAP Kinases - metabolism | raf Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Oncogene Protein p21(ras) - metabolism | Diphenylamine - analogs & derivatives | Mitogen-Activated Protein Kinase Kinases - metabolism | Peripheral Nervous System Neoplasms - enzymology | Mice, Mutant Strains | Female | Benzamides - pharmacology | Neurofibromatosis 1 - genetics | Peripheral Nervous System Neoplasms - pathology | raf Kinases - genetics | Child | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Peripheral Nervous System Neoplasms - drug therapy | Transcriptome - drug effects | Transcriptome - genetics | Xenograft Model Antitumor Assays | Neurofibromatosis 1 - enzymology | Neurofibromatosis 1 - pathology | Animals | Neurofibromatosis 1 - drug therapy | Mice | Peripheral Nervous System Neoplasms - genetics | Oncogene Protein p21(ras) - genetics | Physiological aspects | Causes of | Neural stimulation | Enzyme inhibitors | Properties | Neurofibromatosis
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2013, Volume 14, Issue 10, pp. 20443 - 20458
Gallic acid is one of the major flavonoids found in plants. It acts as an antioxidant, and seems to have anti-inflammatory, anti-viral, and anti-cancer... 
Dct | Gallic acid | Wnt/β-catenin | MEK/ERK | TRP1 | Melanogenesis | Tyrosinase | Microphthalmia-associated transcription factor (MITF) | PI3K/Akt | CARCINOMA CELLS | catenin | melanogenesis | MITF | Akt | MICROPHTHALMIA GENE-PRODUCT | tyrosinase | gallic acid | HUMAN MELANOCYTES | CHEMISTRY, MULTIDISCIPLINARY | MEK | PHOSPHATIDYLINOSITOL-3-KINASE PATHWAY | PI3K | microphthalmia-associated transcription factor (MITF) | MELANOMA-CELLS | Wnt | NEURAL CREST | TRANSCRIPTION FACTOR | CAMP | ERK | Gallic Acid - pharmacology | Melanoma, Experimental - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Wnt Proteins - metabolism | Melanins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Melanins - biosynthesis | Monophenol Monooxygenase - metabolism | MAP Kinase Signaling System - genetics | Wnt Proteins - genetics | Monophenol Monooxygenase - genetics | Intramolecular Oxidoreductases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Microphthalmia-Associated Transcription Factor - metabolism | Oxidoreductases - metabolism | Oxidoreductases - genetics | Signal Transduction - genetics | Melanins - genetics | Down-Regulation - drug effects | beta Catenin - metabolism | Down-Regulation - genetics | beta Catenin - genetics | Phosphatidylinositol 3-Kinases - genetics | Animals | MAP Kinase Signaling System - drug effects | Melanoma, Experimental - genetics | Intramolecular Oxidoreductases - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Mice | Microphthalmia-Associated Transcription Factor - genetics
Journal Article
Journal Article
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 09/2016, Volume 36, Issue 36, pp. 9505 - 9511
Synchronous optogenetic activation of striatal cholinergic interneurons ex vivo produces a disynaptic inhibition of spiny projection neurons composed of biophysically distinct GABAAfast and GABAAslow components... 
Striatum | Neurogliaform | Optogenetics | Cholinergic interneuron | GABA-A | Cholinergic Agents - pharmacology | Channelrhodopsins | GABAergic Neurons - physiology | Nerve Net - drug effects | Corpus Striatum - cytology | Excitatory Postsynaptic Potentials - drug effects | Nerve Net - physiology | Choline O-Acetyltransferase - metabolism | Cholinergic Neurons - physiology | DNA-Binding Proteins - metabolism | Neuropeptide Y - metabolism | Neural Inhibition - physiology | Inhibitory Postsynaptic Potentials - genetics | Excitatory Postsynaptic Potentials - genetics | Choline O-Acetyltransferase - genetics | Nerve Growth Factor - metabolism | Receptors, Serotonin, 5-HT3 - genetics | Bacterial Proteins - genetics | GABAergic Neurons - drug effects | Mice, Transgenic | Cholinergic Neurons - drug effects | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Patch-Clamp Techniques | Animals | Receptors, Serotonin, 5-HT3 - metabolism | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | In Vitro Techniques | Neural Inhibition - drug effects | Inhibitory Postsynaptic Potentials - drug effects | Luminescent Proteins - metabolism | GABA-Aslow | neurogliaform | striatum | cholinergic interneuron | optogenetics
Journal Article
Neurobiology of disease, ISSN 0969-9961, 2014, Volume 69, pp. 65 - 75
Journal Article
PloS one, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0164752
.... Suppression of SNAI2 suffices to up-regulate several cancer stem genes. Although unrelated to the metastatic ability, SNAI2 inhibition... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | NEURAL CREST DEVELOPMENT | APOPTOSIS | METASTASIS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | STEM-CELL | SLUG SNAI2 | E-CADHERIN | EXPRESSION | RNA, Small Interfering - genetics | Transcription, Genetic - drug effects | Neoplastic Stem Cells - drug effects | Drug Resistance, Multiple - drug effects | Humans | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Neoplasm Metastasis | Snail Family Transcription Factors - genetics | ATP-Binding Cassette Transporters - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Liver Neoplasms - pathology | Cell Proliferation - genetics | Drug Resistance, Multiple - genetics | Gene Silencing | Up-Regulation - genetics | Verapamil - pharmacology | Up-Regulation - drug effects | Animals | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Snail Family Transcription Factors - deficiency | Cell Proliferation - drug effects | Mice | Transcription, Genetic - genetics | ATP-Binding Cassette Transporters - antagonists & inhibitors | Drug resistance in microorganisms | Genetic aspects | Metastasis | Hepatoma | Genetic transcription | Stem cells | Mesenchyme | Laboratories | Epithelial cells | Liver | Genes | Genomics | Hepatocellular carcinoma | Drug resistance | Metastases | Liver cancer | Education | Hepatology | Tumorigenesis | Inhibition | Efflux | Phenotypes | Multidrug resistance | Breast cancer | Gene expression | Medicine | Medical prognosis | Tumor suppressor genes | Verapamil | Snail protein | Prostate | Transporter | ABC transporter | Tumors | Cancer | Apoptosis
Journal Article