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PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e60529
Background: Pancreatic neuritis is a histopathological hallmark of pancreatic neuropathy and correlates to abdominal neuropathic pain sensation in pancreatic... 
INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INNERVATION | DEGRANULATION | SUBSTANCE-P | EXPRESSION | CORRELATE | IRRITABLE-BOWEL-SYNDROME | NERVE GROWTH-FACTOR | HYPERSENSITIVITY | PLASTICITY | Neuralgia - complications | Adenocarcinoma - pathology | Humans | Middle Aged | Receptor, PAR-1 - analysis | Male | Neuritis - pathology | Neuralgia - pathology | T-Lymphocytes, Cytotoxic - pathology | Neuralgia - immunology | Pancreas - innervation | Female | Macrophages - immunology | Pancreatitis, Chronic - immunology | T-Lymphocytes, Cytotoxic - immunology | Mast Cells - immunology | Macrophages - pathology | Adenocarcinoma - immunology | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreas - immunology | Adenocarcinoma - complications | Pancreatitis, Chronic - pathology | Neuritis - complications | Neuritis - immunology | Pancreatitis, Chronic - complications | Mast Cells - pathology | Pancreatic Neoplasms - immunology | Receptor, PAR-2 - analysis | Aged | Pancreatic Neoplasms - complications | Proteases | Lymphocytes | Pancreatic cancer | Nerves | Pancreatitis | Inflammation | Macrophages | Adenocarcinoma | CD8 antigen | Helper cells | Cytotoxicity | Lymphocytes T | Colorimetry | Neuropathy | CD45 antigen | Ethics | Enteric nervous system | Pain | Rodents | Surgery | Neuritis | Elastase | Lesions | Pancreas | Growth factors | Pain perception | Leukocytes (eosinophilic) | Hypersensitivity | Patients | CD4 antigen | Chymase | Beta cells | White blood cells | CD20 antigen | Mast cells | Immunoreactivity | Tumors | Eosinophils
Journal Article
European Spine Journal, ISSN 0940-6719, 12/2009, Volume 18, Issue 12, pp. 1978 - 1985
Altered dorsal root ganglion (DRG) function is associated with neuropathic pain following spinal nerve injury. However, compression of the cauda equina and... 
Tumor necrosis factor alpha | Surgical Orthopedics | Medicine & Public Health | Neurosurgery | Dorsal root ganglion | Neuropathic pain | Apoptosis | NUCLEUS PULPOSUS | SELECTIVE-INHIBITION | MECHANICAL ALLODYNIA | NECROSIS-FACTOR-ALPHA | RAT SCIATIC-NERVE | MOTOR DYSFUNCTION | CLINICAL NEUROLOGY | DISC HERNIATION | GANGLION APOPTOSIS | TNF-ALPHA | ORTHOPEDICS | CHRONIC-CONSTRICTION-INJURY | Tumor Necrosis Factor-alpha - metabolism | Pain Threshold - physiology | Spondylosis - pathology | Spinal Nerves - pathology | Caspase 3 - metabolism | Nerve Degeneration - physiopathology | Ganglia, Spinal - physiopathology | Peripheral Nervous System Diseases - physiopathology | Neuralgia - pathology | Spondylosis - physiopathology | Hyperalgesia - pathology | Radiculopathy - pathology | Low Back Pain - physiopathology | Spinal Nerve Roots - physiopathology | Low Back Pain - etiology | Sensory Receptor Cells - physiology | Neuralgia - etiology | Radiculopathy - physiopathology | Female | Peripheral Nervous System Diseases - pathology | Neuralgia - physiopathology | Low Back Pain - pathology | Lumbar Vertebrae - physiopathology | Disease Models, Animal | Biomarkers - metabolism | Peripheral Nervous System Diseases - etiology | Spinal Nerves - physiopathology | Sensory Receptor Cells - pathology | Biomarkers - analysis | Rats | Behavior, Animal - physiology | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Hyperalgesia - physiopathology | Tumor Necrosis Factor-alpha - analysis | Animals | Ganglia, Spinal - pathology | Caspase 3 - analysis | Hyperalgesia - etiology | Lumbar Vertebrae - pathology | Apoptosis - physiology | Spinal Nerve Roots - pathology | Nerve Degeneration - etiology | Pain Measurement | Medical colleges | Cell research | Comparative analysis | Neurons | Tumors | Original
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, p. 42304
Multiple sclerosis (MS)-induced neuropathic pain deteriorates quality of life in patients but is often refractory to treatment. In experimental autoimmune... 
MYELIN OLIGODENDROCYTE GLYCOPROTEIN | NERVOUS-SYSTEM | MULTIPLE-SCLEROSIS | PRIMARY AFFERENT NEURONS | MULTIDISCIPLINARY SCIENCES | MUSCLE | DORSAL-ROOT GANGLIA | SENSING ION CHANNELS | SPINAL-CORD | ANIMAL-MODEL | SUBSTANCE-P | Analgesics - pharmacology | Neuralgia - complications | Pregabalin - therapeutic use | Demyelinating Diseases - complications | Encephalomyelitis, Autoimmune, Experimental - immunology | Motor Activity - drug effects | Neuralgia - pathology | Gray Matter - drug effects | Hyperalgesia - pathology | Myelin Sheath - metabolism | Peripheral Nervous System - pathology | Peptides - metabolism | Microglia - pathology | Neuralgia - physiopathology | Demyelinating Diseases - pathology | Lumbar Vertebrae - physiopathology | Analgesics - therapeutic use | Demyelinating Diseases - physiopathology | Encephalomyelitis, Autoimmune, Experimental - physiopathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Sensory Receptor Cells - pathology | Microglia - drug effects | Hyperalgesia - complications | Pregabalin - pharmacology | Peripheral Nervous System - physiopathology | Lumbar Vertebrae - drug effects | Neuralgia - drug therapy | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Encephalomyelitis, Autoimmune, Experimental - complications | Ganglia, Spinal - pathology | Gray Matter - pathology | Peripheral Nervous System - injuries | Lumbar Vertebrae - pathology | Ganglia, Spinal - drug effects | Acid Sensing Ion Channels - metabolism | Ganglia, Spinal - metabolism | Sodium channels | Spinal cord | Multiple sclerosis | Central nervous system | Acidity | Neuropathy | Peripheral neuropathy | Pertussis toxin | Experimental allergic encephalomyelitis | Quality of life | Pertussis | Side effects | Pain | Analgesics | Rodents | Ion channels | Acidosis
Journal Article
Molecular pain, ISSN 1744-8069, 2016, Volume 12, p. 174480691664678
Background Chronic pain is often accompanied by short-term memory deficit and depression. Currently, it is believed that short-term memory deficit and... 
short-term memory deficit | peripheral nerve injury | depression | microglia | interleukin-1β | Neuropathic pain | MIRROR-IMAGE PAIN | NECROSIS-FACTOR-ALPHA | BLOOD-BRAIN-BARRIER | SRC-FAMILY KINASES | SYNAPTIC PLASTICITY | NEUROSCIENCES | SPINAL DORSAL-HORN | interleukin-1 beta | LONG-TERM POTENTIATION | GROWTH-FACTOR | HIPPOCAMPAL NEUROGENESIS | WORKING-MEMORY | Microglia - metabolism | Sciatic Nerve - injuries | Injections, Intravenous | Depression - pathology | Neuralgia - metabolism | Memory Disorders - metabolism | Male | Neuralgia - pathology | Hyperalgesia - pathology | Interleukin-1beta - blood | Behavior, Animal | Depression - etiology | Depression - metabolism | Time Factors | Interleukin-1beta - metabolism | Peripheral Nerve Injuries - metabolism | Receptors, Interleukin-1 - deficiency | Gene Deletion | Neuralgia - etiology | Microglia - pathology | Sciatic Nerve - drug effects | Memory Disorders - pathology | Hyperalgesia - metabolism | Microglia - drug effects | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | Rats | Antibodies, Neutralizing - pharmacology | Rats, Sprague-Dawley | Receptors, Interleukin-1 - metabolism | Peripheral Nerve Injuries - etiology | Spinal Cord Dorsal Horn - drug effects | Animals | Memory Disorders - etiology | Spinal Cord Dorsal Horn - pathology | Signal Transduction - drug effects
Journal Article
Journal Article
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