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1. Full Text Hepatitis E virus infection beyond the liver?
by Pischke, S and Hartl, J and Pas, SD and Lohse, AW and Jacobs, BC and van der Eijk, AA
Journal of Hepatology, ISSN 0168-8278, 2016, Volume 66, Issue 5, pp. 1082 - 1095
Abstract Hepatitis E virus (HEV) infections are not limited to the liver but may also affect other organs. Several diseases, including Guillain-Barré syndrome,... 
Gastroenterology and Hepatology | HEV | Antiviral agents | Hepatitis E | Extrahepatic manifestations | Ribavirin | PSYCHIATRIC-PATIENTS | GUILLAIN-BARRE-SYNDROME | CULTURED-CELLS | ORGAN TRANSPLANT RECIPIENTS | NEURALGIC AMYOTROPHY | ACUTE VIRAL-HEPATITIS | RENAL-FAILURE | ACUTE-PANCREATITIS | 7-YEAR-OLD BOY | GASTROENTEROLOGY & HEPATOLOGY | Animals | Thrombocytopenia - etiology | Pancreatitis - etiology | Humans | Guillain-Barre Syndrome - etiology | Hepatitis, Autoimmune - etiology | Monoclonal Gammopathy of Undetermined Significance - etiology | Brachial Plexus Neuritis - etiology | Kidney Diseases - etiology | Mental Disorders - etiology | Hepatitis E - complications | Guillain-Barre syndrome | Analysis | Liver | Thyroid diseases | Pancreatitis | Meningitis | Lymphomas | Kidney diseases | Health aspects
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2. Full Text Neurological manifestations of dengue infection
by Li, Guo-Hong and Ning, Zhi-Jie and Liu, Yi-Ming and Li, Xiao-Hong
Frontiers in Cellular and Infection Microbiology, ISSN 2235-2988, 10/2017, Volume 7, p. 449
Dengue counts among the most commonly encountered arboviral diseases, representing the fastest spreading tropical illness in the world. It is prevalent in 128... 
Neuropathogenesis | Prevention | Treatment | Dengue | Neurological complications | In children | Manifestations | manifestations | treatment | neurological complications | EPIDURAL HEMATOMA | GUILLAIN-BARRE-SYNDROME | MICROBIOLOGY | IMMUNOLOGY | neuropathogenesis | dengue | in children | UNUSUAL COMPLICATION | HEMORRHAGIC-FEVER | ACUTE DISSEMINATED ENCEPHALOMYELITIS | HYPOKALEMIC PARALYSIS | JAPANESE ENCEPHALITIS-VIRUS | CENTRAL-NERVOUS-SYSTEM | TRANSVERSE MYELITIS | ISCHEMIC-STROKE | prevention | Stroke - virology | Encephalitis, Japanese - virology | Myositis - etiology | Brain Diseases - virology | Neuropathology | Cerebellar Diseases - etiology | Humans | Meningitis - etiology | Meningitis - virology | Dengue Virus - pathogenicity | Ophthalmic Nerve - virology | Neuritis - virology | Brain Diseases - etiology | Encephalitis, Japanese - etiology | Child | Central Nervous System - virology | Cerebellar Diseases - virology | Neuritis - etiology | Peripheral Nervous System Diseases - etiology | Dengue - virology | Nervous System Diseases - therapy | Peripheral Nervous System Diseases - virology | Encephalomyelitis, Acute Disseminated - virology | Dengue - complications | Nervous System Diseases - prevention & control | Stroke - etiology | Myositis - virology | Guillain-Barre Syndrome - etiology | Nervous System Diseases - etiology | Guillain-Barre Syndrome - virology | Nervous System Diseases - virology | Encephalomyelitis, Acute Disseminated - etiology | Research | Children | Health aspects | Risk factors
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3. Full Text Inflammation as a causative factor in the aetiology of Parkinson's disease
by Whitton, P S
British Journal of Pharmacology, ISSN 0007-1188, 04/2007, Volume 150, Issue 8, pp. 963 - 976
Parkinson's disease (PD) is a progressive neurodegenerative disorder affecting mainly the elderly, although a small proportion of PD patients develop the... 
inflammation | Parkinson's disease | neuroinflamation | Inflammation | Neuroinflamation | INTERCELLULAR-ADHESION MOLECULE-1 | NITRIC-OXIDE SYNTHASE | 1-METHYL-4-PHENYL-1,2,3,6-TETRAHYDROPYRIDINE MOUSE MODEL | SYNERGISTIC DOPAMINERGIC NEUROTOXICITY | BLOOD-BRAIN-BARRIER | TUMOR-NECROSIS-FACTOR | P55 TNF-RECEPTOR | IN-VITRO MODEL | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | LIPOPOLYSACCHARIDE-INDUCED NEUROTOXICITY | Neuritis - metabolism | Microglia - metabolism | Neurons - pathology | Reactive Oxygen Species - metabolism | Complement Activation | Substantia Nigra - pathology | Humans | Lipopolysaccharides - metabolism | Neuritis - pathology | Parkinson Disease - drug therapy | Substantia Nigra - metabolism | Antiparkinson Agents - therapeutic use | Anti-Inflammatory Agents - therapeutic use | Granulocytes - pathology | Neurons - metabolism | Parkinson Disease - metabolism | Granulocytes - metabolism | Dopamine - metabolism | Disease Models, Animal | Parkinson Disease - pathology | Neuritis - drug therapy | Blood-Brain Barrier - metabolism | Blood-Brain Barrier - pathology | Animals | Neuritis - complications | Nitric Oxide - metabolism | Cytokines - biosynthesis | Parkinson Disease - etiology | Review
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4. Full Text Different etiologies and prognoses of optic neuritis in demyelinating diseases
by Akaishi, Tetsuya and Nakashima, Ichiro and Takeshita, Takayuki and Kaneko, Kimihiko and Mugikura, Shunji and Sato, Douglas Kazutoshi and Takahashi, Toshiyuki and Nakazawa, Toru and Aoki, Masashi and Fujihara, Kazuo
Journal of Neuroimmunology, ISSN 0165-5728, 2016, Volume 299, pp. 152 - 157
Abstract We compared the clinical features of optic neuritis (ON) that are frequently observed in various central nervous system demyelinating diseases,... 
Neurology | Allergy and Immunology | Visual prognosis | Multiple sclerosis | Neuromyelitis optica spectrum disorder | Optic MRI | Anti-MOG autoantibody | Optic neuritis | VISUAL-ACUITY | MRI | ANTIBODY | IMMUNOLOGY | NEUROSCIENCES | MULTIPLE-SCLEROSIS | NEUROMYELITIS-OPTICA | Demyelinating Diseases - diagnostic imaging | Demyelinating Diseases - blood | Prognosis | Cross-Sectional Studies | Follow-Up Studies | Humans | Middle Aged | Optic Neuritis - diagnostic imaging | Demyelinating Diseases - complications | Male | Vision Disorders - diagnostic imaging | Aquaporin 4 - blood | Young Adult | Optic Neuritis - blood | Optic Neuritis - etiology | Vision Disorders - blood | Adolescent | Adult | Female | Aged | Retrospective Studies | Vision Disorders - etiology | Autoimmunity | Viral antibodies | Aquaporins | Antibodies | Development and progression | Diagnostic imaging | Myelin proteins | Medical colleges | Autoantibodies | Ophthalmology
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5. Full Text Etiology of vertigo in children
by Balatsouras, Dimitrios G and Kaberos, Antonis and Assimakopoulos, Dimitrios and Katotomichelakis, Michael and Economou, Nicolas C and Korres, Stavros G
International Journal of Pediatric Otorhinolaryngology, ISSN 0165-5876, 2006, Volume 71, Issue 3, pp. 487 - 494
Summary Objective To detect the most common causes of vertigo in children. Methods Fifty-four children (20 boys and 34 girls) aged 3–16 years, who presented... 
Otolaryngology | Pediatrics | Vestibular disease | Neurotology | Vertigo | neurotology | vertigo | VESTIBULAR NEURITIS | OTORHINOLARYNGOLOGY | BENIGN PAROXYSMAL VERTIGO | vestibular disease | PEDIATRICS | DIZZINESS | POSITIONAL VERTIGO | CHILDHOOD | MIGRAINE | Severity of Illness Index | Brain Neoplasms - diagnostic imaging | Humans | Brain Neoplasms - pathology | Child, Preschool | Brain Injuries - complications | Male | Tomography, X-Ray Computed | Virus Diseases - virology | Brain Neoplasms - complications | Virus Diseases - complications | Electronystagmography | Magnetic Resonance Imaging | Brain Injuries - diagnostic imaging | Vertigo - etiology | Adolescent | Female | Otitis Media - complications | Brain Injuries - pathology | Child | Vertigo - physiopathology | Vertigo - diagnosis | Children
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6. Full Text Neuroinflammation, mitochondrial defects and neurodegeneration in mucopolysaccharidosis III type C mouse model
by Martins, Carla and Hulkova, Helena and Dridi, Larbi and Dormoy-Raclet, Virginie and Grigoryeva, Lubov and Choi, Yoo and Langford-Smith, Alexander and Wilkinson, Fiona L and Ohmi, Kazuhiro and DiCristo, Graziella and Hamel, Edith and Ausseil, Jerǒme and Cheillan, David and Moreau, Alain and Svobodova, Eva and Hàjkovà, Zuzana and Tesařovà, Markéta and Hansíkovà, Hana and Bigger, Brian W and Hrebícek, Martin and Pshezhetsky, Alexey V
Brain, ISSN 0006-8950, 02/2015, Volume 138, Issue 2, pp. 336 - 355
Severe progressive neurological paediatric disease mucopolysaccharidosis III type C is caused by mutations in the HGSNAT gene leading to deficiency of... 
Mucopolysaccharidosis | Glycosaminoglycans | Heparan sulphate | Acetyl-CoA: α-glucosaminide N-acetyltransferase | Knockout mouse model | glycosaminoglycans | acetyl-CoA: alpha-glucosaminide N-acetyltransferase | MICROGLIAL ACTIVATION | ACETYL-COA | SANFILIPPO-SYNDROME | mucopolysaccharidosis | SYNDROME TYPE-B | knockout mouse model | LYSOSOMAL STORAGE DISEASES | GLUCOSAMINIDE N-ACETYLTRANSFERASE | NEUROSCIENCES | CLINICAL NEUROLOGY | CELL-DEATH | NIEMANN-PICK | heparan sulphate | HEPARAN-SULFATE | BRAIN | Mitochondrial Diseases - pathology | Neurodegenerative Diseases - pathology | Glycosaminoglycans - metabolism | Microtubule-Associated Proteins - metabolism | Neurodegenerative Diseases - etiology | Neurodegenerative Diseases - psychology | Mice, Inbred C57BL | Mucopolysaccharidosis III - pathology | Proteostasis Deficiencies - pathology | Neuritis - pathology | Acetyltransferases - genetics | Mucopolysaccharidosis III - psychology | Mitochondrial Diseases - etiology | Behavior, Animal | Mucopolysaccharidosis III - complications | Animals | Neurologic Examination | Acetyltransferases - deficiency | Gangliosides - metabolism | Mice | Energy Metabolism - physiology | Neuritis - etiology | Life Sciences | acetyl-CoA: α-glucosaminide N-acetyltransferase | Original
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7. Full Text Importance of background rates of disease in assessment of vaccine safety during mass immunisation with pandemic H1N1 influenza vaccines
by Black, Steven, Prof and Eskola, Juhani, Prof and Siegrist, Claire-Anne, Prof and Halsey, Neal, Prof and MacDonald, Noni, Prof and Law, Barbara, Prof and Miller, Elizabeth, FRCPath and Andrews, Nick, MSc and Stowe, Julia, BA and Salmon, Daniel, PhD and Vannice, Kirsten, MHS and Izurieta, Hector S, MD and Akhtar, Aysha, MD and Gold, Mike, Prof and Oselka, Gabriel, Prof and Zuber, Patrick, MD and Pfeifer, Dina, MD and Vellozzi, Claudia, MD
Lancet, The, ISSN 0140-6736, 2009, Volume 374, Issue 9707, pp. 2115 - 2122
Summary Because of the advent of a new influenza A H1N1 strain, many countries have begun mass immunisation programmes. Awareness of the background rates of... 
Internal Medicine | POPULATION | MEDICINE, GENERAL & INTERNAL | STATES | MULTIPLE-SCLEROSIS | SUDDEN-DEATH | GUILLAIN-BARRE-SYNDROME | RISK | OPTIC NEURITIS | PREVALENCE | ANAPHYLAXIS | EPIDEMIOLOGY | Influenza A Virus, H1N1 Subtype - immunology | Myelitis, Transverse - etiology | Influenza Vaccines - standards | Humans | Influenza Vaccines - adverse effects | Child, Preschool | Male | Guillain-Barre Syndrome | Pregnancy | Optic Neuritis - etiology | Adolescent | Mass Vaccination - adverse effects | Adult | Female | Multiple Sclerosis - etiology | Obstetric Labor, Premature - etiology | Neuritis - etiology | Influenza vaccines | Prevention | Safety and security measures | Demographic aspects | Vaccination | Swine influenza | Health aspects | Immunization | Pandemics | Swine flu | Mortality | Industrialized nations | Data collection | Vaccines | Public health | Geography | Spontaneous abortion | Guillain-Barre syndrome | Optic neuritis | Poliomyelitis | Influenza | Measles | Population | Neuritis | Influenza A | Safety | Females | Age
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8. The theory of sympathetic neuritis
by Myddelton, Geoffrey Cheadle
1950, 42 p. diagrs.
Book
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9. Full Text Air pollution, a rising environmental risk factor for cognition, neuroinflammation and neurodegeneration: The clinical impact on children and beyond
by Calderón-Garcidueñas, L and Leray, E and Heydarpour, P and Torres-Jardón, R and Reis, J
Revue Neurologique, ISSN 0035-3787, 01/2016, Volume 172, Issue 1, pp. 69 - 80
Air pollution (indoors and outdoors) is a major issue in public health as epidemiological studies have highlighted its numerous detrimental health consequences... 
Multiple sclerosis | Parkinson's disease | Nitrogen oxide gases | Neurodegeneration | Particulate matter | Children's brain development | Air pollution | Ozone | Alzheimer's disease | OXIDATIVE STRESS | POLYCYCLIC AROMATIC-HYDROCARBONS | ALZHEIMERS-DISEASE | MULTIPLE-SCLEROSIS DISEASE | VITAMIN-D STATUS | CLINICAL NEUROLOGY | SYSTEMIC INFLAMMATION | TAU PHOSPHORYLATION | SERUM 25-HYDROXYVITAMIN-D | PARKINSONS-DISEASE | Alzheimer Disease - etiology | Neurodegenerative Diseases - etiology | Humans | Risk Factors | Air Pollution - adverse effects | Cognition Disorders - etiology | Neuritis - epidemiology | Multiple Sclerosis - etiology | Child | Cognition Disorders - epidemiology | Neurodegenerative Diseases - epidemiology | Neuritis - etiology | Parkinson Disease - etiology | Life Sciences
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10. Full Text Etiologies of acute demyelinating optic neuritis: an observational study of 110 patients
by Deschamps, R and Lecler, A and Lamirel, C and Aboab, J and Gueguen, A and Bensa, C and Vignal, C and Gout, O
European Journal of Neurology, ISSN 1351-5101, 06/2017, Volume 24, Issue 6, pp. 875 - 879
Background and purpose New criteria for the diagnosis of multiple sclerosis (MS) and discovery of myelin oligodendrocyte glycoprotein (MOG) or aquaporin‐4... 
anti‐aquaporin‐4 | myelin oligodendrocyte glycoprotein | multiple sclerosis | neuromyelitis optica | optic neuritis | anti-aquaporin-4 | AUTOANTIBODIES | MULTIPLE-SCLEROSIS | MOG | NEUROMYELITIS-OPTICA | IGG | NEUROSCIENCES | CLINICAL NEUROLOGY | Myelin-Oligodendrocyte Glycoprotein - immunology | Aquaporin 4 - immunology | Humans | Middle Aged | Male | Optic Neuritis - diagnosis | Young Adult | Autoantibodies - immunology | Optic Neuritis - etiology | Adolescent | Adult | Female | Retrospective Studies | Optic Neuritis - pathology | Optic Neuritis - immunology | Databases, Factual | Medical research | Multiple sclerosis | Aquaporins | Analysis | Optic neuritis | Medicine, Experimental | Development and progression | Myelin proteins | Aquaporin 4 | Oligodendrocyte-myelin glycoprotein | Myelin | Immunoglobulin G | Glycoprotein | Antibodies | Criteria | Patients | Etiology | Demyelination |