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Anticancer research, ISSN 0250-7005, 2016, Volume 36, Issue 2, pp. 599 - 609
Polo-like kinase 1 (PLK1) controls the main cell-cycle checkpoints, suggesting utility of its inhibition for cancer treatment, including of highly... 
neuroblastoma | childhood tumors | Cell-cycle checkpoint | polo-like kinase 1 combination | rhabdomyosarcoma | TARGET | POLO-LIKE-KINASE-1 | IDENTIFICATION | COMBINATION | CANCER-THERAPY | ONCOLOGY | PLK EXPRESSION | BI 6727 | PROGNOSTIC-SIGNIFICANCE | Apoptosis - drug effects | Humans | Neuroblastoma - enzymology | Sarcoma, Ewing - pathology | Rhabdomyosarcoma - pathology | Pteridines - administration & dosage | Bone Neoplasms - pathology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Cell Cycle Proteins - antagonists & inhibitors | Flow Cytometry | Medulloblastoma - pathology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Vincristine - administration & dosage | Female | Rhabdomyosarcoma - drug therapy | Bone Neoplasms - drug therapy | Tumor Cells, Cultured | Child | Neuroblastoma - pathology | Protein-Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Bone Neoplasms - enzymology | Cell Cycle Proteins - metabolism | Neoplasms - enzymology | Blotting, Western | Neoplasms - drug therapy | Sarcoma, Ewing - drug therapy | Drug Synergism | Xenograft Model Antitumor Assays | Rhabdomyosarcoma - enzymology | Animals | Mice, Nude | Neuroblastoma - drug therapy | Cell Proliferation - drug effects | Mice | Cell Cycle - drug effects | Medulloblastoma - drug therapy | Neoplasms - pathology | Medulloblastoma - enzymology | Sarcoma, Ewing - enzymology
Journal Article
Annals of Diagnostic Pathology, ISSN 1092-9134, 2015, Volume 19, Issue 4, pp. 239 - 242
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2005, Volume 102, Issue 47, pp. 17213 - 17218
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 11/2009, Volume 8, Issue 11, pp. 3024 - 3035
Rhabdomyosarcoma, consisting of alveolar (aRMS) and embryonal (eRMS) subtypes, is the most common type of sarcoma in children. Currently, there are no targeted... 
polo-like kinase 1 | siRNA library | kinases | rhabdomyosarcoma | phosphatases | CANCER-CELLS | SURVIVAL | GENE-EXPRESSION SIGNATURE | APOPTOSIS | ALVEOLAR RHABDOMYOSARCOMAS | CHILDRENS-ONCOLOGY-GROUP | ONCOLOGY | PATHWAY | IN-VIVO | TUMOR-GROWTH | PAX3-FKHR FUSION PROTEIN | Protein Kinases - metabolism | RNA, Small Interfering - genetics | Glioblastoma - enzymology | Protein Kinases - genetics | Bone Neoplasms - therapy | Sarcoma, Ewing - therapy | Humans | Neuroblastoma - enzymology | Child, Preschool | Rhabdomyosarcoma - pathology | Apoptosis - genetics | Immunoblotting | Neuroblastoma - therapy | Cell Cycle Proteins - antagonists & inhibitors | Transfection | Glioblastoma - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Bone Neoplasms - genetics | Cell Growth Processes - genetics | Child | Protein-Serine-Threonine Kinases - metabolism | Phosphoric Monoester Hydrolases - antagonists & inhibitors | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Phosphoric Monoester Hydrolases - genetics | Bone Neoplasms - enzymology | Osteosarcoma - enzymology | Neuroblastoma - genetics | Cell Cycle Proteins - metabolism | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Glioblastoma - therapy | Rhabdomyosarcoma - enzymology | Animals | Cell Line, Tumor | Mice | Rhabdomyosarcoma - genetics | Osteosarcoma - genetics | Phosphoric Monoester Hydrolases - metabolism | RNA, Small Interfering - administration & dosage | Sarcoma, Ewing - genetics | Osteosarcoma - therapy | Rhabdomyosarcoma - therapy | Sarcoma, Ewing - enzymology | Genetic Therapy - methods
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2003, Volume 87, Issue 6, pp. 1427 - 1435
We describe here a new component of the phosphatidylinositol 3‐kinase/Akt signaling pathway that directly impacts mitochondria. Akt (protein kinase B) was... 
insulin‐like growth factor‐1 | ATP synthase | Akt | glycogen synthase kinase‐3β | mitochondria | phosphatidylinositol 3‐kinase | Insulin-like growth factor-1 | Mitochondria | Phosphatidylinositol 3-kinase | Glycogen synthase kinase-3β | SURVIVAL | TRANSLOCATION | CELLS | LOCALIZATION | APOPTOSIS | PROTEIN-KINASE-B | glycogen synthase kinase-3 beta | PHOSPHORYLATION | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | insulin-like growth factor-1 | NEUROSCIENCES | GLYCOGEN-SYNTHASE KINASE-3-BETA | WITHDRAWAL | phosphatidylinositol 3-kinase | Mitochondria - enzymology | Insulin-Like Growth Factor I - pharmacology | Embryo, Mammalian | Humans | Neuroblastoma - enzymology | Protein-Serine-Threonine Kinases | Cytosol - drug effects | Porins - metabolism | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | Threonine - metabolism | Kidney | Cell Nucleus - enzymology | Electron Transport Complex IV - metabolism | Immunoblotting - methods | Cytosol - enzymology | Drug Interactions | Time Factors | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Cell Membrane - metabolism | Phosphorylation - drug effects | Cell Membrane - drug effects | Ionophores - pharmacology | Superoxide Dismutase - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Brain Neoplasms - enzymology | Cytochromes c - metabolism | Cell Fractionation - methods | Enzyme Inhibitors - pharmacology | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - methods | Mitochondria - drug effects | Enzyme Activation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Serine - metabolism | Mitochondrial Proton-Translocating ATPases - metabolism | Precipitin Tests - methods | Proto-Oncogene Proteins c-akt | Androstadienes - pharmacology | Wortmannin | Voltage-Dependent Anion Channel 1 | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - instrumentation | Cell Nucleus - drug effects | glycogen synthase kinase-3β
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2005, Volume 280, Issue 21, pp. 20493 - 20502
The restoration of energy balance during ischemia is critical to cellular survival; however, relatively little is known concerning the regulation of neuronal... 
AORTIC ENDOTHELIAL-CELLS | NEURONAL-ACTIVITY | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | N-TERMINAL KINASE | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | GLUCOSE-TRANSPORT | FATTY-ACID SYNTHASE | NEUROBLASTOMA-CELLS | Fatty Acid Synthases - antagonists & inhibitors | Immunohistochemistry | Neuroprotective Agents - therapeutic use | Nerve Tissue Proteins - deficiency | Nitric Oxide Synthase - physiology | Stroke - complications | Male | AMP-Activated Protein Kinases | Middle Cerebral Artery | Aminoimidazole Carboxamide - pharmacology | Glucose - administration & dosage | Brain Ischemia - enzymology | Ribonucleotides - pharmacology | Nitric Oxide Synthase Type I | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Hippocampus - enzymology | Neurons - metabolism | Disease Models, Animal | Cerebral Cortex - enzymology | Nerve Tissue Proteins - physiology | Constriction | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | 4-Butyrolactone - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Stroke - drug therapy | Enzyme Activation - drug effects | Multienzyme Complexes - antagonists & inhibitors | Enzyme Inhibitors - therapeutic use | Mice, Knockout | Oxygen - administration & dosage | Stroke - etiology | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Energy Metabolism | Nitric Oxide Synthase - deficiency | Multienzyme Complexes - physiology | Neurons - enzymology | Mice | 4-Butyrolactone - analogs & derivatives
Journal Article
Annals of Oncology, ISSN 0923-7534, 09/2016, Volume 27, Issue suppl_3, pp. iii4 - iii15
Journal Article
Neuropharmacology, ISSN 0028-3908, 09/2017, Volume 123, pp. 465 - 476
Journal Article