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PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, pp. e0146129 - e0146129
Parkinson's disease (PD) is characterized by progressive motor impairment attributed to progressive loss of dopaminergic neurons in the substantia nigra (SN) pars compacta... 
NEURODEGENERATIVE DISORDERS | PROTEIN | ALZHEIMERS-DISEASE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | FLUID NITRATE LEVELS | CEREBROSPINAL-FLUID | ISCHEMIA-MODIFIED ALBUMIN | ASSOCIATION | BRAIN | Catalase - blood | Oxidative Stress | Reactive Nitrogen Species - blood | Dopaminergic Neurons - pathology | Humans | Middle Aged | Superoxide Dismutase - blood | Male | Sulfhydryl Compounds - blood | Case-Control Studies | Pars Compacta - pathology | 5'-Nucleotidase - blood | Adenosine Deaminase - blood | Ferritins - blood | Dopaminergic Neurons - metabolism | Female | Ascorbic Acid - blood | Iron - blood | Parkinson Disease - pathology | Adenosine Triphosphatases - blood | Oxidation-Reduction | Reactive Oxygen Species - blood | Thiobarbituric Acid Reactive Substances - metabolism | Pars Compacta - metabolism | Biomarkers - blood | Advanced Oxidation Protein Products - blood | Aged | Parkinson Disease - blood | Peroxidase - blood | Transferrin - metabolism | Nucleotidase | Health sciences | Oxidative stress | Reactive oxygen species | Transferrin | Parkinson's disease | Ascorbic acid | Thiols | Pathogenesis | Substantia nigra | Ferritin | Parkinsons disease | Homeostasis | Superoxide dismutase | Iron | Biochemistry | Parameter identification | Proteins | Antioxidants | Ischemia | Catalase | Neurodegeneration | Rodents | Vitamin C | Peroxidase | Oxidation | Life sciences | Thiobarbituric acid | Malnutrition | Movement disorders | Dopamine receptors | Medical research | Adenosine | Dopamine | Neurodegenerative diseases | Inflammation | Adenosine deaminase | Patients | Serum levels | Neurology | Brain research | Hospitals | Biomarkers | Protein thiols | Cancer | Index Medicus | Fluorescence recovery after photobleaching
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 2, p. e90523
Alzheimer's disease (AD) is characterized by neurotoxic amyloid-beta plaque formation in brain parenchyma and cerebral blood vessels known as cerebral amyloid angiopathy (CAA... 
BRAIN-BARRIER | PHOSPHOLIPASE C-GAMMA-2 | ACTIVATION | VESSELS | MULTIDISCIPLINARY SCIENCES | AMYLOID-BETA-PEPTIDE | CALCIUM | RISK | ANGIOPATHY | PRECURSOR PROTEIN | EXPRESSION | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Amyloid beta-Peptides - pharmacology | Humans | Middle Aged | Amyloid - ultrastructure | Cerebral Amyloid Angiopathy - metabolism | Young Adult | Amyloid - metabolism | Amyloid beta-Peptides - metabolism | Adult | Blood Platelets - drug effects | Cerebral Amyloid Angiopathy - genetics | Cerebral Amyloid Angiopathy - blood | Disease Models, Animal | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Microscopy, Electron | Disease Progression | Animals | Blood Platelets - metabolism | Blood Platelets - ultrastructure | Alzheimer Disease - metabolism | Platelet Activation - drug effects | Alzheimer Disease - genetics | Microscopy, Fluorescence | Alzheimer Disease - blood | Stroke (Disease) | Medical research | Advertising executives | Analysis | Atherosclerosis | Medicine, Experimental | Development and progression | Genetic engineering | Neovascularization | Alzheimer's disease | Thrombosis | Blood clot | Occlusion | Brain | Reactive oxygen species | Peptides | Disease | Neuropathology | Pathogenesis | Memory | Parenchyma | Nervous system | Kinases | Blood | Cerebrovascular system | Cerebral amyloid angiopathy | Neurotoxicity | Vascular diseases | Ischemia | Blood platelets | Enzymatic activity | Hemostasis | Cerebral blood flow | Physiology | Bioindicators | Hemostatics | Thromboembolism | Plaques | Stroke | Neurodegenerative diseases | Transgenic mice | Blood vessels | Inflammation | Blood flow | Studies | Neurology | Pathology | Brain research | Arteriosclerosis | Biomarkers | Platelets | Apoptosis
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