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American Journal of Psychiatry, ISSN 0002-953X, 07/2014, Volume 171, Issue 7, pp. 758 - 767
Journal Article
Trends in Neurosciences, ISSN 0166-2236, 2012, Volume 35, Issue 11, pp. 700 - 709
.... Caspase-3 activation, a crucial event of neuronal cell death program, is also a feature of many chronic neurodegenerative diseases... 
Neurology | synaptic plasticity | synaptic loss | Alzheimer's disease | long-term depression | neuronal differentiation | long-term potentiation | Neuronal differentiation | Synaptic loss | Long-term depression | Long-term potentiation | Synaptic plasticity | ALZHEIMERS-DISEASE | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | PROGRAMMED CELL-DEATH | MITOCHONDRIAL-MEMBRANE PERMEABILIZATION | PRECURSOR PROTEIN | HUNTINGTONS-DISEASE | PARKINSONS-DISEASE | Nerve Net - embryology | Mitochondria - enzymology | Caspase Inhibitors - pharmacology | Humans | Huntington Disease - pathology | Neurons - cytology | Nerve Net - enzymology | Molecular Targeted Therapy | Alzheimer Disease - pathology | Caspase 3 - physiology | Nervous System - growth & development | Nervous System - embryology | Huntington Disease - enzymology | Disease Models, Animal | Nerve Net - growth & development | Long-Term Synaptic Depression - physiology | Nerve Tissue Proteins - physiology | Nervous System - enzymology | Parkinson Disease - pathology | Embryonic Development - physiology | Neural Tube - physiology | Alzheimer Disease - enzymology | Receptors, N-Methyl-D-Aspartate - physiology | Mice, Knockout | Animals | Caspase Inhibitors - therapeutic use | Neurons - enzymology | Parkinson Disease - enzymology | Apoptosis Regulatory Proteins - physiology | Mice | Apoptosis - physiology | Enzyme Activation | Nervous system diseases | Neurosciences | Neural networks | Proteases | Neurons | Depression, Mental | Apoptosis | Neurodegenerative diseases | Proteinase | Central nervous system | Caspase-3 | Neurological diseases | Axons | Mitochondria | Neural tube | Plasticity (synaptic) | Reviews | Differentiation
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, p. e0174206
... common phenotypes including: retinitis pigmentosa, renal cystic disease, polydactyly, situs inversus, mental retardation, hypoplasia of the corpus callosum, Dandy... 
CILIARY | SONIC HEDGEHOG | KINESIN-II | MULTIDISCIPLINARY SCIENCES | MOUSE | DISEASE | ECTODERM | NEURAL CREST | REPRESSOR FUNCTIONS | SIGNALING CENTER | PRIMARY CILIUM | Skull - metabolism | Face - abnormalities | Male | Neural Plate - metabolism | Cilia - genetics | Neural Crest - metabolism | Skull - embryology | Animals | Gene Expression Regulation, Developmental | Neural Crest - embryology | Gene Deletion | Neural Plate - embryology | Tumor Suppressor Proteins - genetics | Adaptor Proteins, Signal Transducing - genetics | Skull - abnormalities | Kinesin - genetics | Female | Face - embryology | Mice | Craniofacial Abnormalities - genetics | Physiological aspects | Genetic aspects | Research | Craniofacial abnormalities | Gene expression | Cilia and ciliary motion | Heart | Pediatrics | Transcription factors | Parkinson's disease | Craniosynostosis | Hyperplasia | Disorders | AKT protein | Arrestin | Proteins | Bone growth | Etiology | Surgery | Fibroblasts | Genetics | Diagnosis | Bone (endochondral) | Movement disorders | Liver diseases | Neurodegenerative diseases | Developmental biology | Plastic surgery | Mammals | Embryos | Ablation | Hospitals | Aberration | Mutation | Kidney transplantation | Cargo | Animal models | Retinitis pigmentosa | Genes | Biology | Tissues | Basal bodies | Cell surface | Defects | Morphogenesis | Division | Children | Cilia | Septum | Mutants | Skull | Position (location) | Differentiation | Combinatorial analysis
Journal Article
Science, ISSN 0036-8075, 02/2018, Volume 359, Issue 6375, pp. 555 - 559
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2015, Volume 47, Issue 8, pp. 926 - 932
... peripheral neuropathy to identify additional genes involved in disease, on the basis of the hypothesis that causative genes would uncover new factors in common... 
RECRUITMENT | CELLS | MITOCHONDRIAL DYNAMICS | FUSION | NEUROPATHY | ZEBRAFISH | UGO1P | OPA1 | GENETICS & HEREDITY | OUTER | GTPASE | Humans | Optic Atrophy, Autosomal Dominant - metabolism | Embryo, Nonmammalian - metabolism | Phosphate Transport Proteins - metabolism | Cercopithecus aethiops | Embryo, Nonmammalian - embryology | Male | Mitochondrial Proteins - genetics | Zebrafish - embryology | Charcot-Marie-Tooth Disease - genetics | RNA Interference | Mitochondrial Proteins - metabolism | HEK293 Cells | Muscle Proteins - metabolism | Female | Membrane Proteins - metabolism | Optic Atrophy, Autosomal Dominant - pathology | Phosphate Transport Proteins - genetics | Charcot-Marie-Tooth Disease - metabolism | Microscopy, Electron, Transmission | Genetic Predisposition to Disease - genetics | Animals, Genetically Modified | Membrane Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | Sequence Analysis, DNA | Mitochondrial Membranes - metabolism | Muscle Proteins - genetics | Microscopy, Confocal | Exome - genetics | Animals | Pedigree | Embryo, Nonmammalian - ultrastructure | Zebrafish - metabolism | Optic Atrophy, Autosomal Dominant - genetics | Saccharomyces cerevisiae Proteins - metabolism | Protein Binding | Mutation | COS Cells | Development and progression | Genetic aspects | Optic atrophy | Gene mutations | Identification and classification | Health aspects | Proteins | Neurodegeneration | Genes | Neurological disorders
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45069
Aging is the main risk factor for Alzheimer's disease (AD); however, the aspects of the aging process that predispose the brain to the development of AD are largely unknown... 
NATURAL OLIGOMERS | CELLS | IN-VITRO | ACTIVATION | BIOMARKER | COLLAGENASE | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | AMYLOID-BETA-PROTEIN | P16(INK4A) | CELLULAR SENESCENCE | Brain - embryology | Cricetulus | Amyloid beta-Peptides - pharmacology | Humans | Middle Aged | Astrocytes - pathology | Immunoblotting | Male | Peptide Fragments - pharmacology | Alzheimer Disease - pathology | Brain - metabolism | Cellular Senescence | Aged, 80 and over | Adult | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | CHO Cells | Biomarkers - metabolism | Astrocytes - drug effects | Cricetinae | Cells, Cultured | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Aged | Matrix Metalloproteinase 1 - metabolism | Microscopy, Fluorescence | Astrocytes - metabolism | Advertising executives | Interleukins | Neurons | Alzheimer's disease | Risk factors | Neurophysiology | Brain | Oxidative stress | Senescence | Peptides | Disease | Laboratories | Interleukin | INK4a protein | Central nervous system | Homeostasis | Galactosidase | Matrix metalloproteinase | Interleukin 6 | Proteins | Cortex (frontal) | Rodents | Cell cycle | Fibroblasts | Aging | Metalloproteinase | Age | Interstitial collagenase | Cytokines | Astrocytes | Fetuses | Health risks | p16 Protein | Inflammation | Patients | Medicine | Pathology | β-Amyloid | In vivo methods and tests | Dementia
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50705
... to the family of polyQ expansion disorders, also named polyQ diseases, a group of neurodegenerative disorders comprising spinobulbar muscular atrophy (SBMA) [2... 
EXPANDED POLYGLUTAMINE | CONE-ROD DYSTROPHY | MACHADO-JOSEPH DISEASE | MULTIDISCIPLINARY SCIENCES | SPINOCEREBELLAR ATAXIA-7 | STAGGERER MUTANT MICE | CAG REPEAT EXPANSION | HISTONE ACETYLTRANSFERASE ACTIVITY | BINDING PROTEIN GENE | HOMEOBOX GENE | TRINUCLEOTIDE REPEAT | Humans | Nerve Tissue Proteins - deficiency | Photoreceptor Cells, Vertebrate - drug effects | Cerebellum - drug effects | Ataxin-7 | Neurons - cytology | Peptide Fragments - pharmacology | Zebrafish - embryology | Cerebellum - embryology | Nerve Tissue Proteins - chemistry | Neurons - drug effects | Zebrafish Proteins - chemistry | Zebrafish Proteins - metabolism | Muscles - embryology | Photoreceptor Cells, Vertebrate - cytology | Nerve Tissue Proteins - genetics | Zebrafish - genetics | Nerve Tissue Proteins - metabolism | Spinal Cord - embryology | Zebrafish Proteins - deficiency | Animals | Cell Differentiation - drug effects | Zebrafish - metabolism | Cerebellum - cytology | Photoreceptor Cells, Vertebrate - metabolism | Zebrafish Proteins - genetics | Embryonic Development - drug effects | Embryonic development | Nervous system diseases | Genetic transcription | Neurons | Analysis | Cerebellum | Disease | Transcription | Laboratories | Genes | Retina | Lethality | Inactivation | Proteins | Embryogenesis | Ataxin | Spinocerebellar ataxia | Ataxia | Trinucleotide repeat diseases | Photoreception | Polyglutamine | Deactivation | Neurodegenerative diseases | Deformation mechanisms | Granule cells | Zebrafish | Functional analysis | Embryos | Hereditary diseases | Embryonic growth stage | Depletion | Granular materials | Photoreceptors | Mutation | Differentiation
Journal Article