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Cell, ISSN 0092-8674, 09/2014, Volume 159, Issue 1, pp. 200 - 214
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 07/2017, Volume 59, Issue 2, pp. 633 - 641
Both common and rare polymorphisms within ABCA7 have been associated with Alzheimer’s disease (AD). In particular, the rare AD associated polymorphism... 
Brain | Splicing | Neurodegenerative diseases | Ribonucleic acids | Alleles | Genetics | mRNA | Aberration | Alzheimer's disease | Polymorphism | ABCA7 | SNP | Alzheimer’s
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 06/2017, Volume 58, Issue 3, pp. 631 - 659
Alzheimer’s disease (AD), the main form of dementia in the elderly, is the most common progressive neurodegenerative disease characterized by rapidly... 
Elderly people | Neurodegenerative diseases | Genes | Cognitive ability | Genomes | Gene sequencing | Biomaterials | Biomedical materials | Etiology | Apolipoprotein E | Searching | Dementia disorders | Genetics | Heritability | Mutation | Elderly | Alzheimer's disease | Secretase | Geriatrics
Journal Article
Science, ISSN 0036-8075, 2014, Volume 344, Issue 6183, pp. 519 - 523
Journal Article
Molecular Systems Biology, ISSN 1744-4292, 2011, Volume 7, Issue 1, pp. 494 - n/a
Proteins targeting the same subcellular localization tend to participate in mutual protein–protein interactions (PPIs) and are often functionally associated.... 
comorbidity | human disease | subcellular localization | cellular networks | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | DISORDERS | IDENTIFICATION | INTERACTOME | INTERACTION NETWORK | PHENOTYPE NETWORK | CONSTRUCTION | MUTATIONS | EXPRESSION | MAP | Disease - genetics | Comorbidity | Humans | Molecular Epidemiology - methods | Data Collection - methods | Cell Compartmentation | Disease - etiology | Proteins - genetics | Systems Biology - methods | Disease - classification | Algorithms | Proteins - metabolism | Databases, Protein | Subcellular Fractions - chemistry | Cluster Analysis | Health care | Enrichment | Cluster analysis | Modules | Genes | Correlation coefficients | Spatial discrimination | Data bases | Risk factors | Proteins | Mitochondria | Databases | Pathways | Mathematical analysis | Etiology | Compartments | Risk assessment | Genetics | Localization | Protein transport | Alzheimer's disease | Correlation coefficient | Translocation | Progressions | Integration | Phenotypes | Neurodegenerative diseases | Angiotensin I | Diabetes mellitus | Health risks | Metabolism | Clustering | Organelles | Patients | Molecular chains | Golgi apparatus | Diseases | Government programs | Proteomics | Angiotensin | Metabolic pathways | Mutation | Position (location) | Alzheimers disease | Protein interaction | Endoplasmic reticulum | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 09/2017, Volume 47, Issue 3, pp. 566 - 581.e9
Microglia play a pivotal role in the maintenance of brain homeostasis but lose homeostatic function during neurodegenerative disorders. We identified a... 
Alzheimer’s disease | multiple sclerosis | transcriptional regulation | neurodegeneration | TREM2 | APOE | amyotrophic lateral sclerosis | microglia | Alzheimer's disease | GENE-EXPRESSION SIGNATURE | ACTIVATION | MULTIPLE-SCLEROSIS | ALZHEIMERS-DISEASE | MOUSE MODEL | MACROPHAGE | MICE | IMMUNOLOGY | DEFICIENCY | APOLIPOPROTEIN-E | CELL-DEATH | Microglia - metabolism | Apolipoproteins E - deficiency | Membrane Glycoproteins - metabolism | Humans | Cerebral Cortex - pathology | Transcriptome | Apoptosis - genetics | Monocytes - metabolism | Gene Expression Profiling | Monocytes - immunology | Phagocytosis - genetics | Neurodegenerative Diseases - immunology | Apolipoproteins E - metabolism | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Microglia - immunology | Superoxide Dismutase-1 - metabolism | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Female | Neurons - metabolism | Disease Models, Animal | Gene Targeting | Plaque, Amyloid - pathology | Signal Transduction | Gene Expression Regulation | Phagocytosis - immunology | Immune Tolerance | Mice, Transgenic | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Mice, Knockout | Encephalomyelitis, Autoimmune, Experimental | Phenotype | Animals | Apoptosis - immunology | Apolipoproteins E - genetics | Alzheimer Disease - metabolism | Superoxide Dismutase-1 - genetics | Plaque, Amyloid - metabolism | Mice | Alzheimer Disease - genetics | Transforming Growth Factor beta - metabolism | Receptors, Immunologic - metabolism | Cluster Analysis | Nervous system diseases | Multiple sclerosis | Neurons | Analysis | Amyotrophic lateral sclerosis | Genetic aspects | Genetic transcription | Apolipoproteins | Brain | Animal models | Disease | Transcription | Genes | Homeostasis | Sclerosis | Proteins | Restoration | Neurodegeneration | Apolipoprotein E | Rodents | Plaques | Myeloid cells | Neurodegenerative diseases | Microglia | Neurological diseases | Molecular modelling | β-Amyloid | Phagocytosis | Apoptosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7388, pp. 222 - 226
Cognitive decline is a debilitating feature of most neurodegenerative diseases of the central nervous system, including Alzheimer's disease(1). The causes... 
REMOTE MEMORIES | LONG-TERM-MEMORY | CDK5 | CHROMATIN | PHOSPHORYLATION | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | CLINICAL-IMPLICATIONS | SYNAPTIC PLASTICITY | EXPRESSION | TRANSGENIC MICE | Memory Disorders - physiopathology | Alzheimer Disease - complications | Memory Disorders - genetics | Peptide Fragments - toxicity | Humans | Neuronal Plasticity - drug effects | Receptors, Glucocorticoid - metabolism | Hippocampus - drug effects | RNA Polymerase II - metabolism | Memory Disorders - complications | Promoter Regions, Genetic - drug effects | Promoter Regions, Genetic - genetics | Gene Knockdown Techniques | Brain - metabolism | Neuronal Plasticity - genetics | Epigenesis, Genetic - drug effects | Phosphorylation - drug effects | Disease Models, Animal | Hydrogen Peroxide - toxicity | Histone Deacetylase 2 - genetics | Alzheimer Disease - physiopathology | Amyloid beta-Peptides - toxicity | Brain - physiopathology | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - complications | Brain - drug effects | Gene Expression Regulation - drug effects | Hippocampus - metabolism | Acetylation - drug effects | Animals | Neurodegenerative Diseases - physiopathology | Histone Deacetylase 2 - deficiency | Mice | Histone Deacetylase 2 - metabolism | Histones - metabolism | Alzheimer Disease - genetics | Epigenetic inheritance | Complications and side effects | Causes of | Nervous system | Genetic aspects | Degeneration | Cognition disorders | Health aspects | Epigenetics | Genetics | RNA polymerase | Kinases | Neurodegeneration | Cyclin-dependent kinases | Index Medicus
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 07/2011, Volume 89, Issue 1, pp. 162 - 167
Journal Article