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Cancer Research, ISSN 0008-5472, 03/2010, Volume 70, Issue 6, pp. 2224 - 2234
Journal Article
Nature Communications, ISSN 2041-1723, 11/2015, Volume 6, Issue 1, p. 8784
Journal Article
Journal Article
ANTICANCER RESEARCH, ISSN 0250-7005, 02/2016, Volume 36, Issue 2, pp. 713 - 719
Journal Article
Oncogene, ISSN 0950-9232, 04/2014, Volume 33, Issue 14, pp. 1799 - 1808
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2013, Volume 335, Issue 1, pp. 1 - 8
Highlights ► Dysregulation of the PI3K/Akt/mTOR pathway can occur at multiple nodes in pNETs, providing several potential drug targets. ► PI3K pathway... 
Hematology, Oncology and Palliative Medicine | mTOR | Inhibitor | Akt | PI3K | Pancreatic | Neuroendocrine tumors | MTOR | MAMMALIAN TARGET | PROTEIN-KINASE-C | ISLET-CELL CARCINOMA | MTOR PATHWAY | ENDOCRINE TUMORS | REVEALS ALTERATIONS | RAD001 EVEROLIMUS | GROWTH-FACTOR RECEPTOR | AKT INHIBITION | ONCOLOGY | LOW-GRADE | Sirolimus - analogs & derivatives | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Humans | Pancreatic Neoplasms - enzymology | Clinical Trials as Topic | Phosphatidylinositol 3-Kinases - metabolism | Molecular Targeted Therapy | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Neuroendocrine Tumors - enzymology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Pancreatic Neoplasms - drug therapy | TOR Serine-Threonine Kinases - antagonists & inhibitors | Protein Kinase Inhibitors - administration & dosage | Sirolimus - administration & dosage | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Neuroendocrine Tumors - drug therapy | Proto-Oncogene Proteins c-akt - metabolism | Everolimus | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Proteins | Studies | Epidermal growth factor | Disease | Pathogenesis | Insulin-like growth factors | Tumorigenesis | Metastasis | Kinases | Gene expression | Vascular endothelial growth factor | Tumors
Journal Article
Medical Science Monitor, ISSN 1234-1010, 11/2018, Volume 24, pp. 8125 - 8140
Journal Article
Human Pathology, ISSN 0046-8177, 09/2018, Volume 79, pp. 66 - 76
Journal Article
World Journal of Gastroenterology, ISSN 1007-9327, 12/2015, Volume 21, Issue 47, pp. 13240 - 13249
AIM: To study histidine decarboxylase (HDC) expression in normal and neoplastic gastric neuroendocrine cells in relationship to the main histamine metabolite.... 
Immunohistochemistry | Enterochromaffin-like cells | Gastric neuroendocrine tumours | Urinary excretion of the main histamine metabolite methylimidazoleacetic acid | Vesicular monoamine transporter 2 | High performance liquid chromatography | Histidine decarboxylase | SYSTEM | GHRELIN | ENDOCRINE TUMORS | VESICULAR MONOAMINE TRANSPORTER-2 | RAT STOMACH | CARCINOID-TUMORS | GROWTH | ECL CELLS | HISTAMINE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Urinalysis | Histidine Decarboxylase - analysis | Humans | Middle Aged | Male | Stomach Neoplasms - pathology | Chromatography, High Pressure Liquid | Ghrelin - analysis | Neuroendocrine Tumors - enzymology | Vesicular Monoamine Transport Proteins - analysis | Young Adult | Neuroendocrine Cells - enzymology | Biomarkers, Tumor - urine | Aged, 80 and over | Adult | Female | Enterochromaffin Cells - pathology | Stomach Neoplasms - enzymology | Neuroendocrine Cells - pathology | Adenocarcinoma - enzymology | Neuroendocrine Tumors - urine | Adenocarcinoma - secondary | Enterochromaffin Cells - enzymology | Renal Elimination | Neuroendocrine Tumors - secondary | Fluorescent Antibody Technique | Aged | Imidazoles - urine | Adenocarcinoma - urine | Stomach Neoplasms - urine | Basic Study | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Enterochromaffin-like cells; High performance liquid chromatography; Gastric neuroendocrine tumours; Histidine decarboxylase; Immunohistochemistry; Urinary excretion of the main histamine metabolite methylimidazoleacetic acid; Vesicular monoamine transporter 2
Journal Article
Pancreas, ISSN 0885-3177, 2017, Volume 46, Issue 6, pp. 758 - 763
Alkylating agents have activity in well-differentiated pancreatic neuroendocrine tumors (WD panNETs). In glioblastoma multiforme, decreased activity of... 
Pancreatic neuroendocrine tumors | Alkylating agent | Biomarker | Neuroendocrine tumors | Immunohistochemistry | Dacarbazine - adverse effects | Dacarbazine - therapeutic use | Humans | Middle Aged | DNA Repair Enzymes - genetics | Drug Resistance, Neoplasm | Male | Neuroendocrine Tumors - enzymology | Pancreatic Neoplasms - drug therapy | Young Adult | DNA Methylation | Neoplasm Grading | Time Factors | Tumor Suppressor Proteins - genetics | Dacarbazine - analogs & derivatives | Adult | Female | Cell Differentiation | Child | DNA Modification Methylases - analysis | Databases, Factual | Neuroendocrine Tumors - pathology | Promoter Regions, Genetic | Pharmacogenetics | Biomarkers, Tumor - analysis | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Pancreatic Neoplasms - enzymology | Pancreatic Neoplasms - genetics | Treatment Outcome | DNA Repair Enzymes - analysis | Sequence Analysis, DNA | Disease Progression | Neuroendocrine Tumors - genetics | Antineoplastic Agents, Alkylating - therapeutic use | DNA Modification Methylases - genetics | Adolescent | Neuroendocrine Tumors - drug therapy | Aged | Biomarkers, Tumor - genetics | Antineoplastic Agents, Alkylating - adverse effects | Tumor Suppressor Proteins - analysis | pancreatic neuroendocrine tumors | biomarker | MGMT | neuroendocrine tumors | alkylating agent
Journal Article
Human Pathology, ISSN 0046-8177, 2017, Volume 65, pp. 41 - 52
Epigenetic factors contribute to carcinogenesis, tumor promotion and chemoresistance. Histone deacetylases (HDACs) are epigenetic regulators that primarily... 
Pathology | Expression analysis | Proliferation | Overall survival | Disease-free survival | HDAC | Pancreatic neuroendocrine tumor | CANCER-CELLS | DUCTAL ADENOCARCINOMA | ACETYLATION | PATHOLOGY | MAINTENANCE | CLINICOPATHOLOGICAL PARAMETERS | GROWTH | 1 EXPRESSION | HUMAN HEPATOCELLULAR-CARCINOMA | PROGRESSION | Immunohistochemistry | Histone Deacetylases - analysis | Predictive Value of Tests | Up-Regulation | Cell Proliferation | Histone Deacetylase 2 - analysis | Mitosis | Humans | Middle Aged | Male | Neuroendocrine Tumors - enzymology | Neoplasm Grading | Time Factors | Aged, 80 and over | Adult | Female | Pancreatic Neoplasms - mortality | Neuroendocrine Tumors - pathology | Sirtuin 1 - analysis | Biomarkers, Tumor - analysis | Pancreatic Neoplasms - pathology | Risk Factors | Kaplan-Meier Estimate | Pancreatic Neoplasms - enzymology | Disease-Free Survival | Histone Deacetylase 1 - analysis | Neuroendocrine Tumors - mortality | Aged | Neuroendocrine Tumors - therapy | Pancreatic Neoplasms - therapy | Epigenetic inheritance | Chromatin | Genetic engineering | Patient outcomes | Tumors | Enzymes | Immunoglobulins | Statistical analysis | Breast cancer | Multivariate analysis | Gene expression | Studies | Liver cancer | Survival analysis | Cell growth | Medical prognosis | Rodents | Cell cycle | Epigenetics | Tumorigenesis | Diabetes | Apoptosis
Journal Article