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Journal Article
Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 298 - 305
Journal Article
Cell, ISSN 0092-8674, 04/2013, Volume 153, Issue 3, pp. 666 - 677
Journal Article
Cancer Cell, ISSN 1535-6108, 10/2016, Volume 30, Issue 4, pp. 563 - 577
The transition from castration-resistant prostate adenocarcinoma (CRPC) to neuroendocrine prostate cancer (NEPC) has emerged as an important mechanism of... 
castration-resistant prostate adenocarcinoma | prostate cancer organoid | N-Myc | genetically engineered mouse | neuroendocrine prostate cancer | Aurora kinase A | EZH2 | REARRANGEMENT | INACTIVATION | GENOMICS | PROTEIN | ONCOLOGY | ADENOCARCINOMA | MOUSE MODEL | SMALL-CELL CARCINOMA | PTEN | DIFFERENTIATION | IDENTIFICATION | CELL BIOLOGY | Enhancer of Zeste Homolog 2 Protein - antagonists & inhibitors | Prostatic Neoplasms - metabolism | Humans | N-Myc Proto-Oncogene Protein - genetics | Male | Genes, myc | Prostatic Neoplasms, Castration-Resistant - genetics | Prostatic Neoplasms, Castration-Resistant - pathology | Heterografts | Prostatic Neoplasms - genetics | Transcription, Genetic | N-Myc Proto-Oncogene Protein - biosynthesis | Prostatic Neoplasms - drug therapy | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Signal Transduction | Neuroendocrine Tumors - metabolism | Mice, Transgenic | Prostatic Neoplasms, Castration-Resistant - drug therapy | Pyrimidines - pharmacology | Prostatic Neoplasms, Castration-Resistant - metabolism | Neuroendocrine Tumors - genetics | Azepines - pharmacology | Animals | N-Myc Proto-Oncogene Protein - metabolism | Neuroendocrine Tumors - drug therapy | Mice | Protein Kinase Inhibitors - pharmacology | Development and progression | Genetic aspects | Genetic engineering | Genetic transcription | Prostate cancer | Genetically modified organisms
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2014, Volume 20, Issue 11, pp. 2846 - 2850
A subset of patients with advanced castration-resistant prostate cancer may eventually evolve into an androgen receptor (AR)-independent phenotype, with a... 
METASTATIC PROSTATE | FUSION | ONCOLOGY | ADENOCARCINOMA | SMALL-CELL CARCINOMA | NEUROENDOCRINE CARCINOMA | MODEL | EXPRESSION | PROGRESSION | CD44 | P53 | Male | Carcinoma, Neuroendocrine | Prostatic Neoplasms, Castration-Resistant | Humans | AR negative | anaplastic prostate cancer | neuroendocrine prostate cancer | androgen independent
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2016, Volume 29, Issue 4, pp. 536 - 547
MYCN amplification and overexpression are common in neuroendocrine prostate cancer (NEPC). However, the impact of aberrant N-Myc expression in prostate... 
INACTIVATION | ORIGIN | ONCOLOGY | ADENOCARCINOMA | C-MYC | STEM-CELL | DIFFERENTIATION | IDENTIFICATION | CARCINOMA | EXPRESSION | BASAL-CELLS | CELL BIOLOGY | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Laser Capture Microdissection | Male | Antineoplastic Agents - therapeutic use | Genes, myc | Aurora Kinase A - antagonists & inhibitors | Molecular Targeted Therapy | Recombinant Fusion Proteins - metabolism | Exome | Neoplasm Metastasis | Proto-Oncogene Proteins c-myc - physiology | Prostatic Neoplasms - genetics | Aurora Kinase A - physiology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Adenocarcinoma - genetics | Neoplasm Proteins - genetics | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Transduction, Genetic | Neoplasm Invasiveness | Phenylurea Compounds - therapeutic use | Epithelial Cells - pathology | Proto-Oncogene Proteins c-akt - physiology | Mice, SCID | Azepines - therapeutic use | Neuroendocrine Tumors - genetics | Orchiectomy | Xenograft Model Antitumor Assays | Animals | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Cell Line, Tumor | Mice, Inbred NOD | Enzyme Activation | Molecular biology | Health aspects | Prostate cancer | Stem cells | Medical genetics | Cell death
Journal Article
by Kim, J and Jin, H and Zhao, J.C and Yang, Y.A and Li, Y and Yang, X and Dong, X and Yu, J
Oncogene, ISSN 0950-9232, 07/2017, Volume 36, Issue 28, pp. 4072 - 4080
Journal Article