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Journal Article
Annals of Neurology, ISSN 0364-5134, 03/1999, Volume 45, Issue 3, pp. 358 - 368
The distribution and density of neurofibrillary tangles and amyloid plaques was studied in a unique series of cases whose premortem cognitive status had been... 
POPULATION | DEMENTIA | CERAD | PATTERN | DIFFERENT AGE CATEGORIES | FREQUENCY | NEUROPATHOLOGY | NEURONAL LOSS | NEUROSCIENCES | LESIONS | CLINICAL NEUROLOGY | Neurofibrillary Tangles - pathology | Age Distribution | Plaque, Amyloid - pathology | Humans | Middle Aged | Aged, 80 and over | Brain Mapping | Female | Male | Aged | Alzheimer Disease - pathology
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 10/2017, Volume 134, Issue 4, pp. 619 - 628
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2000, Volume 25, Issue 4, pp. 402 - 405
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2010, Volume 30, Issue 49, pp. 16559 - 16566
Journal Article
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 2005, Volume 1739, Issue 2, pp. 216 - 223
Neurofibrillary tangles (NFT) are comprised of the microtubule-associated protein tau, in the form of filamentous aggregates. In addition to the well-known... 
Neurofibrillary tangle | Tau | Alzheimer's disease | NEUROFIBRILLARY TANGLES | PROTEIN-TAU | MICROTUBULE | BIOCHEMISTRY & MOLECULAR BIOLOGY | neurofibrillary tangle | tau | MILD COGNITIVE IMPAIRMENT | NEURODEGENERATIVE DISEASES | CELL-DEATH | IN-VITRO | BIOPHYSICS | NEURONAL DEATH | CASPASE-CLEAVAGE | PAIRED HELICAL FILAMENTS | Monomers
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 09/2017, Volume 139, Issue 38, pp. 13393 - 13403
The elucidation of the cause of Alzheimer’s disease remains one of the greatest questions in neurodegenerative research. The lack of highly reliable low-cost... 
MOUSE-BRAIN | BRAIN-BARRIER PERMEABILITY | NEUROFIBRILLARY TANGLES | PROTEIN | ALZHEIMERS-DISEASE | PAIRED HELICAL FILAMENTS | A-BETA | BASIS-SETS | MODEL | CHEMISTRY, MULTIDISCIPLINARY | OKADAIC ACID
Journal Article
Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 04/2015, Volume 74, Issue 4, pp. 319 - 344
ABSTRACTTo understand neuroinflammation-related gene regulation during normal aging and in sporadic Alzheimer disease (sAD), we performed functional genomics... 
Neuroinflammation | Tau | Sporadic Alzheimer disease | Cytokines | Genomics | Beta-amyloid | HUMAN BRAIN | AMYLOID-BETA-PROTEIN | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | TAU PATHOLOGY |