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Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 12/2013, Volume 28, Issue 12, pp. 2476 - 2489
Journal Article
Cell Reports, ISSN 2211-1247, 03/2016, Volume 14, Issue 8, pp. 1979 - 1990
To identify genes and signaling pathways that initiate Neurofibromatosis type 1 (NF1) neurofibromas, we used unbiased insertional mutagenesis screening, mouse... 
CELLS | ACTIVATION | GENE | PANCREATIC-CANCER | NF1 | NATURAL ANTISENSE TRANSCRIPTS | BETA-CATENIN | TYPE-1 | NERVE SHEATH TUMORS | PROGRESSION | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Carcinogenesis - metabolism | Neurofibromatosis 1 - metabolism | Neurofibromin 1 - metabolism | DNA Helicases - genetics | STAT3 Transcription Factor - genetics | Disease Models, Animal | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Carcinogenesis - genetics | Schwann Cells - pathology | Schwann Cells - metabolism | beta Catenin - metabolism | DNA Helicases - metabolism | Mice, Nude | Peripheral Nervous System Neoplasms - metabolism | Mice | Histones - metabolism | Neurofibromin 1 - genetics | RNA, Small Interfering - metabolism | Neoplasm Transplantation | Phosphorylation | Gene Expression Regulation, Neoplastic | Glycogen Synthase Kinase 3 beta - genetics | DNA-Binding Proteins - metabolism | Female | Neurofibromatosis 1 - genetics | Nuclear Proteins - genetics | Peripheral Nervous System Neoplasms - pathology | STAT3 Transcription Factor - metabolism | N-Terminal Acetyltransferase A - genetics | Glycogen Synthase Kinase 3 beta - antagonists & inhibitors | Nuclear Proteins - metabolism | N-Terminal Acetyltransferase A - antagonists & inhibitors | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Glycogen Synthase Kinase 3 beta - metabolism | Neural Stem Cells - pathology | beta Catenin - genetics | Carcinogenesis - pathology | Transcription Factors - metabolism | Neurofibromatosis 1 - pathology | Animals | Histones - genetics | N-Terminal Acetyltransferase A - metabolism | Mutagenesis, Insertional | STAT3 Transcription Factor - antagonists & inhibitors | Neural Stem Cells - metabolism | Peripheral Nervous System Neoplasms - genetics
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 01/2014, Volume 20, Issue 2, pp. 358 - 371
Journal Article
Cell Reports, ISSN 2211-1247, 01/2017, Volume 18, Issue 3, pp. 659 - 672
Journal Article
IUBMB Life, ISSN 1521-6543, 12/2017, Volume 69, Issue 12, pp. 918 - 928
Transforming growth factor (TGF)‐β inhibited membrane associated protein, TIMAP, is the member of the myosin phosphatase targeting protein (MYPT) family of... 
PPP1R16B | endothelial cells | MYPT family | TIMAP | angiogenesis | endothelial barrier | protein phosphatase 1 | RHO-ASSOCIATED KINASE | SIGNALING PATHWAYS | ERM PROTEINS | LIGHT-CHAIN PHOSPHATASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | BARRIER FUNCTION | 67-KDA LAMININ RECEPTOR | TARGETING SUBUNIT | CELL BIOLOGY | CONVERTING ENZYME-1 | TUMOR-SUPPRESSOR | MYOSIN PHOSPHATASE | Neoplasms - metabolism | Neurofibromin 2 - genetics | Humans | Glycogen Synthase Kinase 3 beta - genetics | Receptors for Activated C Kinase - genetics | Neoplasm Proteins - metabolism | Stroke - genetics | Stroke - pathology | Cyclic AMP-Dependent Protein Kinases - genetics | Neoplasms - genetics | Protein Phosphatase 1 - genetics | Receptors for Activated C Kinase - metabolism | Conserved Sequence | Membrane Proteins - metabolism | Hypertension - genetics | Neoplasm Proteins - genetics | Neurofibromin 2 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Gene Expression Regulation | Glycogen Synthase Kinase 3 beta - metabolism | Hypertension - pathology | Hypertension - metabolism | Protein Phosphatase 1 - metabolism | Stroke - metabolism | Animals | GTP Phosphohydrolases - metabolism | Endothelial Cells - cytology | GTP Phosphohydrolases - genetics | Protein Binding | Neoplasms - pathology | Proteins | Endothelin | Phosphatases | Myosin | Endothelium
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, p. 5893
Journal Article