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Nature Neuroscience, ISSN 1097-6256, 11/2012, Volume 15, Issue 11, pp. 1488 - 1497
FUS/TLS (fused in sarcoma/translocated in liposarcoma) and TDP-43 are integrally involved in amyotrophic lateral sclerosis (ALS) and frontotemporal dementia.... 
NEURODEGENERATIVE DISEASE | GENE | AMYOTROPHIC-LATERAL-SCLEROSIS | FAMILY PROTEINS | FUS PATHOLOGY | MUTATIONS | FRONTOTEMPORAL LOBAR DEGENERATION | BINDING | NEUROSCIENCES | BRAIN | NASCENT TRANSCRIPTION | RNA, Small Interfering - genetics | Protein Binding - genetics | Oligonucleotide Array Sequence Analysis | Humans | tau Proteins - metabolism | Gene Expression Profiling | RNA, Messenger - metabolism | Kv Channel-Interacting Proteins - metabolism | Brain - metabolism | Frontotemporal Dementia - metabolism | RNA Splicing - genetics | Frontotemporal Dementia - genetics | RNA-Binding Protein FUS - deficiency | Amyotrophic Lateral Sclerosis - genetics | Cell Cycle Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | RNA-Binding Protein FUS - genetics | Mice, Knockout | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Shal Potassium Channels - metabolism | Brain - pathology | Mice | Neurofilament Proteins - metabolism | RNA, Small Interfering - metabolism | Immunoprecipitation | Spinal Cord - metabolism | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | tau Proteins - genetics | Cell Cycle Proteins - genetics | Female | RNA Precursors - metabolism | Excitatory Amino Acid Transporter 2 - genetics | Membrane Proteins - metabolism | Frontotemporal Dementia - pathology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | RNA Precursors - genetics | Protein Structure, Tertiary - genetics | RNA-Binding Protein FUS - metabolism | DNA-Binding Proteins - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Histone-Lysine N-Methyltransferase - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Neural Cell Adhesion Molecules - metabolism | Neural Stem Cells - metabolism | Cell Line, Transformed | Amyotrophic lateral sclerosis | Development and progression | Genetic aspects | Messenger RNA | Health aspects
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 11/2012, Volume 124, Issue 5, pp. 733 - 747
While the pathogenesis of amyotrophic lateral sclerosis (ALS) remains to be clearly delineated, there is mounting evidence that altered RNA metabolism is a... 
Pathology | Neurosciences | Ubiquitination | Medicine & Public Health | Amyotrophic lateral sclerosis | Superoxide dismutase | RGNEF | C9orf72 | RNA binding proteins | HEXANUCLEOTIDE REPEAT EXPANSION | GENE-MUTATIONS | INCLUSIONS | AMYOTROPHIC-LATERAL-SCLEROSIS | PATHOLOGY | FRONTOTEMPORAL LOBAR DEGENERATION | LENGTH POLYGLUTAMINE EXPANSIONS | NEUROSCIENCES | CLINICAL NEUROLOGY | MESSENGER-RNA | PATHOLOGICAL TDP-43 | EXPRESSION | SOD1 MUTATIONS | Superoxide Dismutase - genetics | Immunoprecipitation | Membrane Glycoproteins - metabolism | Organic Chemicals | Sequestosome-1 Protein | Humans | Male | Transcription Factor TFIIIA - metabolism | DNA-Binding Proteins - metabolism | Guanine Nucleotide Exchange Factors - metabolism | Spinal Cord - pathology | Female | C9orf72 Protein | Superoxide Dismutase - metabolism | Peripherins | Gene Expression Regulation - genetics | Amyotrophic Lateral Sclerosis - genetics | RNA-Binding Protein FUS - genetics | RNA-Binding Protein FUS - metabolism | DNA-Binding Proteins - genetics | Mutation - genetics | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Proteins - genetics | Microscopy, Confocal | Amyotrophic Lateral Sclerosis - pathology | Proteins - metabolism | Amyotrophic Lateral Sclerosis - classification | Superoxide Dismutase-1 | Adaptor Proteins, Signal Transducing - metabolism | Neurofilament Proteins - metabolism | Intermediate Filament Proteins - metabolism | RNA-Binding Proteins - metabolism | Immunohistochemistry | Ubiquitin | RNA | Neurons | Chemical properties | Binding proteins | Intermediate filament proteins | Protein binding | Motor neurons | Antibodies | Colorimetry | Nucleotides | Metabolism | FUS protein | RNA-binding protein | Inclusion bodies | Neurofilaments | Peripherin | Mutation
Journal Article
Nature Neuroscience, ISSN 1097-6256, 11/2003, Volume 6, Issue 11, pp. 1162 - 1168
The generation of neurons from stem cells involves the activity of proneural basic helix-loop-helix (bHLH) proteins, but the mechanism by which these proteins... 
VENTRAL NEURAL-TUBE | SOX2 | NERVOUS-SYSTEM | STEM-CELLS | BHLH PROTEIN | GENE-EXPRESSION | NEURONAL DIFFERENTIATION | CHICK-EMBRYO | ACHAETE-SCUTE | FATE | NEUROSCIENCES | Helix-Loop-Helix Motifs - physiology | Homeodomain Proteins - metabolism | PAX7 Transcription Factor | Phosphopyruvate Hydratase - metabolism | DNA-Binding Proteins - metabolism | Stem Cells | Tubulin - metabolism | Time Factors | Gene Expression Regulation, Developmental | Neurons - metabolism | Bromodeoxyuridine - metabolism | Membrane Glycoproteins | Spinal Cord - cytology | SOXB1 Transcription Factors | Cell Differentiation - physiology | Basic Helix-Loop-Helix Transcription Factors | DNA-Binding Proteins - physiology | HMGB Proteins | High Mobility Group Proteins - metabolism | Drosophila | Embryonic Induction | Neuropeptides - metabolism | Nuclear Proteins - metabolism | Electroporation - methods | Chick Embryo | Proto-Oncogene Proteins c-myc - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Spinal Cord - embryology | Vertebrates | Animals | Eye Proteins - metabolism | Spinal Cord - physiology | Avian Proteins | LIM-Homeodomain Proteins | High Mobility Group Proteins - physiology | Mice | Nuclear Proteins - physiology | Neurofilament Proteins - metabolism | Physiological aspects | Research | DNA binding proteins | Neurons | Cell cycle
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2007, Volume 313, Issue 10, pp. 2098 - 2109
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2006, Volume 26, Issue 39, pp. 10006 - 10019
alpha-Internexin, a neuronal intermediate filament protein implicated in neurodegenerative disease, coexists with the neurofilament (NF) triplet proteins... 
Neurofilament | α-internexin | Tropical spastic paraparesis | Cytoskeleton | Neurofilament inclusion disease | Axonal transport | Intermediate filament | neurofilament inclusion disease | SLOW AXONAL-TRANSPORT | MARIE-TOOTH-DISEASE | cytoskeleton | CELL-SURFACE EXPRESSION | tropical spastic paraparesis | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | NEUROSCIENCES | INTERMEDIATE-FILAMENT PROTEIN | intermediate filament | NF-L | INCLUSION DISEASE | neurofilament | axonal transport | alpha-internexin | TRANSGENIC MICE | Intermediate Filament Proteins - ultrastructure | Multiprotein Complexes | Recombinant Fusion Proteins - analysis | Recombinant Fusion Proteins - physiology | Male | Structure-Activity Relationship | Spinal Cord - ultrastructure | Microscopy, Immunoelectron | Nerve Degeneration - metabolism | Neurofilament Proteins - genetics | Spinal Cord - chemistry | Transfection | Neurofilament Proteins - analysis | Axons - ultrastructure | Intermediate Filament Proteins - genetics | Female | Retinal Ganglion Cells - chemistry | Retinal Ganglion Cells - ultrastructure | Mice, Inbred C57BL | Intermediate Filament Proteins - physiology | Rats | Axons - chemistry | Intermediate Filament Proteins - analysis | Nerve Degeneration - pathology | Intermediate Filaments - chemistry | Mice, Knockout | Protein Interaction Mapping | Protein Transport | Microscopy, Confocal | Animals | Neurofilament Proteins - physiology | Intermediate Filament Proteins - deficiency | Intermediate Filaments - ultrastructure | Neurofilament Proteins - deficiency | Neurofilament Proteins - ultrastructure | Mice | Microscopy, Fluorescence | Crosses, Genetic
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 15, pp. 9753 - 9766
Journal Article
NeuroMolecular Medicine, ISSN 1535-1084, 3/2006, Volume 8, Issue 1, pp. 217 - 241
We review the putative functions and malfunctions of proteins encoded by genes mutated in Charcot-Marie-Tooth disease (CMT; inherited motor and sensory... 
myelin | Neurology | Neurosciences | Biomedicine | Schwann cell | Hereditary neuropathies | Internal Medicine | axon | Axon | Myelin | Cytoskeletal Proteins - genetics | Demyelinating Diseases - genetics | Early Growth Response Protein 2 - genetics | Humans | Protein Tyrosine Phosphatases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Charcot-Marie-Tooth Disease - genetics | Heat-Shock Proteins - genetics | Neurofilament Proteins - genetics | Protein Tyrosine Phosphatases - genetics | Mitochondrial Proteins - metabolism | SOXE Transcription Factors | Dynamin II - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | Connexins - genetics | Charcot-Marie-Tooth Disease - pathology | Protein Tyrosine Phosphatases, Non-Receptor | Myelin Proteins - genetics | Connexins - metabolism | Myelin Proteolipid Protein - genetics | Lamin Type A - genetics | Charcot-Marie-Tooth Disease - physiopathology | Mutation | Myelin Proteins - metabolism | Neurofilament Proteins - metabolism | GTP Phosphohydrolases | rab GTP-Binding Proteins - genetics | Mitochondrial Proteins - genetics | Myelin Sheath - metabolism | DNA-Binding Proteins - metabolism | Myelin Proteolipid Protein - metabolism | Cell Cycle Proteins - genetics | Kinesin - genetics | Cytoskeletal Proteins - metabolism | Membrane Proteins - metabolism | Demyelinating Diseases - pathology | Nuclear Proteins - genetics | rab GTP-Binding Proteins - metabolism | Demyelinating Diseases - physiopathology | High Mobility Group Proteins - metabolism | Heat-Shock Proteins - metabolism | Dynamin II - genetics | Nuclear Proteins - metabolism | Lamin Type A - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Endocytosis - physiology | Kinesin - metabolism | Protein Transport | Nerve Tissue Proteins - metabolism | Proteins - genetics | Transcription Factors - metabolism | Proteins - metabolism | Myelin Sheath - genetics | Myelin P0 Protein - genetics | Early Growth Response Protein 2 - metabolism | Myelin P0 Protein - metabolism | High Mobility Group Proteins - genetics | Gene mutations | Neurons | Proteolysis | Charcot-Marie-Tooth disease | Aquaporins | Development and progression | Protein biosynthesis | Genetic transcription | Peripheral nerves | Schwann cells | Lysosomes | Connexin 32 | Neuropathy | Sox10 protein | EGR-2 protein | Axons | Reviews | Demyelination | Dynamin | Neurofilaments | Myelin P0 protein | Peripheral myelin protein 22
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 6, pp. 1794 - 1818
Multiple sclerosis involves demyelination and axonal degeneration of the central nervous system. The molecular mechanisms of axonal degeneration are relatively... 
collapsin response mediator protein 2 | Nogo receptor | axonal degeneration | experimental autoimmune encephalomyelitis | Nogo-A | ENCEPHALOMYELITIS | FUNCTIONAL RECOVERY | RESPONSE MEDIATOR PROTEIN-2 | ALZHEIMERS-DISEASE | AXONAL REGENERATION | RHO-KINASE | NEUROSCIENCES | LESIONS | CLINICAL NEUROLOGY | MYELIN OLIGODENDROCYTE GLYCOPROTEIN | HYPERPHOSPHORYLATION | INHIBITOR | Humans | Middle Aged | Myelin Proteins - deficiency | Encephalomyelitis, Autoimmune, Experimental - immunology | tau Proteins - metabolism | Male | Green Fluorescent Proteins - genetics | Receptors, Cell Surface - antagonists & inhibitors | Nerve Degeneration - metabolism | Retinal Ganglion Cells - pathology | Time Factors | Nogo Receptor 1 | GPI-Linked Proteins - antagonists & inhibitors | Disease Models, Animal | Transduction, Genetic | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Myelin Proteins - antagonists & inhibitors | Silver Staining | Myelin-Oligodendrocyte Glycoprotein | Glycoproteins - adverse effects | Mutation - genetics | CD3 Complex - metabolism | Receptors, Cell Surface - immunology | Mice, Knockout | Analysis of Variance | Axons - pathology | Encephalomyelitis, Autoimmune, Experimental - complications | Receptors, Cell Surface - deficiency | Cell Line, Tumor | Mice | Peptide Fragments - adverse effects | Nerve Degeneration - etiology | Neurofilament Proteins - metabolism | Optic Nerve - metabolism | Optic Nerve - pathology | Phosphorylation | Immunoprecipitation | Spinal Cord - metabolism | Demyelinating Diseases - metabolism | Retinal Ganglion Cells - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Tubulin - metabolism | GPI-Linked Proteins - deficiency | Spinal Cord - pathology | Axons - ultrastructure | Myelin Proteins - immunology | Adult | Female | Demyelinating Diseases - etiology | Demyelinating Diseases - pathology | Neuroblastoma - pathology | Severity of Illness Index | Antibodies - therapeutic use | Green Fluorescent Proteins - metabolism | GPI-Linked Proteins - immunology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Axons - metabolism | Nerve Tissue Proteins - genetics | Multiple Sclerosis - complications | Nerve Tissue Proteins - metabolism | Animals | Multiple Sclerosis - pathology | Original
Journal Article
Journal Article