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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2017, Volume 114, Issue 3, pp. E396 - E405
Astrocytes are believed to bridge interactions between infiltrating lymphocytes and neurons during brain ischemia, but the mechanisms for this action are... 
Ischemic brain injury | Interleukin-15 | Inflammation | Astrocyte | Lymphocytes | FUNCTIONAL RECOVERY | NATURAL-KILLER-CELLS | ACTIVATION | lymphocytes | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | IL-15 | interleukin-15 | INTERFERON-GAMMA | inflammation | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | astrocyte | T-CELLS | ischemic brain injury | Humans | Brain Ischemia - metabolism | Male | Mice, Transgenic - metabolism | Brain Injuries - metabolism | Brain Ischemia - immunology | Glial Fibrillary Acidic Protein - immunology | Glial Fibrillary Acidic Protein - metabolism | Interleukin-15 - immunology | Brain - metabolism | Astrocytes - immunology | Brain Injuries - immunology | Neuroglia - immunology | Aged, 80 and over | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Female | Neurons - metabolism | Stroke - immunology | Disease Models, Animal | Mice, Inbred C57BL | Neurons - immunology | Mice, Transgenic - immunology | Immunity, Cellular - immunology | Stroke - metabolism | Animals | Up-Regulation - immunology | Interleukin-15 - metabolism | Neuroglia - metabolism | Mice | Killer Cells, Natural - metabolism | Promoter Regions, Genetic - immunology | CD8-Positive T-Lymphocytes - immunology | Brain - immunology | Astrocytes - metabolism | Physiological aspects | Brain | Interleukins | Astrocytes | Health aspects | Injuries | Traumatic brain injury | Cytokines | Propagation | Ischemia | Cells | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Nature, ISSN 0028-0836, 07/2016, Volume 535, Issue 7612, pp. 440 - 443
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2008, Volume 106, Issue 1, pp. 158 - 169
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2014, Volume 193, Issue 4, pp. 1911 - 1919
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 05/2017, Volume 24, Issue 5, pp. 832 - 843
Journal Article
JNEN: Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 11/2002, Volume 61, Issue 11, pp. 1013 - 1021
The family of Toll-like receptors (TLRs) plays a key role in controlling innate immune responses to a wide variety of pathogen-associated molecules. In this... 
Toll-like receptors | Multiple sclerosis | Astrocytes | Central nervous system | Oligodendrocytes | Microglia | CELL-WALL COMPONENTS | SIGNALING PATHWAYS | ACTIVATION | DIFFERENTIAL EXPRESSION | RECOGNITION | multiple sclerosis | INNATE IMMUNE-RESPONSE | oligodendrocytes | PATHOLOGY | COOPERATION | NEUROSCIENCES | CLINICAL NEUROLOGY | central nervous system | astrocytes | microglia | NF-KAPPA-B | Immunohistochemistry | Microglia - metabolism | Oligodendroglia - metabolism | Toll-Like Receptor 2 | Toll-Like Receptor 3 | Central Nervous System - metabolism | Toll-Like Receptor 4 | Membrane Glycoproteins - metabolism | Humans | Middle Aged | Astrocytes - pathology | Central Nervous System - pathology | Male | Central Nervous System Infections - immunology | RNA, Messenger - metabolism | Central Nervous System Infections - metabolism | Central Nervous System - immunology | Toll-Like Receptors | Oligodendroglia - immunology | Microglia - immunology | Astrocytes - immunology | Encephalitis - metabolism | Neuroglia - immunology | Aged, 80 and over | Immune System - immunology | Female | Membrane Glycoproteins - immunology | Multiple Sclerosis - metabolism | Drosophila Proteins | Gene Expression Regulation - immunology | Cells, Cultured | Encephalitis - pathology | Receptors, Cell Surface - metabolism | Encephalitis - immunology | Receptors, Cell Surface - immunology | Membrane Glycoproteins - genetics | Oligodendroglia - pathology | Immune System - metabolism | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Neuroglia - metabolism | Aged | Central Nervous System Infections - pathology | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
Journal of Neurology, Neurosurgery and Psychiatry, ISSN 0022-3050, 06/2017, Volume 88, Issue 6, pp. 465 - 473
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 1, pp. 176 - 187.e1
Background & Aims Postoperative ileus (POI) is a common consequence of abdominal surgery that increases the risk of postoperative complications and morbidity.... 
Gastroenterology and Hepatology | Inflammatory Response | Gastrointestinal Dysmotility | Pathogen Recognition | Innate Immunity | CELLS | STERILE INFLAMMATION | MUSCULARIS | GASTROINTESTINAL-TRACT | BACTERIAL TRANSLOCATION | IL-1-ALPHA | INFLAMMATORY-BOWEL-DISEASE | NITRIC-OXIDE | NEURONS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Ileus - metabolism | Interleukin-18 - immunology | Myenteric Plexus - immunology | Receptors, Interleukin-1 Type I - immunology | Postoperative Complications - metabolism | Myenteric Plexus - metabolism | Muscle, Smooth - immunology | Postoperative Complications - immunology | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1 - immunology | Myeloid Differentiation Factor 88 - immunology | Neuroglia - immunology | Toll-Like Receptors - metabolism | Ileus - immunology | Disease Models, Animal | Interleukin-1 - metabolism | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Myeloid Differentiation Factor 88 - genetics | Mice, Transgenic | Muscle, Smooth - metabolism | Gastrointestinal Motility - immunology | Animals | Toll-Like Receptors - genetics | Receptors, Interleukin-1 Type I - genetics | Neuroglia - metabolism | Mice | Interleukin-18 - genetics | Myeloid Differentiation Factor 88 - metabolism | Interleukin-18 - metabolism | Medical colleges | Intestines | Obstructions | Neurosciences | Interleukins | Index Medicus | Abridged Index Medicus
Journal Article
Molecular and Cellular Neuroscience, ISSN 1044-7431, 11/2003, Volume 24, Issue 3, pp. 623 - 631
We have recently shown that the inflammatory process during experimental allergic encephalomyelitis (EAE), the animal model of MS, attracts transplanted NPC... 
PRIMARY DEMYELINATION | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | STEM-CELLS | ADULT-RAT | OLIGODENDROCYTE PROGENITORS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | NECROSIS-FACTOR-ALPHA | SUBVENTRICULAR ZONE | NEUROSCIENCES | TRANSGENIC MICE | Stem Cells - immunology | Apoptosis - drug effects | Rats, Inbred Lew | Stem Cells - metabolism | Cell Lineage - drug effects | Neuroglia - drug effects | Cell Movement - immunology | Multiple Sclerosis - physiopathology | Cell Division - immunology | Inflammation Mediators - pharmacology | Neuroglia - immunology | Tumor Necrosis Factor-alpha - immunology | Neurons - metabolism | Neurons - drug effects | Spheroids, Cellular | Cytokines - immunology | Multiple Sclerosis - metabolism | Animals, Newborn | Inflammation Mediators - immunology | Biomarkers - analysis | Cells, Cultured | Neurons - immunology | Rats | Cell Division - drug effects | Nerve Tissue Proteins - metabolism | Cell Differentiation - immunology | Cell Movement - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Apoptosis - immunology | Cell Differentiation - drug effects | Models, Biological | Interferon-gamma - immunology | Multiple Sclerosis - immunology | Stem Cells - drug effects | Neuroglia - metabolism | Cytokines - pharmacology | Interferon-gamma - pharmacology | Cell Lineage - immunology | Interferon | Interleukins | Biological response modifiers | Growth | Cell death | Analysis | Jewish schools | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 11, pp. 4923 - 4934
The Alzheimer's disease (AD) process is understood to involve the accumulation of amyloid plaques and tau tangles in the brain. However, attempts at targeting... 
CASCADE HYPOTHESIS | IMMUNE-RESPONSE | A-BETA IMMUNIZATION | ANTIBODY-PRODUCTION | MOUSE MODEL | AMYLOID-BETA-PROTEIN | CENTRAL-NERVOUS-SYSTEM | NEUROSCIENCES | PEPTIDE | OLIGOMERS | TRANSGENIC MICE | Surface Plasmon Resonance | Dose-Response Relationship, Immunologic | Age Factors | Antibodies, Anti-Idiotypic - pharmacology | Humans | Macaca fascicularis | Male | Epitopes, B-Lymphocyte - immunology | Immunologic Memory - drug effects | Alzheimer Disease - pathology | Neuroglia - drug effects | Brain - metabolism | Vaccination - methods | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Peptide Fragments - immunology | Amyloid beta-Peptides - metabolism | Neuroglia - immunology | Female | Epitopes, T-Lymphocyte - immunology | Alzheimer Disease - immunology | Immunologic Memory - immunology | Disease Models, Animal | Antibody Formation - immunology | Peptide Fragments - metabolism | Guinea Pigs | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Plaque, Amyloid - pathology | Antibodies, Anti-Idiotypic - blood | Alzheimer Disease - therapy | Cells, Cultured | Vaccines - immunology | Mice, Transgenic | Mutation - genetics | Protein Binding - immunology | Peptide Fragments - chemistry | Amyloid beta-Protein Precursor - genetics | Animals | Analysis of Variance | Epitopes, B-Lymphocyte - metabolism | Brain - pathology | Amyloid beta-Peptides - immunology | Plaque, Amyloid - metabolism | T-Lymphocytes - immunology | Mice | Amyloid beta-Peptides - chemistry | Alzheimer Disease - genetics | Index Medicus
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2006, Volume 21, Issue 5, pp. 599 - 616
Abstract Chronic constriction injury (CCI) of the sciatic nerve in rodents produces mechanical and thermal hyperalgesia and is a common model of neuropathic... 
Psychiatry | Allergy and Immunology | MHC II | CD4+ macrophages | OX-42 (CD11b) | CD8+ T-cells | ED1 (CD68) | Neuroinflammation | Astrocyte | Macrophage | Microglia | MHC I | MICROGLIAL ACTIVATION | PRIMARY AFFERENT NEURONS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | macrophage | FACTOR RECEPTOR-1 | PAINFUL PERIPHERAL NEUROPATHY | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | NEUROSCIENCES | SENSORY NEURONS | neuroinflammation | CD4+macrophages | DIFFERENTIAL ACTIVATION | FACIAL MOTOR NUCLEUS | CD8+T-cells | SODIUM-CHANNEL EXPRESSION | microglia | astrocyte | Adaptation, Physiological | Gait - immunology | Sciatic Nerve - injuries | Male | Ganglia, Spinal - cytology | Activating Transcription Factor 3 - immunology | Sciatic Nerve - immunology | Axotomy | Sciatic Nerve - metabolism | Constriction, Pathologic - immunology | Inflammation Mediators - metabolism | Neuroglia - immunology | Neurons, Afferent - immunology | Sciatic Nerve - cytology | Spinal Cord - cytology | Calcitonin Gene-Related Peptide - metabolism | Macrophages - immunology | Ganglia, Spinal - immunology | Motor Neurons - immunology | Rats | Activating Transcription Factor 3 - metabolism | Rats, Sprague-Dawley | Motor Neurons - metabolism | Spinal Cord - immunology | Macrophages - metabolism | Animals | Analysis of Variance | Lymphocyte Subsets | Neuroglia - metabolism | Lumbar Vertebrae | Neurons, Afferent - metabolism | Index Medicus
Journal Article