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Journal of Neuroscience, ISSN 0270-6474, 09/2012, Volume 32, Issue 39, pp. 13454 - 13469
Abnormal deposition and intercellular propagation of alpha-synuclein plays a central role in the pathogenesis of disorders such as Parkinson's Disease (PD) and... 
IMMUNIZATION | PROTEIN | PLASMA | BIOMARKER | ALZHEIMERS-DISEASE | MOUSE MODEL | PATHOLOGY | FC-GAMMA-RIIA | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | PARKINSONS-DISEASE | Synaptic Transmission - physiology | Antibodies - metabolism | Lewy Body Disease - immunology | Humans | alpha-Synuclein - immunology | Extracellular Space - drug effects | Nerve Degeneration - genetics | Cell Communication - physiology | Culture Media, Conditioned - pharmacology | Phosphopyruvate Hydratase - metabolism | Amyloid - ultrastructure | Antigens, CD - metabolism | Neuroglia - drug effects | Brain - metabolism | Lewy Body Disease - genetics | Extracellular Space - metabolism | Amyloid - metabolism | Microfilament Proteins - metabolism | alpha-Synuclein - genetics | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Cell Line | Microscopy, Electron, Transmission | Cytokines - metabolism | Mice, Transgenic | Nerve Degeneration - immunology | Cathepsin D - metabolism | Extracellular Space - immunology | Antibodies - pharmacology | Caveolin 1 - metabolism | Lewy Body Disease - metabolism | Platelet-Derived Growth Factor - metabolism | Animals | Analysis of Variance | Brain - pathology | Immunization, Passive | Neuroglia - metabolism | Mice | Chromatography, Gel | alpha-Synuclein - metabolism | Nerve Degeneration - drug therapy | Astrocytes - metabolism | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Index Medicus | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Neuron, ISSN 0896-6273, 06/2018, Volume 98, Issue 6, pp. 1141 - 1154.e7
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2014, Volume 111, Issue 43, pp. 15579 - 15584
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 2010, Volume 78, Issue 3, pp. 466 - 477
Parkinson's disease (PD) is the second most common neurodegenerative disease characterized by a progressive loss of dopamine (DA) neurons in the substantia... 
RESPONSES | IN-VITRO | MICROGLIAL ACTIVATION | PHOSPHORYLATION | GENE-EXPRESSION | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | NECROSIS-FACTOR-ALPHA | NADPH OXIDASE ACTIVITY | PARKINSONS-DISEASE | DEGENERATION | Microglia - metabolism | Mesencephalon - cytology | Reactive Oxygen Species - metabolism | Embryo, Mammalian | Rats, Inbred F344 | NADPH Oxidases - metabolism | Lipopolysaccharides - antagonists & inhibitors | Anti-Inflammatory Agents - metabolism | Nerve Degeneration - metabolism | Neuroprotective Agents - metabolism | Microglia - physiology | Neuroprotective Agents - pharmacology | Stilbenes | Dopamine - physiology | Anti-Inflammatory Agents - therapeutic use | Polyphenols | Neurons - metabolism | Dopamine - metabolism | Phenols - pharmacology | Anti-Inflammatory Agents - pharmacology | NADPH Oxidases - antagonists & inhibitors | Rats | Reactive Oxygen Species - therapeutic use | Lipopolysaccharides - pharmacology | Mitogen-Activated Protein Kinases - pharmacology | Neurotoxicity Syndromes - metabolism | Mice | NADPH Oxidases - physiology | Neuroprotective Agents - therapeutic use | Mesencephalon - metabolism | Reactive Oxygen Species - pharmacology | Lipopolysaccharides - metabolism | Parkinson Disease - drug therapy | Substantia Nigra - metabolism | Flavonoids - therapeutic use | Neuroglia - drug effects | Nerve Degeneration - prevention & control | Neurons - physiology | Female | Flavonoids - pharmacology | Parkinson Disease - metabolism | Neurons - drug effects | Microglia - drug effects | Mice, Inbred C57BL | Dopamine - pharmacology | Phenols - metabolism | Pregnancy | Animals | Flavonoids - metabolism | Phenols - therapeutic use | Neuroglia - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 08/2014, Volume 63, Issue 8, pp. 1300 - 1312
Objective Enteric glia activation has been reported to amplify intestinal inflammation via the enteroglial-specific S100B protein. This neurotrophin promotes... 
NITRIC-OXIDE PRODUCTION | INHIBITION | CROHNS-DISEASE | MACROPHAGES | ULCERATIVE-COLITIS | FATTY-ACID AMIDE | S100B PROTEIN | N-PALMITOYL-ETHANOLAMINE | GLIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rectum - pathology | Humans | Middle Aged | Palmitic Acids - therapeutic use | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Nitric Oxide - analysis | Colon, Sigmoid - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Neuroglia - drug effects | Colitis - chemically induced | Female | Indoles - pharmacology | Colitis - drug therapy | S100 Calcium Binding Protein beta Subunit - metabolism | Colitis, Ulcerative - drug therapy | Palmitic Acids - pharmacology | Colon, Sigmoid - pathology | Endocannabinoids - therapeutic use | Neutrophil Infiltration - drug effects | Severity of Illness Index | PPAR alpha - antagonists & inhibitors | Signal Transduction | Colitis, Ulcerative - metabolism | Cells, Cultured | Colitis, Ulcerative - pathology | Toll-Like Receptor 4 - metabolism | Dinoprostone - metabolism | Nerve Tissue Proteins - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anilides - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Rectum - chemistry | Ethanolamines - therapeutic use | Neuroglia - metabolism | Mice | PPAR alpha - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Influence | Amides | Inflammation | Neuroglia | Analysis | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 10/2018, Volume 73, pp. 520 - 532
Functional pain syndromes, such as fibromyalgia and temporomandibular disorder, are associated with enhanced catecholamine tone and decreased levels of... 
Allodynia | Epinephrine | Mitogen-activated protein kinase (MAPK) | Catecholamines | Neuroinflammation | Astrocyte | Extracellular signal-regulated kinases (ERK) | Chronic pain | Hyperalgesia | Microglia | Beta-adrenergic receptor (βAR) | AR | p38 | Catechol-O-methyltransferase (COMT) | Beta 2-adrenergic receptor (β | Idiopathic pain | Norepinephrine | Tumor necrosis factor alpha (TNFα) | Beta 3-adrenergic receptor (β | Functional pain | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Phosphorylation | Spinal Cord - metabolism | Receptors, Adrenergic, beta-2 - physiology | Receptors, Adrenergic, beta-3 - drug effects | Male | Receptors, Adrenergic, beta-2 - drug effects | Pain - metabolism | Catechol O-Methyltransferase Inhibitors - metabolism | Catechol O-Methyltransferase - metabolism | Temporomandibular Joint Disorders - metabolism | Temporomandibular Joint Disorders - physiopathology | Interleukin-1beta - metabolism | Catechols - pharmacology | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Receptors, Adrenergic, beta - metabolism | Interleukin-6 - metabolism | Receptors, Adrenergic, beta-3 - physiology | Mitogen-Activated Protein Kinases | Fibromyalgia - metabolism | Cytokines - metabolism | Hyperalgesia - metabolism | Rats | Imidazoles - pharmacology | Receptors, Adrenergic, beta-3 - metabolism | Rats, Sprague-Dawley | Receptors, Adrenergic, beta-2 - metabolism | Animals | Pain - physiopathology | Etanercept - pharmacology | Neuroglia - metabolism | Pyridines - pharmacology | Fibromyalgia - physiopathology | Propanolamines - pharmacology
Journal Article
Glia, ISSN 0894-1491, 01/2016, Volume 64, Issue 1, pp. 90 - 104
The glycosphingolipid SSEA‐4 and the glycoprotein YKL‐40 have both been associated with human embryonic and neural stem cell differentiation. We investigated... 
human neural stem cells | cerebral cortex | corticogenesis | development | astrocytes | Development | Cerebral cortex | Corticogenesis | Astrocytes | Human neural stem cells | HUMAN TELENCEPHALON | LISSENCEPHALIC PRIMATE | RADIAL GLIA | NEUROSCIENCES | IN-VITRO | HUMAN FOREBRAIN | MAMMALIAN CEREBRAL-CORTEX | GLYCOPROTEIN YKL-40 | EMBRYONIC STEM-CELLS | BRAIN BARRIERS | PRECURSOR CELLS | Immunohistochemistry | Neocortex - embryology | Antigens, Nuclear - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Calbindins - metabolism | Neurons - cytology | Neural Stem Cells - cytology | Antigens, CD - metabolism | Chitinase-3-Like Protein 1 | DNA-Binding Proteins - metabolism | Lectins - metabolism | Neocortex - metabolism | Neuroglia - cytology | Stage-Specific Embryonic Antigens - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neurons - metabolism | Tumor Suppressor Proteins - metabolism | Fatty Acid-Binding Protein 7 | Neuropeptides - metabolism | S100 Proteins - metabolism | Adipokines - metabolism | Oligodendrocyte Transcription Factor 2 | T-Box Domain Proteins - metabolism | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Carrier Proteins - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Neocortex - cytology | Neuroglia - metabolism | Stem Cell Niche - physiology | Neural Stem Cells - metabolism | Localization | Rodents | Stem cells | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 7602 - 8
Verteporfin (VP), a light-activated drug used in photodynamic therapy for the treatment of choroidal neovascular membranes, has also been shown to be an... 
TRANSCRIPTION FACTORS | BREAST-CANCER | PHOTODYNAMIC THERAPY | CANCER CELLS | MULTIDISCIPLINARY SCIENCES | HIPPO PATHWAY | OVARIAN-CANCER | YAP-TEAD | EXPRESSION | OPTIC PATHWAY GLIOMA | NERVE GLIOMA | Neuroglia - pathology | Humans | Verteporfin - pharmacology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Octamer Transcription Factor-3 - genetics | Photosensitizing Agents - pharmacology | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Survivin - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - agonists | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Receptor Protein-Tyrosine Kinases - metabolism | Cysteine-Rich Protein 61 - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Cell Line, Tumor | Connective Tissue Growth Factor - genetics | Proto-Oncogene Proteins c-myc - genetics | Connective Tissue Growth Factor - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Neoplastic | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | Cysteine-Rich Protein 61 - metabolism | Neuroglia - drug effects | DNA-Binding Proteins - metabolism | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Nuclear Proteins - genetics | Proto-Oncogene Proteins - metabolism | Survivin - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Connective Tissue Growth Factor - antagonists & inhibitors | Cysteine-Rich Protein 61 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Octamer Transcription Factor-3 - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - metabolism | Transcription Factors - metabolism | Receptor Protein-Tyrosine Kinases - genetics | Nuclear Proteins - antagonists & inhibitors | Octamer Transcription Factor-3 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Neuroglia - metabolism | Cell Proliferation - drug effects | Adaptor Proteins, Signal Transducing - metabolism | TOR protein | Photodynamic therapy | Brain tumors | Oct-4 protein | Glioblastoma | c-Myc protein | MAP kinase | Connective tissue growth factor | AKT protein | Survivin | Myc protein | Tumor cell lines | Ovarian cancer | Axl protein | Yes-associated protein | Cell activation | Glioma cells | CYR61 protein | Retinoblastoma | Pluripotency | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e36949 - e36949
Many molecular and cellular abnormalities detected in the diabetic retina support a role for IL-1 beta-driven neuroinflammation in the pathogenesis of diabetic... 
ADHESION MOLECULE-1 | PROTEIN-KINASE-C | MICROGLIAL ACTIVATION | ENDOTHELIAL-CELLS | BIOLOGY | GENE-EXPRESSION | RETINOPATHY | INDUCTION | INTERLEUKIN-1 INDUCES INTERLEUKIN-1 | MULLER CELLS | BRAIN | Microglia - metabolism | Up-Regulation | Retina - metabolism | Rats, Wistar | Diabetes Mellitus, Experimental - genetics | Male | Interleukin-1beta - genetics | Glial Fibrillary Acidic Protein - metabolism | Hyperglycemia - genetics | Glucose - administration & dosage | Interleukin-1beta - metabolism | Protein Kinase C - metabolism | Diabetes Mellitus, Experimental - metabolism | Interleukin-1beta - antagonists & inhibitors | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rats | Protein Kinase C - antagonists & inhibitors | Random Allocation | Rats, Sprague-Dawley | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Hyperglycemia - metabolism | alpha-Macroglobulins - metabolism | Animals | Endothelium - metabolism | Ceruloplasmin - metabolism | Glucose - metabolism | Neuroglia - metabolism | Astrocytes - metabolism | Ceruloplasmin | Diabetic retinopathy | Protein kinase C | Retinopathy | Pathogenesis | Streptozocin | Retina | Immunoblotting | Glucose | Hyperglycemia | Ischemia | Rodents | Enzyme-linked immunosorbent assay | Enzymes | Cytokines | Astrocytes | Abnormalities | Diabetes mellitus | Blood vessels | Inflammation | IL-1β | Endothelial cells | Endothelium | Microglia | Polymerase chain reaction | Brain research | Diabetes | Glia | Laboratory animals | Subcultures | Apoptosis | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2005, Volume 8, Issue 6, pp. 723 - 729
The cerebellum is critical for motor coordination and cognitive function and is the target of transformation in medulloblastoma, the most common malignant... 
IN-VITRO | WHITE-MATTER | SONIC HEDGEHOG | NEURONS | CNS | CENTRAL-NERVOUS-SYSTEM | GROWTH-FACTOR | PRECURSOR CELLS | NEUROSCIENCES | MOUSE CEREBELLUM | BRAIN-TUMORS | Oligodendroglia - metabolism | Multipotent Stem Cells - metabolism | Glycoproteins - metabolism | Peptides - genetics | Nerve Growth Factors - metabolism | Spheroids, Cellular - cytology | Cerebellum - physiology | Cell Lineage - drug effects | Hedgehog Proteins | Nerve Growth Factors - pharmacology | Stem Cell Transplantation | Neuroglia - cytology | Multipotent Stem Cells - drug effects | Peptides - metabolism | Interneurons - cytology | Spheroids, Cellular - drug effects | Trans-Activators - pharmacology | Oligodendroglia - cytology | Medulloblastoma - genetics | Cell Differentiation - physiology | Basic Helix-Loop-Helix Transcription Factors | Animals, Newborn | Astrocytes - cytology | Biomarkers - metabolism | Glycoproteins - genetics | Cell Separation | Cerebellum - metabolism | Spheroids, Cellular - metabolism | Medulloblastoma - physiopathology | Mice, Transgenic | Medulloblastoma - metabolism | Transcription Factors - genetics | AC133 Antigen | Cell Lineage - physiology | Nerve Tissue Proteins - metabolism | Animals | Interneurons - metabolism | Cell Differentiation - drug effects | Multipotent Stem Cells - cytology | Cerebellum - cytology | Neuroglia - metabolism | Trans-Activators - metabolism | Mice | Antigens, CD | Astrocytes - metabolism | Cerebellum | Cell proliferation | Stem cell research | Neural transmission | Regulation | Research | Structure | Index Medicus
Journal Article