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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2013, Volume 127, Issue 1, pp. 109 - 135
... broad definition of pathology, we here review the “neuropa for any individual. In fact, stress appears to be increas - thology of stress” and focus on structural... 
Brain | Pituitary ACTH Hypersecretion | Animals | Pituitary-Adrenal System | Neurons | Humans | Mental Disorders | Neuroglia | Stress, Psychological | Hypothalamo-Hypophyseal System | Alzheimer Disease | Mood disorder | PTSD | Pathology | Neurosciences | Medicine & Public Health | Amygdala | HPA axis | Prefrontal cortex | Hippocampus | CORTICOTROPIN-RELEASING HORMONE | CHRONIC PSYCHOSOCIAL STRESS | RECEPTOR MESSENGER-RNA | EARLY-LIFE STRESS | ADULT HIPPOCAMPAL NEUROGENESIS | PATHOLOGY | NEUROSCIENCES | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | CLINICAL NEUROLOGY | PROLONGED GLUCOCORTICOID EXPOSURE | MEDIAL PREFRONTAL CORTEX | MAJOR DEPRESSIVE DISORDER | NEURAL PROGENITOR CELLS | Neurons - pathology | Alzheimer Disease - physiopathology | Mental Disorders - pathology | Pituitary ACTH Hypersecretion - pathology | Pituitary-Adrenal System - physiopathology | Hypothalamo-Hypophyseal System - pathology | Neuroglia - pathology | Brain - physiopathology | Neuroglia - physiology | Pituitary-Adrenal System - pathology | Mental Disorders - immunology | Stress, Psychological - immunology | Alzheimer Disease - pathology | Mental Disorders - physiopathology | Stress, Psychological - pathology | Pituitary ACTH Hypersecretion - physiopathology | Neurons - physiology | Brain - pathology | Brain - immunology | Hypothalamo-Hypophyseal System - physiopathology | Stress, Psychological - physiopathology | Cognition | Corticosteroids | Post-traumatic stress disorder | Depressive Disorders | Neuroendocrinology | Cognitive Disorders | Neuroanatomy | Brain Imaging | Stressors | Prefrontal Cortex | Review
Journal Article
Brain (London, England : 1878), ISSN 1460-2156, 2013, Volume 136, Issue 8, pp. 2510 - 2526
Journal Article
The Lancet (British edition), ISSN 0140-6736, 2016, Volume 388, Issue 10047, pp. 898 - 904
Summary Background Zika virus is an arthropod-borne virus that is a member of the family Flaviviridae transmitted mainly by mosquitoes of the genus Aedes .... 
Internal Medicine | ETIOLOGY | MEDICINE, GENERAL & INTERNAL | STATES | VIRUS-INFECTION | ARTHROGRYPOSIS | UPDATE | TISSUES | BRAIN | PCR | Pregnancy Trimester, Second | Limb Deformities, Congenital - virology | Pregnancy Complications, Infectious - virology | Neuroglia - pathology | Zika Virus Infection - congenital | Humans | Abortion, Spontaneous - virology | Brain - virology | Infant | Male | Microcephaly - virology | Pregnancy Trimester, First | Pregnancy Trimester, Third | Antigens, Viral - isolation & purification | Autopsy | Microcephaly - pathology | Fatal Outcome | Brazil | Adult | Female | RNA, Viral - isolation & purification | Zika Virus - immunology | Zika Virus Infection - pathology | Zika Virus - isolation & purification | Reverse Transcriptase Polymerase Chain Reaction | Pregnancy Complications, Infectious - pathology | Syndrome | Immunohistochemistry - methods | Pregnancy | Ultrasonography, Prenatal | Limb Deformities, Congenital - diagnostic imaging | Placenta - pathology | Brain - pathology | Neuroglia - virology | Placenta - virology | Virus diseases | Mosquitoes | Lung diseases | Birth defects | Conjunctivitis | Health aspects | Hyperthermia | Abortion | Fever | Zoonoses | Diseases | Disease transmission | Genetic disorders | Analysis | Pathology | Zika virus | Congenital diseases | Maternal & child health | Newborn babies | Health risk assessment
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2015, Volume 125, Issue 5, pp. 1767 - 1779
Amyotrophic lateral sclerosis (ALS) is a devastating degenerative disease characterized by progressive loss of motor neurons in the motor cortex, brainstem,... 
RNA-BINDING PROTEINS | MEDICINE, RESEARCH & EXPERIMENTAL | C9ORF72 HEXANUCLEOTIDE REPEAT | PRION-LIKE DOMAINS | HEAVY NEUROFILAMENT SUBUNIT | REGULATORY T-LYMPHOCYTES | FAST AXONAL-TRANSPORT | CU/ZN SUPEROXIDE-DISMUTASE | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | FRONTOTEMPORAL LOBAR DEGENERATION | Genetic Therapy | RNA-Binding Proteins - genetics | Axonal Transport | Superoxide Dismutase - genetics | Oxidative Stress | Humans | Molecular Targeted Therapy | Motor Neurons - pathology | DNA-Binding Proteins - metabolism | Nerve Tissue Proteins - chemistry | Ubiquitination | Proteolysis | Axons - ultrastructure | Neuroglia - immunology | Genetic Association Studies | Amyotrophic Lateral Sclerosis - therapy | Amyotrophic Lateral Sclerosis - genetics | Mice, Transgenic | Superoxide Dismutase - deficiency | Inflammation | Cytoskeleton - ultrastructure | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Protein Aggregation, Pathological | Nerve Tissue Proteins - metabolism | Motor Neurons - chemistry | Amyotrophic Lateral Sclerosis - pathology | Animals | Amyotrophic Lateral Sclerosis - metabolism | Mice | Protein Processing, Post-Translational | Superoxide Dismutase-1 | RNA-Binding Proteins - metabolism | Gene mutations | Physiological aspects | Amyotrophic lateral sclerosis | Genetic aspects | Research | Binding proteins | Health aspects | Risk factors | Pathology | Mutation | Rodents | Review
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2015, Volume 130, Issue 4, pp. 537 - 555
A massive expansion of a GGGGCC repeat upstream of the C9orf72 coding region is the most common known cause of amyotrophic lateral sclerosis and frontotemporal... 
Pathology | Neurosciences | Neurotoxicity | DPR inclusions | Medicine & Public Health | FTLD | ALS | Repeat disorders | C9orf72 | DEMENTIA | EXPANSIONS | PATHOLOGY | FRONTOTEMPORAL LOBAR DEGENERATION | NEUROSCIENCES | CLINICAL NEUROLOGY | MARINESCO BODIES | EXPANDED C9ORF72 | ANTISENSE TRANSCRIPTS | DISEASE | HEXANUCLEOTIDE REPEAT | RNA FOCI | Frontotemporal Lobar Degeneration - pathology | Neurons - pathology | Cell Nucleolus - pathology | Spinal Cord - metabolism | Neuroglia - pathology | Humans | Middle Aged | Brain - metabolism | DNA Repeat Expansion | Motor Neuron Disease - metabolism | Frontotemporal Lobar Degeneration - metabolism | Motor Neuron Disease - genetics | Spinal Cord - pathology | Cell Nucleolus - metabolism | Adult | C9orf72 Protein | Neurons - metabolism | Inclusion Bodies - metabolism | Motor Neuron Disease - complications | Gene Silencing | Rats | Frontotemporal Lobar Degeneration - complications | Motor Neuron Disease - pathology | Proteins - genetics | Animals | Proteins - metabolism | Brain - pathology | Inclusion Bodies - pathology | Neuroglia - metabolism | Aged | Frontotemporal Lobar Degeneration - genetics | Adaptor Proteins, Signal Transducing - metabolism | Cohort Studies | Proteins | Amyotrophic lateral sclerosis | Genetic aspects | Genetic transcription | Comparative analysis | Index Medicus | Original Paper
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2014, Volume 127, Issue 5, pp. 645 - 665
Journal Article