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Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 221 - 234
Caffeine administered to preterm infants has been shown to reduce rates of cerebral palsy and cognitive delay, compared to placebo. We investigated the... 
Methylxanthine | Oxidative stress | Postnatal neurogenesis | Developmental brain | Free radicals | Hyperoxia | Preterm infants | PROGENITOR CELLS | A(2A) RECEPTOR ANTAGONISTS | FUNCTIONAL IMPLICATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT HIPPOCAMPAL NEUROGENESIS | TNF-ALPHA PRODUCTION | NEURAL STEM-CELLS | INTENSIVE-CARE-UNIT | ENDOCRINOLOGY & METABOLISM | DENTATE GYRUS | EXPRESSION | Cell Proliferation | Homeodomain Proteins - metabolism | Cerebral Cortex - pathology | Ki-67 Antigen - metabolism | SOXB1 Transcription Factors - metabolism | Dentate Gyrus - drug effects | SOXB1 Transcription Factors - genetics | Hyperoxia - metabolism | Caffeine - pharmacology | Neurons - metabolism | Cerebral Cortex - drug effects | Repressor Proteins - metabolism | Animals, Newborn | Biomarkers - metabolism | Gene Expression | Tumor Suppressor Proteins - metabolism | Dentate Gyrus - metabolism | Rats | Repressor Proteins - genetics | Neuropeptides - metabolism | T-Box Domain Proteins - metabolism | Hyperoxia - pathology | Eye Proteins - metabolism | Ki-67 Antigen - genetics | Dentate Gyrus - pathology | Paired Box Transcription Factors - genetics | Neurons - pathology | Microtubule-Associated Proteins - genetics | Rats, Wistar | Antigens, Nuclear - metabolism | Microtubule-Associated Proteins - metabolism | Gray Matter - drug effects | Cerebral Cortex - metabolism | Gray Matter - metabolism | Tumor Suppressor Proteins - genetics | Nestin - genetics | Eye Proteins - genetics | Neuropeptides - genetics | Neurons - drug effects | PAX6 Transcription Factor | Hyperoxia - prevention & control | Nerve Tissue Proteins - genetics | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Antigens, Nuclear - genetics | Gray Matter - pathology | Paired Box Transcription Factors - metabolism | Antioxidants | Neurons | Analysis | Caffeine | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 2006, Volume 21, Issue 4, pp. 521 - 531
Oxygen (O ) deprivation, or hypoxia, has profound effects on cell metabolism and growth. Cells can adapt to low O in part through activation of... 
RNA | CELLCYCLE | SIGNALING | MAMMALIAN TARGET | ACTIVATED PROTEIN-KINASE | SIGNALING PATHWAY | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | TSC2 | PROLIFERATION | TUBEROUS-SCLEROSIS | MTOR | INITIATION-FACTOR EIF2-ALPHA | CELL BIOLOGY | Protein Kinases - metabolism | Protein Biosynthesis | eIF-2 Kinase - metabolism | Humans | Multienzyme Complexes - metabolism | Ras Homolog Enriched in Brain Protein | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Hypoxia-Inducible Factor 1 - metabolism | AMP-Activated Protein Kinases | Oxygen - metabolism | Peptide Elongation Factor 2 - antagonists & inhibitors | Tumor Suppressor Proteins - genetics | Eukaryotic Initiation Factor-2 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Eukaryotic Initiation Factor-2 - antagonists & inhibitors | Cell Line | Tumor Suppressor Proteins - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Gene Expression Regulation | Neuropeptides - metabolism | Adaptor Proteins, Signal Transducing | Animals | Carrier Proteins - metabolism | Energy Metabolism | Monomeric GTP-Binding Proteins - metabolism | Hypoxia | Signal Transduction - physiology | TOR Serine-Threonine Kinases | Enzyme Activation | Peptide Elongation Factor 2 - metabolism | Messenger RNA | Growth | Energy conservation | Oncology, Experimental | Physiological aspects | Research | Cancer | Index Medicus | eEF2 | Rheb | mTOR | AMPK | eIF2
Journal Article
Cell Metabolism, ISSN 1550-4131, 02/2013, Volume 17, Issue 2, pp. 236 - 248
Neuropepetide Y (NPY) is best known for its powerful stimulation of food intake and its effects on reducing energy expenditure. However, the pathways involved... 
ENERGY-BALANCE | BODY-WEIGHT | MESSENGER-RNA | ADAPTIVE THERMOGENESIS | FOOD-INTAKE | ENDOCRINOLOGY & METABOLISM | BROWN ADIPOSE-TISSUE | NORMAL RATS | NEUROPEPTIDE-Y INFUSION | DIET-INDUCED OBESITY | VENTROMEDIAL HYPOTHALAMUS | CELL BIOLOGY | Thermogenesis - genetics | Tyrosine 3-Monooxygenase - metabolism | Temperature | Receptors, Neuropeptide Y - metabolism | Arcuate Nucleus of Hypothalamus - cytology | Male | Neurons - cytology | Neuropeptide Y - genetics | Paraventricular Hypothalamic Nucleus - enzymology | RNA, Messenger - metabolism | Sympathetic Nervous System - metabolism | Neuropeptide Y - metabolism | Paraventricular Hypothalamic Nucleus - cytology | Mitochondrial Proteins - metabolism | Diet, High-Fat | Models, Animal | Energy Metabolism - genetics | Pro-Opiomelanocortin - genetics | Arcuate Nucleus of Hypothalamus - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Feeding Behavior | Signal Transduction - genetics | Agouti-Related Protein - metabolism | Pro-Opiomelanocortin - metabolism | Tyrosine 3-Monooxygenase - genetics | Animals | Ion Channels - metabolism | Oxygen Consumption - genetics | Neurons - enzymology | Glutamate Decarboxylase - metabolism | Adipose Tissue, Brown - metabolism | Mice | Uncoupling Protein 1 | Neuropeptide Y | Tyrosine | Neurosciences | Neurons | Eating disorders | Phenols | Universities and colleges | Hydroxylases | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2006, Volume 116, Issue 7, pp. 1983 - 1993
Many homeostatic processes, including appetite and food intake, are controlled by neuroendocrine circuits involving the CNS. The CNS also directly regulates... 
MEDICINE, RESEARCH & EXPERIMENTAL | SKELETAL-MUSCLE | AGOUTI-RELATED PROTEIN | PERIPHERAL INSULIN-RESISTANCE | LIPID-METABOLISM | BODY-WEIGHT GAIN | ENERGY HOMEOSTASIS | FOOD-INTAKE | BROWN ADIPOSE-TISSUE | NORMAL RATS | NEUROPEPTIDE-Y | Brain - anatomy & histology | Fatty Acid Synthases - metabolism | Ion Channels | Rats, Wistar | Adipose Tissue - cytology | Homeostasis | Adipocytes - cytology | Male | Neuropeptide Y - genetics | Mitochondrial Proteins | Peptide Hormones - genetics | Acetyl-CoA Carboxylase - genetics | Brain - metabolism | Neuropeptide Y - metabolism | Peptide Hormones - metabolism | Adipose Tissue - metabolism | Ghrelin | Membrane Proteins - metabolism | Fatty Acid Synthases - genetics | Lipoprotein Lipase - metabolism | Peptide Hormones - administration & dosage | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Rats | Stearoyl-CoA Desaturase - genetics | Mice, Knockout | Carrier Proteins - genetics | Eating - drug effects | Animals | Carrier Proteins - metabolism | Adipocytes - metabolism | Glucose - metabolism | Stearoyl-CoA Desaturase - metabolism | Mice | Uncoupling Protein 1 | Uncoupling Protein 3 | Acetyl-CoA Carboxylase - metabolism | Energy Metabolism - drug effects | Adipose tissues | Medical research | Central nervous system | Cell metabolism | Medicine, Experimental | Research | Index Medicus | Abridged Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2017, Volume 19, Issue 10, pp. 1274 - 1285
Metastatic cancer is a systemic disease, and metastasis determinants might elicit completely different effects in various target organs. Here we show that... 
CELLS | INVASION | WNT/BETA-CATENIN | PATHWAY | GENES | DICKKOPF-1 | INDUCTION | BETA-CATENIN | CARCINOMA | FAMILY | CELL BIOLOGY | Humans | Lung Neoplasms - metabolism | Transforming Growth Factor beta1 - metabolism | Bone Neoplasms - secondary | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Molecular Targeted Therapy | Neutrophil Infiltration | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Transfection | Lung Neoplasms - secondary | RNA Interference | Time Factors | Female | Antineoplastic Agents - pharmacology | Bone Neoplasms - genetics | Neuropeptides - genetics | Neutrophils - metabolism | Calcium Signaling | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Latent TGF-beta Binding Proteins - metabolism | Neutrophils - pathology | Lung Neoplasms - genetics | Macrophages - pathology | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Neuropeptides - metabolism | Mice, SCID | Breast Neoplasms - drug therapy | Cell Movement - drug effects | Osteoblasts - pathology | Macrophages - metabolism | Animals | Breast Neoplasms - genetics | Wnt Signaling Pathway - drug effects | Lung Neoplasms - prevention & control | Bone Neoplasms - prevention & control | Breast Neoplasms - pathology | Cyclooxygenase 2 - metabolism | Mice, Inbred NOD | Mice, Inbred BALB C | HeLa Cells | Osteoblasts - metabolism | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Wnt protein | Lung | Lung cancer | Metastasis | Macrophages | Osteoblasts | Metastases | Recruitment | Calcium signalling | Biomedical materials | Biocompatibility | Secretion | Organs | Rac1 protein | JNK protein | Breast cancer | Bone cancer | Lungs | Breast | Bone | Dkk1 protein | Organotropism | Cancer | Tumors | Ca2+/calmodulin-dependent protein kinase II | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 04/2011, Volume 12, Issue 4, pp. 295 - 303
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 46, pp. 12107 - 12119
Recent studies in adult and weanling rats show that dietary fat, in close association with circulating lipids, can stimulate expression of hypothalamic... 
Lipids | Maternal high-fat diet | Hypothalamus | Food intake | Orexigenic peptides | Neurogenesis | orexigenic peptides | lipids | EMBRYONIC-DEVELOPMENT | LEPTIN RECEPTOR ISOFORMS | CELL GENERATION | DOPAMINERGIC-NEURONS | DIABETIC MOTHER RATS | NEUROSCIENCES | NEUROPEPTIDE-Y | CIRCULATING TRIGLYCERIDES | PARAVENTRICULAR NUCLEUS | MESSENGER-RNA | maternal high-fat diet | neurogenesis | hypothalamus | food intake | Dietary Fats - metabolism | Hypothalamus - secretion | Opioid Peptides - secretion | Cell Proliferation | Fetal Nutrition Disorders - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Galanin - secretion | Melanins - metabolism | Galanin - metabolism | Melanins - secretion | Pituitary Hormones - metabolism | Female | Intracellular Signaling Peptides and Proteins - secretion | Obesity - etiology | Neurons - metabolism | Neuropeptides - secretion | Animals, Newborn | Dietary Fats - adverse effects | Hyperphagia - etiology | Orexins | Pituitary Hormones - secretion | Rats | Neuropeptides - metabolism | Obesity - physiopathology | Prenatal Exposure Delayed Effects - physiopathology | Hyperphagia - metabolism | Hypothalamus - physiopathology | Opioid Peptides - metabolism | Rats, Sprague-Dawley | Prenatal Exposure Delayed Effects - metabolism | Obesity - metabolism | Pregnancy | Hypothalamic Hormones - secretion | Animals | Hypothalamus - metabolism | Hypothalamic Hormones - metabolism | Neurogenesis - physiology | Hyperphagia - physiopathology | Appetite Regulation - physiology | Body Weight - physiology | Fetal Nutrition Disorders - physiopathology | Index Medicus
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2010, Volume 141, Issue 2, pp. 290 - 303
Journal Article
Journal Article