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International journal of environmental research and public health, ISSN 1660-4601, 08/2013, Volume 10, Issue 8, pp. 3384 - 3408
In recent years concern has risen about the increasing prevalence of Autism Spectrum Disorders (ASD). Accumulating evidence shows that exposure to neurotoxic... 
Hydrocarbons, Chlorinated/metabolism | Organophosphates/metabolism | gamma-Aminobutyric Acid/metabolism | Glutamic Acid/metabolism | Serotonin/metabolism | Humans | Child Development Disorders, Pervasive/epidemiology | Dopamine/metabolism | Environmental Pollutants/metabolism | Child | Neurotransmitter Agents/metabolism | Neurotoxins/metabolism | ASD | Dopamine | Organophosphate | Serotonin | Neurotoxic exposure | GABA | Neurotransmitter | Phthalate | Organochlorine | Glutamate | Autism spectrum disorder | Public, Environmental & Occupational Health | Environmental Sciences & Ecology | Life Sciences & Biomedicine | Science & Technology | Environmental Sciences | Hydrocarbons, Chlorinated - metabolism | Organophosphates - metabolism | Child Development Disorders, Pervasive - epidemiology | Child Development Disorders, Pervasive - metabolism | Hydrocarbons, Chlorinated - toxicity | gamma-Aminobutyric Acid - metabolism | Neurotransmitter Agents - metabolism | Child Development Disorders, Pervasive - etiology | Neurotoxins - toxicity | Environmental Pollutants - metabolism | Child Development Disorders, Pervasive - genetics | Serotonin - metabolism | Neurotoxins - metabolism | Glutamic Acid - metabolism | Environmental Pollutants - toxicity | Organophosphates - toxicity | Dopamine - metabolism | Autism | Neurotransmitters | Neurotoxicity | Human exposure | Index Medicus | organophosphate | serotonin | neurotoxic exposure | phthalate | Review | autism spectrum disorder | organochlorine | neurotransmitter | dopamine | glutamate
Journal Article
Molecular neurobiology, ISSN 1559-1182, 11/2015, Volume 53, Issue 9, pp. 6426 - 6443
Artemether, a lipid-soluble derivative of artemisinin has been reported to possess anti-inflammatory properties. In this study, we have investigated the... 
Neurology | NF-κB | Neurosciences | Biomedicine | BV2 microglia | HT22 hippocampal neurons | Nrf2 | Neurobiology | Artemether | Neuroinflammation | Cell Biology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neuroprotective Agents - therapeutic use | Inflammation - pathology | Microglia - metabolism | Heme Oxygenase-1 - metabolism | Transcriptional Activation - genetics | Antioxidants - metabolism | Transcriptional Activation - drug effects | NF-kappa B - metabolism | Lipopolysaccharides | Artemisinins - pharmacology | Cell Nucleus - metabolism | Neuroprotective Agents - pharmacology | Inflammation - drug therapy | Amyloid beta-Peptides - metabolism | Microglia - pathology | NF-E2-Related Factor 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Artemisinins - chemistry | Cell Line | Cell Survival - drug effects | Microglia - drug effects | Prostaglandin-E Synthases - metabolism | Neurotoxins - toxicity | Antimalarials - therapeutic use | Antimalarials - pharmacology | Dinoprostone - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Artemisinins - therapeutic use | MAP Kinase Signaling System - drug effects | Aspartic Acid Endopeptidases - metabolism | NF-E2-Related Factor 2 - metabolism | Antimalarials - chemistry | Cyclooxygenase 2 - metabolism | Interleukin-6 - biosynthesis | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Cell Nucleus - drug effects | Nitric Oxide - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Nitric Oxide Synthase Type II - metabolism | Nervous system diseases | Neurons | Nitric oxide | Heme | Amyloid beta-protein | Synthetic prostaglandins E | Mitogens | Protein kinases | Proteins | Enzymes | Inflammation | Kinases | Index Medicus
Journal Article
Journal of the neurological sciences, ISSN 0022-510X, 12/2012, Volume 323, Issue 1-2, pp. 1 - 8
...), Parkinson's disease (PD), Huntington's disease (HD) and amyotrophic lateral sclerosis (ALS). Several studies have demonstrated that altered kynurenine metabolism plays an important role in the pathogenesis of this group of diseases... 
Neurology | Aging | Alzheimer's disease (AD) | Amyotrophic lateral sclerosis (ALS) | Huntington's disease (HD) | Parkinson's disease (PD) | Kynurenine pathway | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism | Neuroprotective Agents - therapeutic use | Microglia - metabolism | Kynurenine - therapeutic use | Humans | Neurodegenerative Diseases - prevention & control | Parkinson Disease - drug therapy | Kynurenic Acid - metabolism | Molecular Targeted Therapy | Neurodegenerative Diseases - drug therapy | Quinolinic Acid - antagonists & inhibitors | Tryptophan - metabolism | Amyotrophic Lateral Sclerosis - drug therapy | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Alzheimer Disease - prevention & control | Neurons - metabolism | Parkinson Disease - metabolism | Kynurenine - metabolism | Huntington Disease - drug therapy | Receptors, G-Protein-Coupled - drug effects | Receptors, G-Protein-Coupled - physiology | Receptors, N-Methyl-D-Aspartate - drug effects | Kynurenine - pharmacology | Enzyme Inhibitors - pharmacology | Alzheimer Disease - drug therapy | Neurodegenerative Diseases - metabolism | Enzyme Inhibitors - therapeutic use | Huntington Disease - metabolism | Kynurenine - analogs & derivatives | Receptors, N-Methyl-D-Aspartate - physiology | Parkinson Disease - prevention & control | Animals | Quinolinic Acid - metabolism | Alzheimer Disease - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Neurotoxins - metabolism | Kynurenic Acid - antagonists & inhibitors | Aging - metabolism | Enzymes | Nervous system diseases | Enzyme inhibitors | Parkinson's disease | Metabolites | Huntington's chorea | Analysis | Physiological aspects | Tryptophan | Amyotrophic lateral sclerosis | Health aspects | Alzheimer's disease | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2009, Volume 29, Issue 2, pp. 529 - 542
.... Reactive oxygen species (ROS) generated by the mitochondrial metabolism have been postulated to be the central source of molecular damages and imbalance between levels of intracellular ROS and antioxidant defenses... 
Aging | Antioxidant | Bmi1 | Neuronal cell death | ROS | p53 | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cell Proliferation | Reactive Oxygen Species - metabolism | Age Factors | Embryo, Mammalian | Microtubule-Associated Proteins - metabolism | Homeodomain Proteins - metabolism | Peroxiredoxins - metabolism | Cell Survival - genetics | Apoptosis - genetics | Phosphopyruvate Hydratase - metabolism | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - cytology | Tumor Suppressor Protein p53 - genetics | Peroxiredoxins - genetics | Nuclear Proteins - deficiency | beta-Galactosidase - metabolism | Neurons - metabolism | Neurons - drug effects | Repressor Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Cells, Cultured | Hydrogen Peroxide - pharmacology | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | PAX6 Transcription Factor | Proto-Oncogene Proteins - deficiency | Mice, Knockout | Neurotoxins - pharmacology | Animals | Polycomb Repressive Complex 1 | Analysis of Variance | Eye Proteins - metabolism | Mice | Histones - metabolism | Lipid Peroxidation - genetics | Paired Box Transcription Factors - metabolism | Index Medicus | neuronal cell death | antioxidant | aging
Journal Article
Brain pathology (Zurich, Switzerland), ISSN 1750-3639, 01/2008, Volume 18, Issue 1, pp. 52 - 61
Journal Article
Biochemical journal, ISSN 0264-6021, 09/2013, Volume 454, Issue 2, pp. 303 - 310
To identify high-affinity interactions between long-chain α-neurotoxins and nicotinic receptors, we determined the crystal structure of the complex between... 
Molecular recognition | Neurotoxin | Nicotinic acetylcholine receptor | Crystal structure | Reptilian Proteins - chemistry | Nicotinic Agonists - metabolism | Cobra Neurotoxin Proteins - metabolism | Pyridines - chemistry | Humans | Recombinant Fusion Proteins - metabolism | Nicotinic Agonists - chemistry | Receptors, Nicotinic - chemistry | Carrier Proteins - chemistry | Protein Interaction Domains and Motifs | Binding Sites | Peptide Fragments - genetics | Bridged Bicyclo Compounds, Heterocyclic - metabolism | Bungarotoxins - metabolism | Neurotoxins - chemistry | Peptide Fragments - metabolism | Receptors, Nicotinic - metabolism | Bridged Bicyclo Compounds, Heterocyclic - chemistry | Bungarotoxins - chemistry | Lymnaea | Models, Molecular | Mutant Proteins - metabolism | Recombinant Fusion Proteins - chemistry | Cobra Neurotoxin Proteins - chemistry | Bungarus | Carrier Proteins - genetics | Peptide Fragments - chemistry | Animals | Carrier Proteins - metabolism | Pyridines - metabolism | Mutant Proteins - chemistry | Ligands | Neurotoxins - metabolism | Protein Conformation | Receptors, Nicotinic - genetics | Amino Acid Substitution | Reptilian Proteins - metabolism | alpha7 Nicotinic Acetylcholine Receptor | Index Medicus | BASIC BIOLOGICAL SCIENCES | crystal structure | neurotoxin | molecular recognition | nicotinic acetylcholine receptor
Journal Article