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The Journal of neuroscience, ISSN 1529-2401, 2006, Volume 26, Issue 18, pp. 4930 - 4939
Increased expression of glial fibrillary acidic protein (GFAP) represents astroglial activation and gliosis during neurodegeneration. However, the molecular... 
cGMP | Neurotoxins | Astroglial activation | Guanylate cyclase | Nitric oxide | GFAP | VIRUS TYPE-1 HIV-1 | nitric oxide | ACTIVATION | HUMAN ASTROGLIA | neurotoxins | MICROGLIAL CELLS | RAT PRIMARY ASTROCYTES | astroglial activation | NEUROSCIENCES | PEPTIDE | SYNTHASE | guanylate cyclase | LONG-TERM POTENTIATION | NF-KAPPA-B | Free Radical Scavengers - pharmacology | Cyclic GMP - pharmacology | Glial Fibrillary Acidic Protein - genetics | Gene Expression - drug effects | Nitric Oxide - pharmacology | Male | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - cytology | Dose-Response Relationship, Drug | Tetrazolium Salts | RNA, Messenger - biosynthesis | Drug Interactions | Cyclic GMP - analogs & derivatives | Time Factors | Cyclic N-Oxides - pharmacology | Reverse Transcriptase Polymerase Chain Reaction - methods | Thiazoles | Animals, Newborn | Astrocytes - drug effects | Cell Survival - drug effects | Lysine - analogs & derivatives | Fluorescent Antibody Technique - methods | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Thionucleotides - pharmacology | Electrophoretic Mobility Shift Assay - methods | Animals | Lysine - pharmacology | HIV Envelope Protein gp120 - pharmacology | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Cytokines - pharmacology
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2007, Volume 27, Issue 6, pp. 1445 - 1455
Excitotoxicity is associated with stroke, brain trauma, and a number of neurodegenerative disorders. In the brain, during excitotoxic insults, neurons undergo... 
Anion channel | Varicosity | Cortical neuron | Volume regulation | Excitotoxicity | Necrosis | cortical neuron | RAT | anion channel | NMDA RECEPTOR ACTIVATION | DEATH | NEUROSCIENCES | DENDRITE MORPHOLOGY | MORPHOLOGICAL-CHANGES | DEGENERATION | CORTICAL-NEURONS | necrosis | GLUTAMATE | varicosity | CALCIUM | excitotoxicity | volume regulation | REVERSIBLE CHANGES | Apoptosis - drug effects | 2-Amino-5-phosphonovalerate - pharmacology | Nitrobenzoates - pharmacology | Potassium Channels - physiology | Ion Channels - drug effects | Dendrites - ultrastructure | Ion Channels - physiology | Sodium Chloride Symporter Inhibitors - pharmacology | Cerebral Cortex - cytology | Phloretin - pharmacology | Cells, Cultured - pathology | Quinine - pharmacology | Somatosensory Cortex - cytology | Tetrodotoxin - pharmacology | Neurons - drug effects | Chlorides - physiology | 4-Aminopyridine - pharmacology | Potassium Channel Blockers - pharmacology | Sodium Chloride Symporters - physiology | Dendrites - drug effects | Cells, Cultured - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | GABA-A Receptor Antagonists | Mice, Inbred C57BL | Cell Size | Neurotoxins - toxicity | Excitatory Amino Acid Antagonists - pharmacology | Bicuculline - pharmacology | Benzothiadiazines - pharmacology | Receptors, N-Methyl-D-Aspartate - physiology | Bumetanide - pharmacology | Ion Channels - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Glycolates - pharmacology | Picrotoxin - pharmacology | Mice | Dizocilpine Maleate - pharmacology | N-Methylaspartate - toxicity
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2012, Volume 122, Issue 5, pp. 941 - 951
J. Neurochem. (2012) 122, 941–951. In vitro and in vivo models of Parkinson’s disease (PD) suggest that increased oxidant production leads to mitochondrial... 
hydroethidine | mitochondria | glycolysis | reactive oxygen species | NADPH OXIDASE | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOPAMINERGIC-NEURONS | PARAQUAT | NEUROSCIENCES | BRAIN MITOCHONDRIA | REACTIVE OXYGEN | ANIMAL-MODELS | CELL-CULTURE | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Insecticides - pharmacology | Rats | Herbicides - pharmacology | Adrenergic Agents - pharmacology | Dose-Response Relationship, Drug | Neurotoxins - pharmacology | Animals | Oligomycins - pharmacology | Oxidopamine - pharmacology | Time Factors | Dopamine Agents - pharmacology | Oxygen Consumption - drug effects | Adenosine Triphosphate - metabolism | Dopaminergic Neurons - metabolism | Proton Ionophores - pharmacology | Rotenone - pharmacology | Superoxides - metabolism | Dopaminergic Neurons - drug effects | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Paraquat - pharmacology | Cell Line, Transformed | Energy Metabolism - drug effects | Glucose metabolism | Herbicides | Parkinson's disease | Cell death | Analysis | Superoxide | Mitochondrial DNA | Universities and colleges | Cells | Neurochemistry | Mitochondria | Neurotransmitters | Bioenergetics | Parkinsons disease | Apoptosis | Index Medicus | Dopamine | Neurodegenerative diseases | 6-Hydroxydopamine | Acidification | Data processing | Oxygen consumption | Rotenone | Toxicants | MPP super(+) | Oxidants | Glycolysis | Paraquat | Oxidation | Movement disorders
Journal Article
Acta Pharmacologica Sinica, ISSN 1671-4083, 12/2008, Volume 29, Issue 12, pp. 1419 - 1424
Aim: Endothelium-dependent relaxations to certain neurohumoral substances are mediated by pertussis toxin-sensitive Gi/o protein. Our experiments were designed... 
CELLS | DEPENDENT RELAXATION | ANANDAMIDE | MESENTERIC-ARTERY | NATRIURETIC PEPTIDE | CHEMISTRY, MULTIDISCIPLINARY | endothelium-derived hyperpolarizing factor | Gi/o protein | pig coronary artery | L-ARGININE | pertussis toxin | PHARMACOLOGY & PHARMACY | RECEPTORS | EXPRESSION | EDHF | Endothelium-Dependent Relaxing Factors - pharmacology | Hemostatics - pharmacology | Peptide Fragments - pharmacology | Substance P - pharmacology | Apamin - pharmacology | Serotonin - pharmacology | Dose-Response Relationship, Drug | Thrombin - pharmacology | Charybdotoxin - pharmacology | Neurotransmitter Agents - pharmacology | Dinoprost - pharmacology | Bradykinin - pharmacology | Ionophores - pharmacology | Pertussis Toxin - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Ketanserin - pharmacology | Coronary Vessels - drug effects | Vasodilator Agents - pharmacology | Coronary Vessels - physiology | Enzyme Inhibitors - pharmacology | Serotonin Antagonists - pharmacology | Calcimycin - pharmacology | Neurotoxins - pharmacology | Animals | Muscle Relaxation - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Sus scrofa | Biological Factors - pharmacology | Tocolytic agents | Swine | Tachykinins | Bradykinin | Thrombin | Indomethacin | Carbonates | Endothelium-derived relaxing factors | Peptide hormones | Arteries | Endothelium | Index Medicus
Journal Article
Neurochemical research, ISSN 1573-6903, 2017, Volume 42, Issue 9, pp. 2495 - 2504
...Neurochem Res (2017) 42:2495–2504 DOI 10.1007/s11064-017-2314-9 ORIGINAL PAPER Forty Y ears of Sodium Channels: Structure, Function, Pharmacology, and Epilepsy... 
Neurochemistry | Biochemistry, general | Neurology | Neurosciences | Biomedicine | Sodium channel | Epilepsy | Ion channel structure | Cell Biology | Local anesthetics | ALPHA-SUBUNIT | BIOCHEMISTRY & MOLECULAR BIOLOGY | NA+ CHANNELS | MOLECULAR DETERMINANTS | SCORPION TOXIN | NEUROSCIENCES | MOUSE SPINAL-CORD | VOLTAGE SENSOR | DRAVET-SYNDROME | AMINO-ACID-RESIDUES | SEVERE MYOCLONIC EPILEPSY | RAT-BRAIN | Protein Structure, Tertiary | Protein Structure, Secondary | Humans | Epilepsy - metabolism | Sodium Channels - physiology | Action Potentials - physiology | Neurotoxins - pharmacology | Ion Channel Gating - physiology | Animals | Sodium Channels - chemistry | Neurotoxins - metabolism | Protein Subunits - chemistry | Protein Subunits - physiology | Action Potentials - drug effects | Ion Channel Gating - drug effects | Animal genetics | Biological products | Neural circuitry | Neurons | Analysis | GABA | Pharmacology | Mice | Children | Drug therapy | Diseases | Brain | Leadership | Electric potential | Spinal cord | Disease | Laboratories | Circuits | Biological evolution | Cognitive ability | Insecticides | Action potential | Neuroblastoma | Inactivation | Crystallography | Channels | Neuroblastoma cells | γ-Aminobutyric acid | Synaptic transmission | Occupational health | Sodium channels (voltage-gated) | Atomic structure | Probes | Neurotoxins | Deactivation | Excitability | Morbidity | Resistance | Autism | Molecular modelling | Sodium | Neural networks | Conductance | Toxins | Mutation | Structure-function relationships | Index Medicus
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2012, Volume 74, Issue 2, pp. 384 - 396
Functional neural circuit formation during development involves massive elimination of redundant synapses. In the cerebellum, one-to-one connection from... 
DEVELOPING PURKINJE-CELLS | LONG-TERM DEPRESSION | POSTNATAL-DEVELOPMENT | MOTOR COORDINATION | NEUROSCIENCES | GLUTAMATE-RECEPTOR DELTA-2 | MICE LACKING | CORTICAL CIRCUITS | MOUSE CEREBELLUM | CLIMBING FIBER SYNAPSE | PARALLEL FIBER | Receptors, Glutamate - metabolism | Electric Stimulation | Calcium Channels - metabolism | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Biophysical Phenomena - drug effects | Dendrites - ultrastructure | Green Fluorescent Proteins - genetics | Purkinje Cells - physiology | Diazepam - pharmacology | Calbindins | Cytochromes c - pharmacology | Protein Kinase C - metabolism | Biophysical Phenomena - genetics | Chromones - pharmacology | Excitatory Postsynaptic Potentials - genetics | GABA-A Receptor Antagonists - pharmacology | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Mice, Transgenic | Excitatory Amino Acid Antagonists - pharmacology | Bicuculline - pharmacology | Microscopy, Confocal | Patch-Clamp Techniques | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Analysis of Variance | Cerebellum - cytology | Cerebellum - growth & development | Mice | Membrane Potentials - genetics | Age Factors | Gene Expression Regulation, Developmental - genetics | Agatoxins - pharmacology | Receptors, Metabotropic Glutamate - metabolism | Excitatory Postsynaptic Potentials - drug effects | Quinoxalines - pharmacology | Purkinje Cells - cytology | GABA Modulators - pharmacology | Phospholipase C beta - metabolism | Microscopy, Electron, Transmission | Synapses - physiology | Mice, Inbred C57BL | Glutamate Decarboxylase - genetics | Biophysics | Membrane Potentials - physiology | Nerve Tissue Proteins - metabolism | Neurotoxins - pharmacology | S100 Calcium Binding Protein G - metabolism | Animals | In Vitro Techniques | Brain | Medical colleges | Enzymes | Neurosciences | Neurons | GABA | Glutamate decarboxylase | Universities and colleges | Diazepam | Antianxiety agents | Kinases | Rodents | Molecular weight
Journal Article
Journal of Neurophysiology, ISSN 0022-3077, 12/2005, Volume 94, Issue 6, pp. 4121 - 4130
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 08/2013, Volume 33, Issue 8, pp. 1892 - 1901
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 2, p. e32432
Journal Article