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Journal of Neurochemistry, ISSN 0022-3042, 09/2012, Volume 122, Issue 5, pp. 941 - 951
J. Neurochem. (2012) 122, 941951. Abstract In vitro and in vivo models of Parkinsons disease (PD) suggest that increased oxidant production leads to... 
hydroethidine | mitochondria | glycolysis | reactive oxygen species | NADPH OXIDASE | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOPAMINERGIC-NEURONS | PARAQUAT | NEUROSCIENCES | BRAIN MITOCHONDRIA | REACTIVE OXYGEN | ANIMAL-MODELS | CELL-CULTURE | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Insecticides - pharmacology | Rats | Herbicides - pharmacology | Adrenergic Agents - pharmacology | Dose-Response Relationship, Drug | Neurotoxins - pharmacology | Animals | Oligomycins - pharmacology | Oxidopamine - pharmacology | Time Factors | Dopamine Agents - pharmacology | Oxygen Consumption - drug effects | Adenosine Triphosphate - metabolism | Dopaminergic Neurons - metabolism | Proton Ionophores - pharmacology | Rotenone - pharmacology | Superoxides - metabolism | Dopaminergic Neurons - drug effects | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Paraquat - pharmacology | Cell Line, Transformed | Energy Metabolism - drug effects | Glucose metabolism | Herbicides | Parkinson's disease | Cell death | Analysis | Superoxide | Mitochondrial DNA | Universities and colleges | Cells | Neurochemistry | Mitochondria | Neurotransmitters | Bioenergetics | Parkinsons disease | Apoptosis | Index Medicus | Dopamine | Neurodegenerative diseases | 6-Hydroxydopamine | Acidification | Data processing | Oxygen consumption | Rotenone | Toxicants | MPP super(+) | Oxidants | Glycolysis | Paraquat | Oxidation | Movement disorders
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2007, Volume 27, Issue 6, pp. 1445 - 1455
Excitotoxicity is associated with stroke, brain trauma, and a number of neurodegenerative disorders. In the brain, during excitotoxic insults, neurons undergo... 
Anion channel | Varicosity | Cortical neuron | Volume regulation | Excitotoxicity | Necrosis | cortical neuron | anion channel | EPILEPTIC FOCUS | DEATH | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | DENDRITE MORPHOLOGY | MORPHOLOGICAL-CHANGES | necrosis | RAT CORTICAL CULTURES | varicosity | GLUTAMATE-RECEPTOR ACTIVATION | CELL-CULTURE | excitotoxicity | volume regulation | REVERSIBLE CHANGES | BRAIN NEURONS | Apoptosis - drug effects | 2-Amino-5-phosphonovalerate - pharmacology | Nitrobenzoates - pharmacology | Potassium Channels - physiology | Ion Channels - drug effects | Dendrites - ultrastructure | Ion Channels - physiology | Sodium Chloride Symporter Inhibitors - pharmacology | Cerebral Cortex - cytology | Phloretin - pharmacology | Cells, Cultured - pathology | Quinine - pharmacology | Somatosensory Cortex - cytology | Tetrodotoxin - pharmacology | Neurons - drug effects | Chlorides - physiology | 4-Aminopyridine - pharmacology | Potassium Channel Blockers - pharmacology | Sodium Chloride Symporters - physiology | Dendrites - drug effects | Cells, Cultured - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | GABA-A Receptor Antagonists | Mice, Inbred C57BL | Cell Size | Neurotoxins - toxicity | Excitatory Amino Acid Antagonists - pharmacology | Bicuculline - pharmacology | Benzothiadiazines - pharmacology | Receptors, N-Methyl-D-Aspartate - physiology | Bumetanide - pharmacology | Ion Channels - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Glycolates - pharmacology | Picrotoxin - pharmacology | Mice | Dizocilpine Maleate - pharmacology | N-Methylaspartate - toxicity | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2006, Volume 26, Issue 18, pp. 4930 - 4939
Increased expression of glial fibrillary acidic protein (GFAP) represents astroglial activation and gliosis during neurodegeneration. However, the molecular... 
cGMP | Neurotoxins | Astroglial activation | Guanylate cyclase | Nitric oxide | GFAP | VIRUS TYPE-1 HIV-1 | nitric oxide | ACTIVATION | HUMAN ASTROGLIA | neurotoxins | MICROGLIAL CELLS | RAT PRIMARY ASTROCYTES | astroglial activation | NEUROSCIENCES | PEPTIDE | SYNTHASE | guanylate cyclase | LONG-TERM POTENTIATION | NF-KAPPA-B | Free Radical Scavengers - pharmacology | Cyclic GMP - pharmacology | Glial Fibrillary Acidic Protein - genetics | Gene Expression - drug effects | Nitric Oxide - pharmacology | Male | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - cytology | Dose-Response Relationship, Drug | Tetrazolium Salts | RNA, Messenger - biosynthesis | Drug Interactions | Cyclic GMP - analogs & derivatives | Time Factors | Cyclic N-Oxides - pharmacology | Reverse Transcriptase Polymerase Chain Reaction - methods | Thiazoles | Animals, Newborn | Astrocytes - drug effects | Cell Survival - drug effects | Lysine - analogs & derivatives | Fluorescent Antibody Technique - methods | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Thionucleotides - pharmacology | Electrophoretic Mobility Shift Assay - methods | Animals | Lysine - pharmacology | HIV Envelope Protein gp120 - pharmacology | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Cytokines - pharmacology | Index Medicus
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 01/1999, Volume 11, Issue 1, pp. 327 - 334
Evidence has accumulated that Zn 2+ plays a central role in neurodegenerative processes following brain injuries including ischaemia or epilepsy. In the... 
cortical cell culture | apoptosis | ischaemia | necrosis | zinc | oxidative stress | Ischaemia | Oxidative stress | Cortical cell culture | Zinc | Apoptosis | Necrosis | GLUTAMATE NEUROTOXICITY | ZINC NEUROTOXICITY | DEATH | NEUROSCIENCES | INHIBITION | NITRIC-OXIDE | NMDA | RECEPTORS | BRAIN | METHYL-D-ASPARTATE | Neurons - pathology | Apoptosis - drug effects | Cerebral Cortex - cytology | Nerve Degeneration - chemically induced | Nerve Degeneration - metabolism | Brain-Derived Neurotrophic Factor - pharmacology | Zinc - pharmacokinetics | Neurons - ultrastructure | Chromans - pharmacology | Amino Acid Chloromethyl Ketones - pharmacology | Lipid Peroxidation - drug effects | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Free Radicals - metabolism | Female | Neurons - metabolism | Mitochondrial Swelling | Dopamine Agonists - pharmacology | Insulin - pharmacology | Kainic Acid - pharmacology | Cells, Cultured | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Antioxidants - pharmacology | Microscopy, Electron | Hypoglycemic Agents - pharmacology | Drug Synergism | Neurotoxins - pharmacology | Pregnancy | Animals | Cysteine Proteinase Inhibitors - pharmacology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Zinc - toxicity | Mice | Oxidative Stress - drug effects | Dizocilpine Maleate - pharmacology | Oligopeptides - pharmacology | Index Medicus
Journal Article
The Journal of urology, 01/2019, Volume 201, Issue 1, pp. 37 - 38
Journal Article
Acta Pharmacologica Sinica, ISSN 1671-4083, 12/2008, Volume 29, Issue 12, pp. 1419 - 1424
Aim: Endothelium-dependent relaxations to certain neurohumoral substances are mediated by pertussis toxin-sensitive Gi/o protein. Our experiments were designed... 
CELLS | DEPENDENT RELAXATION | ANANDAMIDE | MESENTERIC-ARTERY | NATRIURETIC PEPTIDE | CHEMISTRY, MULTIDISCIPLINARY | endothelium-derived hyperpolarizing factor | Gi/o protein | pig coronary artery | L-ARGININE | pertussis toxin | PHARMACOLOGY & PHARMACY | RECEPTORS | EXPRESSION | EDHF | Endothelium-Dependent Relaxing Factors - pharmacology | Hemostatics - pharmacology | Peptide Fragments - pharmacology | Substance P - pharmacology | Apamin - pharmacology | Serotonin - pharmacology | Dose-Response Relationship, Drug | Thrombin - pharmacology | Charybdotoxin - pharmacology | Neurotransmitter Agents - pharmacology | Dinoprost - pharmacology | Bradykinin - pharmacology | Ionophores - pharmacology | Pertussis Toxin - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Ketanserin - pharmacology | Coronary Vessels - drug effects | Vasodilator Agents - pharmacology | Coronary Vessels - physiology | Enzyme Inhibitors - pharmacology | Serotonin Antagonists - pharmacology | Calcimycin - pharmacology | Neurotoxins - pharmacology | Animals | Muscle Relaxation - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Sus scrofa | Biological Factors - pharmacology | Tocolytic agents | Swine | Tachykinins | Bradykinin | Thrombin | Indomethacin | Carbonates | Endothelium-derived relaxing factors | Peptide hormones | Arteries | Endothelium | Index Medicus
Journal Article
Journal of Neurophysiology, ISSN 0022-3077, 12/2005, Volume 94, Issue 6, pp. 4121 - 4130
Journal Article
Neuron, ISSN 0896-6273, 04/2012, Volume 74, Issue 2, pp. 384 - 396
Functional neural circuit formation during development involves massive elimination of redundant synapses. In the cerebellum, one-to-one connection from... 
DEVELOPING PURKINJE-CELLS | LONG-TERM DEPRESSION | POSTNATAL-DEVELOPMENT | MOTOR COORDINATION | NEUROSCIENCES | GLUTAMATE-RECEPTOR DELTA-2 | MICE LACKING | CORTICAL CIRCUITS | MOUSE CEREBELLUM | CLIMBING FIBER SYNAPSE | PARALLEL FIBER | Receptors, Glutamate - metabolism | Electric Stimulation | Calcium Channels - metabolism | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Biophysical Phenomena - drug effects | Dendrites - ultrastructure | Green Fluorescent Proteins - genetics | Purkinje Cells - physiology | Diazepam - pharmacology | Calbindins | Cytochromes c - pharmacology | Protein Kinase C - metabolism | Biophysical Phenomena - genetics | Chromones - pharmacology | Excitatory Postsynaptic Potentials - genetics | GABA-A Receptor Antagonists - pharmacology | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Mice, Transgenic | Excitatory Amino Acid Antagonists - pharmacology | Bicuculline - pharmacology | Microscopy, Confocal | Patch-Clamp Techniques | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Analysis of Variance | Cerebellum - cytology | Cerebellum - growth & development | Mice | Membrane Potentials - genetics | Age Factors | Gene Expression Regulation, Developmental - genetics | Agatoxins - pharmacology | Receptors, Metabotropic Glutamate - metabolism | Excitatory Postsynaptic Potentials - drug effects | Quinoxalines - pharmacology | Purkinje Cells - cytology | GABA Modulators - pharmacology | Phospholipase C beta - metabolism | Microscopy, Electron, Transmission | Synapses - physiology | Mice, Inbred C57BL | Glutamate Decarboxylase - genetics | Biophysics | Membrane Potentials - physiology | Nerve Tissue Proteins - metabolism | Neurotoxins - pharmacology | S100 Calcium Binding Protein G - metabolism | Animals | In Vitro Techniques | Brain | Medical colleges | Enzymes | Neurosciences | Neurons | GABA | Glutamate decarboxylase | Universities and colleges | Diazepam | Antianxiety agents | Kinases | Rodents | Molecular weight | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 08/2012, Volume 122, Issue 3, pp. 582 - 593
Parkinson’s disease‐like symptoms inducing compound affects hippocampus. Cognitive deficits observed in PD paradigms might be related to the hippocampus. Acute... 
CA1 | MPTP | synaptic plasticity | hippocampus | dopamine | Parkinson’s disease | Parkinson's disease | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | LTP | METABOTROPIC GLUTAMATE RECEPTORS | CA1 NEURONS | NEUROSCIENCES | IN-VITRO | SLICES | PROTEASOME | MICE | LONG-TERM POTENTIATION | PARKINSONS-DISEASE | Sulpiride - pharmacology | Electric Stimulation | Neural Pathways - drug effects | Neuronal Plasticity - drug effects | Male | Excitatory Postsynaptic Potentials - drug effects | Quinoxalines - pharmacology | Long-Term Potentiation - drug effects | Time Factors | Tetrodotoxin - pharmacology | Neurons - drug effects | Receptors, Dopamine - physiology | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Mice, Inbred C57BL | Valine - analogs & derivatives | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Benzazepines - pharmacology | Biophysics | Hippocampus - cytology | Neurotoxins - pharmacology | Patch-Clamp Techniques | Animals | Dopamine Antagonists - pharmacology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Mice | Valine - pharmacology | In Vitro Techniques | Neurochemistry | Neurology | Dopamine | Index Medicus | Neurodegenerative diseases | Glutamic acid receptors (ionotropic) | Brain slice preparation | Long-term potentiation | Cognitive ability | Plasticity (presynaptic) | N-Methyl-D-aspartic acid receptors | Antagonists | Glutamic acid receptors | Vesicles | Dopamine D1 receptors | Plasticity (synaptic) | Synaptic transmission | Dopamine D2 receptors | Hippocampus | Movement disorders | Dopamine receptors
Journal Article
The Journal of Sexual Medicine, ISSN 1743-6095, 05/2008, Volume 5, Issue 5, pp. 1114 - 1125
In addition to nitric oxide (NO), it is thought that an endothelium-derived hyperpolarizing factor (EDHF) plays an important role in the relaxation of penile... 
Acetylcholine | CNP | Human Penile Arteries | EDHF | Endothelium | endothelium | CA2+-ACTIVATED K+ CHANNELS | INDUCED VASODILATION | human penile arteries | NONADRENERGIC NONCHOLINERGIC NEUROTRANSMISSION | MEDIATED RELAXATION | SMOOTH-MUSCLE | CORPUS CAVERNOSUM | ENDOTHELIUM-DEPENDENT RELAXATIONS | NITRIC-OXIDE | UROLOGY & NEPHROLOGY | acetylcholine | ERECTILE DYSFUNCTION | GUANYLYL CYCLASE-B | Natriuretic Agents - pharmacology | Humans | Middle Aged | Male | Apamin - pharmacology | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - drug effects | Arteries - physiology | Charybdotoxin - pharmacology | Natriuretic Peptide, C-Type - drug effects | Vascular Resistance - drug effects | Adult | Natriuretic Peptide, C-Type - pharmacology | Penis - blood supply | Muscle, Smooth, Vascular - physiology | Ouabain - pharmacology | Membrane Potentials - drug effects | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Chlorides - pharmacology | Enzyme Inhibitors - pharmacology | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Arteries - drug effects | Membrane Potentials - physiology | Indomethacin - pharmacology | Barium Compounds - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Peptides - pharmacology | Neurotoxins - pharmacology | Nitroarginine - pharmacology | Natriuretic Peptide, C-Type - metabolism | Adolescent | Blood vessels | Endothelium-derived relaxing factors | Dilatation | Arteries | Natriuretic peptides | Index Medicus
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 08/2009, Volume 24, Issue 3-4, pp. 141 - 152
Microglia are the intrinsic immune cells of the brain. As such, they are crucially involved in neuro-protection as well as neuro-degeneration. Their activation... 
Original Paper | Brain macrophage | Margatoxin | Kir2.1 | Potassium channels | Kv1.3 | Patch-clamp | Microglia | PHYSIOLOGY | TGF-BETA | DISTINCT | COMPONENTS | CELL BIOLOGY | BRAIN MACROPHAGES | ION CHANNELS | DEPENDENT POTASSIUM CHANNELS | UP-REGULATION | EXPRESSION | CONTRIBUTES | Microglia - metabolism | Potassium Channels - physiology | Electrophysiology | Apamin - pharmacology | Dose-Response Relationship, Drug | Electrodes | Microglia - physiology | Cations - pharmacology | Charybdotoxin - pharmacology | 4-Aminopyridine - pharmacology | Potassium Channel Blockers - pharmacology | Pyrazoles - pharmacology | Membrane Potentials - drug effects | Microglia - cytology | Cell Line | Calcium - pharmacology | Fluorescent Antibody Technique, Direct | Mice, Inbred C57BL | Potassium Channels, Inwardly Rectifying | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Barium - pharmacology | Elapid Venoms - pharmacology | Membrane Potentials - physiology | Potassium Channels - drug effects | Neurotoxins - pharmacology | Scorpion Venoms - pharmacology | Transforming Growth Factor beta - pharmacology | Patch-Clamp Techniques | Animals | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Genistein - pharmacology | Lipopolysaccharides - pharmacology | Mice | Drug Combinations | Interferon-gamma - pharmacology | Index Medicus | Life Sciences | Biochemistry, Molecular Biology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2013, Volume 33, Issue 8, pp. 1892 - 1901
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/1997, Volume 17, Issue 21, pp. 8147 - 8155
Dopamine has been shown to stimulate phosphorylation of DARPP-32, a phosphoprotein highly enriched in medium-sized spiny neurons of the neostriatum. Here, we... 
Phosphorylation | Raclopride | Dopamine | Calcineurin | Neostriatum | DARPP-32 | D2 receptor | CELLS | MOLECULAR-BIOLOGY | RAT | INNERVATED BRAIN-REGIONS | PROTEIN PHOSPHATASE INHIBITOR-1 | calcineurin | NEUROSCIENCES | K RU NEUROSCIENCES | raclopride | ADENOSINE 3'-5'-MONOPHOSPHATE-REGULATED PHOSPHOPROTEIN | NEURONAL PHOSPHOPROTEIN | neostriatum | RECEPTORS | dopamine | phosphorylation | STRIATAL NEURONS | EXPRESSION | Cyclosporine - pharmacology | Male | Nucleus Accumbens - physiology | Calcineurin Inhibitors | Fenoldopam - pharmacology | Calcium - physiology | Protein Processing, Post-Translational - drug effects | Phosphoproteins | Receptors, Dopamine D2 - drug effects | Tetrodotoxin - pharmacology | Dopamine Agonists - pharmacology | Receptors, N-Methyl-D-Aspartate - drug effects | Colforsin - pharmacology | 8-Bromo Cyclic Adenosine Monophosphate - pharmacology | Mice, Inbred C57BL | Receptors, Dopamine D1 - drug effects | Dopamine - pharmacology | Chelating Agents - pharmacology | Receptors, N-Methyl-D-Aspartate - physiology | Dopamine and cAMP-Regulated Phosphoprotein 32 | Nerve Tissue Proteins - metabolism | Neurotoxins - pharmacology | Animals | Egtazic Acid - pharmacology | Signal Transduction - drug effects | Receptors, Dopamine D1 - physiology | Corpus Striatum - drug effects | Corpus Striatum - physiology | Receptors, Dopamine D2 - physiology | Mice | Quinpirole - pharmacology | Antipsychotic Agents - pharmacology | Nucleus Accumbens - drug effects | Salicylamides - pharmacology | Calcineurin - metabolism | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2005, Volume 25, Issue 6, pp. 1324 - 1334
Mitochondria release proteins that propagate both caspase-dependent and caspase-independent cell death pathways. AIF (apoptosis-inducing factor) is an... 
Neuron | Caspase independent | Kainic acid | Excitotoxicity | AIF | Apoptosis | caspase independent | POLY(ADP-RIBOSE) POLYMERASE | ACTIVATION | kainic acid | neuron | apoptosis | DNA-DEGRADATION | NEUROSCIENCES | NUCLEAR TRANSLOCATION | CAENORHABDITIS-ELEGANS | NMDA | excitotoxicity | FACTOR AIF | MITOCHONDRIAL RELEASE | CEREBRAL-ISCHEMIA | BRAIN | Proto-Oncogene Proteins c-bcl-2 - physiology | Apoptosis Inducing Factor | Cells, Cultured - cytology | Recombinant Fusion Proteins - physiology | Male | Neurons - cytology | Seizures - metabolism | Hippocampus - drug effects | Cerebral Cortex - cytology | N-Methylaspartate - pharmacology | Seizures - chemically induced | Seizures - pathology | Membrane Proteins - physiology | Mice, Mutant Strains | Neurons - metabolism | Kainic Acid - toxicity | Neurons - drug effects | Drug Resistance | Apoptotic Protease-Activating Factor 1 | Cells, Cultured - drug effects | Benzodiazepines - pharmacology | Glutamic Acid - pharmacology | Flavoproteins - genetics | Membrane Proteins - genetics | Kainic Acid - pharmacology | Hippocampus - pathology | bcl-2-Associated X Protein | Benzothiadiazines - pharmacology | Caspase Inhibitors | Mice, Knockout | Flavoproteins - physiology | Neurotoxins - pharmacology | Proteins - genetics | Hippocampus - metabolism | Animals | Glycine - pharmacology | Convulsants - toxicity | Cerebellum - cytology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Cells, Cultured - metabolism | Mice | Apoptosis - physiology | Dizocilpine Maleate - pharmacology | Camptothecin - pharmacology | Index Medicus | Neurobiology of Disease
Journal Article