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American Journal of Medical Genetics Part C: Seminars in Medical Genetics, ISSN 1552-4868, 02/2011, Volume 157, Issue 1, pp. 33 - 44
Derivatives of cobalamin (vitamin B12) are required for activity of two enzymes in humans. Adenosylcobalamin is required for activity of mitochondrial... 
methylmalonic acidemia | vitamin B12 | cobalamin | homocystinuria | transcobalamin | Methylmalonic acidemia | Cobalamin | Transcobalamin | Homocystinuria | Vitamin B12 | HEREDITARY MEGALOBLASTIC-ANEMIA | HOMOCYSTINURIA CBLC | COMBINED METHYLMALONIC ACIDURIA | INTRINSIC-FACTOR GENE | METHIONINE SYNTHASE REDUCTASE | CBLB COMPLEMENTATION GROUP | METHYLCOBALAMIN DEFICIENCY | GENETICS & HEREDITY | TRANSCOBALAMIN-I HAPTOCORRIN | VITAMIN-B-12 METABOLISM | MOUSE GASTRULATION | Malabsorption Syndromes - diagnosis | Methylmalonic Acid - blood | Humans | Vitamin B 12 - analogs & derivatives | Hyperhomocysteinemia - metabolism | Malabsorption Syndromes - metabolism | Neonatal Screening | Amino Acid Metabolism, Inborn Errors - diagnosis | Methylmalonyl-CoA Mutase - metabolism | Amino Acid Metabolism, Inborn Errors - metabolism | Homocysteine - urine | Anemia, Megaloblastic | Proteinuria - diagnosis | Vitamin B 12 Deficiency - metabolism | Infant, Newborn | Methylmalonic Acid - urine | Cobamides - metabolism | Vitamin B 12 Deficiency - diagnosis | Metabolism, Inborn Errors - metabolism | Homocysteine - blood | Proteinuria - metabolism | Methylmalonyl-CoA Mutase - deficiency | Metabolism, Inborn Errors - diagnosis | Vitamin B 12 Deficiency - genetics | Hyperhomocysteinemia - diagnosis | Vitamin B 12 - metabolism | Thrombocytopenia | Urine | homocysteine | Enzymes | 5-Methyltetrahydrofolate-homocysteine S-methyltransferase | Anemia | adenosylcobalamin | Blood | Psychosis | Mitochondria | Dementia disorders | Complementation | Degeneration | Metabolic acidosis | Neutropenia
Journal Article
Journal Article
Journal Article
Journal of lipid research, ISSN 0022-2275, 2005, Volume 46, Issue 6, pp. 1182 - 1195
Journal Article
Haematologica, ISSN 0390-6078, 07/2015, Volume 100, Issue 7, pp. 927 - 934
We previously demonstrated vast expansion of hypoxic areas in the leukemic microenvironment and provided a rationale for using hypoxia-activated prodrugs.... 
STEM-CELLS | INHIBITION | PR-104A | ACUTE MYELOGENOUS LEUKEMIA | BONE-MARROW | I TRIAL | KETO REDUCTASE 1C3 | HIF-1-ALPHA | HEMATOLOGY | CANCER-THERAPY | CHEMOTHERAPY | Recurrence | Humans | Middle Aged | Male | Antineoplastic Agents, Alkylating - administration & dosage | Carbonic Anhydrases - metabolism | Thrombocytopenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Anemia - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Bone Marrow - metabolism | Enterocolitis - genetics | Antigens, Neoplasm - metabolism | Bone Marrow - drug effects | Biomarkers - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - complications | Gene Expression | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Leukemia, Myeloid, Acute - pathology | Anemia - chemically induced | Hypoxia - complications | Thrombocytopenia - chemically induced | Thrombocytopenia - pathology | Nitrogen Mustard Compounds - adverse effects | Remission Induction | Hypoxia - pathology | Antineoplastic Agents, Alkylating - adverse effects | Enterocolitis - pathology | Prodrugs - administration & dosage | Hypoxia - drug therapy | Neutropenia - metabolism | Nitrogen Mustard Compounds - administration & dosage | Carbonic Anhydrases - genetics | Anemia - pathology | Leukemia, Myeloid, Acute - complications | Prodrugs - metabolism | Leukemia, Myeloid, Acute - drug therapy | Neutropenia - pathology | Adult | Female | Neutropenia - chemically induced | Neutropenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Carbonic Anhydrase IX | Enterocolitis - chemically induced | Antigens, Neoplasm - genetics | Nitroimidazoles - pharmacology | Thrombocytopenia - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Anemia - metabolism | Prodrugs - adverse effects | Hypoxia - genetics | Nitrogen Mustard Compounds - metabolism | Bone Marrow - pathology | Aged | Enterocolitis - metabolism | Antineoplastic Agents, Alkylating - metabolism | Leukemia, Myeloid, Acute - genetics
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2008, Volume 295, Issue 6, pp. 2242 - 2249
We hypothesized that neutralization of TNF-α at the time of reperfusion exerts a salubrious role on endothelial function and reduces the production of reactive... 
Microcirculation | Free radicals | Coronary disease | Nitric oxide | Endothelium | RHEUMATOID-ARTHRITIS | endothelium | free radicals | nitric oxide | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | HEART-FAILURE | MYOCARDIAL-ISCHEMIA | LUNG INJURY | NECROSIS-FACTOR-ALPHA | microcirculation | CORONARY MICROEMBOLIZATION | coronary disease | THERAPY | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | ETANERCEPT | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Oxidative Stress | Neutropenia - metabolism | Coronary Vessels - physiopathology | Tumor Necrosis Factor-alpha - genetics | NADPH Oxidases - metabolism | Male | RNA, Messenger - metabolism | Muscle, Smooth, Vascular - physiopathology | Vasodilation | Coronary Vessels - metabolism | Superoxides - metabolism | Tumor Necrosis Factor-alpha - immunology | Female | Disease Models, Animal | Endothelium, Vascular - immunology | Myocardial Reperfusion Injury - immunology | Coronary Circulation | Xanthine Oxidase - metabolism | Endothelium, Vascular - physiopathology | Coronary Vessels - immunology | Myocardial Reperfusion Injury - physiopathology | Antibodies - administration & dosage | Myocardial Reperfusion Injury - metabolism | Animals | Endothelium, Vascular - metabolism | Peroxynitrous Acid - metabolism | Mice | Nitric Oxide - metabolism | Myocardial Reperfusion Injury - prevention & control | Neutropenia - physiopathology | Peroxidase - metabolism | Translational Physiology
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2012, Volume 42, Issue 9, pp. 2395 - 2408
Journal Article
Shock, ISSN 1073-2322, 08/2005, Volume 24, Issue 2, pp. 132 - 138
Journal Article
Journal Article
Cancer, ISSN 0008-543X, 06/2017, Volume 123, Issue 11, pp. 1979 - 1988
BACKGROUND Advanced cholangiocarcinoma carries a poor prognosis, and no standard treatment exists beyond first‐line gemcitabine/platinum‐based chemotherapy. A... 
vascular endothelial growth factor receptor 2 (VEGFR2) | phase 2 | soluble MET | cholangiocarcinoma | cabozantinib | FACTOR-RECEPTOR | BILIARY-TRACT CANCER | INTRAHEPATIC CHOLANGIOCARCINOMA | INTERLEUKIN-6 | OVEREXPRESSION | ONCOLOGY | C-MET | PROGNOSTIC-SIGNIFICANCE | INHIBITOR | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Proto-Oncogene Proteins c-met - metabolism | Anilides - therapeutic use | Humans | Middle Aged | Placenta Growth Factor - metabolism | Male | Vascular Endothelial Growth Factor A - metabolism | Cholangiocarcinoma - metabolism | Intestinal Perforation - chemically induced | Angiopoietin-2 - metabolism | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Neoplasm Metastasis | Hyperbilirubinemia - chemically induced | Hypertension - chemically induced | Biomarkers, Tumor - metabolism | Female | Neutropenia - chemically induced | Interleukin-6 - metabolism | Bile Duct Neoplasms - drug therapy | Pyridines - therapeutic use | Bile Duct Neoplasms - metabolism | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Epistaxis - chemically induced | Survival Rate | Disease-Free Survival | Cholangiocarcinoma - pathology | Chemokine CXCL12 - metabolism | Cholangiocarcinoma - drug therapy | Protein Kinase Inhibitors - therapeutic use | Intestinal Fistula - chemically induced | Aged | Bile Duct Neoplasms - pathology | Matrix Metalloproteinase 1 - metabolism | Cancer patients | Research | Biological markers | Health aspects | Drugs | Plasma | Gemcitabine | Toxicity | Perforation | Angiopoietin | Interleukin | Population studies | Matrix metalloproteinase | Metastases | Reduction (metal working) | Confidence intervals | Interleukin 6 | Hyperbilirubinemia | Platinum | Intestine | Quality | Bioindicators | Metalloproteinase | Inhibition | Vascular endothelial growth factor receptor 2 | Vascular endothelial growth factor | Drug dosages | Cholangiocarcinoma | Age | Neutropenia | Hypertension | SDF-1 protein | Patients | Survival | Chemotherapy | Placenta | Inhibitors | Antiangiogenics | Medical prognosis | Biomarkers | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Blood, ISSN 0006-4971, 02/2017, Volume 129, Issue 6, pp. 715 - 722
Chronic neutrophilic leukemia (CNL) is a distinct myeloproliferative neoplasm with a high prevalence (> 80%) of mutations in the colony-stimulating factor 3... 
CHRONIC MYELOGENOUS LEUKEMIA | ATYPICAL CML | STIMULATING-FACTOR-RECEPTOR | CLONAL HEMATOPOIESIS | SEVERE CONGENITAL NEUTROPENIA | MYELOPROLIFERATIVE NEOPLASMS | ACUTE MYELOID-LEUKEMIA | G-CSF RECEPTOR | HEMATOLOGY | SETBP1 MUTATIONS | CHRONIC MYELOMONOCYTIC LEUKEMIA | Genomics | Humans | Antineoplastic Agents - therapeutic use | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | DNA-Binding Proteins - metabolism | Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative - drug therapy | Receptors, Colony-Stimulating Factor - genetics | Receptors, Colony-Stimulating Factor - metabolism | Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative - genetics | Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative - pathology | Leukemia, Neutrophilic, Chronic - pathology | Nuclear Proteins - genetics | Neutrophils - metabolism | Repressor Proteins - metabolism | Neutrophils - pathology | Proto-Oncogene Proteins - metabolism | Neutrophils - drug effects | Gene Expression Regulation, Leukemic - drug effects | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | DNA-Binding Proteins - genetics | Disease Progression | Splicing Factor U2AF - genetics | Carrier Proteins - genetics | Carrier Proteins - metabolism | Leukemia, Neutrophilic, Chronic - drug therapy | Cell Proliferation - drug effects | Leukemia, Neutrophilic, Chronic - metabolism | Leukemia, Neutrophilic, Chronic - genetics | Mutation | Leukemia, Myeloid, Chronic, Atypical, BCR-ABL Negative - metabolism | Clonal Evolution | Practice Guidelines as Topic | Splicing Factor U2AF - metabolism | Myeloproliferative Neoplasms | Review Series
Journal Article