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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 19, pp. 8748 - 8753
Balanced induction of proinflammatory and type I IFN responses upon activation of Toll-like receptors (TLRs... 
Pathogens | Receptors | Bacterial infections | Genes | Internalization | Neutrophils | Bacteria | Mast cells | Infections | Macrophages | Salmonella | Vesicular stomatitis virus | Innate immunity | Endolysosome | Listeria | LISTERIA-MONOCYTOGENES | endolysosome | MULTIDISCIPLINARY SCIENCES | vesicular stomatitis virus | SUSCEPTIBILITY | RECEPTOR | LUNG INFECTION | INDUCTION | IFN | innate immunity | ROLES | AUTOIMMUNITY | RESISTANCE | MICE | Neutrophils - cytology | Transcription, Genetic - drug effects | Mast Cells - ultrastructure | Interferon-beta - immunology | Vesiculovirus - drug effects | Vesiculovirus - immunology | Listeria monocytogenes - immunology | Acids - metabolism | Endosomes - metabolism | Mast Cells - microbiology | Listeria monocytogenes - ultrastructure | Salmonella typhimurium - ultrastructure | Endosomes - drug effects | Biological Transport - drug effects | Salmonella typhimurium - drug effects | Inflammation Mediators - immunology | Mast Cells - immunology | Intracellular Space - drug effects | Salmonella typhimurium - immunology | Subcellular Fractions - drug effects | Endocytosis - drug effects | Neutrophils - drug effects | Toll-Like Receptors - immunology | Neutrophils - immunology | Toll-Like Receptor 4 - immunology | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Vesiculovirus - ultrastructure | Animals | Interferon-beta - pharmacology | Intracellular Space - microbiology | Lipopolysaccharides - pharmacology | Cell Compartmentation - drug effects | Mast Cells - cytology | Mice | Mice, Inbred BALB C | Listeria monocytogenes - drug effects | Autoimmunity | Stomatitis | Lysosomes | Interferon | Dosage and administration | Research | Biological response modifiers | Properties | Drug therapy | Antibacterial agents | Biological Sciences
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 09/2016, Volume 311, Issue 3, pp. H725 - H734
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e58371
Background: The present study investigates the effects and mechanisms of alpha-Lipoic acid (LA... 
ISCHEMIA-REPERFUSION INJURY | CELLS | OXIDATIVE STRESS | HEME OXYGENASE-1 EXPRESSION | NRF2 | PHOSPHORYLATION | PROTEIN-KINASE | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | ERYTHROID 2-RELATED FACTOR-2 | Tumor Necrosis Factor-alpha - metabolism | Thioctic Acid - administration & dosage | Apoptosis - drug effects | Male | Phosphatidylinositol 3-Kinases - metabolism | Protein Transport - drug effects | Heme Oxygenase-1 - genetics | Myocardial Reperfusion Injury - pathology | Cell Nucleus - metabolism | Myocardial Infarction - pathology | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Neutrophils - pathology | Creatine Kinase - blood | Ventricular Function, Left - drug effects | Rats | Thioctic Acid - pharmacology | Cardiotonic Agents - pharmacology | Myocardial Reperfusion Injury - physiopathology | Gene Expression Regulation - drug effects | Myocardial Reperfusion Injury - metabolism | Animals | Cardiotonic Agents - administration & dosage | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | L-Lactate Dehydrogenase - blood | NF-E2-Related Factor 2 - metabolism | Myocytes, Cardiac - metabolism | Myocardial infarction | Lipoic acid | Oxidative stress | Phosphorylation | Dehydrogenases | Heart surgery | AKT protein | Myocardial ischemia | Activation | Kinases | Heart function | Accumulation | Antioxidants | Signal transduction | Reperfusion | Ischemia | Rodents | Inhibition | Pretreatment | Cardiology | Heart diseases | Translocation | Coronary artery | Cardiomyocytes | Inflammation | Tumor necrosis factor-α | Nuclear transport | 1-Phosphatidylinositol 3-kinase | Injury prevention | Acids | Nitric oxide | Myocardium | In vivo methods and tests | Diabetes | Laboratory animals | Apoptosis
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 2/2014, Volume 355, Issue 2, pp. 375 - 396
.... We test the hypothesis that angiostatin inhibits neutrophil activation, induces apoptosis and blocks recruitment in vivo and in vitro... 
Human Genetics | Intravital microscopy | Lipid raft | Biomedicine | Neutrophil polarization | Proteomics | Integrin α v β 3 | Molecular Medicine | Apoptosis | Integrin α | COMPLEX | LEUKOCYTE RECRUITMENT | CHEMOTAXIS | PLASMINOGEN | CELL BIOLOGY | Integrin alpha(v)beta | RECEPTOR ANNEXIN-II | REACTIVE OXYGEN | ENDOTHELIAL-CELLS | ACTIN CYTOSKELETON | BETA INTEGRINS | MAPK PATHWAY | Inflammation - pathology | Neutrophils - cytology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Membrane Microdomains - metabolism | Humans | Neutrophil Activation - drug effects | Muscles - pathology | Protein Subunits - metabolism | Proton-Translocating ATPases - metabolism | Microtubules - metabolism | Microtubules - drug effects | Oxidation-Reduction - drug effects | N-Formylmethionine Leucyl-Phenylalanine - pharmacology | Cell Polarity - drug effects | Membrane Proteins - metabolism | Muscles - metabolism | Neutrophil Infiltration - drug effects | Neutrophils - enzymology | Endocytosis - drug effects | Neutrophils - drug effects | Mitochondria - metabolism | CD18 Antigens - metabolism | Cell Movement - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | MAP Kinase Signaling System - drug effects | Membrane Microdomains - drug effects | Angiostatins - pharmacology | Muscles - drug effects | Lipopolysaccharides - pharmacology | Mice | Heat shock proteins | Integrins | Angiogenesis | Microscopy | Cell adhesion & migration
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 1322 - 14
...). Here the authors show neutrophil activation and NETosis are elevated in patients with HIT, and are essential for thrombosis in HIT mouse models. 
ANTIBODIES | CLEARANCE | DIAGNOSIS | PLATELETS | DNA | MULTIDISCIPLINARY SCIENCES | MOUSE | MONOCYTES | EXTRACELLULAR TRAP FORMATION | FC-GAMMA-RIIA | Protein-Arginine Deiminases - genetics | Thrombosis - chemically induced | Blood Platelets - immunology | Protein-Arginine Deiminase Type 4 | Humans | Platelet Activation - immunology | Neutrophil Activation - drug effects | Platelet Factor 4 - immunology | Heparin - adverse effects | Receptors, IgG - genetics | Extracellular Traps - immunology | Immunoglobulin G - biosynthesis | Extracellular Traps - chemistry | Extracellular Traps - drug effects | Thrombosis - prevention & control | Blood Platelets - drug effects | Platelet Factor 4 - genetics | Antigen-Antibody Complex - biosynthesis | Citrullination | Microfluidic Analytical Techniques | Neutrophil Activation - immunology | Protein-Arginine Deiminases - immunology | Histones - immunology | Signal Transduction | Neutrophils - drug effects | Thrombocytopenia - immunology | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Neutrophils - immunology | Thrombosis - immunology | Mice, Transgenic | Thrombocytopenia - chemically induced | Receptors, IgG - immunology | Thrombocytopenia - pathology | Thrombosis - pathology | Animals | Histones - genetics | Mice | Platelet Activation - drug effects | Protein-Arginine Deiminases - antagonists & inhibitors | Thrombocytopenia | Antigens | Immune response | Pathogenesis | Immunoglobulin G | Neutrophils | Antibodies | Leukocytes (neutrophilic) | Citrulline | Thrombosis | Morbidity | Catheters | Blood clots | Platelet factor 4 | Cell activation | Side effects | Depletion | Heparin | Thromboembolism | Platelets | Deoxyribonucleic acid--DNA | Histone H3 | Microfluidics | Antigen-antibody complexes
Journal Article
Journal of neuroinflammation, ISSN 1742-2094, 2016, Volume 13, Issue 1, p. 23
...) to mimic local infection of the brain and investigated the role of key complement component C3 in brain vasculature endothelial activation and leukocyte recruitment... 
Intravital microscopy | Adhesion molecule | Complement | Leukocyte recruitment | CNS inflammation | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | CHEMOKINE EXPRESSION | PNEUMOCOCCAL MENINGITIS | IMMUNOLOGY | CEREBROSPINAL-FLUID | NEUROSCIENCES | NEUTROPHIL RECRUITMENT | STREPTOCOCCUS-PNEUMONIAE | BACTERIAL-MENINGITIS | MULTIPLE-SCLEROSIS | INTRACEREBRAL HEMORRHAGE | CENTRAL-NERVOUS-SYSTEM | Cell Adhesion Molecules - genetics | Receptors, Complement - deficiency | Injections, Intraventricular | Male | NF-kappa B - metabolism | Leukocytes - physiology | Complement C3a - genetics | Neutrophil Infiltration - physiology | Time Factors | Receptors, Complement - genetics | Blood-Brain Barrier - physiology | Complement C3a - metabolism | Endothelial Cells - physiology | Neutrophil Infiltration - drug effects | Brain - cytology | Cell Adhesion - genetics | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Cells, Cultured | Microvessels - drug effects | Mice, Transgenic | Cell Adhesion - drug effects | Blood-Brain Barrier - drug effects | Cell Adhesion Molecules - metabolism | Brain - drug effects | Gene Expression Regulation - drug effects | Microvessels - physiology | Animals | Lipopolysaccharides - pharmacology | Leukocytes - drug effects | Mice | Mice, Inbred BALB C | Endothelial Cells - drug effects | Complications and side effects | Development and progression | Nervous system diseases | Research | Endothelium
Journal Article
Journal Article
Journal Article
Journal Article
Science translational medicine, ISSN 1946-6242, 2019, Volume 11, Issue 486, p. eaav0537
Journal Article