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1. Full Text Distinct Infiltration of Neutrophils in Lesion Shoulders in ApoE−/− Mice
by Rotzius, Pierre and Thams, Sebastian and Soehnlein, Oliver and Kenne, Ellinor and Tseng, Chi-Nan and Björkström, Niklas K and Malmberg, Karl-Johan and Lindbom, Lennart and Eriksson, Einar E
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 177, Issue 1, pp. 493 - 500
Inflammation and activation of immune cells are key mechanisms in the development of atherosclerosis. Previous data indicate important roles for monocytes and... 
Pathology | ATHEROSCLEROTIC LESIONS | CYTOKINE-ACTIVATED NEUTROPHILS | GREEN FLUORESCENT PROTEIN | MYELOPEROXIDASE | MYOCARDIAL-INFARCTION | MACROPHAGES | ENDOTHELIAL-CELLS | MONOCYTES | ACUTE CORONARY SYNDROMES | PATHOLOGY | EXPRESSION | Neutrophils - cytology | Atherosclerosis - pathology | Atherosclerosis - immunology | Apolipoproteins E - deficiency | Monocytes - cytology | Humans | Leukocyte Rolling | Neutrophils - immunology | Aorta - immunology | Green Fluorescent Proteins - genetics | Monocytes - immunology | Plaque, Atherosclerotic - immunology | Gene Knock-In Techniques | Mice, Knockout | Aorta - pathology | Animals | Green Fluorescent Proteins - immunology | Apolipoproteins E - genetics | Bone Marrow Transplantation | Mice | Aorta - cytology | Plaque, Atherosclerotic - pathology | Regular
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2. Full Text Increased inflammatory gene expression in ABC transporter-deficient macrophages: Free cholesterol accumulation, increased signaling via toll-like receptors, and neutrophil infiltration of atherosclerotic lesions
by Yvan-Charvet, Laurent and Welch, Carrie and Pagler, Tamara A and Ranalletta, Mollie and Lamkanfi, Mohamed and Han, Seongah and Ishibashi, Minako and Li, Rong and Wang, Nan and Tall, Alan R
Circulation, ISSN 0009-7322, 10/2008, Volume 118, Issue 18, pp. 1837 - 1847
Background-Two macrophage ABC transporters, ABCA1 and ABCG1, have a major role in promoting cholesterol efflux from macrophages. Peritoneal macrophages... 
Lipoproteins | ABC transporters | Inflammation | Cholesterol | Atherosclerosis | atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | INDUCED APOPTOSIS | CELLULAR CHOLESTEROL | NECROSIS-FACTOR-ALPHA | ABCG1(-/-) BONE-MARROW | HIGH-DENSITY-LIPOPROTEIN | APOLIPOPROTEIN-E | LIPID-ACCUMULATION | EFFLUX | inflammation | ABCA1-DEFICIENT MACROPHAGES | cholesterol | PERIPHERAL VASCULAR DISEASE | MICE | lipoproteins | ATP Binding Cassette Transporter, Sub-Family G, Member 1 | Lipoproteins - genetics | Membrane Microdomains - immunology | Signal Transduction - immunology | ATP-Binding Cassette Transporters - genetics | Mice, Mutant Strains | Lipoproteins - metabolism | ATP-Binding Cassette Transporters - metabolism | Mice, Inbred DBA | ATP Binding Cassette Transporter 1 | Macrophages - immunology | Receptors, LDL - genetics | Atherosclerosis - immunology | Atherosclerosis - physiopathology | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Neutrophils - immunology | Receptors, LDL - metabolism | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Cholesterol - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Atherosclerosis - metabolism | Macrophages - metabolism | Animals | Lipopolysaccharides - pharmacology | Ligands | Macrophages - drug effects | Mice | ATP-Binding Cassette Transporters - immunology | Lipoproteins - immunology | Physiological aspects | Research | Macrophages | Gene expression | Toll-receptors
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3. Full Text Leukocyte infiltration in experimental stroke
by Grønberg, Nina Vindegaard and Johansen, Flemming Fryd and Kristiansen, Uffe and Hasseldam, Henrik
Journal of Neuroinflammation, ISSN 1742-2094, 09/2013, Volume 10, Issue 1, pp. 115 - 115
Stroke is one of the leading causes of death worldwide. At present, the only available treatment is thrombolysis, which should be initiated no later than 4.5... 
T-cells | Stroke | Neutrophil granulocytes | Leukocytes | Macrophages | Middle cerebral artery occlusion (MCAO) | MICROGLIAL ACTIVATION | ARTERY OCCLUSION | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | IMMUNOLOGY | NEUROSCIENCES | HEMORRHAGIC TRANSFORMATION | INFLAMMATORY CELLS | STRAIN DIFFERENCES | REGULATORY T-CELLS | NEUTROPHIL DEPLETION | BORNE CELL RECRUITMENT | Stroke - pathology | Animals | Macrophages - pathology | Neutrophils - immunology | Granulocytes - pathology | T-Lymphocytes - immunology | T-Lymphocytes - pathology | Granulocytes - immunology | Macrophages - immunology | Stroke - immunology | Disease Models, Animal | Neutrophils - pathology | Stroke (Disease) | Prevention | Brain research | Inflammation | T cells | Health aspects | Studies | Statistical analysis | Free radicals | Ischemia | Rodents | Experiments | Veins & arteries | Cerebral blood flow
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4. Full Text Peripheral and central neuronal ATF3 precedes CD4+ T-cell infiltration in EAE
by Frezel, Noémie and Sohet, Fabien and Daneman, Richard and Basbaum, Allan I and Braz, Joao M
Experimental Neurology, ISSN 0014-4886, 09/2016, Volume 283, Issue Pt A, pp. 224 - 234
Experimental allergic encephalomyelitis (EAE), an animal model of multiple sclerosis produced by immunization with myelin oligodendrocyte glycoprotein (MOG)... 
Spinal cord | Myelin oligodendrocyte glycoprotein | Demyelination | Dorsal root ganglion | Experimental allergic encephalomyelitis | ATF3 | Pertussis toxin | CELLS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DORSAL-ROOT GANGLIA | SPINAL-CORD | CNS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | NEUROPEPTIDE-Y | PAIN | MULTIPLE-SCLEROSIS | BORDETELLA-PERTUSSIS | Freund's Adjuvant - toxicity | Ganglia - metabolism | Peptide Fragments - toxicity | CD4-Positive T-Lymphocytes - physiology | Neutrophil Infiltration - physiology | Brain - metabolism | TRPV Cation Channels - metabolism | Myelin-Oligodendrocyte Glycoprotein - toxicity | Female | Neurons - metabolism | Neurons - drug effects | Neutrophil Infiltration - drug effects | Disease Models, Animal | Pertussis Toxin - pharmacology | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Neurotransmitter Agents - metabolism | Macrophage Colony-Stimulating Factor - metabolism | Activating Transcription Factor 3 - metabolism | Disease Progression | Nerve Tissue Proteins - metabolism | Ganglia - drug effects | Animals | Brain - pathology | Mice | Multiple sclerosis | T cells | Neurons | Analysis | Myelin proteins | Neurosciences
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5. Full Text TRPM2-mediated Ca2+ influx induces chemokine production in monocytes that aggravates inflammatory neutrophil infiltration
by Yamamoto, Shinichiro and Shimizu, Shunichi and Kiyonaka, Shigeki and Takahashi, Nobuaki and Wajima, Teruaki and Hara, Yuji and Negoro, Takaharu and Hiroi, Toshihito and Kiuchi, Yuji and Okada, Takaharu and Kaneko, Shuji and Lange, Ingo and Fleig, Andrea and Penner, Reinhold and Nishi, Miyuki and Takeshima, Hiroshi and Mori, Yasuo
Nature Medicine, ISSN 1078-8956, 07/2008, Volume 14, Issue 7, pp. 738 - 747
Reactive oxygen species (ROS) induce chemokines responsible for the recruitment of inflammatory cells to sites of injury or infection. Here we show that the... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ULCERATIVE-COLITIS | TRP CHANNELS | ADP-RIBOSE | BOWEL-DISEASE | CELL BIOLOGY | REACTIVE OXYGEN | GENE-EXPRESSION | SODIUM-INDUCED COLITIS | HYDROGEN-PEROXIDE | NF-KAPPA-B | Oxidants - pharmacology | Calcium - metabolism | Interleukin-8 - biosynthesis | Humans | Hydrogen Peroxide - pharmacology | Monocytes - metabolism | Neutrophil Infiltration | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Time Factors | Calcium Signaling - drug effects | Models, Biological | U937 Cells | TRPM Cation Channels - genetics | TRPM Cation Channels - metabolism | Medical research | Biochemistry | Cellular biology | Rodents | Inflammatory diseases | Index Medicus
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6. Full Text Astrocytes are primed by chronic neurodegeneration to produce exaggerated chemokine and cell infiltration responses to acute stimulation with the cytokines IL-1β and TNF-α
by Hennessy, Edel and Griffin, Eádaoin W and Cunningham, Colm
Journal of Neuroscience, ISSN 0270-6474, 06/2015, Volume 35, Issue 22, pp. 8411 - 8422
Microgliosis and astrogliosis are standard pathological features of neurodegenerative disease. Microglia are primed by chronic neurodegeneration such that... 
Chemokine | Neuroinflammation | Astrocyte | Neurodegeneration | Cytokine | Microglia | cytokine | RECRUITMENT | chemokine | ACTIVATION | neurodegeneration | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | INDUCTION | MODEL | NEUROSCIENCES | neuroinflammation | INFLAMMATORY RESPONSE | astrocyte | microglia | NF-KAPPA-B | EXPRESSION | BRAIN | Tumor Necrosis Factor-alpha - metabolism | Astrocytes - drug effects | Interleukin-1beta - pharmacology | Prion Diseases - drug therapy | Mice, Inbred C57BL | Hippocampus - pathology | Prion Diseases - pathology | Monocytes - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Interleukin-1beta - metabolism | T-Lymphocytes - drug effects | Female | Chemokines - metabolism | Mice | Chronic Disease | Neutrophil Infiltration - drug effects | Disease Models, Animal
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7. Full Text Neutrophil infiltration and chemokines
by Kobayashi, Yoshiro
Critical Reviews in Immunology, ISSN 1040-8401, 2006, Volume 26, Issue 4, pp. 307 - 315
Neutrophil infiltration is mainy mediated by ELR+ CXC chemokines and is regulated through interactions between various stimulants and cells, which are perhaps... 
Neutrophil | IL-8 | MIP-2 | CXCR2 | Macrophage | ISCHEMIA/REPERFUSION INJURY | A-INDUCED HEPATITIS | MIGRATION IN-VIVO | REPERFUSION INJURY | INDUCED LUNG INJURY | macrophage | IMMUNOLOGY | neutrophil | LATE APOPTOTIC CELLS | MACROPHAGE-INFLAMMATORY PROTEIN-2 | FACTOR-ALPHA | TUMOR-NECROSIS-FACTOR | KC | CXC-CHEMOKINES | Animals | Reperfusion Injury - prevention & control | Chemokines - antagonists & inhibitors | Humans | Reperfusion Injury - immunology | Neutrophils - immunology | Chemokines - physiology | Inflammation - immunology | Neutrophil Infiltration | Chemokines - genetics
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8. Full Text Chronic plus binge ethanol feeding synergistically induces neutrophil infiltration and liver injury in mice: A critical role for E‐selectin
by Bertola, Adeline and Park, Ogyi and Gao, Bin
Hepatology, ISSN 0270-9139, 11/2013, Volume 58, Issue 5, pp. 1814 - 1823
Chronic plus binge ethanol feeding acts synergistically to induce liver injury in mice, but the mechanisms underlying this phenomenon remain unclear. Here, we... 
PATHOGENESIS | LEUKOCYTE RECRUITMENT | A-INDUCED HEPATITIS | SEVERE ALCOHOLIC HEPATITIS | INFLAMMATION | ENDOTHELIAL-CELLS | DISEASE | ADHESION MOLECULES | GROWTH-FACTOR | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Interleukin-1beta - physiology | Liver - pathology | Humans | Mice, Inbred C57BL | Alanine Transaminase - blood | Cytochrome P-450 CYP2E1 - genetics | Neutrophil Infiltration | Liver Diseases, Alcoholic - etiology | Animals | E-Selectin - genetics | Tumor Necrosis Factor-alpha - physiology | Mice | E-Selectin - physiology | Vascular Cell Adhesion Molecule-1 - analysis | Intercellular Adhesion Molecule-1 - analysis | Leucocytes | Cytokines | Ethanol | Liver | Rodents | Hepatology | Cell adhesion & migration
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9. Full Text IL‐1α and inflammasome‐independent IL‐1β promote neutrophil infiltration following alum vaccination
by Oleszycka, Ewa and Moran, Hannah B.T and Tynan, Graham A and Hearnden, Claire H and Coutts, Graham and Campbell, Matthew and Allan, Stuart M and Scott, Christopher J and Lavelle, Ed C
FEBS Journal, ISSN 1742-464X, 01/2016, Volume 283, Issue 1, pp. 9 - 24
Despite its long record of successful use in human vaccines, the mechanisms underlying the immunomodulatory effects of alum are not fully understood. Alum is a... 
cathepsin S | aluminium adjuvant | inflammasome | IL-1 | IMMUNE-RESPONSE | ACTIVATION | PROTEIN | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADJUVANT ACTIVITY | NLRP3 INFLAMMASOME | NALP3 INFLAMMASOME | CUTTING EDGE | CRYSTALS | URIC-ACID | Interleukin-1alpha - immunology | Inflammasomes | Mice, Inbred C57BL | Vaccination | Interleukin-1beta - immunology | Alum Compounds - pharmacology | Animals | Macrophages - drug effects | Mice | Alum Compounds - administration & dosage | Macrophages - immunology | Neutrophil Infiltration - drug effects | Neutrophil Infiltration - immunology
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10. Full Text Immune cell infiltration as an indicator of the immune microenvironment of pancreatic cancer
by Ino, Y and Yamazaki-Itoh, R and Shimada, K and Iwasaki, M and Kosuge, T and Kanai, Y and Hiraoka, N
British Journal of Cancer, ISSN 0007-0920, 03/2013, Volume 108, Issue 4, pp. 914 - 923
Background: The host immune reaction is represented by immune/inflammatory cell infiltrates. Here we systematically analysed tumour-infiltrating... 
pancreas cancer | immune microenvironment | immune/inflammatory cell infiltration | prognosis | SURVIVAL | MACROPHAGE PLASTICITY | TUMOR | PHENOTYPE | NEUTROPHILS | ONCOLOGY | SUPPRESSOR-CELLS | DIFFERENTIATION | POLARIZATION | T-CELLS | PROGRESSION | Prognosis | Humans | Middle Aged | Pancreatic Neoplasms - pathology | Tumor Microenvironment | Male | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Disease-Free Survival | Pancreatic Neoplasms - immunology | Survival Analysis | Female | Pancreatic Neoplasms - mortality | Macrophages - immunology | Lymphocytes, Tumor-Infiltrating - immunology | CD4 antigen | inflammatory cell infiltration | immune | Molecular Diagnostics
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11. Full Text Chronic interleukin-1β expression in mouse brain leads to leukocyte infiltration and neutrophil-independent blood-brain barrier permeability without overt neurodegeneration
by Shaftel, Solomon S and Carlson, Thaddeus J and Olschowka, John A and Kyrkanides, Stephanos and Matousek, Sarah B and O'Banion, M. Kerry
Journal of Neuroscience, ISSN 0270-6474, 08/2007, Volume 27, Issue 35, pp. 9301 - 9309
The proinflammatory cytokine interleukin-1 beta( IL-1 beta) plays a significant role in leukocyte recruitment to the CNS. Although acute effects of IL-1 beta... 
Neurotoxicity | Blood- Brain barrier | Interleukin-1β | Hippocampus | CXCR2 | Neutrophils | blood-brain barrier | MEDIATED HOST-DEFENSE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | hippocampus | NEURONAL INJURY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CHEMOKINE RECEPTOR-2 LIGANDS | NEUROSCIENCES | IL-8 RECEPTOR | TUMOR-NECROSIS-FACTOR | interleukin-1 beta | neurotoxicity | MULTIPLE-SCLEROSIS | GLIAL REACTION | CENTRAL-NERVOUS-SYSTEM | neutrophils | blood–brain barrier | interleukin-1β
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12. Full Text The dual CCR2/CCR5 chemokine receptor antagonist Cenicriviroc reduces macrophage infiltration and disease severity in Duchenne muscular dystrophy (DMDmdx-4Cv) mice
by Liang, Feng and Giordano, Christian and Shang, Dong and Li, Qian and Petrof, Basil J
PLoS ONE, ISSN 1932-6203, 03/2018, Volume 13, Issue 3, p. e0194421
Duchenne muscular dystrophy (DMD) is characterized by progressive muscle weakness which is ultimately fatal, most often due to involvement of the diaphragm.... 
RECRUITMENT | ACTIVATION | CC CLASS CHEMOKINES | TGF-BETA | PHARMACOKINETICS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MUSCLE DEGENERATION | EXPRESSION | MDX MICE | DEFICIENCY | Severity of Illness Index | CD8-Positive T-Lymphocytes - pathology | Diaphragm - pathology | Macrophages - pathology | Isometric Contraction - drug effects | Muscular Dystrophy, Duchenne - drug therapy | Receptors, CCR2 - immunology | Muscular Dystrophy, Duchenne - pathology | Imidazoles - pharmacology | CD4-Positive T-Lymphocytes - pathology | Isometric Contraction - immunology | Muscle Strength - immunology | Receptors, CCR2 - antagonists & inhibitors | CD4-Positive T-Lymphocytes - immunology | Animals | Muscular Dystrophy, Duchenne - immunology | Mice, Inbred mdx | Diaphragm - immunology | Receptors, CCR5 | Mice | Muscle Strength - drug effects | Muscular Dystrophy, Duchenne - genetics | CD8-Positive T-Lymphocytes - immunology | Macrophages - immunology | Care and treatment | Immune response | Duchenne muscular dystrophy | Development and progression | Genetic aspects | Health aspects | Chemokine receptors | CC chemokine receptors | Therapy | Flow cytometry | Disease | Laboratories | CD8 antigen | Medical services | Homology | Lymphocytes T | Macrophages | Muscular dystrophy | Fibers | Necrosis | Reduction | CCR2 protein | Lymphocytes | Rodents | Human immunodeficiency virus--HIV | Duchenne's muscular dystrophy | Gangrene | Diaphragms | CCR5 protein | Muscles | Leukocytes (neutrophilic) | Inflammation | Muscle contraction | Nitric-oxide synthase | CD4 antigen | Regeneration | Musculoskeletal system | Cytometry | Respiratory diseases | Fibrosis | Ligands | Infiltration | Dystrophy | Diaphragm | Chemokines | Monocyte chemoattractant protein 1 | HIV | Human immunodeficiency virus
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13. Full Text Pulmonary Neutrophil Infiltration in Murine Sepsis: Role of Inducible Nitric Oxide Synthase
by Razavi, Habib M and Wang, Le Feng and Weicker, Sean and Rohan, Marta and Law, Cedrin and McCormack, David G and Mehta, Sanjay
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 08/2004, Volume 170, Issue 3, pp. 227 - 233
Nitric oxide (NO) derived from inducible NO synthase (NOS) contributes to the pathophysiology of acute lung injury (ALI). The effect of NOS on pulmonary... 
DEFORMABILITY | CELLS | PERMEABILITY | MECHANISMS | reciprocal bone marrow transplant chimeras | acute lung injury | inducible nitric oxide synthase | SEQUESTRATION | MYELOPEROXIDASE | neutrophil infiltration | RESPIRATORY SYSTEM | NITROTYROSINE | INHIBITOR | EXPRESSION | sepsis | CRITICAL CARE MEDICINE | Nitric Oxide - biosynthesis | Nitric Oxide - immunology | Acute Disease | Systemic Inflammatory Response Syndrome - physiopathology | Mice, Inbred C57BL | Male | Bronchopulmonary Sequestration - immunology | Random Allocation | Cell Movement - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Nitric Oxide Synthase - deficiency | Bronchopulmonary Sequestration - enzymology | Endothelium, Vascular - metabolism | Lung Diseases - complications | Lung Diseases - physiopathology | Mice | Nitric Oxide Synthase - metabolism | Systemic Inflammatory Response Syndrome - complications | Lung Diseases - pathology | Disease Models, Animal | Neutrophil Infiltration - immunology | Acute lung injury | Sepsis | Reciprocal bone marrow transplant chimeras | Neutrophil infiltration | Inducible nitric oxide synthase
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14. Full Text Detailed analysis of inflammatory cell infiltration in colorectal cancer
by Väyrynen, J.P and Tuomisto, A and Klintrup, K and Mäkelä, J and Karttunen, T.J and Mäkinen, M.J
British Journal of Cancer, ISSN 0007-0920, 10/2013, Volume 109, Issue 7, pp. 1839 - 1847
Background: Higher-grade inflammatory infiltrate is a promising marker for better prognosis in colorectal cancer (CRC). However, the knowledge on the... 
immune cell |