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Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, pp. e0137305 - e0137305
Cytokines such as TNF alpha can polarize microglia/macrophages into different neuroinflammatory types. Skewing of the phenotype towards a cytotoxic state is... 
LEUKOCYTES | MICROGLIA | NEUROINFLAMMATION | ALZHEIMERS-DISEASE | HIPPOCAMPAL-LESIONS | BEHAVIOR | MULTIDISCIPLINARY SCIENCES | A-BETA | INNATE | PRECURSOR PROTEIN | BRAIN | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Isoindoles - administration & dosage | Humans | Biological Availability | Male | Thiones - therapeutic use | tau Proteins - chemistry | Alzheimer Disease - pathology | Thiones - adverse effects | Brain - metabolism | Dose-Response Relationship, Drug | Isoindoles - therapeutic use | Female | Safety | Alzheimer Disease - immunology | Isoindoles - pharmacology | Cognition - classification | Macrophages - immunology | Neutrophil Infiltration - drug effects | Disease Models, Animal | Thioamides - administration & dosage | Cell Line | Immunity, Innate - drug effects | Microglia - drug effects | Administration, Oral | Isoindoles - adverse effects | Mice, Inbred C57BL | Solubility | Alzheimer Disease - drug therapy | Thioamides - pharmacology | Mice, Transgenic | Brain - drug effects | Phenotype | Animals | Protein Structure, Secondary - drug effects | Thiones - administration & dosage | Alzheimer Disease - metabolism | Brain - pathology | Macrophages - drug effects | Thioamides - therapeutic use | Mice | Thiones - pharmacology | Thioamides - adverse effects | Brain - immunology | Protein Multimerization - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Brain | Flow cytometry | Memory | Cognitive ability | Cytotoxicity | Nervous system | Systematic review | Cognition | Biochemistry | Arthritis | Macrophages | Neuromodulation | Modulators | Learning | Rodents | Tumor necrosis factor-TNF | Amyloid | Alzheimer's disease | Immune system | Phenotypes | Cytokines | Neutrophils | Inflammation | Bioavailability | Tumor necrosis factor-α | Microglia | Pathology | Cytometry | Tau protein | Infiltration | Alzheimers disease | Phagocytosis | Animal cognition | Index Medicus
Journal Article
PLOS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0174544 - e0174544
Background Alcohol-induced intestinal dysbiosis disrupts homeostatic gut-liver axis function and is essential in the development of alcoholic liver disease.... 
CROSS-TALK | MICROFLORA | DANGER SIGNALS | ACTIVATION | MULTIDISCIPLINARY SCIENCES | INJURY | ETHANOL | STEATOHEPATITIS | LIVER-DISEASE | URIC-ACID | PROTECTS | Inflammation - chemically induced | Liver - pathology | Ethanol - toxicity | Gene Expression - drug effects | Bacteria - drug effects | Ethanol - administration & dosage | Fatty Liver - chemically induced | Hepatitis, Alcoholic - etiology | Fatty Liver, Alcoholic - genetics | Liver - drug effects | Bacteria - classification | Female | Central Nervous System Depressants - toxicity | Neutrophil Infiltration - drug effects | Fatty Liver - genetics | Fatty Liver, Alcoholic - etiology | Liver - metabolism | Mice, Inbred C57BL | Gastrointestinal Microbiome - genetics | Reverse Transcriptase Polymerase Chain Reaction | Bacteria - genetics | Neutrophil Infiltration - genetics | Animals | Gastrointestinal Microbiome - drug effects | Central Nervous System Depressants - administration & dosage | Inflammation - genetics | RNA, Ribosomal, 16S - genetics | Anti-Bacterial Agents - pharmacology | Hepatitis, Alcoholic - genetics | Anti-Bacterial Agents - administration & dosage | Hepatitis | Care and treatment | Microbiota (Symbiotic organisms) | Cytokines | Analysis | Physiological aspects | Inflammation | Health aspects | Medical research | Liver diseases | Ethanol | Laboratories | Alcoholism | Neutrophils | Lipids | Gene expression | Medical schools | Alcohol use | Medicine | Immunology | Antibiotics | Rodents | Gastroenterology | Ligands | Bacteria | Deoxyribonucleic acid--DNA | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Experimental Neurology, ISSN 0014-4886, 09/2016, Volume 283, Issue Pt A, pp. 224 - 234
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 1560 - 1560
Journal Article
Experimental Neurology, ISSN 0014-4886, 2007, Volume 204, Issue 1, pp. 220 - 233
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2012, Volume 188, Issue 1, pp. 462 - 469
Psoriasis is a common chronic autoimmune skin disease of unknown cause that involves dysregulated interplay between immune cells and keratinocytes. IL-22 is a... 
ROR-GAMMA-T | DENDRITIC CELLS | DOUBLE-BLIND | INTERLEUKIN-22 | CYTOKINE | IMMUNOLOGY | EXPRESSION | 5-PERCENT CREAM | ACTINIC KERATOSIS | INNATE IMMUNITY | TH17 CELLS | Adjuvants, Immunologic - pharmacology | Chemokine CCL3 - immunology | Dermatitis - immunology | Skin - metabolism | Immunity, Innate - genetics | Receptors, Antigen, T-Cell, gamma-delta - genetics | Adjuvants, Immunologic - adverse effects | Interleukins - genetics | Chemokine CCL3 - genetics | Psoriasis - pathology | T-Lymphocytes - metabolism | Interleukins - immunology | Psoriasis - metabolism | Antigens, Differentiation - metabolism | Chemokine CCL3 - metabolism | Interleukins - biosynthesis | T-Lymphocytes - pathology | Aminoquinolines - pharmacology | Neutrophils - metabolism | Neutrophil Infiltration - drug effects | Skin - pathology | Disease Models, Animal | Neutrophil Infiltration - immunology | Neutrophils - pathology | Skin - immunology | Psoriasis - immunology | Immunity, Innate - drug effects | Receptors, Antigen, T-Cell, gamma-delta - metabolism | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Neutrophils - immunology | Receptors, Antigen, T-Cell, gamma-delta - immunology | Neutrophil Infiltration - genetics | Mice, Knockout | Dermatitis - etiology | Immunity, Innate - immunology | Animals | Psoriasis - chemically induced | T-Lymphocytes - immunology | Antigens, Differentiation - genetics | Mice | Aminoquinolines - adverse effects | Antigens, Differentiation - immunology | Dermatitis - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 09/2016, Volume 5, Issue 9, p. n/a
Background-The gut microbiome is essential for physiological host responses and development of immune functions. The impact of gut microbiota on blood pressure... 
arterial hypertension | angiotensin | monocytes | vascular dysfunction | inflammation | gut microbiota | Gut microbiota | Arterial hypertension | Monocytes | Inflammation | Vascular dysfunction | Angiotensin II | INTERLEUKIN 17 | MACROPHAGE INFILTRATION | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | INTESTINAL MICROBIOTA | INDUCED CARDIAC DAMAGE | GERM-FREE MICE | DEFICIENT MICE | BLOOD-PRESSURE | angiotensin II | REGULATORY T-CELLS | Hypertension - microbiology | Reactive Oxygen Species - metabolism | Blood Vessels - metabolism | Endothelium, Vascular - drug effects | Gastrointestinal Microbiome - physiology | RNA, Messenger - metabolism | Arterial Pressure - drug effects | NADPH Oxidase 2 - drug effects | Neutrophil Infiltration - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | RNA, Messenger - drug effects | Angiotensin II - pharmacology | Aorta - drug effects | Nitric Oxide Synthase Type II - drug effects | Chemokine CCL2 - genetics | Cell Adhesion - drug effects | Animals | Nitric Oxide Synthase Type II - genetics | Blood Vessels - drug effects | Germ-Free Life | Leukocytes - drug effects | Mice | Aorta - cytology | NADPH Oxidase 2 - genetics | Chemokine CCL2 - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - drug effects | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35