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Circulation (New York, N.Y.), ISSN 0009-7322, 10/2008, Volume 118, Issue 18, pp. 1837 - 1847
Background-Two macrophage ABC transporters, ABCA1 and ABCG1, have a major role in promoting cholesterol efflux from macrophages. Peritoneal macrophages... 
Lipoproteins | ABC transporters | Inflammation | Cholesterol | Atherosclerosis | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | ATP Binding Cassette Transporter, Sub-Family G, Member 1 | Lipoproteins - genetics | Membrane Microdomains - immunology | Signal Transduction - immunology | ATP-Binding Cassette Transporters - genetics | Mice, Mutant Strains | Lipoproteins - metabolism | ATP-Binding Cassette Transporters - metabolism | Mice, Inbred DBA | ATP Binding Cassette Transporter 1 | Macrophages - immunology | Receptors, LDL - genetics | Atherosclerosis - immunology | Atherosclerosis - physiopathology | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Neutrophils - immunology | Receptors, LDL - metabolism | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Cholesterol - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Atherosclerosis - metabolism | Macrophages - metabolism | Animals | Lipopolysaccharides - pharmacology | Ligands | Macrophages - drug effects | Mice | ATP-Binding Cassette Transporters - immunology | Lipoproteins - immunology | Physiological aspects | Research | Macrophages | Gene expression | Index Medicus | Abridged Index Medicus | cholesterol | Toll-receptors | inflammation
Journal Article
Nature immunology, ISSN 1529-2916, 04/2012, Volume 13, Issue 5, pp. 449 - 456
Intestinal phagocytes transport oral antigens and promote immune tolerance, but their role in innate immune responses remains unclear. Here we found that... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Inflammasomes - metabolism | Humans | Receptors, Interleukin-1 - genetics | Salmonella Infections - genetics | Salmonella Infections - immunology | Clonal Anergy | Caspase 1 - metabolism | Monocytes - metabolism | Monocytes - immunology | Intestines - immunology | Host-Pathogen Interactions - immunology | Signal Transduction - immunology | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Apoptosis Regulatory Proteins - genetics | Pseudomonas - immunology | Toll-Like Receptors - metabolism | Neutrophil Infiltration - immunology | Neutrophils - pathology | Apoptosis Regulatory Proteins - immunology | Flagellin - immunology | Receptors, Interleukin-1 - immunology | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Neutrophils - immunology | Salmonella - genetics | Signal Transduction - genetics | Phagocytes - immunology | Neutrophil Infiltration - genetics | Mice, Knockout | Pseudomonas Infections - immunology | Salmonella - immunology | Animals | Intestines - microbiology | Phagocytes - microbiology | Inflammasomes - immunology | Interleukin-6 - immunology | Interleukin-6 - biosynthesis | Mice | Mice, Inbred BALB C | Tumor Necrosis Factor-alpha - biosynthesis | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 01/2012, Volume 188, Issue 1, pp. 462 - 469
Psoriasis is a common chronic autoimmune skin disease of unknown cause that involves dysregulated interplay between immune cells and keratinocytes. IL-22 is a... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Adjuvants, Immunologic - pharmacology | Chemokine CCL3 - immunology | Dermatitis - immunology | Skin - metabolism | Immunity, Innate - genetics | Receptors, Antigen, T-Cell, gamma-delta - genetics | Adjuvants, Immunologic - adverse effects | Interleukins - genetics | Chemokine CCL3 - genetics | Psoriasis - pathology | T-Lymphocytes - metabolism | Interleukins - immunology | Psoriasis - metabolism | Antigens, Differentiation - metabolism | Chemokine CCL3 - metabolism | Interleukins - biosynthesis | T-Lymphocytes - pathology | Aminoquinolines - pharmacology | Neutrophils - metabolism | Neutrophil Infiltration - drug effects | Skin - pathology | Disease Models, Animal | Neutrophil Infiltration - immunology | Neutrophils - pathology | Skin - immunology | Psoriasis - immunology | Immunity, Innate - drug effects | Receptors, Antigen, T-Cell, gamma-delta - metabolism | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Neutrophils - immunology | Receptors, Antigen, T-Cell, gamma-delta - immunology | Neutrophil Infiltration - genetics | Mice, Knockout | Dermatitis - etiology | Immunity, Innate - immunology | Animals | Psoriasis - chemically induced | T-Lymphocytes - immunology | Antigens, Differentiation - genetics | Mice | Aminoquinolines - adverse effects | Antigens, Differentiation - immunology | Dermatitis - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 01/2014, Volume 289, Issue 2, pp. 1174 - 1182
In the current study, we examined the role of CD14 in regulating LPS activation of corneal epithelial cells and Pseudomonas aeruginosa corneal infection. Our... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Pseudomonas aeruginosa - immunology | Epithelial Cells - metabolism | Interferon-beta - immunology | Protein-Tyrosine Kinases - metabolism | Humans | Cornea - immunology | Intracellular Signaling Peptides and Proteins - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Pseudomonas aeruginosa - physiology | Lipopolysaccharides - immunology | Protein-Tyrosine Kinases - immunology | Host-Pathogen Interactions - immunology | Lipopolysaccharide Receptors - metabolism | Cornea - cytology | Chemokine CCL5 - metabolism | Phosphorylation - immunology | Phosphorylation - drug effects | Syk Kinase | Neutrophil Infiltration - drug effects | Endocytosis - immunology | Interferon Regulatory Factor-3 - immunology | Neutrophil Infiltration - immunology | Cornea - microbiology | Cell Line | Endocytosis - drug effects | Mice, Inbred C57BL | Cells, Cultured | Lipopolysaccharide Receptors - immunology | Toll-Like Receptor 4 - immunology | Pseudomonas Infections - microbiology | Toll-Like Receptor 4 - metabolism | Blotting, Western | Mice, Knockout | Pseudomonas Infections - immunology | Animals | Interferon-beta - metabolism | Epithelial Cells - immunology | Epithelial Cells - microbiology | Lipopolysaccharides - pharmacology | Interferon Regulatory Factor-3 - metabolism | Lipopolysaccharide Receptors - genetics | Mice | Chemokine CCL5 - immunology | Index Medicus | Cornea | Immunology | Toll-like Receptors (TLRs) | Epithelial Cell | Pseudomonas aeruginosa | Inflammation | Neutrophil | Lipopolysaccharide (LPS)
Journal Article
American journal of physiology: Gastrointestinal and liver physiology, ISSN 0193-1857, 07/2015, Volume 309, Issue 1, pp. G30 - G41
Although previous studies reported the involvement of the TLR4-TRIF pathway in alcohol-induced liver injury, the role of TLR2 and TLR9 signaling in alcohol-mediated neutrophil infiltration and liver... 
Chemokine | ALD | Binge ethanol feeding | MyD88 | Gastroenterology & Hepatology | Physiology | Life Sciences & Biomedicine | Science & Technology | Up-Regulation | Liver - pathology | Toll-Like Receptor 2 - genetics | Toll-Like Receptor 9 - genetics | Hepatic Stellate Cells - metabolism | Hepatocytes - metabolism | Neutrophil Infiltration | Toll-Like Receptor 2 - deficiency | Liver - immunology | Liver Diseases, Alcoholic - genetics | Hepatic Stellate Cells - immunology | Time Factors | Female | Kupffer Cells - metabolism | Chemokine CXCL1 - metabolism | Disease Models, Animal | Liver Diseases, Alcoholic - pathology | Ethanol | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Cells, Cultured | Liver Diseases, Alcoholic - prevention & control | Binge Drinking | Receptors, Interleukin-8B - immunology | Toll-Like Receptor 2 - metabolism | Hepatocytes - immunology | Liver Diseases, Alcoholic - metabolism | Mice, Knockout | Chemokine CXCL1 - immunology | Animals | Liver Diseases, Alcoholic - immunology | Receptors, Interleukin-8B - metabolism | Toll-Like Receptor 9 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Kupffer Cells - immunology | Medical research | Liver diseases | Liver | Physiological aspects | TLR9 | Medicine, Experimental | Research | Drinking of alcoholic beverages | Health aspects | TLR2 | Analysis | Index Medicus | chemokine | binge ethanol feeding | Liver and Biliary Tract
Journal Article