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European Journal of Pharmacology, ISSN 0014-2999, 2011, Volume 666, Issue 1, pp. 53 - 60
Journal Article
Critical Care Medicine, ISSN 0090-3493, 08/2004, Volume 32, Issue 8, pp. 1695 - 1702
Journal Article
Archiv der Pharmazie, ISSN 0365-6233, 02/2018, Volume 351, Issue 2, pp. 1700269 - n/a
The elastase inhibition ability of novel aryl, substituted aryl, and heteroaryl oxime ester derivatives was investigated for the first time, compared to that... 
oxime ester derivatives | elastase inhibition | ursolic acid | CHEMISTRY, MEDICINAL | PHARMACOLOGY & PHARMACY | NEUTROPHIL ELASTASE | CHEMISTRY, MULTIDISCIPLINARY | Enzymes | Esters | Saturated fatty acids | Analysis | Isomerization | Cosmetics industry
Journal Article
Respiratory Research, ISSN 1465-9921, 01/2013, Volume 14, Issue 1, pp. 8 - 8
Background: Chronic asthma is often associated with neutrophilic infiltration in the airways. Neutrophils contain elastase, a potent secretagogue in the... 
Hyperresponsiveness | Elastase | Neutrophil | Airway | Asthma | FIBROSIS | LUNG | RESPONSIVENESS | SERINE PROTEASES | MITE ALLERGEN | PROTEASE-ACTIVATED RECEPTORS | RESPIRATORY SYSTEM | EXPRESSION | T-CELLS | REQUIREMENT | Bronchoalveolar Lavage Fluid | Immunohistochemistry | Allergens | Glycine - analogs & derivatives | Glycine - therapeutic use | Inflammation - pathology | Ovalbumin - immunology | Bronchial Hyperreactivity - drug therapy | Leukocyte Elastase - antagonists & inhibitors | Respiratory Hypersensitivity - pathology | Neutrophil Infiltration - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, PAR-2 - immunology | Respiratory Hypersensitivity - drug therapy | Flow Cytometry | Inflammation - drug therapy | Female | Bronchi - pathology | Receptor, PAR-2 - biosynthesis | Cytokines - metabolism | Cells, Cultured | Sulfonamides - pharmacology | Proteinase Inhibitory Proteins, Secretory - pharmacology | Bronchial Hyperreactivity - pathology | Leukocyte Elastase - physiology | Animals | Glycine - pharmacology | Sulfonamides - therapeutic use | Chemokines - metabolism | Mice | Mice, Inbred BALB C | Leukocyte Elastase - immunology | Animal experimentation | Albumin | Inflammation | T cells | Transforming growth factors | Eosinophils | Airway management | Lungs | Cytokines | Pathogenesis | Rodents | Gene expression | Research
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2017, Volume 195
Using different length heparin polymers: pentasaccharide, low molecular weight heparin (LMWH) (15 saccharides), 2-O, 3-O desulfated heparin (ODSH, ~45... 
Proteases | Neutrophils | Cystic fibrosis | Polymers | Deoxyribonucleic acid--DNA
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 01/2017, Volume 195
[...]we tested whether ODSH inhibited NE activity in de-identified CF sputum samples. [...]post-DNase treatment, ODSH inhibited NE in a concentration-dependent... 
Neutrophils | Cystic fibrosis | Deoxyribonucleic acid--DNA
Journal Article
by Kumar, H and Choi, H and Jo, MJ and Joshi, HP and Muttigi, M and Bonanomi, D and Kim, SB and Ban, E and Kim, A and Lee, SH and Kim, KT and Sohn, S and Zeng, X and Han, I
ACTA NEUROPATHOLOGICA COMMUNICATIONS, ISSN 2051-5960, 08/2018, Volume 6, Issue 1, pp. 73 - 18
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 09/2013, Volume 110, Issue 37, pp. 15007 - 15012
The rationale of alpha 1-alpha ntitrypsin (AAT) augmentation therapy to treat progressive emphysema in AAT-deficient patients is based on inhibition of... 
immunomodulation | CELLS | ACUTE-PHASE PROTEIN | alpha 1-antitrypsin | ALPHA-ANTITRYPSIN DEFICIENCY | MULTIDISCIPLINARY SCIENCES | MODEL | RELEASE | inflammation | DISEASE | MICE | MARROW-TRANSPLANTATION | HUMAN NEUTROPHIL | AUGMENTATION THERAPY
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2013, Volume 110, Issue 37, pp. 15007 - 15012
Journal Article
Journal Article
Journal Article
Brain Research, ISSN 0006-8993, 2000, Volume 858, Issue 1, pp. 55 - 60
It has been reported that activated neutrophils are involved in the development of cerebral damage induced by ischemia. Activated neutrophils release a lot of... 
Elastase inhibitor | Edema | Ischemic cerebral damage | Neutrophil elastase
Journal Article
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY, ISSN 1040-0605, 09/2007, Volume 293, Issue 3, pp. L762 - L768
Neutrophil elastase inhibition of cell cycle progression in airway epithelial cells in vitro is mediated by p27(kip1). Am J Physiol Lung Cell Mol Physiol 293:... 
PHYSIOLOGY | PROTEIN-KINASE | airway epithelial cells | CDK INHIBITOR | cell cycle arrest | DEPENDENT KINASE INHIBITOR | p27(kip1) | LEUKOCYTE ELASTASE | GROWTH ARREST | RESPIRATORY SYSTEM | neutrophil elastase | TRANSFORMING GROWTH-FACTOR-BETA-1 | GENE-EXPRESSION | EXTRACELLULAR-MATRIX | SMOOTH-MUSCLE CELLS | PULMONARY-FIBROSIS
Journal Article