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Annals of Hematology, ISSN 0939-5555, 2/2018, Volume 97, Issue 2, pp. 309 - 317
This retrospective study attempts to establish if a correlation exists between osteoporosis and hematopoiesis before and after adjuvant chemotherapy in the... 
T-score | Hematology | TBS | Anemia | Bone marrow microenvironment | Oncology | Breast cancer | Graded toxicity of chemotherapy | Osteoporosis | Chemotherapy | Platelet | Medicine & Public Health | Trabecular bone score | Hematopoiesis | Stress hematopoiesis | Osteoblast | Neutrophil | Bone marrow adipocytes | Febrile neutropenia | SCORE TBS | BONE-MINERAL DENSITY | COLONY-STIMULATING FACTOR | POSTMENOPAUSAL WOMEN | HEMATOLOGY | STEM-CELLS | PREMENOPAUSAL WOMEN | COMPLEMENTARY APPROACH | MARROW NICHE | BLOOD-CELL COUNTS | CLINICAL-PRACTICE | Blood Platelets - immunology | Bone Density - immunology | Breast Neoplasms - immunology | Cell Count | Humans | Middle Aged | Adipocytes - drug effects | Antineoplastic Agents - administration & dosage | Hematopoiesis - drug effects | Bone Diseases, Metabolic - drug therapy | Bone Diseases, Metabolic - complications | Bone Marrow Cells - immunology | Osteoporosis - diagnostic imaging | Chemotherapy, Adjuvant | Neutropenia - immunology | Body Mass Index | Osteoporosis - immunology | Lumbar Vertebrae - diagnostic imaging | Neutrophils - drug effects | Bone Marrow Cells - pathology | Lumbar Vertebrae - drug effects | Absorptiometry, Photon | Breast Neoplasms - drug therapy | Hematopoiesis - immunology | Neutropenia - diagnostic imaging | Lumbar Vertebrae - pathology | Blood Platelets - pathology | Osteoporosis - drug therapy | Antineoplastic Agents - adverse effects | Neutropenia - pathology | Adult | Bone Marrow Cells - drug effects | Female | Retrospective Studies | Breast Neoplasms - diagnostic imaging | Blood Platelets - drug effects | Neutropenia - chemically induced | Adipocytes - immunology | Neutrophils - pathology | Bone Diseases, Metabolic - diagnostic imaging | Bone Diseases, Metabolic - immunology | Osteoblasts - drug effects | Neutrophils - immunology | Osteoblasts - immunology | Lumbar Vertebrae - immunology | Adipocytes - pathology | Breast Neoplasms - complications | Osteoblasts - pathology | Bone Density - drug effects | Osteoporosis - complications | Aged | Oncology, Experimental | Adjuvant treatment | Research | Cancer | Neutrophils | Index Medicus | Original
Journal Article
PLoS pathogens, ISSN 1553-7374, 2015, Volume 11, Issue 5, p. e1004917
Pulmonary cavities, the hallmark of tuberculosis (TB), are characterized by high mycobacterial load and perpetuate the spread of M. tuberculosis. The mechanism... 
MYCOBACTERIUM-TUBERCULOSIS | ACTIVATED PROTEIN-KINASE | MICROBIOLOGY | IN-VITRO | VIROLOGY | INTERFERON-GAMMA | INFECTION | EXTRACELLULAR TRAPS | NF-KAPPA-B | LUNG INFLAMMATION | T-CELLS | PARASITOLOGY | INNATE IMMUNITY | AMP-Activated Protein Kinases - metabolism | Tuberculosis, Pulmonary - metabolism | Respiratory Mucosa - drug effects | Humans | Mycobacterium tuberculosis - immunology | NF-kappa B - metabolism | Tuberculosis, Pulmonary - drug therapy | Host-Pathogen Interactions - drug effects | Mycobacterium tuberculosis - drug effects | Respiratory Mucosa - pathology | Proteolysis - drug effects | Protein Processing, Post-Translational - drug effects | Matrix Metalloproteinase 8 - metabolism | Respiratory Mucosa - immunology | Mycobacterium tuberculosis - physiology | Adult | Lung - metabolism | Phosphorylation - drug effects | Neutrophils - metabolism | Extracellular Matrix Proteins - metabolism | Neutrophil Infiltration - drug effects | Neutrophils - pathology | Lung - pathology | Immunity, Innate - drug effects | Neutrophils - enzymology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Neutrophils - immunology | Tuberculosis, Pulmonary - pathology | Matrix Metalloproteinase 8 - chemistry | Tuberculosis, Pulmonary - immunology | Active Transport, Cell Nucleus - drug effects | Lung - drug effects | Respiratory Mucosa - metabolism | Sputum - enzymology | Lung - immunology | Cohort Studies | Proteins | Studies | Pathology | Tuberculosis | Biomedical research | Disease | Biopsy | Infections | Kinases | Metabolism | Experiments
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 5, pp. 1607 - 1620
Chronic inflammation, a hallmark of obliterative bronchiolitis, is known to induce lymphangiogenesis. We therefore studied the role of lymphangiogenic vascular... 
Pathology | CELLS | LUNG TRANSPLANTATION | ACTIVATION | TGF-BETA | RAT TRACHEAL ALLOGRAFTS | FACTOR-C | INFLAMMATION | LYMPHANGIOGENESIS | PATHOLOGY | UP-REGULATION | ENDOTHELIAL GROWTH-FACTOR | Inflammation - pathology | Epithelial Cells - metabolism | Cyclosporine - pharmacology | Dendritic Cells - immunology | Epithelial Cells - drug effects | Trachea - drug effects | Bronchiolitis Obliterans - metabolism | Transplantation, Homologous | Dose-Response Relationship, Drug | Trachea - transplantation | Graft Rejection - complications | Inflammation - complications | Signal Transduction - immunology | Th17 Cells - drug effects | Dendritic Cells - drug effects | Adaptive Immunity - immunology | Bronchiolitis Obliterans - pathology | Graft Rejection - pathology | Macrophages - immunology | Epithelium - drug effects | Immunoglobulins - pharmacology | Epithelium - pathology | Epithelium - metabolism | Immunity, Innate - drug effects | Graft Rejection - physiopathology | Neutrophils - drug effects | Bronchiolitis Obliterans - immunology | Rats | Epithelial Cells - pathology | Inflammation - immunology | Chemotaxis - drug effects | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Down-Regulation - drug effects | Lymphangiogenesis - drug effects | Trachea - pathology | Up-Regulation - drug effects | Immunity, Innate - immunology | Bronchiolitis Obliterans - physiopathology | Adaptive Immunity - drug effects | Animals | Vascular Endothelial Growth Factor C - antagonists & inhibitors | Signal Transduction - drug effects | Vascular Endothelial Growth Factor C - metabolism | Th17 Cells - immunology | Macrophages - drug effects | Graft Rejection - immunology
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 19, pp. 8748 - 8753
Balanced induction of proinflammatory and type I IFN responses upon activation of Toll-like receptors (TLRs) determines the outcome of microbial infections and... 
Pathogens | Receptors | Bacterial infections | Genes | Internalization | Neutrophils | Bacteria | Mast cells | Infections | Macrophages | Salmonella | Vesicular stomatitis virus | Innate immunity | Endolysosome | Listeria | LISTERIA-MONOCYTOGENES | endolysosome | MULTIDISCIPLINARY SCIENCES | vesicular stomatitis virus | SUSCEPTIBILITY | RECEPTOR | LUNG INFECTION | INDUCTION | IFN | innate immunity | ROLES | AUTOIMMUNITY | RESISTANCE | MICE | Neutrophils - cytology | Transcription, Genetic - drug effects | Mast Cells - ultrastructure | Interferon-beta - immunology | Vesiculovirus - drug effects | Vesiculovirus - immunology | Listeria monocytogenes - immunology | Acids - metabolism | Endosomes - metabolism | Mast Cells - microbiology | Listeria monocytogenes - ultrastructure | Salmonella typhimurium - ultrastructure | Endosomes - drug effects | Biological Transport - drug effects | Salmonella typhimurium - drug effects | Inflammation Mediators - immunology | Mast Cells - immunology | Intracellular Space - drug effects | Salmonella typhimurium - immunology | Subcellular Fractions - drug effects | Endocytosis - drug effects | Neutrophils - drug effects | Toll-Like Receptors - immunology | Neutrophils - immunology | Toll-Like Receptor 4 - immunology | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Vesiculovirus - ultrastructure | Animals | Interferon-beta - pharmacology | Intracellular Space - microbiology | Lipopolysaccharides - pharmacology | Cell Compartmentation - drug effects | Mast Cells - cytology | Mice | Mice, Inbred BALB C | Listeria monocytogenes - drug effects | Autoimmunity | Stomatitis | Lysosomes | Interferon | Dosage and administration | Research | Biological response modifiers | Properties | Drug therapy | Antibacterial agents | Biological Sciences
Journal Article
International journal of molecular sciences, ISSN 1661-6596, 07/2017, Volume 18, Issue 7, p. 1394
...), which play important roles in the progression of sepsis. In this study, we investigated the effects and mechanism of a novel histone deacetylase (HDAC8) inhibitor, (E... 
Histone deacetylase | Endotoxemia | Matrix metalloproteinases-9 (MMP-9) | Lipopolysaccharide (LPS) | lipopolysaccharide (LPS) | ACETYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | endotoxemia | TRANSCRIPTION | NEUTROPHILS | POTENTIAL ROLES | histone deacetylase | CHEMISTRY, MULTIDISCIPLINARY | HISTONE DEACETYLASES | INNATE IMMUNE-RESPONSES | GENE-EXPRESSION | NF-KAPPA-B | ASSOCIATION | matrix metalloproteinases-9 (MMP-9) | SEVERE SEPSIS | Tumor Necrosis Factor-alpha - metabolism | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | YY1 Transcription Factor - metabolism | Interleukin-6 | RNA, Messenger - biosynthesis | Sepsis - drug therapy | Matrix Metalloproteinase 9 - metabolism | THP-1 Cells - drug effects | Tubulin - metabolism | Histone Deacetylase 1 - drug effects | Acetylation | Repressor Proteins - metabolism | RNA, Messenger - drug effects | JNK Mitogen-Activated Protein Kinases - drug effects | Cell Survival - drug effects | Chromosomal Proteins, Non-Histone - metabolism | Cytokines - metabolism | Down-Regulation | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Chondroitin Sulfate Proteoglycans - metabolism | Histone Deacetylases - metabolism | Cyclooxygenase 2 - drug effects | Tumor Necrosis Factor-alpha - pharmacology | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | MAP Kinase Signaling System - drug effects | Cytokines - drug effects | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Matrix Metalloproteinase 9 - drug effects | Histone Deacetylase Inhibitors - pharmacology | Histone Deacetylases - drug effects | Repressor Proteins - drug effects | Mice
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2012, Volume 38, Issue 3, pp. 367 - 371
Abstract Introduction The purpose of this study was to evaluate the biocompatibility of calcium aluminate cement (EndoBinder) in subcutaneous tissue of rats.... 
Endocrinology & Metabolism | Dentistry | tissue reaction | Biocompatibility | mineral trioxide aggregate | calcium aluminate cement | RESORPTION | SUBCUTANEOUS CONNECTIVE-TISSUE | PULP | PROROOT MTA | SOLUBILITY | DENTIN SIALOPROTEIN | ROOT-CANAL SEALER | DENTISTRY, ORAL SURGERY & MEDICINE | RADIOPACITY | HYDROXIDE | Inflammation - pathology | Neovascularization, Physiologic - drug effects | Rats, Wistar | Giant Cells - drug effects | Connective Tissue - drug effects | Microvessels - pathology | Male | Phagocytes - pathology | Necrosis | Plasma Cells - pathology | Plasma Cells - drug effects | Collagen - drug effects | Time Factors | Intubation - instrumentation | Calcium Compounds - pharmacology | Neutrophils - pathology | Silicates - pharmacology | Subcutaneous Tissue - pathology | Leukocytes, Mononuclear - drug effects | Connective Tissue - pathology | Macrophages - pathology | Phagocytes - drug effects | Neutrophils - drug effects | Microvessels - drug effects | Rats | Materials Testing | Biocompatible Materials - pharmacology | Fibroblasts - pathology | Polyethylene - chemistry | Leukocytes, Mononuclear - pathology | Oxides - pharmacology | Lymphocytes - pathology | Animals | Fibroblasts - drug effects | Lymphocytes - drug effects | Macrophages - drug effects | Root Canal Filling Materials - pharmacology | Aluminum Compounds - pharmacology | Edema - pathology | Drug Combinations | Giant Cells - pathology | Subcutaneous Tissue - drug effects
Journal Article
Toxicological sciences, ISSN 1096-0929, 2010, Volume 115, Issue 2, pp. 307 - 321
Journal Article
Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 11037
Excessive activation of blood coagulation and neutrophil accumulation have been described in several human cancers. However, whether hypercoagulation and... 
COLON-CANCER | COMPLEMENT | ACTIVATION | RISK-FACTORS | VENOUS THROMBOEMBOLISM | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | MOLECULAR-WEIGHT HEPARIN | MOUSE MODEL | EXTRACELLULAR DNA TRAPS | TISSUE FACTOR
Journal Article
Clinical pharmacology and therapeutics, ISSN 0009-9236, 10/2018, Volume 104, Issue 4, pp. 675 - 686
.... coli in healthy humans and found that anabasum (5 mg) exerted a potent anti‐inflammatory effect equivalent to that of prednisolone in terms of inhibiting neutrophil infiltration, the hallmark of acute inflammation... 
LUNG | CYCLIC-AMP | MACROPHAGE PHAGOCYTOSIS | CHRONIC GRANULOMATOUS-DISEASE | APOPTOTIC CELLS | IN-VIVO | RESOLUTION | PHARMACOLOGY & PHARMACY | INFECTION | MEDIATORS | AJULEMIC ACID | Skin - microbiology | Skin Diseases, Bacterial - microbiology | Skin Diseases, Bacterial - immunology | Dermatitis - immunology | Skin - metabolism | Dermatitis - microbiology | Escherichia coli - drug effects | Humans | Middle Aged | Skin Diseases, Bacterial - metabolism | Male | Skin Diseases, Bacterial - drug therapy | Dose-Response Relationship, Drug | Young Adult | Anti-Inflammatory Agents - adverse effects | Dronabinol - analogs & derivatives | Escherichia coli - pathogenicity | Inflammation Mediators - metabolism | Adult | Anti-Inflammatory Agents - administration & dosage | Neutrophils - microbiology | Neutrophils - metabolism | Neutrophil Infiltration - drug effects | Cytokines - immunology | Skin - immunology | Escherichia coli Infections - drug therapy | Inflammation Mediators - immunology | Dronabinol - adverse effects | Phagocytosis - drug effects | Cytokines - metabolism | Neutrophils - drug effects | Neutrophils - immunology | Escherichia coli Infections - microbiology | Escherichia coli Infections - metabolism | Prednisolone - pharmacology | Host-Pathogen Interactions | Adolescent | Lipid Metabolism - drug effects | Escherichia coli Infections - immunology | Dronabinol - administration & dosage | Skin - drug effects | Dermatitis - drug therapy | Dermatitis - metabolism | Research
Journal Article