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Analyst, ISSN 0003-2654, 07/2014, Volume 139, Issue 16, pp. 4056 - 4063
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 1560 - 1560
Journal Article
PLoS Pathogens, ISSN 1553-7366, 05/2015, Volume 11, Issue 5, pp. e1004917 - e1004917
Pulmonary cavities, the hallmark of tuberculosis (TB), are characterized by high mycobacterial load and perpetuate the spread of M. tuberculosis. The mechanism... 
MYCOBACTERIUM-TUBERCULOSIS | ACTIVATED PROTEIN-KINASE | MICROBIOLOGY | IN-VITRO | VIROLOGY | INTERFERON-GAMMA | INFECTION | EXTRACELLULAR TRAPS | NF-KAPPA-B | LUNG INFLAMMATION | T-CELLS | PARASITOLOGY | INNATE IMMUNITY | AMP-Activated Protein Kinases - metabolism | Tuberculosis, Pulmonary - metabolism | Respiratory Mucosa - drug effects | Humans | Mycobacterium tuberculosis - immunology | NF-kappa B - metabolism | Tuberculosis, Pulmonary - drug therapy | Host-Pathogen Interactions - drug effects | Mycobacterium tuberculosis - drug effects | Respiratory Mucosa - pathology | Proteolysis - drug effects | Protein Processing, Post-Translational - drug effects | Matrix Metalloproteinase 8 - metabolism | Respiratory Mucosa - immunology | Mycobacterium tuberculosis - physiology | Adult | Lung - metabolism | Phosphorylation - drug effects | Neutrophils - metabolism | Extracellular Matrix Proteins - metabolism | Neutrophil Infiltration - drug effects | Neutrophils - pathology | Lung - pathology | Immunity, Innate - drug effects | Neutrophils - enzymology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Neutrophils - immunology | Tuberculosis, Pulmonary - pathology | Matrix Metalloproteinase 8 - chemistry | Tuberculosis, Pulmonary - immunology | Active Transport, Cell Nucleus - drug effects | Lung - drug effects | Respiratory Mucosa - metabolism | Sputum - enzymology | Lung - immunology | Cohort Studies | Index Medicus | Proteins | Studies | Pathology | Tuberculosis | Biomedical research | Disease | Biopsy | Infections | Kinases | Metabolism | Experiments
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | Index Medicus | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2012, Volume 18, Issue 11, pp. 1651 - 1657
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 09/2016, Volume 5, Issue 9, p. n/a
Background-The gut microbiome is essential for physiological host responses and development of immune functions. The impact of gut microbiota on blood pressure... 
arterial hypertension | angiotensin | monocytes | vascular dysfunction | inflammation | gut microbiota | Gut microbiota | Arterial hypertension | Monocytes | Inflammation | Vascular dysfunction | Angiotensin II | INTERLEUKIN 17 | MACROPHAGE INFILTRATION | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | INTESTINAL MICROBIOTA | INDUCED CARDIAC DAMAGE | GERM-FREE MICE | DEFICIENT MICE | BLOOD-PRESSURE | angiotensin II | REGULATORY T-CELLS | Hypertension - microbiology | Reactive Oxygen Species - metabolism | Blood Vessels - metabolism | Endothelium, Vascular - drug effects | Gastrointestinal Microbiome - physiology | RNA, Messenger - metabolism | Arterial Pressure - drug effects | NADPH Oxidase 2 - drug effects | Neutrophil Infiltration - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | RNA, Messenger - drug effects | Angiotensin II - pharmacology | Aorta - drug effects | Nitric Oxide Synthase Type II - drug effects | Chemokine CCL2 - genetics | Cell Adhesion - drug effects | Animals | Nitric Oxide Synthase Type II - genetics | Blood Vessels - drug effects | Germ-Free Life | Leukocytes - drug effects | Mice | Aorta - cytology | NADPH Oxidase 2 - genetics | Chemokine CCL2 - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - drug effects | Index Medicus
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 05/2012, Volume 56, Issue 5, pp. 2314 - 2325
Classifications Services AAC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
IN-VITRO | POLYMORPHONUCLEAR LEUKOCYTES | PSEUDOMONAS-AERUGINOSA VIRULENCE | COMPOUND | ANTIBIOTIC-RESISTANCE | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | CHRONIC WOUNDS | CYSTIC-FIBROSIS | LUNG INFECTION | VIBRIO-HARVEYI | BACTERIAL BIOFILMS | Plant Extracts - chemistry | Biofilms - growth & development | Tobramycin - administration & dosage | Garlic - chemistry | Respiratory Tract Infections - microbiology | Anti-Bacterial Agents - therapeutic use | Disulfides - therapeutic use | Pseudomonas aeruginosa - pathogenicity | Disulfides - pharmacology | Genes, Reporter | Tobramycin - therapeutic use | Virulence Factors - antagonists & inhibitors | Glycolipids - antagonists & inhibitors | Biofilms - drug effects | Quorum Sensing - drug effects | Pseudomonas Infections - drug therapy | Neutrophils - drug effects | Respiratory Tract Infections - drug therapy | Anti-Bacterial Agents - isolation & purification | Pseudomonas aeruginosa - drug effects | Pseudomonas Infections - microbiology | Quorum Sensing - genetics | Drug Synergism | Gene Expression Regulation, Bacterial - drug effects | Animals | Pseudomonas aeruginosa - genetics | Chemical Fractionation | Anti-Bacterial Agents - pharmacology | Mice | Disulfides - isolation & purification | Index Medicus | Drugs | Animal models | Communication systems | quorum sensing | Lung | Antimicrobial agents | virulence factors | Necrosis | Infection | Pathogenicity | Biofilms | DNA microarrays | Leukocytes (polymorphonuclear) | Tobramycin | rhamnolipids | Mechanisms of Action | Physiological Effects
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2006, Volume 116, Issue 12, pp. 3211 - 3219
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 02/2015, Volume 107, Issue 2, pp. dju413 - dju413
Background: Positive findings from the phase III MPACT trial led to the regulatory approval of nab-paclitaxel plus gemcitabine as a treatment option for... 
LYMPHOCYTE RATIO | 1ST-LINE TREATMENT | RATIO PREDICTS SURVIVAL | NEUTROPHIL | ONCOLOGY | BEVACIZUMAB | GUIDELINES | CARBOHYDRATE ANTIGEN 19-9 | COMBINATION | ERLOTINIB | TUMOR-MARKER | Prognosis | Antigens, Tumor-Associated, Carbohydrate - blood | Follow-Up Studies | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Liver Neoplasms - mortality | Pancreatic Neoplasms - drug therapy | Albumins - adverse effects | Adult | Deoxycytidine - adverse effects | Female | Pancreatic Neoplasms - mortality | Liver Neoplasms - secondary | Paclitaxel - administration & dosage | Drug Administration Schedule | Deoxycytidine - administration & dosage | Neutrophils - drug effects | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Liver Neoplasms - drug therapy | Paclitaxel - adverse effects | Albumins - administration & dosage | Treatment Outcome | International Cooperation | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Biomarkers, Tumor - blood | Disease-Free Survival | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Lymphocytes - drug effects | Aged | Deoxycytidine - analogs & derivatives | Adenocarcinoma - mortality | Antimitotic agents | Pancreatic cancer | Analysis | Clinical trials | Product development | Metastasis | Antineoplastic agents | Care and treatment | Patient outcomes | Development and progression | Dosage and administration | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2009, Volume 15, Issue 11, pp. 1318 - 1321
Journal Article
Nature Communications, ISSN 2041-1723, 03/2016, Volume 7, Issue 1, pp. 11037 - 11037
Excessive activation of blood coagulation and neutrophil accumulation have been described in several human cancers. However, whether hypercoagulation and... 
COLON-CANCER | COMPLEMENT | ACTIVATION | RISK-FACTORS | VENOUS THROMBOEMBOLISM | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | MOLECULAR-WEIGHT HEPARIN | MOUSE MODEL | EXTRACELLULAR DNA TRAPS | TISSUE FACTOR | Index Medicus
Journal Article