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Cell metabolism, ISSN 1550-4131, 2013, Volume 17, Issue 5, pp. 695 - 708
Diabetes is a major risk factor for atherosclerosis. Although atherosclerosis is initiated by deposition of cholesterol-rich lipoproteins in the artery wall,... 
DIABETES-MELLITUS | PLAQUE REGRESSION | INSULIN-RESISTANCE | ADVANCED GLYCATION ENDPRODUCTS | ENDOCRINOLOGY & METABOLISM | LEUKOCYTE COUNT | RECEPTOR | STEM-CELL PROLIFERATION | APOE(-/-) MICE | DEFICIENT MICE | G-CSF | CELL BIOLOGY | Leukocytes - pathology | Humans | Diabetes Mellitus, Type 1 - metabolism | Male | Monocytes - metabolism | NF-kappa B - metabolism | Leukocytosis - metabolism | Coronary Disease - metabolism | Bone Marrow - metabolism | Hyperglycemia - pathology | Monocytes - pathology | Diabetes Mellitus, Experimental - metabolism | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Leukocytosis - pathology | Neutrophils - pathology | Myeloid Progenitor Cells - metabolism | Atherosclerosis - pathology | Myelopoiesis - physiology | Cytokines - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Myeloid Progenitor Cells - pathology | Atherosclerosis - metabolism | Hyperglycemia - metabolism | Coronary Disease - pathology | Animals | Bone Marrow - pathology | Glycation End Products, Advanced - metabolism | Glucose - metabolism | Mice | Leukocytes - metabolism | Receptors, Immunologic - metabolism | Hyperglycemia | Type 1 diabetes | Atherosclerosis | Development and progression | Universities and colleges | Blood lipids | Coronary heart disease | Medicine, Preventive | Cholesterol | Preventive health services
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 7, p. e0200847
.... They remain viable and functional for 4 weeks expressing typical markers of liver function such as synthesis of albumin, urea, and alpha-1 p450 drug metabolism... 
OVEREXPRESSION | HOMEOSTASIS | LIPID-METABOLISM | MULTIDISCIPLINARY SCIENCES | DISEASE | SENSITIVITY | SYSTEMS | LIPOPROTEIN-LIPASE | Glucose Transporter Type 4 - metabolism | Signal Transduction | Humans | Liver - metabolism | Organoids - cytology | MicroRNAs - metabolism | Necrosis - metabolism | Non-alcoholic Fatty Liver Disease - metabolism | Hepatocytes - metabolism | Insulin Receptor Substrate Proteins - metabolism | Inflammation - metabolism | Insulin - metabolism | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 8 - metabolism | Organoids - metabolism | Chemokine CCL3 - metabolism | Chemokine CCL2 - metabolism | Kupffer Cells - metabolism | Liver - cytology | Interleukin-6 - metabolism | Care and treatment | MicroRNA | Liver diseases | Development and progression | Insulin resistance | Inflammation | Research | Laboratories | Liver | Kupffer cells | Fluorescence | Reversing | Lipids | Interleukin 6 | Liver cancer | Fatty liver | Organoids | Rodents | Gastroenterology | Drug metabolism | Lipoprotein (low density) receptors | Inhibition | Lipid metabolism | Stellate cells | Cytokines | Incubation | Internal medicine | CCL3 protein | Regression analysis | Gene expression | Metabolism | Ribonucleic acid--RNA | Insulin | Patients | Fatty acids | Endothelial cells | Gelatinase B | Steatosis | Medicine | Urea | Signaling | Hepatocytes | Tumor necrosis factor | Pharmacy | Fibrosis | Neutrophil collagenase | Diabetes | Chemokines | Monocyte chemoattractant protein 1 | Metabolic disorders | RNA | Ribonucleic acid
Journal Article
Blood, ISSN 0006-4971, 02/2009, Volume 113, Issue 7, pp. 1526 - 1534
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1158 - 1172
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2009, Volume 206, Issue 1, pp. 249 - 258
Psoriasis is a type I interferon-driven T cell–mediated disease characterized by the recruitment of plasmacytoid dendritic cells (pDC) into the skin. The... 
PATHOGENESIS | MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | DERMATITIS | INFLAMMATION | LYMPH-NODES | IN-VIVO | CYTOKINE | IMMUNOLOGY | PRECURSORS | T-CELLS | PROTEASES | CD8-Positive T-Lymphocytes - cytology | Neutrophils - cytology | Membrane Glycoproteins - metabolism | Skin - metabolism | Humans - metabolism | Antigens, CD - metabolism | Chemotaxis, Leukocyte - physiology | Lectins, C-Type - metabolism | Chemotaxis, Leukocyte - drug effects | Psoriasis - pathology | Psoriasis - metabolism | CD8-Positive T-Lymphocytes - metabolism | Receptors, Chemokine - genetics | Neutrophils - metabolism | Antibodies, Monoclonal - immunology | Fibroblasts - metabolism | Tretinoin - pharmacology | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Chemokines - genetics | Blotting, Western | Intercellular Adhesion Molecule-1 - metabolism | Calcitriol - pharmacology | Fibroblasts - drug effects | Chemokines - metabolism | Fibroblasts - cytology | Receptors, Immunologic - genetics | Chemokine CXCL10 - metabolism | Dermatitis, Atopic - genetics | Gene Expression - drug effects | Culture Media, Conditioned - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Lectins, C-Type - genetics | Psoriasis - genetics | Adult | Dendritic Cells - metabolism | Skin - pathology | Intercellular Signaling Peptides and Proteins | Receptors, Chemokine - metabolism | Cells, Cultured | Dermatitis, Atopic - pathology | Receptors, Chemokine - immunology | Membrane Glycoproteins - genetics | Chemokine CXCL10 - genetics | CD8-Positive T-Lymphocytes - drug effects | Dendritic Cells - cytology | Dermatitis, Atopic - metabolism | Receptors, Immunologic - metabolism
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2012, Volume 37, Issue 2, pp. 276 - 289
To initiate adaptive immunity, dendritic cells (DCs) move from parenchymal tissues to lymphoid organs by migrating along stromal scaffolds that display the... 
MIGRATION | LYMPHATIC VESSELS | INVASION | GTPASES | RAC | ROLES | IN-VIVO | RHO | NEUTROPHILS | IMMUNOLOGY | SECONDARY LYMPHOID ORGANS | Skin - cytology | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Skin - metabolism | Dendritic Cells - immunology | Humans | Actins - metabolism | Lectins, C-Type - immunology | rhoA GTP-Binding Protein - metabolism | Lectins, C-Type - genetics | Cell Movement - physiology | Lectins, C-Type - metabolism | Flow Cytometry | Female | Endothelium, Lymphatic - metabolism | Dendritic Cells - metabolism | Green Fluorescent Proteins - metabolism | Adaptive Immunity - physiology | Antigen-Presenting Cells - metabolism | Endothelial Cells - metabolism | Tissue Culture Techniques | Mice, Inbred C57BL | Cells, Cultured | Lymph Nodes - metabolism | Lymph Nodes - cytology | Mice, Knockout | Pregnancy | Microscopy, Confocal | Animals | Blood Platelets - metabolism | Platelet Activation | Endothelium, Lymphatic - cytology | Signal Transduction - physiology | Mice | Myosin Light Chains - metabolism | Proto-Oncogene Proteins c-vav - metabolism | rac1 GTP-Binding Protein - metabolism | Medical colleges | Nervous system diseases | Dendritic cells | Actin | Developmental biology | Myosin | Lectins | Muscle proteins | T cells | Endothelium | Telecommunications services industry | Communications industry | Scholarships & fellowships | Phosphorylation | Statistical analysis | Motility | Blood platelets | Microscopy | Migration | Chemokines
Journal Article
Clinical and experimental immunology, ISSN 0009-9104, 2010, Volume 162, Issue 1, pp. 100 - 107
Summary Pyoderma gangrenosum (PG) is a rare, immune‐mediated inflammatory skin disease presenting with painful ulcers having undermined edges. Less commonly,... 
cytokines | pyoderma gangrenosum | matrix metalloproteinases | neutrophilic dermatoses | Sweet's syndrome | INTERLEUKIN-17 | PYODERMA-GANGRENOSUM | SWEETS-SYNDROME | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | EXPRESSION | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Humans | Middle Aged | Male | Vascular Endothelial Growth Factor A - metabolism | Antigens, CD - metabolism | Young Adult | Matrix Metalloproteinase 9 - metabolism | Sweet Syndrome - metabolism | Inflammation Mediators - metabolism | Adult | Female | Interleukin-8 - metabolism | Neutrophils - metabolism | Neutrophils - pathology | Pyoderma Gangrenosum - metabolism | Cytokines - metabolism | Matrix Metalloproteinase 2 - metabolism | Receptors, Cell Surface - metabolism | Sweet Syndrome - pathology | CD3 Complex - metabolism | Pyoderma Gangrenosum - pathology | Interleukin-17 - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Adolescent | Aged | Matrix Metalloproteinases - metabolism | Peroxidase - metabolism | Skin diseases | Cytokines | T cells | Macrophages | Vascular endothelial growth factor | Wounds | CD3 antigen | Inflammation | Leukocytes (neutrophilic) | Lymphocytes T | Matrix metalloproteinase | Gelatinase B | Inflammatory diseases | Interleukin 17 | Gelatinase A | CD163 antigen | Ulcers | Peroxidase | Immunoreactivity | Chemokines | Interleukin 8 | Tumor necrosis factor- alpha | Translational Studies
Journal Article
Cell metabolism, ISSN 1550-4131, 2014, Volume 19, Issue 5, pp. 821 - 835
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Proteins | Studies | Rodents | Mortality | Gangrene | Homeostasis | Mitochondrial DNA | Laboratory animals | Deoxyribonucleic acid--DNA | Apoptosis
Journal Article