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Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 07/1999, Volume 55, Issue 8, pp. 1015 - 1028
Nitric oxide (NO) is a recently discovered mediator produced by mammalian cells. It plays a key role in neurotransmission, control of blood pressure, and... 
L-Arginine | nitric oxide | peroxynitrite | superoxide radical | Nω-hydroxy-L-arginine | nitric oxide synthase | urea cycle | arginase | Peroxynitrite | N(ω)-hydroxy-L-arginine | Nitric oxide | Arginase | Nitric oxide synthase | Urea cycle | Superoxide radical | L-arginine | BIOCHEMISTRY & MOLECULAR BIOLOGY | MARROW-DERIVED MACROPHAGES | CELL BIOLOGY | N-omega-hydroxy-L-arginine | ELECTRON-TRANSFER | SUBUNIT DIMERIZATION | SUPEROXIDE GENERATION | IN-VIVO | RABBIT ALVEOLAR MACROPHAGES | SELECTIVE INHIBITORS | MOLECULAR-CLONING | MACROPHAGE NO SYNTHASE | HYDROXY-L-ARGININE | Electron Transport | Nitric Oxide Synthase - chemistry | Humans | Arginase - metabolism | Nitric Oxide Synthase - antagonists & inhibitors | Isoenzymes - chemistry | Biopterin - analogs & derivatives | Arginine - analogs & derivatives | Biopterin - metabolism | Calcium - antagonists & inhibitors | Calcium - physiology | Isoenzymes - metabolism | Arginase - antagonists & inhibitors | Nitric Oxide Donors - pharmacology | Nitric Oxide - biosynthesis | Calmodulin - metabolism | Free Radicals | Enzyme Inhibitors - pharmacology | Enzyme Induction | Animals | Mice | Nitric Oxide Synthase - metabolism | Enzyme Activation | Calmodulin - antagonists & inhibitors | Arginine - metabolism | Isoenzymes - antagonists & inhibitors | Physiological aspects | Enzymes | Biosynthesis | Cellular biology | Life Sciences | Biochemistry, Molecular Biology
Journal Article
1996, ISBN 0124355552, 373
This unique sourcebook serves as a comprehensive reference, bridging the well-established chemistry of nitric oxide and the new and exciting role of nitric... 
Physiological effect | Nitric oxide | Biology, life sciences
eBook
Journal Article
Circulation, ISSN 0009-7322, 05/2004, Volume 109, Issue 17, pp. 2058 - 2067
Background - Myocardial ischemia provides a potent stimulus to angiogenesis, and the mobilization and differentiation of endothelial progenitor cells (EPCs)... 
Nitric oxide synthase | C-reactive protein | Cells | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | ANGIOGENESIS | NITRIC-OXIDE SYNTHASE | MARKERS | apoptosis | ISCHEMIA | VASCULAR SMOOTH-MUSCLE | DIABETES-MELLITUS | cells | CORONARY COLLATERAL VESSELS | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | nitric oxide synthase | NEOVASCULARIZATION | HEMATOLOGY | C-Reactive Protein - antagonists & inhibitors | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Humans | Male | Receptors, Cytoplasmic and Nuclear - biosynthesis | Nitric Oxide Synthase - genetics | C-Reactive Protein - pharmacology | Enzyme Induction - drug effects | Nitric Oxide Synthase - biosynthesis | Thiazolidinediones - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Cardiovascular Diseases - etiology | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Nitric Oxide Synthase Type III | Nitric Oxide - physiology | Transcription Factors - biosynthesis | Inflammation | Receptors, Cytoplasmic and Nuclear - agonists | Receptors, Cytoplasmic and Nuclear - genetics | Recombinant Proteins - pharmacology | Transcription Factors - genetics | Gene Expression Regulation - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Transcription Factors - agonists | Endothelial Cells - drug effects
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2010, Volume 246, Issue 1, pp. 8 - 17
Acetaminophen (APAP) overdose, which causes liver injury in animals and humans, activates c-jun N-terminal kinase (JNK). Although it was shown that the JNK... 
Mitochondria | Acetaminophen | JNK | Bax | Hepatotoxicity | Oxidant stress | SP600125 | BAX TRANSLOCATION | CELL INJURY | INDUCED LIVER-INJURY | TUMOR-NECROSIS-FACTOR | MITOCHONDRIAL PERMEABILITY TRANSITION | CULTURED MOUSE HEPATOCYTES | ONCOTIC NECROSIS | PROTECTIVE ROLE | IN-VIVO | PHARMACOLOGY & PHARMACY | TOXICOLOGY | NH2-TERMINAL KINASE | Liver - pathology | Phosphorylation | Liver - enzymology | Apoptosis - drug effects | Analgesics, Non-Narcotic - pharmacology | Nitric Oxide Synthase - drug effects | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Alanine Transaminase - blood | Analgesics, Non-Narcotic - toxicity | Liver - drug effects | Acetaminophen - pharmacology | Enzyme Induction - drug effects | Nitric Oxide Synthase - biosynthesis | JNK Mitogen-Activated Protein Kinases - drug effects | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Subcellular Fractions - drug effects | Acetaminophen - toxicity | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Anthracenes - pharmacology | Blotting, Western | Mitochondria, Liver - drug effects | Peroxynitrous Acid - biosynthesis | Animals | JNK Mitogen-Activated Protein Kinases - physiology | Mice | Oxidative Stress - drug effects | Translocation, Genetic - genetics | Messenger RNA | Glutathione transferase | Nitric oxide | Mitochondrial DNA | Superoxide | Permeability | Index Medicus | OXYGEN COMPOUNDS | NITRIC OXIDE | DIGESTIVE SYSTEM | MITOCHONDRIA | 60 APPLIED LIFE SCIENCES | STRESSES | PHOSPHORUS-GROUP TRANSFERASES | GLANDS | OXIDIZERS | NITROGEN OXIDES | ENZYMES | DRUGS | VERTEBRATES | MICE | NITROGEN COMPOUNDS | RADIOPROTECTIVE SUBSTANCES | MAMMALS | ANIMALS | INJURIES | RODENTS | RESPONSE MODIFYING FACTORS | ORGANIC COMPOUNDS | TRANSFERASES | NITRITES | POLYPEPTIDES | CHALCOGENIDES | ORGANS | DISEASES | PEPTIDES | GLUTATHIONE | LIVER | PHOSPHOTRANSFERASES | OXIDES | PROTEINS | BODY | CELL CONSTITUENTS | Oxidant Stress
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2004, Volume 101, Issue 24, pp. 9121 - 9126
Significant impairment in endothelial-derived nitric oxide is present in the diabetic corpus cavernosum. RhoA/Rho-kinase may suppress endothelial nitric oxide... 
Biological Sciences | Phosphorylation | Transfection | Diabetes complications | Impotence | Smooth muscle | Oxides | Diabetes | Type 1 diabetes mellitus | Vehicles | Endothelium | Neuronal NO synthase | Gene therapy | INDUCED CA2+ SENSITIZATION | endothelium | PHOSPHORYLATION | PHOSPHATASE | MULTIDISCIPLINARY SCIENCES | RHO-KINASE | RATS | gene therapy | SMOOTH-MUSCLE MYOSIN | neuronal NO synthase | CORPUS CAVERNOSUM | RABBIT | GENE-TRANSFER | EXPRESSION | Body Weight | Erectile Dysfunction - enzymology | Penis - enzymology | Diabetes Mellitus, Experimental - enzymology | Nitric Oxide Synthase - antagonists & inhibitors | Male | Diabetes Mellitus, Experimental - blood | Penis - drug effects | Nitric Oxide Synthase Type I | Muscle Relaxants, Central - pharmacology | rho-Associated Kinases | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Adenoviridae - genetics | Nitric Oxide Synthase - biosynthesis | Diabetes Mellitus, Experimental - complications | Cyclic GMP - biosynthesis | Amides - pharmacology | Gene Expression | Enzyme Inhibitors - metabolism | Protein-Serine-Threonine Kinases - physiology | Nitric Oxide Synthase Type III | Protein-Serine-Threonine Kinases - genetics | Intracellular Signaling Peptides and Proteins | Rats | Erectile Dysfunction - etiology | Rats, Sprague-Dawley | Protein-Serine-Threonine Kinases - biosynthesis | Animals | Nitric Oxide Synthase - metabolism | Pyridines - pharmacology | Blood Glucose - metabolism | Myosin-Light-Chain Phosphatase - biosynthesis | Nitric oxide | Research
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2001, Volume 276, Issue 34, pp. 32008 - 32015
Sulforaphane (SFN), an aliphatic isothiocyanate, is a known cancer chemopreventive agent. Aiming to investigate anti-inflammatory mechanisms of SFN, we here... 
SIGNAL-TRANSDUCTION | BACTERIAL LIPOPOLYSACCHARIDE | COLON-CANCER CELLS | INTERFERON-GAMMA | RAW-264.7 MACROPHAGES | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYCLOOXYGENASE-2 GENE | NECROSIS-FACTOR-ALPHA | TRANSCRIPTION FACTOR | DNA-BINDING ACTIVITY | Prostaglandin-Endoperoxide Synthases - biosynthesis | Dinoprostone - biosynthesis | Glutathione - metabolism | Cyclooxygenase 2 | Tumor Necrosis Factor-alpha - genetics | Nitric Oxide Synthase - antagonists & inhibitors | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Nitric Oxide Synthase - genetics | RNA, Messenger - metabolism | Nitric Oxide Synthase Type II | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Cell Nucleus - metabolism | Thiocyanates - pharmacology | Nitric Oxide Synthase - biosynthesis | Anticarcinogenic Agents - pharmacology | Isothiocyanates | Prostaglandin-Endoperoxide Synthases - genetics | Cell Line | Nitric Oxide - biosynthesis | Isoenzymes - genetics | RNA, Messenger - genetics | Prostaglandin-Endoperoxide Synthases - drug effects | DNA - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Protein Transport | Hydrolysis | Macrophages - metabolism | Animals | Tumor Necrosis Factor-alpha - drug effects | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Isoenzymes - drug effects | Mice | Isoenzymes - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | NF-kappa B - drug effects
Journal Article
Circulation, ISSN 0009-7322, 02/2000, Volume 101, Issue 6, pp. 676 - 681
Background-The vasodilatory effect of insulin can be acute or increase with time from 1 to 7 hours, suggesting that insulin may enhance the expression of... 
Proteins | RNA | Diabetes mellitus | Nitric oxide | Insulin | Cells | Endothelium | endothelium | nitric oxide | PROTEIN-KINASE-C | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | STIMULATION | insulin | PHOSPHATIDYLINOSITOL 3-KINASE | SENSITIVITY | RATS | diabetes mellitus | SKELETAL-MUSCLE | cells | proteins | GROWTH | PERIPHERAL VASCULAR DISEASE | INHIBITOR | HEMATOLOGY
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2005, Volume 174, Issue 11, pp. 7160 - 7171
The emergence of an increasing number of Leishmania donovani strains resistant to pentavalent antimonials (SbV), the first line of treatment for visceral... 
CUTANEOUS LEISHMANIASIS | IMMUNE-RESPONSE | TUMOR-NECROSIS-FACTOR | HUMAN MONONUCLEAR PHAGOCYTES | T-CELL RESPONSES | INDIAN KALA-AZAR | INTERFERON-GAMMA | INFANTUM KMP-11 PROTEIN | IFN-GAMMA | IMMUNOLOGY | FACTOR-ALPHA | Antigens, Protozoan - immunology | Leishmaniasis, Visceral - prevention & control | Reactive Oxygen Species - metabolism | Spleen - immunology | Humans | Vaccines, DNA - immunology | Liver Diseases, Parasitic - immunology | Leishmaniasis, Visceral - immunology | Th2 Cells - immunology | Antimony - pharmacology | Nitric Oxide Synthase - genetics | Protozoan Proteins - genetics | Th1 Cells - immunology | Vaccines, DNA - administration & dosage | Liver Diseases, Parasitic - prevention & control | Liver Diseases, Parasitic - pathology | Th1 Cells - metabolism | Interleukin-4 - biosynthesis | Th2 Cells - parasitology | Immunoglobulin G - biosynthesis | Nitric Oxide Synthase - biosynthesis | Leishmaniasis, Visceral - parasitology | Membrane Glycoproteins - immunology | Protozoan Proteins - administration & dosage | T-Lymphocytes, Cytotoxic - immunology | Cell Line | Cricetinae | Leishmania donovani - immunology | Leishmania donovani - pathogenicity | Spleen - cytology | Leishmaniasis, Visceral - pathology | Liver Diseases, Parasitic - parasitology | Th2 Cells - metabolism | Antibodies, Protozoan - biosynthesis | Membrane Glycoproteins - genetics | Spleen - parasitology | Drug Resistance - genetics | Membrane Glycoproteins - administration & dosage | Animals | Spleen - metabolism | T-Lymphocytes, Cytotoxic - parasitology | Th1 Cells - parasitology | Leishmania donovani - genetics | Nitric Oxide Synthase - metabolism | Reactive Nitrogen Species - biosynthesis | Cytotoxicity, Immunologic - immunology | Interleukin-2 - biosynthesis | Protozoan Proteins - immunology
Journal Article