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1. Full Text Catalpol ameliorates advanced glycation end product‐induced dysfunction of glomerular endothelial cells via regulating nitric oxide synthesis by inducible nitric oxide synthase and endothelial nitric oxide synthase
by Sun, Weixiang and Gao, Yuyan and Ding, Yushi and Cao, Ying and Chen, Jing and Lv, Gaohong and Lu, Jinfu and Yu, Bin and Peng, Meilin and Xu, Huiqin and Sun, Yun
IUBMB life, ISSN 1521-6543, 09/2019, Volume 71, Issue 9, pp. 1268 - 1283
catalpol | nitric oxide | advanced glycation end products | endothelial dysfunction | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Iridoid Glucosides - pharmacology | Nitric Oxide - biosynthesis | Endothelial Cells - metabolism | Kidney Diseases - pathology | Humans | Kidney Glomerulus - drug effects | Kidney Diseases - drug therapy | Chemokine CCL2 - genetics | Kidney Glomerulus - pathology | Nitric Oxide Synthase Type III - genetics | Kidney Diseases - genetics | Phosphatidylinositol 3-Kinases - genetics | Macrophages - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Intercellular Adhesion Molecule-1 - genetics | Nitric Oxide - genetics | Oncogene Protein v-akt - genetics | Macrophages - drug effects | Mice | Glycation End Products, Advanced - genetics | Nitric oxide | Endothelium | Monocytes | Advanced glycosylation end products | Monocyte chemoattractant protein | AKT protein | Glycosylation | Macrophages | Endothelial cells | Nitric-oxide synthase | Cell adhesion molecules | 1-Phosphatidylinositol 3-kinase | Cell adhesion & migration | Index Medicus
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2. Full Text Nitric oxide synthase in innate and adaptive immunity: an update
by Bogdan, Christian
Trends in immunology, ISSN 1471-4906, 2015, Volume 36, Issue 3, pp. 161 - 178
Allergy and Immunology | microenvironment | NOS3/eNOS | myeloid cells | nitric oxide synthases (NOS1/nNOS | antimicrobial activity | B cells | NOS2/iNOS | Th17 cells | Antimicrobial activity | Myeloid cells | Microenvironment | Nitric oxide synthases (NOS1/nNOS | Life Sciences & Biomedicine | Immunology | Science & Technology | Adaptive Immunity | Nitric Oxide - biosynthesis | Nitric Oxide - immunology | Signal Transduction | Isoenzymes - genetics | Humans | Gene Expression Regulation | STAT Transcription Factors - immunology | Immunity, Innate | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | Isoenzymes - immunology | Macrophage Activation | Bacteria - immunology | STAT Transcription Factors - genetics | Lymphocytes - immunology | Neoplasms - genetics | Nitric Oxide Synthase Type II - genetics | Nitric Oxide Synthase Type II - immunology | Neoplasms - immunology | Interferon-Stimulated Gene Factor 3, gamma Subunit - immunology | Neoplasms - pathology | Macrophages - immunology | Nitric oxide | Stem cells | Enzymes | Gram-positive bacteria | Cell division | Nitrates | Metastasis | Cell adhesion & migration | Proteins | Lymphocytes | Cell cycle | Hypoxia | Sulfur | Immune system | Index Medicus
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3. Full Text Enhancer-associated long non-coding RNA LEENE regulates endothelial nitric oxide synthase and endothelial function
by Miao, Yifei and Ajami, Nassim E and Huang, Tse-Shun and Lin, Feng-Mao and Lou, Chih-Hong and Wang, Yun-Ting and Li, Shuai and Kang, Jian and Munkacsi, Hannah and Maurya, Mano R and Gupta, Shakti and Chien, Shu and Subramaniam, Shankar and Chen, Zhen
Nature communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 292 - 13
Endothelial Cells - metabolism | Humans | Mice, Inbred C57BL | Cells, Cultured | Male | Gene Expression Profiling | RNA, Long Noncoding - genetics | RNA Polymerase II - metabolism | Nitric Oxide Synthase Type III - genetics | Promoter Regions, Genetic - genetics | Gene Expression Regulation, Enzymologic | Animals | Transcription, Genetic | Enhancer Elements, Genetic - genetics | Nitric Oxide Synthase Type III - metabolism | Post-transcription | Homeostasis | Gene expression | Ribonucleic acid--RNA | DNA-directed RNA polymerase | Endothelial cells | Nitric-oxide synthase | Gene sequencing | Chromatin remodeling | Nitric oxide | Post-translation | Non-coding RNA | RNA polymerase II | Conformation | Index Medicus
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4. Full Text Regulation of endothelial nitric-oxide synthase (NOS) S-glutathionylation by neuronal NOS: Evidence of a functional interaction between myocardial constitutive NOS isoforms
by Idigo, Winifred O and Reilly, Svetlana and Zhang, Mei Hua and Zhang, Yin Hua and Jayaram, Raja and Carnicer, Ricardo and Crabtree, Mark J and Balligand, Jean-Luc and Casadei, Barbara
The Journal of biological chemistry, ISSN 0021-9258, 12/2012, Volume 287, Issue 52, pp. 43665 - 43673
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Phosphorylation - physiology | Xanthine Dehydrogenase - metabolism | Heart Ventricles - cytology | Citrulline - analogs & derivatives | Membrane Glycoproteins - metabolism | Calcium Signaling - physiology | Arginase - metabolism | NADPH Oxidases - metabolism | Xanthine Dehydrogenase - genetics | Receptors, Adrenergic, beta-3 - immunology | Thiourea - pharmacology | Myocytes, Cardiac - enzymology | Isoenzymes - metabolism | Heart Ventricles - enzymology | Nitric Oxide Synthase Type I - antagonists & inhibitors | Receptors, Adrenergic, beta-3 - genetics | Muscle Proteins - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Muscle Proteins - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Phosphorylation - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Myocytes, Cardiac - cytology | Isoenzymes - genetics | Enzyme Inhibitors - pharmacology | Arginase - genetics | NADPH Oxidase 2 | Nitric Oxide Synthase Type III - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Myocardium - cytology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Muscle Proteins - genetics | Myocardium - enzymology | Animals | Citrulline - pharmacology | Calcium Signaling - drug effects | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Isoenzymes - antagonists & inhibitors | Thiourea - analogs & derivatives | Index Medicus | Signal Transduction | Oxidative Stress | Adrenergic Receptor | Nitric-oxide Synthase | Myocardium | Glutathionylation | Nitric Oxide
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5. Full Text Enhanced XOR activity in eNOS-deficient mice: Effects on the nitrate-nitrite-NO pathway and ROS homeostasis
by Peleli, Maria and Zollbrecht, Christa and Montenegro, Marcelo F and Hezel, Michael and Zhong, Jianghong and Persson, Erik G and Holmdahl, Rikard and Weitzberg, Eddie and Lundberg, Jon O and Carlström, Mattias
Free radical biology & medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 472 - 484
Hypertension | Oxidative stress | Endothelial nitric oxide synthase | Reactive oxygen species | Nitrate | Febuxostat | Nitric oxide | Xanthine oxidoreductase | Xanthine oxidase | Superoxide | Nitrite | Uric acid | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Xanthine Dehydrogenase - metabolism | Xanthine Dehydrogenase - antagonists & inhibitors | Xanthine Dehydrogenase - genetics | Nitrates - pharmacology | Male | Nitric Oxide Synthase Type III - deficiency | Febuxostat - pharmacology | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Synthase Type I - antagonists & inhibitors | Nitrites - blood | Superoxides - metabolism | Blood Pressure - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Nitrates - blood | Nitric Oxide - blood | Oxidation-Reduction | Signal Transduction | Nitric Oxide Synthase Type II - deficiency | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Nitric Oxide Synthase Type III - genetics | Nitrites - pharmacology | Mice, Knockout | Nitric Oxide Synthase Type I - deficiency | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | Mice | Nitric Oxide Synthase Type I - genetics | Oxidases | Iron compounds | Homeostasis | Index Medicus | Medicin och hälsovetenskap
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6. Full Text Nitric oxide synthase inhibition decreases l- DOPA-induced dyskinesia and the expression of striatal molecular markers in Pitx3−/− aphakia mice
by Solís, Oscar and Espadas, Isabel and Del-Bel, Elaine A and Moratalla, Rosario
Neurobiology of disease, ISSN 0969-9961, 2014, Volume 73, pp. 49 - 59
Neurology | Striatum | Molsidomine | Pitx3 deficient aphakia mice | Parkinson disease | l-DOPA | Nitrergic system | Histone3 | FosB | Zaprinast | NO/cGMP | Abnormal involuntary movements | L-DOPA | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cell Count | Transcription Factors - deficiency | Antiparkinson Agents - toxicity | Parkinson Disease - drug therapy | Molsidomine - administration & dosage | Corpus Striatum - metabolism | Time Factors | Dyskinesia, Drug-Induced - drug therapy | Disease Models, Animal | Dyskinesia, Drug-Induced - etiology | Gene Expression Regulation - genetics | Benserazide - therapeutic use | Levodopa - toxicity | Signal Transduction - genetics | Parkinson Disease - genetics | Transcription Factors - genetics | Dyskinesia, Drug-Induced - pathology | Enzyme Inhibitors - therapeutic use | Homeodomain Proteins - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Nitric Oxide Donors - administration & dosage | Animals | Signal Transduction - drug effects | Corpus Striatum - drug effects | Dopamine Agents - toxicity | Mice | Indazoles - therapeutic use | Index Medicus
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