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Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 472 - 484
Xanthine oxidoreductase (XOR) is generally known as the final enzyme in purine metabolism and as a source of reactive oxygen species (ROS). In addition, this... 
Hypertension | Oxidative stress | Endothelial nitric oxide synthase | Reactive oxygen species | Nitrate | Febuxostat | Nitric oxide | Xanthine oxidoreductase | Xanthine oxidase | Superoxide | Nitrite | Uric acid | NADPH OXIDASE | METABOLIC SYNDROME | OXIDASE INHIBITOR FEBUXOSTAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOOD-PRESSURE | REACTIVE SPECIES GENERATION | INDUCED HYPERTENSION | SYNTHASE ACTIVITY | ENDOCRINOLOGY & METABOLISM | DIETARY INORGANIC NITRATE | Xanthine Dehydrogenase - metabolism | Xanthine Dehydrogenase - antagonists & inhibitors | Xanthine Dehydrogenase - genetics | Nitrates - pharmacology | Male | Nitric Oxide Synthase Type III - deficiency | Febuxostat - pharmacology | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Synthase Type I - antagonists & inhibitors | Nitrites - blood | Superoxides - metabolism | Blood Pressure - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Nitrates - blood | Nitric Oxide - blood | Oxidation-Reduction | Signal Transduction | Nitric Oxide Synthase Type II - deficiency | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Nitric Oxide Synthase Type III - genetics | Nitrites - pharmacology | Mice, Knockout | Nitric Oxide Synthase Type I - deficiency | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | Mice | Nitric Oxide Synthase Type I - genetics | Oxidases | Iron compounds | Homeostasis | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Cell Biology | Clinical Medicine | Naturvetenskap | Endokrinologi och diabetes | Biologiska vetenskaper | Natural Sciences | Cellbiologi | Endocrinology and Diabetes
Journal Article
Hypertension, ISSN 0194-911X, 09/2009, Volume 54, Issue 3, pp. 652 - 658
Drugs and antibodies that interrupt vascular endothelial growth factor (VEGF) signaling pathways improve outcomes in patients with a variety of cancers by... 
Hypertension | Angiogenesis | Vascular endothelial growth factor | Cancer | TYROSINE KINASE-1 | angiogenesis | vascular endothelial growth factor | VEGF | MOUSE | PERIPHERAL VASCULAR DISEASE | cancer | MONOCLONAL-ANTIBODY | MICE | PREECLAMPSIA | hypertension | MOLECULAR-MECHANISMS | Gene Expression - drug effects | Kidney - enzymology | Antibodies, Monoclonal - adverse effects | Sodium Chloride, Dietary - administration & dosage | Male | Vascular Endothelial Growth Factor Receptor-2 - immunology | RNA, Messenger - metabolism | Aldosterone - urine | Hypertension - etiology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Renin - genetics | Prostaglandins - urine | Nitric Oxide Synthase Type I - antagonists & inhibitors | Blood Pressure - drug effects | Blood Pressure - physiology | Vascular Endothelial Growth Factor Receptor-2 - physiology | Nitric Oxide Synthase Type III - metabolism | Manometry | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - biosynthesis | Kidney - drug effects | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Sodium Chloride, Dietary - adverse effects | Enzyme Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Nitric Oxide Synthase Type I - metabolism | Hybridomas | Mice | Nitric Oxide Synthase Type I - genetics | Vascular Endothelial Growth Factor A - physiology | Index Medicus | nitric oxide
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2012, Volume 287, Issue 52, pp. 43665 - 43673
Myocardial constitutive No production depends on the activity of both endothelial and neuronal NOS (eNOS and nNOS, respectively). Stimulation of myocardial... 
ACTIVATION | XANTHINE OXIDOREDUCTASE | PHOSPHORYLATION | SUPEROXIDE GENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUSCULAR-DYSTROPHY | BETA-ADRENOCEPTOR | DEFICIENT SKELETAL-MUSCLE | VENTRICULAR MYOCYTES | UP-REGULATION | HUMAN HEART | Phosphorylation - physiology | Xanthine Dehydrogenase - metabolism | Heart Ventricles - cytology | Citrulline - analogs & derivatives | Membrane Glycoproteins - metabolism | Calcium Signaling - physiology | Arginase - metabolism | NADPH Oxidases - metabolism | Xanthine Dehydrogenase - genetics | Receptors, Adrenergic, beta-3 - immunology | Thiourea - pharmacology | Myocytes, Cardiac - enzymology | Isoenzymes - metabolism | Heart Ventricles - enzymology | Nitric Oxide Synthase Type I - antagonists & inhibitors | Receptors, Adrenergic, beta-3 - genetics | Muscle Proteins - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Muscle Proteins - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Phosphorylation - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Myocytes, Cardiac - cytology | Isoenzymes - genetics | Enzyme Inhibitors - pharmacology | Arginase - genetics | NADPH Oxidase 2 | Nitric Oxide Synthase Type III - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Myocardium - cytology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Muscle Proteins - genetics | Myocardium - enzymology | Animals | Citrulline - pharmacology | Calcium Signaling - drug effects | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Isoenzymes - antagonists & inhibitors | Thiourea - analogs & derivatives | Signal Transduction | Oxidative Stress | Adrenergic Receptor | Nitric-oxide Synthase | Myocardium | Glutathionylation | Nitric Oxide
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2012, Volume 109, Issue 41, pp. 16624 - 16629
Nitric oxide (NO) generated by neuronal NO synthase (nNOS) initiates penile erection, but has not been thought to participate in the sustained erection... 
Alternative splicing | Phosphorylation | Calcium | Penile erection | Electrical stimulation | Antibodies | Oxides | Smooth muscle | Physiology | Physiological regulation | Phosphoantibody | Smooth muscle relaxation | Gasotransmitter | Cyclic GMP | Endothelial NOS | NNOS | NERVOUS-SYSTEM | ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | cyclic GMP | endothelial NOS | AKT | smooth muscle relaxation | phosphoantibody | IN-VITRO | CORPUS CAVERNOSUM | CA2 | gasotransmitter | GENE-EXPRESSION | MICE | Immunohistochemistry | Penis - physiology | Electric Stimulation | Male | Penis - innervation | Penile Erection - drug effects | Nitric Oxide Synthase Type I - antagonists & inhibitors | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Colforsin - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Rats | Rats, Sprague-Dawley | Serine - metabolism | Blotting, Western | Mice, Knockout | Animals | Penis - metabolism | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Penile Erection - physiology | Penis | Nitric oxide | Physiological aspects | Cyclic adenylic acid | Research | Health aspects | Biological Sciences
Journal Article
Journal Article