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Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 472 - 484
Xanthine oxidoreductase (XOR) is generally known as the final enzyme in purine metabolism and as a source of reactive oxygen species (ROS). In addition, this... 
Hypertension | Oxidative stress | Endothelial nitric oxide synthase | Reactive oxygen species | Nitrate | Febuxostat | Nitric oxide | Xanthine oxidoreductase | Xanthine oxidase | Superoxide | Nitrite | Uric acid | NADPH OXIDASE | METABOLIC SYNDROME | OXIDASE INHIBITOR FEBUXOSTAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOOD-PRESSURE | REACTIVE SPECIES GENERATION | INDUCED HYPERTENSION | SYNTHASE ACTIVITY | ENDOCRINOLOGY & METABOLISM | DIETARY INORGANIC NITRATE | Xanthine Dehydrogenase - metabolism | Xanthine Dehydrogenase - antagonists & inhibitors | Xanthine Dehydrogenase - genetics | Nitrates - pharmacology | Male | Nitric Oxide Synthase Type III - deficiency | Febuxostat - pharmacology | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Synthase Type I - antagonists & inhibitors | Nitrites - blood | Superoxides - metabolism | Blood Pressure - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Nitrates - blood | Nitric Oxide - blood | Oxidation-Reduction | Signal Transduction | Nitric Oxide Synthase Type II - deficiency | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Nitric Oxide Synthase Type III - genetics | Nitrites - pharmacology | Mice, Knockout | Nitric Oxide Synthase Type I - deficiency | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | Mice | Nitric Oxide Synthase Type I - genetics | Oxidases | Iron compounds | Homeostasis | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Cell Biology | Clinical Medicine | Naturvetenskap | Endokrinologi och diabetes | Biologiska vetenskaper | Natural Sciences | Cellbiologi | Endocrinology and Diabetes
Journal Article
Hypertension, ISSN 0194-911X, 09/2009, Volume 54, Issue 3, pp. 652 - 658
Drugs and antibodies that interrupt vascular endothelial growth factor (VEGF) signaling pathways improve outcomes in patients with a variety of cancers by... 
Hypertension | Angiogenesis | Vascular endothelial growth factor | Cancer | TYROSINE KINASE-1 | angiogenesis | vascular endothelial growth factor | VEGF | MOUSE | PERIPHERAL VASCULAR DISEASE | cancer | MONOCLONAL-ANTIBODY | MICE | PREECLAMPSIA | hypertension | MOLECULAR-MECHANISMS | Gene Expression - drug effects | Kidney - enzymology | Antibodies, Monoclonal - adverse effects | Sodium Chloride, Dietary - administration & dosage | Male | Vascular Endothelial Growth Factor Receptor-2 - immunology | RNA, Messenger - metabolism | Aldosterone - urine | Hypertension - etiology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Renin - genetics | Prostaglandins - urine | Nitric Oxide Synthase Type I - antagonists & inhibitors | Blood Pressure - drug effects | Blood Pressure - physiology | Vascular Endothelial Growth Factor Receptor-2 - physiology | Nitric Oxide Synthase Type III - metabolism | Manometry | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - biosynthesis | Kidney - drug effects | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Sodium Chloride, Dietary - adverse effects | Enzyme Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Nitric Oxide Synthase Type I - metabolism | Hybridomas | Mice | Nitric Oxide Synthase Type I - genetics | Vascular Endothelial Growth Factor A - physiology | Index Medicus | nitric oxide
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2016, Volume 36, Issue 1, pp. 78 - 85
Journal Article
Circulation Research, ISSN 0009-7330, 10/2008, Volume 103, Issue 9, pp. 957 - 964
Reduction of nitrite (NO2) provides a major source of nitric oxide (NO) in the circulation, especially in hypoxemic conditions. Our previous studies suggest... 
Blood vessels | Hypoxia | Cardiovascular research | Nitric oxide | HEMOGLOBIN | hypoxia | nitric oxide | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | blood vessels | INDUCED VASODILATION | cardiovascular research | ISCHEMIA-REPERFUSION INJURY | IN-VIVO | S-NITROSOTHIOLS | MEDIATED DECOMPOSITION | PERIPHERAL VASCULAR DISEASE | RED-BLOOD-CELLS | REDUCES NITRITE | HEMATOLOGY | Xanthine Dehydrogenase - metabolism | Rats, Wistar | Xanthine Dehydrogenase - antagonists & inhibitors | Humans | Nitrites - metabolism | Male | omega-N-Methylarginine - pharmacology | Fluorometry | Cell Hypoxia | Nitrite Reductases - antagonists & inhibitors | Nitrites - blood | Erythrocytes - enzymology | Nitric Oxide Synthase Type III - metabolism | Aorta - enzymology | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - blood | Oxidation-Reduction | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Xanthine Dehydrogenase - blood | Erythrocytes - drug effects | Nitric Oxide Synthase Type III - antagonists & inhibitors | Xanthine - metabolism | Nitric Oxide Synthase Type III - blood | Animals | Allopurinol - pharmacology | Nitric Oxide - metabolism | Endothelial Cells - enzymology | Arginine - metabolism | Endothelial Cells - drug effects | Hydrogen-Ion Concentration | Nitric Oxide Synthase Type II - metabolism | Nitrite Reductases - metabolism
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 3, pp. 729 - 736.e5
Background Clinical observations suggest that anaphylaxis is more common in adult women compared with adult men, although the mechanistic basis for this sex... 
Allergy and Immunology | estrogen | Anaphylaxis | nitric oxide synthase | vascular permeability | mast cells | ACTIVATION | IDIOPATHIC ANAPHYLAXIS | SEX-HORMONES | CARDIOVASCULAR-SYSTEM | SENSITIVITY | BODY-TEMPERATURE | RELEASE | IMMUNOLOGY | SHOCK | ALLERGY | NO SYNTHASE | UP-REGULATION | Protein Aggregates | Capillary Permeability | Humans | Receptors, IgG - chemistry | Male | omega-N-Methylarginine - pharmacology | Histamine - immunology | Receptors, IgG - antagonists & inhibitors | Lung - enzymology | Receptors, Estrogen - immunology | Receptors, IgE - chemistry | Estrogen Receptor Antagonists - pharmacology | Anaphylaxis - physiopathology | Anaphylaxis - immunology | Female | Receptors, IgE - immunology | Estradiol - pharmacology | Disease Models, Animal | Estradiol - metabolism | Mast Cells - immunology | Nitric Oxide - biosynthesis | Nitric Oxide Synthase Type III - immunology | Gene Expression | Histamine - pharmacology | Ovariectomy | Receptors, Estrogen - antagonists & inhibitors | Receptors, IgE - antagonists & inhibitors | Lung - physiopathology | Receptors, IgG - immunology | Body Temperature | Anaphylaxis - enzymology | Mast Cells - drug effects | Nitric Oxide Synthase Type III - genetics | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Anaphylaxis - genetics | Animals | Receptors, Estrogen - chemistry | Lung - drug effects | Mast Cells - pathology | Mice | Lung - immunology | Sex discrimination | Nitric oxide | Estrogen | Endothelium | Gender differences | Rodents | Allergies
Journal Article
American Journal of Physiology : Endocrinology and Metabolism, ISSN 0193-1849, 03/2012, Volume 302, Issue 5, pp. E481 - E495
Kietadisorn R, Juni RP, Moens AL. Tackling endothelial dysfunction by modulating NOS uncoupling: new insights into its pathogenesis and therapeutic... 
Endothelial dysfunction | Nitric oxide synthase | Superoxide | Nitric oxide synthase uncoupling | C-REACTIVE PROTEIN | PHYSIOLOGY | HUMAN ATRIAL-FIBRILLATION | NITRIC-OXIDE SYNTHASE | LOW-DENSITY-LIPOPROTEIN | FOLIC-ACID SUPPLEMENTATION | REDUCES ISCHEMIA/REPERFUSION INJURY | endothelial dysfunction | ISCHEMIA-REPERFUSION INJURY | E-KNOCKOUT MICE | ENDOCRINOLOGY & METABOLISM | ACUTE MYOCARDIAL-INFARCTION | nitric oxide synthase | nitric oxide synthase uncoupling | superoxide | CORONARY-ARTERY-DISEASE | Reactive Oxygen Species - metabolism | Humans | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Endothelium, Vascular - drug effects | Vascular Diseases - physiopathology | Enzyme Stability - drug effects | Stem Cells - metabolism | Enzyme Inhibitors - therapeutic use | Vascular Diseases - diagnosis | Nitric Oxide - agonists | Nitric Oxide Synthase Type III - antagonists & inhibitors | Nitric Oxide Synthase Type III - chemistry | Reactive Oxygen Species - antagonists & inhibitors | Animals | Stem Cells - enzymology | Endothelium, Vascular - metabolism | Enzyme Induction - drug effects | Stem Cells - drug effects | Nitric Oxide Synthase Type III - metabolism | Nitric Oxide - metabolism | Vascular Diseases - metabolism | Vascular Diseases - drug therapy | Physiological aspects | Development and progression | Health aspects | Nitric oxide | Atherosclerosis | Endothelium
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 40, pp. 28668 - 28686
Journal Article
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 10/2009, Volume 107, Issue 4, pp. 1249 - 1257
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2012, Volume 287, Issue 52, pp. 43665 - 43673
Myocardial constitutive No production depends on the activity of both endothelial and neuronal NOS (eNOS and nNOS, respectively). Stimulation of myocardial... 
ACTIVATION | XANTHINE OXIDOREDUCTASE | PHOSPHORYLATION | SUPEROXIDE GENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUSCULAR-DYSTROPHY | BETA-ADRENOCEPTOR | DEFICIENT SKELETAL-MUSCLE | VENTRICULAR MYOCYTES | UP-REGULATION | HUMAN HEART | Phosphorylation - physiology | Xanthine Dehydrogenase - metabolism | Heart Ventricles - cytology | Citrulline - analogs & derivatives | Membrane Glycoproteins - metabolism | Calcium Signaling - physiology | Arginase - metabolism | NADPH Oxidases - metabolism | Xanthine Dehydrogenase - genetics | Receptors, Adrenergic, beta-3 - immunology | Thiourea - pharmacology | Myocytes, Cardiac - enzymology | Isoenzymes - metabolism | Heart Ventricles - enzymology | Nitric Oxide Synthase Type I - antagonists & inhibitors | Receptors, Adrenergic, beta-3 - genetics | Muscle Proteins - metabolism | Superoxides - metabolism | NADPH Oxidases - genetics | Muscle Proteins - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Phosphorylation - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Myocytes, Cardiac - cytology | Isoenzymes - genetics | Enzyme Inhibitors - pharmacology | Arginase - genetics | NADPH Oxidase 2 | Nitric Oxide Synthase Type III - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Myocardium - cytology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Muscle Proteins - genetics | Myocardium - enzymology | Animals | Citrulline - pharmacology | Calcium Signaling - drug effects | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Isoenzymes - antagonists & inhibitors | Thiourea - analogs & derivatives | Signal Transduction | Oxidative Stress | Adrenergic Receptor | Nitric-oxide Synthase | Myocardium | Glutathionylation | Nitric Oxide
Journal Article