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Circulation, ISSN 0009-7322, 08/2011, Volume 124, Issue 6, pp. 731 - 740
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 472 - 484
Xanthine oxidoreductase (XOR) is generally known as the final enzyme in purine metabolism and as a source of reactive oxygen species (ROS). In addition, this... 
Hypertension | Oxidative stress | Endothelial nitric oxide synthase | Reactive oxygen species | Nitrate | Febuxostat | Nitric oxide | Xanthine oxidoreductase | Xanthine oxidase | Superoxide | Nitrite | Uric acid | NADPH OXIDASE | METABOLIC SYNDROME | OXIDASE INHIBITOR FEBUXOSTAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOOD-PRESSURE | REACTIVE SPECIES GENERATION | INDUCED HYPERTENSION | SYNTHASE ACTIVITY | ENDOCRINOLOGY & METABOLISM | DIETARY INORGANIC NITRATE | Xanthine Dehydrogenase - metabolism | Xanthine Dehydrogenase - antagonists & inhibitors | Xanthine Dehydrogenase - genetics | Nitrates - pharmacology | Male | Nitric Oxide Synthase Type III - deficiency | Febuxostat - pharmacology | Nitric Oxide Synthase Type II - antagonists & inhibitors | Nitric Oxide Synthase Type I - antagonists & inhibitors | Nitrites - blood | Superoxides - metabolism | Blood Pressure - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Nitrates - blood | Nitric Oxide - blood | Oxidation-Reduction | Signal Transduction | Nitric Oxide Synthase Type II - deficiency | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Nitric Oxide Synthase Type III - genetics | Nitrites - pharmacology | Mice, Knockout | Nitric Oxide Synthase Type I - deficiency | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | Mice | Nitric Oxide Synthase Type I - genetics | Oxidases | Iron compounds | Homeostasis | Index Medicus | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Cell Biology | Clinical Medicine | Naturvetenskap | Endokrinologi och diabetes | Biologiska vetenskaper | Natural Sciences | Cellbiologi | Endocrinology and Diabetes
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2010, Volume 107, Issue 41, pp. 17716 - 17720
The metabolic syndrome is a clustering of risk factors of metabolic origin that increase the risk for cardiovascular disease and type 2 diabetes. A proposed... 
Animals | Nitrites | Oxides | Metabolic diseases | Nitrogen oxides | Nitrates | Mice | Blood pressure | Type 2 diabetes mellitus | Inorganic nitrates | Obesity | Bacteria | S-nitrosothiol | Glucose | Insulin | THERAPEUTICS | glucose | s-nitrosothiol | insulin | bacteria | MITOCHONDRIAL BIOGENESIS | MULTIDISCIPLINARY SCIENCES | MECHANISMS | BLOOD-PRESSURE | PLASMA NITRITE | REDUCTION | INSULIN-RESISTANCE | RESPIRATION | DYSFUNCTION | EXPRESSION | obesity | Nitrogen Oxides - blood | Body Weight | Nitrates - pharmacology | Rats | Metabolic Syndrome - drug therapy | Nitric Oxide Synthase Type III - deficiency | Nitrates - administration & dosage | Analysis of Variance | Mice, Mutant Strains | Triglycerides - blood | Blood Pressure - drug effects | Intra-Abdominal Fat - drug effects | Nitrogen Oxides - metabolism | Dietary Supplements | Type 2 diabetes | Control | Nitric oxide | Physiological aspects | Metabolic syndrome X | Cardiovascular diseases | Risk factors | Dietary minerals | Rodents | Cardiovascular disease | Biosynthesis | Diabetes | Metabolic syndrome | Index Medicus | Vegetables | oxides | Diabetes mellitus | Dietary supplements | Photochemicals | Triglycerides | Nitrite | Fuels | Nitrogen | Nitric-oxide synthase | Diets | Nitrate | Oxidation | Plasma levels | sodium nitrate | Metabolic disorders | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 2009, Volume 120, Issue 10, pp. 888 - 896
Background-Sudden cardiac arrest (CA) is one of the leading causes of death worldwide. We sought to evaluate the impact of hydrogen sulfide (H2S) on the... 
Heart arrest | Nitric oxide synthase | Myocardial contraction | Physiology | Cardiopulmonary resuscitation | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHORYLATION | PROTEIN-KINASE | MEDIATOR | physiology | apoptosis | PERMEABILITY TRANSITION | CELL-DEATH | ISCHEMIA-REPERFUSION INJURY | cardiopulmonary resuscitation | myocardial contraction | INHIBITION | PATHWAY | PERIPHERAL VASCULAR DISEASE | heart arrest | nitric oxide synthase | CARDIOPROTECTION | HEMATOLOGY | Hydrogen Sulfide - metabolism | Up-Regulation | AMP-Activated Protein Kinases - metabolism | Apoptosis - drug effects | Cystathionine gamma-Lyase - metabolism | Male | Mitochondria, Heart - drug effects | Nitric Oxide Synthase Type III - deficiency | Nervous System - physiopathology | Myocytes, Cardiac - enzymology | Nitric Oxide Synthase Type III - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Sulfides - pharmacology | Heart - physiopathology | Heart Arrest - enzymology | Myocardial Contraction | Cardiopulmonary Resuscitation - adverse effects | Signal Transduction | Brain - physiopathology | Mice, Inbred C57BL | Survival Rate | Cardiotonic Agents - pharmacology | Heart Arrest - physiopathology | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Animals | Heart Arrest - mortality | Nervous System - drug effects | Heart - drug effects | Mice | Oxidative Stress - drug effects | In Vitro Techniques | Heart Arrest - therapy | Hydrogen sulfide | Care and treatment | Usage | Patient outcomes | Cardiac arrest | CPR (First aid) | Models | Research | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2015, Volume 35, Issue 11, pp. 2366 - 2373
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2013, Volume 110, Issue 37, pp. 15049 - 15054
Journal Article
Hypertension, ISSN 0194-911X, 07/2012, Volume 60, Issue 1, pp. 231 - 238
Preeclampsia is associated with impaired uteroplacental adaptations during pregnancy and abnormalities in the endothelial NO synthase (eNOS)-NO pathway, but... 
NO | Hypoxia | Endothelial NO synthase | Placenta | Blood flow regulation | Preeclampsia/pregnancy | hypoxia | NATURAL-KILLER-CELLS | PREECLAMPSIA | KNOCKOUT MICE | PATHOGENESIS | PRESSURE | INTRAUTERINE GROWTH-RETARDATION | blood flow regulation | preeclampsia/pregnancy | placenta | PERIPHERAL VASCULAR DISEASE | UTEROPLACENTAL CIRCULATION | NO SYNTHASE | HYPERTENSION | endothelial NO synthase | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Uterus - metabolism | Male | Nitric Oxide Synthase Type III - deficiency | Oxygen - metabolism | Uterine Artery - physiology | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Killer Cells, Natural - immunology | Female | Uterus - blood supply | Vascular Endothelial Growth Factor Receptor-1 - genetics | Microscopy, Electron, Scanning | Enzyme-Linked Immunosorbent Assay | Placenta - immunology | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Nitric Oxide Synthase Type III - genetics | Mice, Knockout | Placenta - metabolism | Pregnancy | Placenta - blood supply | Animals | Blood Flow Velocity | Lymphocyte Count | Mice | Killer Cells, Natural - metabolism | Uterine Artery - ultrastructure | Vascular Endothelial Growth Factor Receptor-1 - blood | Index Medicus
Journal Article