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Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7593, pp. 253 - 257
Journal Article
Nature, ISSN 0028-0836, 11/2017, Volume 551, Issue 7680, pp. 340 - 345
The role of adaptive immunity in early cancer development is controversial. Here we show that chronic inflammation and fibrosis in humans and mice with... 
FIBROSIS | HEPATOCELLULAR-CARCINOMA | GENE | IMMUNOTHERAPY | MULTIDISCIPLINARY SCIENCES | NONALCOHOLIC STEATOHEPATITIS | MICE | DEFICIENT MOUSE | KAPPA-B | TARGETED DELETION | EXPRESSION | Clone Cells - cytology | Inflammation - pathology | Liver Cirrhosis - immunology | Non-alcoholic Fatty Liver Disease - pathology | Cell Proliferation | Humans | Male | CD8 Antigens - deficiency | Liver Neoplasms - etiology | Non-alcoholic Fatty Liver Disease - complications | Immune Tolerance - immunology | Clone Cells - immunology | Liver Cirrhosis - metabolism | Interleukin-10 - metabolism | Female | Liver Neoplasms - pathology | Plasma Cells - metabolism | Carcinoma, Hepatocellular - etiology | Carcinoma, Hepatocellular - immunology | T-Lymphocytes, Cytotoxic - immunology | Immunoglobulin A - metabolism | Liver Cirrhosis - complications | Lymphocyte Activation | Non-alcoholic Fatty Liver Disease - immunology | Gastrointestinal Microbiome | Liver Neoplasms - immunology | Inflammation - immunology | Non-alcoholic Fatty Liver Disease - metabolism | Inflammation - etiology | Disease Progression | B7-H1 Antigen - metabolism | Animals | Immunoglobulin A - immunology | Carcinoma, Hepatocellular - pathology | Liver Cirrhosis - pathology | Mice | T-Lymphocytes, Cytotoxic - cytology | Plasma Cells - immunology | Prevention | Liver cancer | Immunoglobulins | Health aspects | Index Medicus
Journal Article
世界胃肠病学杂志:英文版(电子版), ISSN 1007-9327, 2014, Volume 20, Issue 23, pp. 7381 - 7391
Emerging data have shown a close association between compositional changes in gut microbiota and the development of nonalcoholic fatty liver disease(NAFLD).The... 
disease | stea | fatty | liver | Nonalcoholic | Gut microbiota | Probiotics | Nonalcoholic fatty liver disease | Nonalcoholic steatohepatitis | Toll-like receptor | Prebiotics | METABOLIC SYNDROME | NECROSIS-FACTOR-ALPHA | DIET-INDUCED OBESITY | TOTAL PARENTERAL-NUTRITION | INCREASED INTESTINAL PERMEABILITY | HEPATIC STELLATE CELLS | INSULIN-RESISTANCE | KUPFFER CELLS | TNF-ALPHA | GASTROENTEROLOGY & HEPATOLOGY | PATTERN-RECOGNITION RECEPTORS | Intestines - drug effects | Humans | Bacteria - drug effects | Liver - microbiology | Pediatric Obesity - microbiology | Intestines - metabolism | Intestines - immunology | Bacteria - immunology | Liver - immunology | Liver - drug effects | Toll-Like Receptors - drug effects | Toll-Like Receptors - metabolism | Cytokines - immunology | Inflammation Mediators - immunology | Pediatric Obesity - immunology | Bacteria - metabolism | Signal Transduction | Liver - metabolism | Non-alcoholic Fatty Liver Disease - immunology | Non-alcoholic Fatty Liver Disease - microbiology | Risk Factors | Toll-Like Receptors - immunology | Non-alcoholic Fatty Liver Disease - therapy | Lipid Metabolism | Non-alcoholic Fatty Liver Disease - metabolism | Permeability | Bacterial Translocation | Host-Pathogen Interactions | Animals | Intestines - microbiology | Probiotics - therapeutic use | Immunologic Factors - therapeutic use | Topic Highlight
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 3, pp. 635 - 648.e14
Background & Aims The prevalence of nonalcoholic fatty liver disease (NAFLD) has increased with the obesity pandemic. We analyzed the transcriptional profiles... 
Gastroenterology and Hepatology | Immune Regulation | Gene Expression | Inflammatory Response | IL8 | FREE MASS | MACROPHAGES | BIOELECTRICAL IMPEDANCE | WEIGHT-LOSS | INSULIN-RESISTANCE | MORBID-OBESITY | STEATOHEPATITIS | MICE | GASTROENTEROLOGY & HEPATOLOGY | HEPATIC STEATOSIS | EXPRESSION | Liver Cirrhosis - immunology | Predictive Value of Tests | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Male | Gene Regulatory Networks | Subcutaneous Fat - immunology | Non-alcoholic Fatty Liver Disease - blood | Subcutaneous Fat - metabolism | Adult | Female | Immunophenotyping - methods | Obesity - diagnosis | Panniculitis - immunology | Cytokines - genetics | Macrophages - immunology | Cytokines - blood | Cytokines - immunology | Bariatric Surgery | Inflammation Mediators - immunology | Intra-Abdominal Fat - immunology | Obesity - surgery | Severity of Illness Index | Panniculitis - diagnosis | Obesity - complications | Non-alcoholic Fatty Liver Disease - immunology | Cells, Cultured | Inflammation Mediators - blood | Gene Expression Profiling - methods | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Disease Progression | Macrophages - metabolism | Panniculitis - blood | Phenotype | Belgium | Biopsy | Non-alcoholic Fatty Liver Disease - diagnosis | South Africa | Adipose tissues | Epidemics | Fatty liver | Analysis
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2017, Volume 23, Issue 7, pp. 829 - 838
Nonalcoholic fatty liver disease (NAFLD), a common prelude to cirrhosis and hepatocellular carcinoma, is the most common chronic liver disease worldwide.... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | NAFLD | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | AMBIENT-TEMPERATURE | DIET-INDUCED OBESITY | CELL BIOLOGY | RESPONSES | INSULIN-RESISTANCE | INFLAMMATION | STEATOHEPATITIS NASH | COLD | Intestinal Mucosa - metabolism | Temperature | Obesity - immunology | Humans | Receptors, Interleukin-17 - immunology | Male | Gene Expression Profiling | Receptors, Interleukin-17 - genetics | Gram-Negative Bacteria - immunology | Machine Learning | Jejunum - metabolism | Flow Cytometry | Housing, Animal | Diet, High-Fat | Female | Disease Models, Animal | Cold Temperature | Non-alcoholic Fatty Liver Disease - immunology | Toll-Like Receptor 4 - genetics | Hematopoietic Stem Cells - metabolism | Inflammation | Non-alcoholic Fatty Liver Disease - metabolism | Permeability | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Disease Progression | Mice, Knockout | Obesity - metabolism | Animals | Sex Factors | Corticosterone - metabolism | Mice | Gastrointestinal Microbiome - immunology | Stress, Physiological - immunology | Care and treatment | Immune response | Fatty liver | Microbiota (Symbiotic organisms) | Corticosterone | Analysis | Patient outcomes | Dosage and administration | Health aspects | Animal models | Pathogenesis | Liver | Sex | Hepatocellular carcinoma | Inactivation | High fat diet | Proteins | Pathways | Clonal deletion | Intestine | Rodents | Interrogation | Toll-like receptors | Deletion | Immune system | Liver diseases | Deactivation | TLR4 protein | Housing | Fatty acids | Interleukin 17 | Hemopoiesis | Amplification | Cirrhosis | Molecular modelling | Fibrosis | Models
Journal Article