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Gastroenterology, ISSN 0016-5085, 04/2018, Volume 154, Issue 5, pp. 1449 - 1464.e20
The innate immune system responds not only to bacterial signals, but also to non-infectious danger-associated molecular patterns that activate the NLRP3... 
Immune Regulation | Acute Liver Failure | Biological Clock | Rev-erbα | ACUTE LIVER-FAILURE | IMMUNITY | ACTIVATION | MACROPHAGES | BEHAVIOR | Rev-erb alpha | TRANSCRIPTION | METABOLISM | REV-ERB-ALPHA | GASTROENTEROLOGY & HEPATOLOGY | CLOCK | EXPRESSION | Liver - pathology | Inflammasomes - metabolism | Macrophages, Peritoneal - pathology | Caspase 1 - metabolism | Lipopolysaccharides | Pyrrolidines - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Liver - immunology | Transfection | Liver - drug effects | RNA Interference | Time Factors | Peritonitis - prevention & control | Nuclear Receptor Subfamily 1, Group D, Member 1 - deficiency | Liver Failure, Acute - prevention & control | Chemical and Drug Induced Liver Injury - pathology | Macrophages, Peritoneal - drug effects | Nuclear Receptor Subfamily 1, Group D, Member 1 - metabolism | Disease Models, Animal | Galactosamine | Severity of Illness Index | Chemical and Drug Induced Liver Injury - prevention & control | Genetic Predisposition to Disease | Nuclear Receptor Subfamily 1, Group D, Member 1 - agonists | Nuclear Receptor Subfamily 1, Group D, Member 1 - genetics | Cytokines - metabolism | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Liver - metabolism | Peritonitis - metabolism | Cells, Cultured | Macrophages, Peritoneal - immunology | Thiophenes - pharmacology | Liver Failure, Acute - metabolism | Peritonitis - immunology | Circadian Rhythm | Inflammasomes - genetics | Chemical and Drug Induced Liver Injury - immunology | Macrophage Activation | Mice, Knockout | Phenotype | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammasomes - immunology | Liver Failure, Acute - immunology | Liver Failure, Acute - pathology | Macrophages, Peritoneal - metabolism | Hepatitis | Mitogens | Analysis | Liver | Index Medicus | Abridged Index Medicus | Life Sciences | Human health and pathology | rev-erb-alpha | biological clock | acute liver failure | immune regulation
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2016, Volume 126, Issue 10, pp. 3905 - 3916
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 485, Issue 7396, pp. 123 - 127
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Genotype & phenotype | Rodents | T cell receptors | Gene expression | Inflammatory diseases | Children & youth | Index Medicus | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
Immunity, ISSN 1074-7613, 09/2012, Volume 37, Issue 3, pp. 463 - 474
Natural helper (NH) cells are innate lymphoid cells (ILCs) that produce T helper-2 (Th2)-cell-type cytokines in the lung- and gut-associated lymphoid tissues.... 
PROGENITORS | CYTOKINES | ROR-GAMMA-T | BONE-MARROW | TYPE-2 IMMUNITY | MICE | INNATE LYMPHOID-CELLS | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | Antigens, Ly - genetics | Male | Th2 Cells - immunology | Proto-Oncogene Proteins c-kit - immunology | Interleukin Receptor Common gamma Subunit - immunology | Flow Cytometry | Lymphocytes - immunology | Allergens - immunology | T-Lymphocytes, Helper-Inducer - immunology | Bone Marrow Cells - immunology | Bone Marrow Transplantation | Pneumonia - immunology | Female | Papain - immunology | Proto-Oncogene Proteins c-kit - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Pneumonia - genetics | Lymphocytes - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Nuclear Receptor Subfamily 1, Group F, Member 1 - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Membrane Proteins - immunology | Mice, SCID | Interleukin Receptor Common gamma Subunit - genetics | Th2 Cells - metabolism | Mice, Knockout | Animals | Antigens, Ly - immunology | Mice, Inbred NOD | Mice | Nuclear Receptor Subfamily 1, Group F, Member 1 - genetics | Bone Marrow Cells - metabolism | Cell Lineage - immunology | Allergens | Proteases | Lymphocytes | Oncology, Experimental | Bone marrow | Transplantation | Inflammation | Research | Cancer | Medical research | Lungs | Cytokines | Rodents | Gene expression | Small intestine | Experiments | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 2013, Volume 14, Issue 3, pp. 230 - 237
Regulatory T cells (T-reg cells) develop from progenitor thymocytes after the engagement of T cell antigen receptors (TCRs) with high-affinity ligands, but the... 
COMPLEX | DIFFERENTIAL REQUIREMENT | DENDRITIC CELLS | NUCLEAR RECEPTORS | TRANSCRIPTION FACTOR FOXP3 | IN-VIVO | MOUSE | NUR77 | CLONAL DELETION | IMMUNOLOGY | EXPRESSION | T-Lymphocyte Subsets - immunology | T-Lymphocytes, Regulatory - metabolism | Thymocytes - metabolism | Autoimmunity - genetics | Homeostasis | Nuclear Receptor Subfamily 4, Group A, Member 2 - genetics | T-Lymphocytes, Regulatory - immunology | Receptors, Thyroid Hormone - genetics | RNA Interference | Forkhead Transcription Factors - metabolism | T-Lymphocytes, Regulatory - cytology | Receptors, Antigen, T-Cell - immunology | Cell Differentiation | Promoter Regions, Genetic | DNA-Binding Proteins - physiology | Nerve Tissue Proteins - physiology | Nuclear Receptor Subfamily 4, Group A, Member 1 - genetics | Receptors, Antigen, T-Cell - metabolism | Signal Transduction | Cells, Cultured | DNA-Binding Proteins - genetics | Forkhead Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Receptors, Thyroid Hormone - physiology | Mice, Knockout | Animals | Nuclear Receptor Subfamily 4, Group A, Member 2 - physiology | Receptors, Steroid - genetics | Receptors, Steroid - physiology | T-Lymphocyte Subsets - metabolism | Mice | RNA, Small Interfering | Receptors, Antigen, T-Cell - genetics | Genes, Immediate-Early | Nuclear Receptor Subfamily 4, Group A, Member 1 - physiology | Autoimmunity | Physiological aspects | Genetic aspects | Research | Genetic transcription | T cells | Index Medicus
Journal Article
Journal Article
HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 02/2014, Volume 23, Issue 4, pp. 889 - 905
Primary aldosteronism (PA) is the main cause of secondary hypertension, resulting from adrenal aldosterone-producing adenomas (APA) or bilateral hyperplasia.... 
CANCER-CELLS | FREQUENT EPIGENETIC INACTIVATION | ADRENAL-CORTEX | ZONA GLOMERULOSA | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | KCNJ5 MUTATIONS | BETA-CATENIN | ADRENOCORTICAL TUMORS | SOMATIC MUTATIONS | Adrenal Cortex Neoplasms - complications | Aldosterone - biosynthesis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Hyperaldosteronism - metabolism | Mice, 129 Strain | Nuclear Receptor Subfamily 4, Group A, Member 2 - genetics | Hyperaldosteronism - etiology | Adult | Female | Membrane Proteins - metabolism | Adrenal Cortex Neoplasms - metabolism | Cytochrome P-450 CYP11B2 - metabolism | Wnt Signaling Pathway | Nuclear Receptor Subfamily 4, Group A, Member 1 - genetics | Membrane Proteins - genetics | Down-Regulation | Mice, Inbred C57BL | Aldosterone - blood | Aldosterone - secretion | Adrenocortical Adenoma - metabolism | Mice, Knockout | Animals | Cell Line, Tumor | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Mice | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | Adrenocortical Adenoma - complications | Cytochrome P-450 CYP11B2 - genetics | Index Medicus | Animal genetics | Cytochrome P-450 CYP11B2 | Hyperaldosteronism | Genomics | Nuclear Receptor Subfamily 4, Group A, Member 1 | Nuclear Receptor Subfamily 4, Group A, Member 2 | Aldosterone | Embryology and Organogenesis | Life Sciences | Genetics | Endocrinology and metabolism | Biochemistry, Molecular Biology | Adrenocortical Adenoma | Membrane Proteins | Human health and pathology | Adrenal Cortex Neoplasms | Development Biology | Molecular biology | Cancer
Journal Article