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Cochrane Database of Systematic Reviews, ISSN 1469-493X, 07/2015, Volume 2015, Issue 7, p. CD000371
Journal Article
Journal Article
Journal Article
Cochrane database of systematic reviews (Online), ISSN 1469-493X, 2007, Issue 4, p. CD000371
Journal Article
Journal of Neuroendocrinology, ISSN 0953-8194, 05/2012, Volume 24, Issue 5, pp. 766 - 773
Journal Article
Digestive Diseases and Sciences, ISSN 0163-2116, 01/2016, Volume 61, Issue 1, pp. 198 - 207
The cystic fibrosis (CF) transmembrane conductance regulator (CFTR) gating mutation G551D prevents sufficient ion transport due to reduced channel-open... 
Growth | FECAL ELASTASE-1 | Kalydeco | CFTR POTENTIATOR | Weight gain | CHILDREN | TRANSMEMBRANE CONDUCTANCE REGULATOR | Bicarbonate | FAT MALABSORPTION | ENERGY-EXPENDITURE | THERAPY | Potentiator | DISEASE | QUALITY-OF-LIFE | Cystic fibrosis transmembrane conductance regulator | BICARBONATE SECRETION | Biochemistry, general | Medicine & Public Health | Hepatology | Gastroenterology | Oncology | Transplant Surgery | GASTROENTEROLOGY & HEPATOLOGY | Cystic Fibrosis - physiopathology | Humans | Male | Young Adult | Time Factors | Adult | Female | Surveys and Questionnaires | Weight Gain - drug effects | Child | Genetic Predisposition to Disease | Double-Blind Method | Drug Administration Schedule | Administration, Oral | Cystic Fibrosis - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - agonists | Treatment Outcome | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Membrane Transport Modulators - administration & dosage | Phenotype | Quinolones - administration & dosage | Cystic Fibrosis - genetics | Nutritional Status - drug effects | Adolescent | Cystic Fibrosis - diagnosis | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mutation | Aminophenols - administration & dosage | Cystic Fibrosis - drug therapy | Cystic fibrosis | Care and treatment | Genetic aspects | Carbonates | Body image | Analysis
Journal Article
Nutrients, ISSN 2072-6643, 09/2018, Volume 10, Issue 9, p. 1284
Background: Antihypertensive drugs affect mineral metabolism, inflammation, and the oxidative state. The aim of this study was to evaluate the effects of... 
Antihypertensive pharmacotherapy | Zinc | Mineral metabolism | CATALASE | ANTIHYPERTENSIVE DRUGS | ANTIOXIDANT ENZYMES | COPPER | EXCRETION | CAPTOPRIL | NUTRITION & DIETETICS | mineral metabolism | METABOLISM | antihypertensive pharmacotherapy | RESISTANCE | zinc | EXTRACELLULAR-SUPEROXIDE DISMUTASE | MODULATION | Prospective Studies | Calcium Channel Blockers - adverse effects | Zinc - metabolism | Humans | Middle Aged | Calcium Channel Blockers - therapeutic use | Hair - metabolism | Hypertension - drug therapy | Male | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Time Factors | Angiotensin-Converting Enzyme Inhibitors - adverse effects | Inflammation Mediators - metabolism | Poland | Adult | Female | Blood Pressure - drug effects | Hypertension - diagnosis | Biomarkers - metabolism | Oxidation-Reduction | Treatment Outcome | Antihypertensive Agents - therapeutic use | Hypertension - physiopathology | Antihypertensive Agents - adverse effects | Hypertension - metabolism | Nutritional Status - drug effects | Lipid Metabolism - drug effects | Diuretics - therapeutic use | Aged | Diuretics - adverse effects | Angiotensin-converting enzyme inhibitors | Drugs | Oxidative stress | Calcium antagonists | Calcium | Oxidative metabolism | Erythrocytes | Lipids | Superoxide dismutase | Iron | Antioxidants | Glucose metabolism | Red blood cells | Enzyme inhibitors | Catalase | Peptidyl-dipeptidase A | Drug metabolism | Diuretics | Lipid metabolism | Copper | Drug therapy | Angiotensin II | Hair | Hypertension | Urine | Enzymes | Kidneys | Superoxide | Inflammation | Tumor necrosis factor-α | Metabolism | Patients | Studies | Inhibitors | Converting | Angiotensin | Diabetes | Metabolic disorders
Journal Article
Journal of Molecular Cell Biology, ISSN 1674-2788, 04/2016, Volume 8, Issue 2, pp. 129 - 143
Mitochondria-associated endoplasmic reticulum membranes (MAM) play a key role in mitochondrial dynamics and function and in hepatic insulin action. Whereas... 
MAM | mitochondria dynamics | PP2A | glucose sensing | hepatocytes | pentose phosphate pathway | PROTEIN PHOSPHATASE | HOMEOSTASIS | CARDIOMYOCYTES | GLUCOKINASE | CELL BIOLOGY |