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1. Full Text Polyphenols Protect the Epithelial Barrier Function of Caco‑2 Cells Exposed to Indomethacin through the Modulation of Occludin and Zonula Occludens‑1 Expression
by Carrasco-Pozo, Catalina and Morales, Pamela and Gotteland, Martin
Journal of agricultural and food chemistry, ISSN 0021-8561, 06/2013, Volume 61, Issue 22, pp. 5291 - 5297
polyphenols | occludin | ZO-1 | transepithelial electrical resistance | rotenone | quercetin | tight junction | indomethacin | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Applied | Food Science & Technology | Agriculture, Multidisciplinary | Agriculture | Science & Technology | Fundamental and applied biological sciences. Psychology | Food industries | Control | Protozoa. Invertebrates | Biological control | Biological and medical sciences | Phytopathology. Animal pests. Plant and forest protection | Indomethacin - antagonists & inhibitors | Enterocytes - metabolism | Antioxidants - metabolism | Humans | Stilbenes - analysis | Stilbenes - metabolism | Protein Transport - drug effects | Functional Food - analysis | Occludin - metabolism | Polyphenols - metabolism | Occludin - agonists | Occludin - genetics | Catechin - analysis | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Zonula Occludens-1 Protein - metabolism | Catechin - metabolism | Antioxidants - analysis | Indomethacin - adverse effects | Tight Junctions - drug effects | Caco-2 Cells | Zonula Occludens-1 Protein - agonists | Zonula Occludens-1 Protein - genetics | Permeability - drug effects | Quercetin - analysis | Zonula Occludens-1 Protein - antagonists & inhibitors | Gene Expression Regulation - drug effects | Occludin - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Enterocytes - drug effects | Polyphenols - analysis | Catechin - analogs & derivatives | Quercetin - metabolism | Index Medicus
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2. Full Text Tight junction protein occludin is a porcine epidemic diarrhea virus entry factor
by Luo, Xiaolei and Guo, Longjun and Zhang, Jian and Xu, Yunfei and Gu, Weihong and Feng, Li and Wang, Yue
Journal of virology, ISSN 0022-538X, 05/2017, Volume 91, Issue 10
Tight junction | PEDV | Occludin | Cell Line | Virus Replication - drug effects | Porcine epidemic diarrhea virus - pathogenicity | Occludin - deficiency | Tight Junctions - virology | Cercopithecus aethiops | Cytoplasm - metabolism | Virus Internalization - drug effects | Occludin - metabolism | Occludin - genetics | Tight Junctions - chemistry | Animals | Swine | Epithelial Cells - virology | Porcine epidemic diarrhea virus - physiology | Virus Attachment | Porcine epidemic diarrhea virus - drug effects | RNA, Small Interfering | Vero Cells | Tight Junctions - pathology | Index Medicus
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3. Full Text Completion of the entire hepatitis C virus life cycle in genetically humanized mice
by Dorner, Marcus and Horwitz, Joshua A and Donovan, Bridget M and Labitt, Rachael N and Budell, William C and Friling, Tamar and Vogt, Alexander and Catanese, Maria Teresa and Satoh, Takashi and Kawai, Taro and Akira, Shizuo and Law, Mansun and Rice, Charles M and Ploss, Alexander
Nature (London), ISSN 0028-0836, 2013, Volume 501, Issue 7466, pp. 237 - 241
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cyclophilin A - metabolism | Cell Line | STAT1 Transcription Factor - deficiency | Hepacivirus - immunology | Humans | Mice, Inbred C57BL | Virion - physiology | Hepatitis C - genetics | Mice, Transgenic | Occludin - metabolism | Occludin - genetics | Hepatitis C - immunology | Animals | Genetic Engineering | Viremia - virology | Virus Replication | Hepatitis C - virology | Tetraspanin 28 - metabolism | Hepacivirus - physiology | Cyclophilin A - genetics | Mice | Tetraspanin 28 - genetics | Virion - growth & development | Disease Models, Animal | Genetic aspects | Nucleotide sequencing | Hepatitis C virus | Methods | DNA sequencing | Hepatitis | Infections | Genomes | Rodents | Immune system | Index Medicus
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4. Full Text Defective epithelial barrier in chronic rhinosinusitis: The regulation of tight junctions by IFN-γ and IL-4
by Soyka, Michael B., MD and Wawrzyniak, Paulina, MSc and Eiwegger, Thomas, MD and Holzmann, David, MD and Treis, Angela, MSc and Wanke, Kerstin, MSc and Kast, Jeannette I., BSc and Akdis, Cezmi A., MD
Journal of allergy and clinical immunology, ISSN 0091-6749, 2012, Volume 130, Issue 5, pp. 1087 - 1096.e10
Allergy and Immunology | occludin | Chronic rhinosinusitis | regulation | leaky epithelium | cytokines | tight junctions | claudin | chronic sinusitis | zonula occludens | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Fundamental and applied biological sciences. Psychology | Fundamental immunology | Immunopathology | Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis | Otorhinolaryngology. Stomatology | Non tumoral diseases | Biological and medical sciences | Medical sciences | Upper respiratory tract, upper alimentary tract, paranasal sinuses, salivary glands: diseases, semeiology | Claudin-4 - genetics | Humans | Interleukin-17 - immunology | Occludin - metabolism | Sinusitis - complications | Occludin - genetics | Sinusitis - immunology | Claudin-4 - metabolism | Sinusitis - pathology | Zonula Occludens-1 Protein - metabolism | Zonula Occludens-1 Protein - genetics | Nasal Polyps - immunology | Rhinitis - immunology | Nasal Polyps - pathology | Cells, Cultured | Gene Expression Regulation | Interleukin-4 - immunology | Nasal Polyps - complications | Tight Junctions - immunology | Biopsy | Nasal Mucosa - immunology | Interferon-gamma - immunology | Rhinitis - complications | Primary Cell Culture | Rhinitis - pathology | Chronic Disease | Tight Junctions - pathology | Index Medicus | Abridged Index Medicus
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5. Full Text Disintegration of colonic epithelial tight junction in uremia: A likely cause of CKD-associated inflammation
by Vaziri, Nosratola D and Yuan, Jun and Rahimi, Ardeshir and Ni, Zhenmin and Said, Hyder and Subramanian, Veedamali S
Nephrology, dialysis, transplantation, ISSN 0931-0509, 07/2012, Volume 27, Issue 7, pp. 2686 - 2693
occludin | gastrointestinal tract | intestinal barrier defect | end-stage renal disease | claudin | Transplantation | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Glomerulonephritis | Emergency and intensive care: renal failure. Dialysis management | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Nephropathies. Renovascular diseases. Renal failure | Nephrology. Urinary tract diseases | Biological and medical sciences | Intensive care medicine | Medical sciences | Inflammation - pathology | Epithelial Cells - metabolism | Tight Junction Proteins - genetics | Male | Uremia - metabolism | Renal Insufficiency, Chronic - complications | Immunoenzyme Techniques | Occludin - metabolism | Occludin - genetics | Renal Insufficiency, Chronic - metabolism | Inflammation - metabolism | Claudin-1 - metabolism | Zonula Occludens-1 Protein - metabolism | Real-Time Polymerase Chain Reaction | Tight Junction Proteins - metabolism | Tight Junctions - metabolism | Zonula Occludens-1 Protein - genetics | Colon - pathology | RNA, Messenger - genetics | Rats | Epithelial Cells - pathology | Uremia - complications | Inflammation - etiology | Rats, Sprague-Dawley | Blotting, Western | Colon - metabolism | Renal Insufficiency, Chronic - pathology | Animals | Claudin-1 - genetics | Protein Processing, Post-Translational | Index Medicus | Original
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6. Full Text Contribution of Tight Junction Proteins to Ion, Macromolecule, and Water Barrier in Keratinocytes
by Kirschner, Nina and Rosenthal, Rita and Furuse, Mikio and Moll, Ingrid and Fromm, Michael and Brandner, Johanna M
Journal of investigative dermatology, ISSN 0022-202X, 05/2013, Volume 133, Issue 5, pp. 1161 - 1169
Life Sciences & Biomedicine | Dermatology | Science & Technology | Tight Junction Proteins - genetics | Occludin - deficiency | Zonula Occludens-1 Protein - physiology | Claudin-4 - genetics | Male | Tight Junction Proteins - physiology | Claudin-4 - physiology | Occludin - genetics | Claudin-4 - deficiency | Tight Junctions - physiology | Claudin-1 - physiology | Models, Animal | Zonula Occludens-1 Protein - deficiency | Zonula Occludens-1 Protein - genetics | Cells, Cultured | Tight Junction Proteins - deficiency | Water - metabolism | Keratinocytes - cytology | Ions - metabolism | Claudin-1 - deficiency | Mice, Knockout | Animals | Cell Membrane Permeability - physiology | Keratinocytes - metabolism | Claudin-1 - genetics | Occludin - physiology | Mice | Macromolecular Substances - metabolism | Index Medicus
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7. Full Text Bradykinin increased the permeability of BTB via NOS/NO/ZONAB-mediating down-regulation of claudin-5 and occludin
by Liu, Li-bo and Liu, Xiao-bai and Ma, Jun and Liu, Yun-hui and Li, Zhi-qing and Ma, Teng and Zhao, Xi-he and Xi, Zhuo and Xue, Yi-xue
Biochemical and biophysical research communications, ISSN 0006-291X, 08/2015, Volume 464, Issue 1, pp. 118 - 125
Claudin-5 | NOS | ZONAB | Bradykinin | Blood-tumor barrier | Occludin | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Rats, Wistar | Brain Neoplasms - pathology | Claudin-5 - metabolism | Gene Expression Regulation, Neoplastic | Brain Neoplasms - metabolism | Occludin - metabolism | Glioma - metabolism | Glioma - genetics | Occludin - genetics | Glioma - pathology | Claudin-5 - antagonists & inhibitors | Female | Nitric Oxide Synthase Type III - metabolism | Bradykinin - pharmacology | Tight Junctions - drug effects | NG-Nitroarginine Methyl Ester - pharmacology | Tight Junctions - metabolism | Promoter Regions, Genetic | Permeability - drug effects | Vasodilator Agents - pharmacology | Signal Transduction | Enzyme Inhibitors - pharmacology | Brain Neoplasms - genetics | Rats | Claudin-5 - genetics | Transcription Factors - genetics | Blood-Brain Barrier - drug effects | Nitric Oxide Synthase Type III - genetics | Protein Transport | Occludin - antagonists & inhibitors | Transcription Factors - metabolism | Animals | Nitric Oxide Synthase Type I - metabolism | Protein Binding | Nitric Oxide Synthase Type I - genetics | Nitric Oxide - metabolism | Permeability | Neurophysiology | Index Medicus
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8. Full Text Early Porcine Sapovirus Infection Disrupts Tight Junctions and Uses Occludin as a Coreceptor
by Alfajaro, Mia Madel and Cho, Eun-Hyo and Kim, Deok-Song and Kim, Ji-Yun and Park, Jun-Gyu and Soliman, Mahmoud and Baek, Yeong-Bin and Park, Chul-Ho and Park, Chul-Ho and Kang, Mun-Il and Park, Sang-Ik and Cho, Kyoung-Oh and Cho, Kyoung-Oh
Journal of virology, ISSN 0022-538X, 02/2019, Volume 93, Issue 4
cellular coreceptor | occludin | sapovirus | tight junction | Life Sciences & Biomedicine | Science & Technology | Virology | Tight Junctions - metabolism | Cricetulus | Sapovirus - metabolism | Tight Junctions - virology | Virus Diseases - metabolism | Gastroenteritis - virology | Sapovirus - physiology | Endosomes - metabolism | Occludin - metabolism | LLC-PK1 Cells | Animals | Epithelial Cells - virology | Occludin - physiology | Sapovirus - pathogenicity | Swine - virology | CHO Cells | Index Medicus
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9. Full Text Inflammation-induced Occludin Downregulation Limits Epithelial Apoptosis by Suppressing Caspase-3 Expression
by Kuo, Wei-Ting and Shen, Le and Zuo, Li and Shashikanth, Nitesh and Ong, Ma.Lora Drizella M and Wu, Licheng and Zha, Juanmin and Edelblum, Karen L and Wang, Yitang and Wang, Yingmin and Nilsen, Steven P and Turner, Jerrold R
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 11/2019, Volume 157, Issue 5, pp. 1323 - 1337
Gene Regulation | IBD | Permeability | Cell Death | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Dextran Sulfate | Colitis, Ulcerative - enzymology | Colon - enzymology | Occludin - deficiency | Colitis - genetics | Humans | Caspase 3 - metabolism | Colitis - pathology | Trinitrobenzenesulfonic Acid | Case-Control Studies | Caspase 3 - deficiency | Occludin - metabolism | Occludin - genetics | Colitis - chemically induced | Intestinal Mucosa - enzymology | Caspase 3 - genetics | Zonula Occludens-1 Protein - metabolism | Disease Models, Animal | Caco-2 Cells | Crohn Disease - enzymology | Zonula Occludens-1 Protein - genetics | Signal Transduction | Colon - pathology | Mice, Inbred C57BL | Epithelial Cells - pathology | Colitis, Ulcerative - pathology | Mice, Knockout | Animals | Colitis - enzymology | Crohn Disease - pathology | Epithelial Cells - enzymology | Apoptosis | Intestinal Mucosa - pathology | Medical research | Analysis | Medicine, Experimental | Genetic engineering | Inflammation | Ulcerative colitis | Index Medicus | Abridged Index Medicus | cell death | gene regulation | survival
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10. Full Text Monoclonal antibodies against occludin completely prevented hepatitis C virus infection in a mouse model
by Shimizu, Yoshimi and Shirasago, Yoshitaka and Kondoh, Masuo and Suzuki, Tetsuro and Wakita, Takaji and Hanada, Kentaro and Yagi, Kiyohito and Fukasawa, Masayoshi
Journal of virology, ISSN 0022-538X, 04/2018, Volume 92, Issue 8, p. e02258-17
Monoclonal antibodies | Antiviral agents | Hepatitis C virus | Occludin | Life Sciences & Biomedicine | Science & Technology | Virology | Liver Neoplasms - prevention & control | Liver Neoplasms - virology | Hepacivirus - isolation & purification | Antiviral Agents - pharmacology | Rats, Wistar | Antibodies, Monoclonal - pharmacology | Carcinoma, Hepatocellular - prevention & control | Humans | Male | Carcinoma, Hepatocellular - virology | Virus Internalization | Occludin - antagonists & inhibitors | Animals | Occludin - immunology | Tight Junctions | Hepatitis C - virology | Mice | Tumor Cells, Cultured | Hepatitis C - prevention & control | Disease Models, Animal | Index Medicus
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