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1. Full Text Integrated microRNA–mRNA profiling identifies oncostatinm as a marker of mesenchymal-like ER-negative/HER2-negative breast cancer
by Bottai, Giulia and Diao, Lixia and Baggerly, Keith A and Paladini, Laura and Győrffy, Balázs and Raschioni, Carlotta and Pusztai, Lajos and Calin, George A and Santarpia, Libero
International Journal of Molecular Sciences, ISSN 1661-6596, 01/2017, Volume 18, Issue 1
Molecular subtypes | Immune response | MicroRNAs | Breast cancer | Oncostatin M
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2. Full Text Oncostatin M is overexpressed in skin squamous-cell carcinoma and promotes tumor progression
by Simonneau, Marie and Frouin, Eric and Huguier, Vincent and Jermidi, Cynthia and Jégou, Jean François and Godet, Julie and Barra, Anne and Paris, Isabelle and Levillain, Pierre and Cordier-Dirikoc, Sevda and Pedretti, Nathalie and Bernard, François Xavier and Lecron, Jean Claude and Morel, Franck and Favot, Laure
Oncotarget, ISSN 1949-2553, 11/2018, Volume 9, Issue 92, pp. 36457 - 36473
Cutaneous squamous cell carcinoma (cSCC) is the second most common keratinocyte malignancy and accounts for 20% of skin cancer deaths. Cancer is closely... 
Tumor microenvironment | Oncostatin M | Immunotherapy | Cytokine | Skin cancer
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3. Full Text Oncostatin M, a muscle-secreted myokine, recovers high-glucose-induced impairment of Akt phosphorylation by Fos induction in hippocampal neuron cells
by Won Seok Hyung, William and Gon Lee, Sung and Tae Kim, Keun and Soo Kim, Hyeon
NeuroReport, ISSN 0959-4965, 08/2019, Volume 30, Issue 11, pp. 765 - 770
Oncostatin M is a muscle-secreted myokine that has various effects on neuronal function, however, the underlying molecular mechanism has been poorly defined.... 
high-glucose | PATHOGENESIS | ALZHEIMERS-DISEASE | INSULIN-RESISTANCE | c-Fos | Akt | Oncostatin M | INTERLEUKIN-6 | NEUROSCIENCES | EXERCISE | ERK
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4. Full Text Correction: Oncostatin M Suppresses Activation of IL-17/Th17 via SOCS3 Regulation in CD4 + T Cells
by Son, Hye-Jin and Lee, Seung Hoon and Lee, Seon-Yeong and Kim, Eun-Kyung and Yang, Eun-Ji and Kim, Jae-Kyung and Seo, Hyeon-Beom and Park, Sung-Hwan and Cho, Mi-La
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 06/2017, Volume 198, Issue 12, pp. 4879 - 4879
Cell activation | Lymphocytes | Helper cells | Lymphocytes T | Oncostatin M | Interleukin 17 | CD4 antigen
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5. Full Text Oncostatin M exerts a protective effect against excessive scarring by counteracting the inductive effect of TGFβ1 on fibrosis markers
by Huguier, Vincent and Giot, Jean-Philippe and Simonneau, Marie and Levillain, Pierre and Charreau, Sandrine and Garcia, Martine and Jégou, Jean-François and Bodet, Charles and Morel, Franck and Lecron, Jean-Claude and Favot, Laure
Scientific reports, ISSN 2045-2322, 02/2019, Volume 9, Issue 1, pp. 2113 - 10
Wound healing is a complex physiological process that repairs a skin lesion and produces fibrous tissue. In some cases, this process can lead to hypertrophic... 
Phenotypes | Collagen (type I) | Scars | Wound healing | Secretion | Fibrosis | Fibroblasts | Extracellular matrix | Skin diseases | Inflammation | Oncostatin M | Fibronectin
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6. Oncostatin M treatment increases the responsiveness toward cisplatin‑based chemoradiotherapy in cervical cancer cells in a STAT3‑dependent mannerrussalina stroeder1
by Stroeder, Russalina and Walch-Rückheim, Barbara and Fischbach, Jil and Juhasz-Böss, Ingolf and Rübe, Christian and Solomayer, Erich-Franz and Smola, Sigrun
Oncology Letters, ISSN 1792-1074, 09/2018, Volume 16, Issue 3, pp. 3351 - 3358
Chemotherapy | Human papillomavirus | Cervical carcinogenesis | Oncostatin M | Radiotherapy
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7. Oncostatin M combined with dacarbazine inhibits proliferation of melanoma cell B16
by Ye, Heng and Qi, Chunjian and Qian, Keqing
Chinese Journal of Clinical Oncology, ISSN 1000-8179, 12/2013, Volume 40, Issue 24, pp. 1540 - 1543
Melanoma | DTIC | Oncostatin M
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8. Full Text HIGH expression of OSM and IL-6 are associated with decreased breast cancer survival: Synergistic induction of IL-6 secretion by OSM and IL-1β
by Tawara, Ken and Scott, Hannah and Emathinger, Jacqueline and Wolf, Cody and LaJoie, Dollie and Hedeen, Danielle and Bond, Laura and Montgomery, Paul and Jorcyk, Cheryl
Oncotarget, ISSN 1949-2553, 2019, Volume 10, Issue 21, pp. 2068 - 2085
Chronic inflammation has been recognized as a risk factor for the development and maintenance of malignant disease. Cytokines such as interleukin-6 (IL-6),... 
Breast cancer | Metastasis | Oncostatin M | Interleukin-1β | Interleukin-6 and oncostatin M
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9. Full Text CD169+ macrophages are critical for osteoblast maintenance and promote intramembranous and endochondral ossification during bone repair
by Batoon, Lena and Millard, Susan Marie and Wullschleger, Martin Eduard and Preda, Corina and Wu, Andy Chiu-Ku and Kaur, Simranpreet and Tseng, Hsu-Wen and Hume, David Arthur and Levesque, Jean-Pierre and Raggatt, Liza Jane and Pettit, Allison Robyn
Biomaterials, ISSN 0142-9612, 03/2019, Volume 196, pp. 51 - 66
Osteal macrophages (osteomacs) contribute to bone homeostasis and regeneration. To further distinguish their functions from osteoclasts, which share many... 
Osteoblast | Bone formation | Osteomac | Fracture repair | Bone regeneration | Macrophage | ONCOSTATIN M | CELLS | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | MODEL | INTERNAL FIXATOR | IN-VITRO | NUCLEAR-FACTOR | RECEPTOR ACTIVATOR | MARROW MACROPHAGES | OSTEOCLAST DIFFERENTIATION
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10. Full Text Roseburia intestinalis inhibits oncostatin M and maintains tight junction integrity in a murine model of acute experimental colitis
by Tan, Bei and Luo, Weiwei and Shen, Zhaohua and Xiao, Mengwei and Wu, Shuai and Meng, Xiangrui and Wu, Xing and Yang, Zhenyu and Tian, Li and Wang, Xiaoyan
Scandinavian Journal of Gastroenterology, ISSN 0036-5521, 04/2019, Volume 54, Issue 4, pp. 432 - 440
Objective: Levels of oncostatin M (OSM) and the composition of gut microbiota predict responses to anti-TNF agents used for IBD therapy. Here, the aim was to... 
anti-TNF therapy | Microbiota | colitis | oncostatin M | tight junction | EPITHELIAL BARRIER | THERAPY | INFLAMMATION | MACROPHAGES | DISEASE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION
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11. Full Text UThe Primary Function of gp130 Signaling in Osteoblasts Is To Maintain Bone Formation and Strength, Rather Than Promote Osteoclast Formation
by Johnson, RW and Brennan, HJ and Vrahnas, C and Poulton, IJ and McGregor, NE and Standal, T and Walker, EC and Koh, TT and Nguyen, H and Walsh, NC and Forwood, MR and Martin, TJ and Sims, NA
JOURNAL OF BONE AND MINERAL RESEARCH, ISSN 0884-0431, 06/2014, Volume 29, Issue 6, pp. 1492 - 1505
Interleukin-6 (IL-6) family cytokines act via gp130 in the osteoblast lineage to stimulate the formation of osteoclasts (bone resorbing cells) and the activity... 
CYTOKINES | ENDOCRINE PATHWAYS | RECEPTOR | LEUKEMIA INHIBITORY FACTOR | INTERLEUKIN-6 | MECHANICAL-PROPERTIES | TISSUE SIGNALING | BONE MATRIX | MATRIX MINERALIZATION | ONCOSTATIN-M | OSTEOBLASTS | IN-VIVO | ENDOCRINOLOGY & METABOLISM | MICE | DIFFERENTIATION | CELL | EXPRESSION
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12. Full Text Invariant NKT cells modulate the suppressive activity of IL-10-secreting neutrophils differentiated with serum amyloid A
by Jones, Margaret and Booth, Sarah and Salio, Mariolina and Arscott, Ramon and Karydis, Ioannis and Asher, Ruth and De Santo, Carmela and Cerundolo, Vincenzo and Middleton, Mark
Nature Immunology, ISSN 1529-2908, 11/2010, Volume 11, Issue 11, pp. 1039 - 1046
Neutrophils are the main effector cells during inflammation, but they can also control excessive inflammatory responses by secreting anti-inflammatory... 
CD1D TETRAMERS | RESPONSES | ACTIVATION | RECEPTOR 2 | ROR-GAMMA-T | DENDRITIC CELLS | TUMOR PROGRESSION | ONCOSTATIN-M | IN-VIVO | IMMUNOLOGY | CUTTING EDGE | Neutrophils - cytology | Humans | Mice, Inbred C57BL | Neutrophils - immunology | Serum Amyloid A Protein - immunology | Cell Differentiation - immunology | Animals | Melanoma - immunology | Cell Line, Tumor | Female | Mice | Interleukin-10 - immunology | Natural Killer T-Cells - immunology | Physiological aspects | Immune response | Interleukins | T cells | Health aspects | Melanoma | Index Medicus
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13. Full Text Revisiting the Clinical Scoring System for the Prognosis of Chronic Rhinosinusitis with Nasal Polyps
by Kim, Jin Youp and Han, Young Eun and Seo, Yuju and Choe, Goun and Kim, Min Kyung and Huh, Gene and Cho, Deuktae and Yang, Seung Koo and Kang, Seung Heon and Kim, Dae Woo
Yonsei medical journal, ISSN 0513-5796, 06/2019, Volume 60, Issue 6, pp. 578 - 584
Purpose: To evaluate the Japanese Epidemiological Survey of Refractory Eosinophilic Chronic Rhinosinusitis (JESREC) classification, a clinical scoring system,... 
eosinophils | neutrophils | prognosis | Sinusitis | ONCOSTATIN M | MEDICINE, GENERAL & INTERNAL | INFLAMMATION | ENDOTYPES | ASTHMA | Index Medicus | Original
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14. Full Text ROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and Stroma
by Sanz-Moreno, Victoria and Gaggioli, Cedric and Yeo, Maggie and Albrengues, Jean and Wallberg, Fredrik and Viros, Amaya and Hooper, Steven and Mitter, Richard and Féral, Chloé C and Cook, Martin and Larkin, James and Marais, Richard and Meneguzzi, Guerrino and Sahai, Erik and Marshall, Chris J
Cancer Cell, ISSN 1535-6108, 2011, Volume 20, Issue 2, pp. 229 - 245
Proinflammatory cytokines are frequently observed in the tumor microenvironment, and chronic inflammation is involved in cancer initiation and progression. We... 
CARCINOMA-CELLS | STAT3 ACTIVATION | JAK/STAT PATHWAY | EPITHELIAL-CELLS | ONCOLOGY | JANUS KINASES | MYELOMA CELLS | COLITIS-ASSOCIATED CANCER | ONCOSTATIN-M-RECEPTOR | KINASE INHIBITOR | GROWTH-FACTOR | CELL BIOLOGY | Melanoma - metabolism | Neoplasms - metabolism | Signal Transduction | Stromal Cells - pathology | Humans | Stromal Cells - metabolism | Melanoma - pathology | Actomyosin - metabolism | Janus Kinase 1 - metabolism | rho-Associated Kinases - metabolism | Neoplasms - pathology | STAT3 Transcription Factor - metabolism | Cell Movement | Squamous cell carcinoma | Oncology, Experimental | Melanoma | Development and progression | Inflammation | Research | Cancer | Index Medicus
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15. Full Text Cardiotrophin-1 is an anti-inflammatory cytokine and promotes IL-4-induced M2 macrophage polarization
by Carneros, David and Santamaría, Eva M and Larequi, Eduardo and Vélez-Ortiz, Jose Miguel and Reboredo, Mercedes and Mancheño, Uxua and Perugorria, María Jesús and Navas, Plácido and Romero-Gómez, Manuel and Prieto, Jesús and Hervás-Stubbs, Sandra and Bustos, Matilde
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 06/2019, Volume 33, Issue 6, pp. 7578 - 7587
Macrophages play a central role in tissue remodeling, repair, and resolution of inflammation. Macrophage polarization to M1 or M2 activation status may... 
gp130 | IL-6 | interleukin-6 | STAT3 | ONCOSTATIN M | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | ENDOTOXEMIA | RECEPTOR | MECHANISMS | CELL BIOLOGY | BIOLOGY | MICE | EXPRESSION | ALTERNATIVE ACTIVATION
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16. Full Text Overexpression of OSM and IL-6 impacts the polarization of profibrotic macrophages and the development of bleomycin-induced lung fibrosis
by Ayaub, EA and Dubey, A and Imani, J and Botelho, F and Kolb, MRJ and Richards, CD and Ask, K
SCIENTIFIC REPORTS, ISSN 2045-2322, 10/2017, Volume 7
Although recent evidence indicates that gp130 cytokines, Oncostatin M (OSM) and IL-6 are involved in alternative programming of macrophages, their role in lung... 
ALTERNATIVELY ACTIVATED MACROPHAGES | ONCOSTATIN-M | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | IN-VIVO | HUMAN FIBROBLASTS | MICE | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION | ACUTE EXACERBATION
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17. Full Text Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor–neutralizing therapy in patients with inflammatory bowel disease
by West, Nathaniel R and West, Nathaniel R and Hegazy, Ahmed N and Hegazy, Ahmed N and Owens, Benjamin M J and Owens, Benjamin M. J and Bullers, Samuel J and Bullers, Samuel J and Linggi, Bryan and Linggi, Bryan and Buonocore, Sofia and Buonocore, Sofia and Coccia, Margherita and Coccia, Margherita and Görtz, Dieter and Görtz, Dieter and This, Sébastien and This, Sébastien and Stockenhuber, Krista and Stockenhuber, Krista and Pott, Johanna and Pott, Johanna and Friedrich, Matthias and Friedrich, Matthias and Ryzhakov, Grigory and Ryzhakov, Grigory and Baribaud, Frédéric and Baribaud, Frédéric and Brodmerkel, Carrie and Brodmerkel, Carrie and Cieluch, Constanze and Cieluch, Constanze and Rahman, Nahid and Rahman, Nahid and Müller-Newen, Gerhard and Müller-Newen, Gerhard and Owens, Raymond J and Owens, Raymond J and Kühl, Anja A and Kühl, Anja A and Maloy, Kevin J and Maloy, Kevin J and Plevy, Scott E and Plevy, Scott E and Keshav, Satish and Travis, Simon P. L and Keshav, Satish and Powrie, Fiona and Travis, Simon P L and Powrie, Fiona and Bailey, Adam and Barnes, Ellie and Bird-Lieberman, Beth and Brain, Oliver and Braden, Barbara and Collier, Jane and East, James and Howarth, Lucy and Klenerman, Paul and Leedham, Simon and Palmer, Rebecca and Rodrigues, Astor and Simmons, Alison and Sullivan, Peter and Uhlig, Holm and Oxford IBD Cohort Investigators
Nature Medicine, ISSN 1078-8956, 05/2017, Volume 23, Issue 5, pp. 579 - 589
Inflammatory bowel diseases (IBD), including Crohn's disease (CD) and ulcerative colitis (UC), are complex chronic inflammatory conditions of the... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | ULCERATIVE-COLITIS | MICROBIOME | IBD | INDUCTION | CELL BIOLOGY | MAINTENANCE | IN-VITRO | DESTRUCTION | GENE-EXPRESSION | STROMAL CELLS | SKIN INFLAMMATION | Immunohistochemistry | Colitis - genetics | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Antibodies, Monoclonal - therapeutic use | Immunoblotting | Male | Gene Expression Profiling | Infliximab - therapeutic use | Inflammatory Bowel Diseases - metabolism | Case-Control Studies | Young Adult | Flow Cytometry | Inflammatory Bowel Diseases - genetics | Oncostatin M - metabolism | Adult | Female | Oncostatin M Receptor beta Subunit - metabolism | Oncostatin M Receptor beta Subunit - genetics | Colitis - immunology | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Inflammatory Bowel Diseases - drug therapy | Intercellular Adhesion Molecule-1 - immunology | Inflammation | Mice, Knockout | Animals | Gastrointestinal Agents - therapeutic use | Interleukin-6 - immunology | Aged | Mice | Oncostatin M Receptor beta Subunit - immunology | Oncostatin M - immunology | Chemokines | Oncostatin M - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
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18. Full Text Pulmonary expression of Oncostatin M (OSM) promotes inducible-BALT formation independently of IL-6, despite a role for IL-6 in OSM-driven pulmonary inflammation6
by Botelho, Fernando M and Rangel-Moreno, Javier and Fritz, Dominik and Randall, Troy D and Xing, Zhou and Richards, Carl D
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 8/2013, Volume 191, Issue 3, pp. 1453 - 1464
Inducible Bronchus-Associated Lymphoid Tissue (iBALT) is associated with immune responses to respiratory infections as well as with local pathology derived... 
lung | Oncostatin M | iBALT | interleukin-6
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19. Full Text Neutrophils are a major source of the epithelial barrier disrupting cytokine Oncostatin M in mucosal airways disease
by Pothoven, Kathryn L., PhD and Norton, James E., MSc and Suh, Lydia A., BSc and Carter, Roderick G., BSc and Harris, Kathleen E., BSc and Biyasheva, Assel, PhD and Welch, Kevin, MD and Shintani-Smith, Stephanie, MD and Conley, David B., MD and Liu, Mark C., MD and Kato, Atsushi, PhD and Avila, Pedro C., MD and Hamid, Qutayba, MD, PhD and Grammer, Leslie C., MD and Peters, Anju T., MD and Kern, Robert C., MD and Tan, Bruce K., MD and Schleimer, Robert P., PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 6, pp. 1966 - 1978.e9
Abstract Background We have previously shown that Oncostatin M (OSM) is elevated in nasal polyps of chronic rhinosinusitis (CRS) patients, as well as in... 
Allergy and Immunology | Atopic asthma | Epithelial barrier | Granulocyte-Monocyte Colony Stimulating Factor | Chronic rhinosinusitis | Oncostatin M | Neutrophils
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