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Atherosclerosis, ISSN 0021-9150, 2014, Volume 236, Issue 2, pp. 327 - 337
Abstract Atherosclerosis is a chronic inflammatory process of the vessel wall with systemic correlates. It is now well established that patients' outcome is... 
Cardiovascular | Osteopontin | Coronary artery disease | Atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | ACCELERATED ATHEROSCLEROSIS | PLASMA OSTEOPONTIN | MESSENGER-RNA | THROMBIN-CLEAVED OSTEOPONTIN | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | UP-REGULATION | CORONARY-ARTERY-DISEASE | DENSITY-LIPOPROTEIN INCREASES | Osteopontin - blood | Osteopontin - genetics | Smoking - adverse effects | Osteopontin - deficiency | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Renal Insufficiency, Chronic - complications | Molecular Targeted Therapy | Coronary Artery Disease - blood | Osteopontin - secretion | Peptide Fragments - physiology | Renal Insufficiency, Chronic - metabolism | Atherosclerosis - etiology | Plaque, Atherosclerotic - pathology | Osteopontin - physiology | Integrins - physiology | Diabetic Angiopathies - metabolism | Atherosclerosis - physiopathology | Risk Factors | Cells, Cultured | Rats | Mice, Transgenic | Inflammation | Calcinosis - blood | Protein Isoforms - physiology | Mice, Knockout | Atherosclerosis - blood | Smoking - metabolism | Animals | Coronary Artery Disease - complications | Biomarkers | Calcinosis - prevention & control | Mice | Protein Processing, Post-Translational | Matrix Metalloproteinases - metabolism | Thrombin - metabolism | Blood circulation disorders | Analysis | Integrins
Journal Article
Journal Article
Expert Opinion on Therapeutic Targets, ISSN 1472-8222, 08/2014, Volume 18, Issue 8, pp. 883 - 895
Journal Article
Journal Article
Gut, ISSN 0017-5749, 06/2017, Volume 66, Issue 6, pp. 1123 - 1137
ObjectiveLiver fibrosis is associated with significant collagen-I deposition largely produced by activated hepatic stellate cells (HSCs); yet, the link between... 
Signaling | Hepatic Stellate Cell | Hepatocyte | Basic Sciences | Hepatic Fibrosis | FIBROSIS | RELEASE | HEPATITIS | GASTROENTEROLOGY & HEPATOLOGY | CHROMATIN PROTEIN HMGB1 | Osteopontin - genetics | Carbon Tetrachloride | Humans | NADPH Oxidases - metabolism | Hepatic Stellate Cells - metabolism | Osteopontin - metabolism | Hepatitis C, Chronic - complications | Case-Control Studies | HMGB1 Protein - genetics | HMGB1 Protein - metabolism | Liver Cirrhosis - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Antibodies, Neutralizing | Cytoplasm - chemistry | Liver Cirrhosis - etiology | Gene Expression | Collagen Type I - metabolism | HMGB1 Protein - analysis | Signal Transduction | Receptor for Advanced Glycation End Products - metabolism | Cells, Cultured | Mice, Transgenic | Recombinant Proteins - pharmacology | Disease Progression | Mice, Knockout | Acetylation - drug effects | Animals | Liver Cirrhosis - pathology | Cell Nucleus - chemistry | Hepatocytes - chemistry | Mice | Histone Deacetylase 2 - metabolism | Osteopontin - analysis | Histone Deacetylase 1 - metabolism | Ubiquitin | Cellular signal transduction | Liver diseases | Research | Pathogenesis | Liver | Carbon tetrachloride | Homeostasis | Mobility | Experiments | HMGB1 protein | NAD(P)H oxidase | Proteins | Hepatitis | Signal transduction | Ischemia | Analgesics | Transgenic animals | Hepatology | Rodents | Extracellular matrix | Acetylation | Explants | Stellate cells | Advanced glycosylation end products | Osteopontin | Glycosylation | 1-Phosphatidylinositol 3-kinase | Hepatocytes | Collagen | Fibrosis | Laboratory animals | Hepatitis C | SIGNALING | HEPATIC STELLATE CELL | HEPATOCYTE | HEPATIC FIBROSIS | BASIC SCIENCES
Journal Article
PloS one, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0198790
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 04/2017, Volume 77, Issue 8, pp. 2018 - 2028
Lung cancers with activating KRAS mutations are characterized by treatment resistance and poor prognosis. In particular, the basis for their resistance to... 
SELECTIVE-INHIBITION | GROWTH-FACTOR RECEPTOR | CELLS | METASTASIS | ONCOLOGY | RAS | RADIORESISTANCE | DNA-DAMAGE | KINASE | RADIOTHERAPY | PROMOTES | Osteopontin - genetics | Carcinoma, Non-Small-Cell Lung - radiotherapy | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - radiotherapy | Lung Neoplasms - pathology | Male | Osteopontin - metabolism | Receptor, Epidermal Growth Factor - metabolism | Heterografts | Neoplastic Stem Cells - metabolism | Osteopontin - biosynthesis | Neoplastic Stem Cells - pathology | Female | Neoplastic Stem Cells - radiation effects | Proto-Oncogene Proteins p21(ras) - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | A549 Cells | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Radiation Tolerance - genetics | Animals | Mice, Nude | Mice | Mutation | Radiation effects | Regulators | Chromatin | Target recognition | Epidermal growth factor receptors | Mitosis | Invasiveness | p53 Protein | Lung cancer | DNA damage | Radiation | Osteopontin | Radiation therapy | Cancer therapies | K-Ras protein | Genotype & phenotype | Properties (attributes) | Radiation tolerance | Biocompatibility | Deoxyribonucleic acid--DNA | Cancer | SPP1 | KRAS | Lung Cancer | EGFR
Journal Article
The lancet oncology, ISSN 1470-2045, 2008, Volume 9, Issue 4, pp. 342 - 351
Summary Background Expression of intrinsic markers of tumour hypoxia and angiogenesis are important predictors of radiotherapeutic, and possibly surgical,... 
Hematology, Oncology and Palliative Medicine | PLASMA OSTEOPONTIN | ONCOLOGY | CONFORMAL RADIOTHERAPY | PHASE-III | PATHOLOGICAL STAGE | MICROVESSEL DENSITY | PROGNOSTIC-SIGNIFICANCE | EXPRESSION | NECK-CANCER | ENDOTHELIAL GROWTH-FACTOR | ANDROGEN WITHDRAWAL | Immunohistochemistry | Osteopontin - genetics | Prostatectomy - methods | Prostatic Neoplasms - radiotherapy | Prognosis | Humans | Middle Aged | Male | Reference Values | Osteopontin - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Hypoxia-Inducible Factor 1 - metabolism | Hypoxia-Inducible Factor 1 - genetics | Cell Hypoxia | Aged, 80 and over | Retrospective Studies | Prostatic Neoplasms - pathology | Prostatic Neoplasms - surgery | Risk Assessment | Biomarkers, Tumor - analysis | Proportional Hazards Models | Probability | Treatment Outcome | Prostate-Specific Antigen - blood | Prostatic Neoplasms - mortality | Randomized Controlled Trials as Topic | Survival Analysis | Neovascularization, Pathologic - genetics | Aged | Biomarkers, Tumor - genetics | Neovascularization, Pathologic - metabolism | Biopsy, Needle | Neoplasm Staging | Cohort Studies | Radiotherapy, Conformal - methods | Care and treatment | Analysis | Surgery | Clinical trials | Neovascularization | Radiotherapy | Prostate cancer | Tumors | Cancer
Journal Article