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Gynecologic Oncology, ISSN 0090-8258, 2012, Volume 128, Issue 2, pp. 233 - 238
Journal Article
PLoS medicine, ISSN 1549-1676, 2008, Volume 5, Issue 12, pp. e232 - 1760
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 1280 - 15
Understanding the intrinsic mediators that render CD8(+) T cells dysfunctional in the tumor microenvironment is a requirement to develop more effective cancer... 
MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | KINASE | CHOP | ENDOPLASMIC-RETICULUM STRESS | DIFFERENTIATION | TRANSLATION | SUPPRESSION | EXPRESSION | CONTRIBUTES | CANCER-IMMUNITY | RNA, Small Interfering - genetics | Transcription Factor CHOP - genetics | CD8-Positive T-Lymphocytes - pathology | Humans | eIF-2 Kinase - immunology | T-Box Domain Proteins - immunology | Adoptive Transfer | Ovarian Neoplasms - mortality | Endoplasmic Reticulum Stress - genetics | Activating Transcription Factor 4 - immunology | Ovarian Neoplasms - genetics | Tumor Microenvironment - genetics | Transcription Factor CHOP - immunology | Female | Carcinoma, Ovarian Epithelial - therapy | eIF-2 Kinase - genetics | Carcinoma, Ovarian Epithelial - genetics | Activating Transcription Factor 4 - antagonists & inhibitors | Carcinoma, Ovarian Epithelial - mortality | Activating Transcription Factor 4 - genetics | Gene Expression Regulation, Neoplastic - immunology | Transcription Factor CHOP - antagonists & inhibitors | T-Box Domain Proteins - genetics | eIF-2 Kinase - antagonists & inhibitors | Mice, Knockout | Tumor Microenvironment - immunology | Xenograft Model Antitumor Assays | Carcinoma, Ovarian Epithelial - immunology | Animals | Lymphocytes, Tumor-Infiltrating - pathology | Survival Analysis | Cell Line, Tumor | Ovarian Neoplasms - therapy | Mice | CD8-Positive T-Lymphocytes - immunology | Immunity, Cellular | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | RNA, Small Interfering - metabolism | Ovarian carcinoma | CD8 antigen | Homology | Lymphocytes T | CCAAT/enhancer-binding protein | Immunity | Immunity (cell-mediated) | Stress | Ovarian cancer | Proteins | Clonal deletion | Lymphocytes | Immunotherapy | Endoplasmic reticulum | Tumors | Cancer
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2016, Volume 56, pp. 131 - 143
Abstract The study of cancer initiation, growth, and metastasis has traditionally been focused on cancer cells, and the view that they proliferate due to... 
Hematology, Oncology and Palliative Medicine | Tumour microenvironment | Ovarian cancer | LYMPHOCYTE-ASSOCIATED ANTIGEN-4 | INFILTRATING LYMPHOCYTES | PHASE-II | OPEN-LABEL | RECURRENT OVARIAN | MYELOID CELLS | MAINTENANCE THERAPY | ONCOLOGY | CD8(+) T-LYMPHOCYTES | MACROPHAGE POLARIZATION | EPITHELIAL OVARIAN | Neoplasms, Glandular and Epithelial - blood supply | Humans | Neovascularization, Pathologic | Ovarian Neoplasms - pathology | Neoplasms, Glandular and Epithelial - metabolism | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Neoplasms, Glandular and Epithelial - immunology | Angiogenesis Inhibitors - therapeutic use | Female | Ovarian Neoplasms - metabolism | Ovarian Neoplasms - blood supply | Ovarian Neoplasms - drug therapy | Fibroblasts - metabolism | Tumor Microenvironment - drug effects | Neoplasms, Glandular and Epithelial - pathology | Endothelial Cells - metabolism | Fibroblasts - pathology | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Fibroblasts - drug effects | Neoplasms, Glandular and Epithelial - drug therapy | Carcinoma, Ovarian Epithelial | Cell Proliferation - drug effects | Endothelial Cells - pathology | Endothelial Cells - drug effects | Ovarian Neoplasms - immunology | Chemotherapy | Relapse | RNA | Metastasis | Health aspects | Diseases | Cancer | tumor microenvironment
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 11, p. e0207319
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2016, Volume 143, Issue 2, pp. 433 - 438
Journal Article
Nature cell biology, ISSN 1476-4679, 2018, Volume 20, Issue 8, pp. 954 - 965
BRCA1 deficiencies cause breast, ovarian, prostate and other cancers, and render tumours hypersensitive to poly(ADP-ribose) polymerase (PARP) inhibitors. To... 
PATHWAY CHOICE | STRAND BREAK REPAIR | RESECTION | DAMAGE-RESPONSE | 53BP1 | CLASS-SWITCH RECOMBINATION | FANCONI-ANEMIA | DIFFERENTIAL EXPRESSION ANALYSIS | POLYMERASE-ZETA | TELOMERES | CELL BIOLOGY | Osteosarcoma - drug therapy | Mad2 Proteins - metabolism | Humans | Multiprotein Complexes | Ovarian Neoplasms - pathology | Bone Neoplasms - pathology | DNA Breaks, Double-Stranded | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ovarian Neoplasms - genetics | Telomere-Binding Proteins - genetics | DNA End-Joining Repair | HEK293 Cells | Female | Bone Neoplasms - genetics | Ovarian Neoplasms - metabolism | Bone Neoplasms - drug therapy | BRCA1 Protein - deficiency | Telomere-Binding Proteins - metabolism | Ovarian Neoplasms - drug therapy | Osteosarcoma - metabolism | DNA-Binding Proteins | Tumor Suppressor p53-Binding Protein 1 - metabolism | Recombinational DNA Repair | Tumor Suppressor p53-Binding Protein 1 - genetics | Cisplatin - pharmacology | Breast Neoplasms - drug therapy | Proteins - genetics | Xenograft Model Antitumor Assays | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Proteins - metabolism | Breast Neoplasms - pathology | Mad2 Proteins - genetics | Cell Line, Tumor | Mice | Osteosarcoma - genetics | Cell Cycle Proteins | Osteosarcoma - pathology | Care and treatment | DNA | Cancer cells | Breast cancer | Genetic aspects | Research | Gene expression | Single-stranded DNA | DNA damage | Homologous recombination | Poly(ADP-ribose) | Homology | Genomes | Inactivation | Proteins | Ribose | Null cells | Deoxyribonucleic acid--DNA | BRCA2 protein | CRISPR | Deactivation | BRCA1 protein | Poly(ADP-ribose) polymerase | Adenosine diphosphate | Oligosaccharides | Double-strand break repair | Screens | Cisplatin | Polymerase | Inhibitors | Prostate | Viability | Tumors | Telomere-Binding Proteins / metabolism | Osteosarcoma / genetics | Telomere-Binding Proteins / genetics | BRCA1 Protein / genetics | Cellular Biology | Genetics | Proteins / genetics | Osteosarcoma / drug therapy | Ovarian Neoplasms / genetics | Mad2 Proteins / genetics | Proteins / metabolism | Breast Neoplasms / drug therapy | Breast Neoplasms / metabolism | Tumor Suppressor p53-Binding Protein 1 / genetics | BRCA1 Protein / deficiency | Ovarian Neoplasms / metabolism | Mad2 Proteins / metabolism | Breast Neoplasms / pathology | Bone Neoplasms / genetics | Ovarian Neoplasms / pathology | Bone Neoplasms / pathology | Life Sciences | Bone Neoplasms / drug therapy | Ovarian Neoplasms / drug therapy | Osteosarcoma / metabolism | Biochemistry, Molecular Biology | Breast Neoplasms / genetics | Drug Resistance, Neoplasm / genetics | Osteosarcoma / pathology | Bone Neoplasms / metabolism | Poly(ADP-ribose) Polymerase Inhibitors / pharmacology | Cisplatin / pharmacology | Molecular biology | Tumor Suppressor p53-Binding Protein 1 / metabolism | Cancer
Journal Article