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2004, Lung biology in health and disease, ISBN 0824748743, Volume 187., xvii, 529
Book
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2013, Volume 273, Issue 2, pp. 345 - 354
Kidney ischemia–reperfusion (I/R) injury elicits cellular injury in the proximal tubule, and mitochondrial dysfunction is a pathological consequence of I/R.... 
SRT1720 | Proximal tubule | Mitochondrial biogenesis | Mitochondrial dysfunction | Ischemia–reperfusion | Acute kidney injury | Ischemia-reperfusion | PGC-1-ALPHA | EPITHELIAL POLARITY | SIRT1 | ENERGY-EXPENDITURE | OXIDANT INJURY | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PROMOTES RECOVERY | HISTONE DEACETYLASE | SMALL-MOLECULE ACTIVATORS | CALORIE RESTRICTION | Kidney - pathology | Recovery of Function - drug effects | Reperfusion Injury - drug therapy | Male | Heterocyclic Compounds, 4 or More Rings - pharmacology | Kidney - metabolism | Heterocyclic Compounds, 4 or More Rings - therapeutic use | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Recovery of Function - physiology | Homeostasis - drug effects | Kidney Tubules - pathology | Kidney Tubules - metabolism | Reperfusion Injury - metabolism | Kidney - drug effects | Kidney Tubules - drug effects | Reperfusion Injury - pathology | Sirtuin 1 - biosynthesis | Rats | Transcription Factors - biosynthesis | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Rats, Sprague-Dawley | Animals | Homeostasis - physiology | Index Medicus | HOMEOSTASIS | INJURIES | EPITHELIUM | MITOCHONDRIA | CREATININE | RATS | RECEPTORS | 60 APPLIED LIFE SCIENCES | ATP | POTASSIUM IONS | SODIUM IONS | mitochondrial dysfunction | acute kidney injury | ischemia-reperfusion | proximal tubule | mitochondrial biogenesis
Journal Article
Journal of the Neurological Sciences, ISSN 0022-510X, 2015, Volume 355, Issue 1, pp. 113 - 119
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, pp. e0140025 - e0140025
We examined whether endoplasmic reticulum (ER) stress-induced autophagy provides cytoprotection from renal tubular epithelial cell injury due to oxidants and... 
ACUTE KIDNEY INJURY | SIGNALING PATHWAYS | IN-VITRO | ACTIVATION | UNFOLDED PROTEIN RESPONSE | INHIBITION | EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | PROXIMAL TUBULE | CYTOTOXICITY | CELL-DEATH | Hypoxia - drug therapy | Kidney - pathology | Reperfusion Injury - drug therapy | Male | Protective Agents - therapeutic use | Autophagy - drug effects | Hypoxia - metabolism | Kidney - metabolism | Caspases - metabolism | Adenosine Triphosphate - metabolism | Cytoprotection - drug effects | Renal Insufficiency - complications | Renal Insufficiency - drug therapy | Reperfusion Injury - complications | Reperfusion Injury - metabolism | Cell Line | Unfolded Protein Response - drug effects | Kidney - drug effects | Endoplasmic Reticulum Stress - drug effects | Reperfusion Injury - pathology | Heat-Shock Proteins - metabolism | Mice, Inbred C57BL | Hypoxia - complications | Renal Insufficiency - metabolism | Oxidants - adverse effects | Tunicamycin - therapeutic use | Animals | Hypoxia - pathology | Renal Insufficiency - pathology | Mice | Oxidative Stress - drug effects | Cell death | Reperfusion injury | Cell culture | Hydrogen peroxide | Renal function | Butyl hydroperoxide | Epithelial cells | Chloroquine | Oxidizing agents | Tunicamycin | Cytotoxicity | Activation | Kinases | Autophagy | Proteins | Cell activation | Reperfusion | Ischemia | Oxidants | Protein folding | Antimycin A | Physiology | Inhibition | Injuries | Pharmaceutical sciences | Stresses | Immunoglobulins | Cell survival | Mortality | Caspase | Histology | Stress | Medicine | Cell injury | Hypoxia | Iridium | Endoplasmic reticulum | Phagocytosis | Clear cell-type renal cell carcinoma | Cancer | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of the American Society of Nephrology : JASN, ISSN 1046-6673, 2014, Volume 25, Issue 6, pp. 1157 - 1162
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2010, Volume 247, Issue 3, pp. 169 - 178
Acetaminophen (APAP) overdose can result in serious liver injury and potentially death. Toxicity is dependent on metabolism of APAP to a reactive metabolite... 
Interleukin-1 | Inflammasome | Inflammation | Acetaminophen | Hepatotoxicity | Neutrophils | APOPTOSIS | OXIDANT STRESS | MURINE ENDOTOXIN-SHOCK | CXC CHEMOKINES | TUMOR-NECROSIS-FACTOR | SINUSOIDAL ENDOTHELIAL-CELLS | KUPFFER CELLS | PHARMACOLOGY & PHARMACY | MICE | TOXICOLOGY | ISCHEMIA-REPERFUSION | INDUCED HEPATOTOXICITY
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2013, Volume 288, Issue 6, pp. 3977 - 3988
Journal Article
by Wang, JA and Dore, S
BRAIN, ISSN 0006-8950, 06/2007, Volume 130, pp. 1643 - 1652
Because heme oxygenase (HO) is the rate limiting enzyme in the degradation of the pro-oxidant hemin/heme from blood, here we investigated the contribution of... 
OXIDATIVE STRESS | OXIDANT DAMAGE | antioxidants | TIN-MESOPORPHYRIN | HO-1 | IRON | INDUCTION | blood | NEUROSCIENCES | CLINICAL NEUROLOGY | CELL-DEATH | reactive oxygen species | CENTRAL-NERVOUS-SYSTEM | TUFTSIN FRAGMENT 1-3 | RAT-BRAIN | hemin | CEREBRAL-ISCHEMIA
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 06/2015, Volume 133, Issue 6, pp. 898 - 908
Journal Article