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Hypertension, ISSN 0194-911X, 09/2009, Volume 54, Issue 3, pp. 652 - 658
Drugs and antibodies that interrupt vascular endothelial growth factor (VEGF) signaling pathways improve outcomes in patients with a variety of cancers by... 
Hypertension | Angiogenesis | Vascular endothelial growth factor | Cancer | TYROSINE KINASE-1 | BEVACIZUMAB | angiogenesis | vascular endothelial growth factor | VEGF | MOUSE | PERIPHERAL VASCULAR DISEASE | cancer | MONOCLONAL-ANTIBODY | MICE | hypertension | MOLECULAR-MECHANISMS | Gene Expression - drug effects | Kidney - enzymology | Antibodies, Monoclonal - adverse effects | Sodium Chloride, Dietary - administration & dosage | Male | Vascular Endothelial Growth Factor Receptor-2 - immunology | RNA, Messenger - metabolism | Aldosterone - urine | Hypertension - etiology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Renin - genetics | Prostaglandins - urine | Nitric Oxide Synthase Type I - antagonists & inhibitors | Blood Pressure - drug effects | Blood Pressure - physiology | Vascular Endothelial Growth Factor Receptor-2 - physiology | Nitric Oxide Synthase Type III - metabolism | Manometry | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - biosynthesis | Kidney - drug effects | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Sodium Chloride, Dietary - adverse effects | Enzyme Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Nitric Oxide Synthase Type I - metabolism | Hybridomas | Mice | Nitric Oxide Synthase Type I - genetics | Vascular Endothelial Growth Factor A - physiology | Index Medicus | nitric oxide
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, p. e0161306
In rheumatoid arthritis (RA), nitric oxide (NO) is implicated in inflammation, angiogenesis and tissue destruction. The enzyme inducible nitric oxide synthase... 
TGF-BETA | JAK/STAT PATHWAY | PROTEIN | SYNOVIAL-FLUID | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | NO SYNTHASE | NF-KAPPA-B | UP-REGULATION | MACROPHAGE ACTIVATION | Oligonucleotide Array Sequence Analysis | Synovial Membrane - enzymology | Humans | Arthritis, Rheumatoid - enzymology | Nitric Oxide Synthase Type II - physiology | Synovial Membrane - metabolism | Cytokines - physiology | Arthritis, Rheumatoid - metabolism | STAT1 Transcription Factor - metabolism | Interferons - metabolism | Interleukin-10 - metabolism | Signal Transduction - physiology | Transforming Growth Factor beta - metabolism | Nitric Oxide Synthase Type II - metabolism | Rheumatoid factor | Enzymes | Interleukins | Nitric oxide | Analysis | Genes | Bone morphogenetic proteins | Arthritis | Interferon | Inflammation | Transforming growth factors | Gene expression | Regulations | Biotechnology | Transcription factors | Target recognition | Gene regulation | Transforming growth factor-b | Interleukin | Macrophages | Synovium | Angiogenesis | Signal transduction | Pathways | Rodents | Stat1 protein | Transducers | Cytokines | Data processing | Rac2 protein | Joint diseases | Synoviocytes | Botulinum toxin | Nitric-oxide synthase | Substrates | Studies | Signaling | DNA microarrays | Rheumatoid arthritis | Interleukin 10 | Synovial joints
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 3, pp. 729 - 736.e5
Background Clinical observations suggest that anaphylaxis is more common in adult women compared with adult men, although the mechanistic basis for this sex... 
Allergy and Immunology | estrogen | Anaphylaxis | nitric oxide synthase | vascular permeability | mast cells | ACTIVATION | IDIOPATHIC ANAPHYLAXIS | SEX-HORMONES | CARDIOVASCULAR-SYSTEM | SENSITIVITY | BODY-TEMPERATURE | RELEASE | IMMUNOLOGY | SHOCK | ALLERGY | NO SYNTHASE | UP-REGULATION | Protein Aggregates | Capillary Permeability | Humans | Receptors, IgG - chemistry | Male | omega-N-Methylarginine - pharmacology | Histamine - immunology | Receptors, IgG - antagonists & inhibitors | Lung - enzymology | Receptors, Estrogen - immunology | Receptors, IgE - chemistry | Estrogen Receptor Antagonists - pharmacology | Anaphylaxis - physiopathology | Anaphylaxis - immunology | Female | Receptors, IgE - immunology | Estradiol - pharmacology | Disease Models, Animal | Estradiol - metabolism | Mast Cells - immunology | Nitric Oxide - biosynthesis | Nitric Oxide Synthase Type III - immunology | Gene Expression | Histamine - pharmacology | Ovariectomy | Receptors, Estrogen - antagonists & inhibitors | Receptors, IgE - antagonists & inhibitors | Lung - physiopathology | Receptors, IgG - immunology | Body Temperature | Anaphylaxis - enzymology | Mast Cells - drug effects | Nitric Oxide Synthase Type III - genetics | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Anaphylaxis - genetics | Animals | Receptors, Estrogen - chemistry | Lung - drug effects | Mast Cells - pathology | Mice | Lung - immunology | Sex discrimination | Nitric oxide | Estrogen | Endothelium | Gender differences | Rodents | Allergies
Journal Article
Allergy, ISSN 0105-4538, 04/2017, Volume 72, Issue 4, pp. 627 - 635
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 01/2012, Volume 674, Issue 2-3, pp. 73 - 79
Journal Article
Nitric Oxide, ISSN 1089-8603, 07/2019, Volume 88, pp. 50 - 60
The human inducible nitric oxide synthase (iNOS) gene contains an upstream open reading frame (uORF) in its 5′-untranslated region (5′-UTR) implying a... 
uORF | iNOS | 5′-UTR | NMD | IRES | CAP-Dependent | Codon | Nitric oxide | Genes | Luciferase
Journal Article
Journal Article