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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2016, Volume 36, Issue 6, pp. 1090 - 1100
OBJECTIVE—Hyperlipidemia-induced endothelial cell (EC) activation is considered as an initial event responsible for monocyte recruitment in atherogenesis.... 
atherosclerosis | hyperlipidemia | endothelial cell | mitochondria | reactive oxygen species | PROGENITOR CELLS | VARESPLADIB | PHOSPHOLIPASE A INHIBITOR | CARDIOVASCULAR EVENTS | RANDOMIZED CLINICAL-TRIAL | ACUTE CORONARY SYNDROME | HEART-DISEASE | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | MICE | ATHEROSCLEROTIC PLAQUE | HEMATOLOGY | Hyperlipidemias - genetics | Reactive Oxygen Species - metabolism | Apolipoproteins E - deficiency | Transcription Factor AP-1 - genetics | Atherosclerosis - genetics | Humans | Aorta - metabolism | Transcription Factor AP-1 - metabolism | Aortic Diseases - metabolism | Time Factors | Membrane Potential, Mitochondrial | Calcium Signaling | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Lysophosphatidylcholines - pharmacology | Hyperlipidemias - metabolism | Oxidation-Reduction | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Lysophosphatidylcholines - metabolism | Gene Expression Profiling - methods | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Metabolomics - methods | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Intercellular Adhesion Molecule-1 - metabolism | Phenotype | Animals | Apolipoproteins E - genetics | Intercellular Adhesion Molecule-1 - genetics | Oxidative Stress - drug effects | Endothelial Cells - drug effects | Index Medicus | vascular inflammation
Journal Article
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2012, Volume 302, Issue 3, pp. C597 - C604
Maalouf RM, Eid AA, Gorin YC, Block K, Escobar GP, Bailey S, Abboud HE. Nox4-derived reactive oxygen species mediate cardiomyocyte injury in early type 1... 
Cardiac injury | NADPH oxidases | Oxidative stress | NADPH OXIDASE 4 | APOPTOSIS | ACTIVATION | PHYSIOLOGY | cardiac injury | CELL BIOLOGY | NAD(P)H OXIDASE | FAILING HEART | NOX FAMILY | MESANGIAL CELLS | MYOFIBROBLASTS | EXPRESSION | oxidative stress | REVEALS | Fibronectins - biosynthesis | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Ventricular Myosins - biosynthesis | Humans | Membrane Glycoproteins - biosynthesis | NADPH Oxidases - metabolism | Diabetes Mellitus, Type 1 - metabolism | Male | Diabetes Mellitus, Type 1 - complications | Collagen - biosynthesis | Diabetes Mellitus, Experimental - complications | NADH, NADPH Oxidoreductases - biosynthesis | NADPH Oxidases - biosynthesis | Diabetes Mellitus, Experimental - metabolism | Actins - biosynthesis | Oligonucleotides, Antisense - pharmacology | Cells, Cultured | Rats | Glucose - pharmacology | NADPH Oxidase 4 | NADPH Oxidase 2 | Rats, Sprague-Dawley | NADPH Oxidase 1 | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | Heart Ventricles - metabolism | Complications and side effects | Type 1 diabetes | Cardiomyopathy | Heart cells | NADP (Coenzyme) | Physiological aspects | Development and progression | Research | Health aspects | Heart diseases | Diabetes | Glucose | Rodents | Cells | Index Medicus | Receptors and Signal Transduction
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 05/2006, Volume 13, Issue 5, pp. 730 - 737
Journal Article
Journal Article