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1. Full Text Sema3E-PlexinD1 signaling selectively suppresses disoriented angiogenesis in ischemic retinopathy in mice
by Fukushima, Yoko and Okada, Mitsuhiro and Kataoka, Hiroshi and Hirashima, Masanori and Yoshida, Yutaka and Mann, Fanny and Gomi, Fumi and Nishida, Kohji and Nishikawa, Shin-Ichi and Uemura, Akiyoshi
Journal of Clinical Investigation, ISSN 0021-9738, 05/2011, Volume 121, Issue 5, pp. 1974 - 1985
During development, the retinal vasculature grows toward hypoxic areas in an organized fashion. By contrast, in ischemic retinopathies, new blood vessels grow... 
DIABETIC-RETINOPATHY | MEDICINE, RESEARCH & EXPERIMENTAL | RETINAL VASCULATURE | PLEXIN D1 | OXYGEN-INDUCED RETINOPATHY | VEGF | IN-VIVO | SEMAPHORIN 3E | MOUSE RETINA | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Signal Transduction | Blood Vessels - pathology | Humans | Mice, Inbred C57BL | Glycoproteins - metabolism | Ischemia - metabolism | Mice, Transgenic | Retinal Diseases - metabolism | Nerve Tissue Proteins | Vascular Endothelial Growth Factor A - metabolism | Mice, Inbred ICR | Animals | GTP Phosphohydrolases - metabolism | Pseudopodia - metabolism | Endothelial Cells - cytology | Hypoxia | Ligands | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Mice | Neovascularization, Pathologic - metabolism | Neurons - metabolism | Membrane Glycoproteins | Physiological aspects | Development and progression | Cellular signal transduction | Research | Neovascularization | Vascular endothelial growth factor | Retinal diseases | Index Medicus | Abridged Index Medicus
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2. Full Text Nogo‐A inhibits vascular regeneration in ischemic retinopathy
by Joly, Sandrine and Dejda, Agnieszka and Rodriguez, Léa and Sapieha, Przemyslaw and Pernet, Vincent
Glia, ISSN 0894-1491, 10/2018, Volume 66, Issue 10, pp. 2079 - 2093
Nogo‐A is a potent glial‐derived inhibitor of axon growth in the injured CNS and acts as a negative regulator of developmental angiogenesis by inhibiting... 
Nogo‐A | glia | vascular repair | immunotherapy | oxygen‐induced retinopathy | Nogo-A | oxygen-induced retinopathy | ANGIOGENESIS | MOUSE | AXON GUIDANCE | NEUROSCIENCES | RECOVERY | ADULT-RAT | GROWTH | CORTICOSPINAL TRACT | ANTI-VEGF AGENTS | UP-REGULATION | Neovascularization, Physiologic - drug effects | Nogo Proteins - immunology | Neuroglia - pathology | Retinal Diseases - metabolism | Vision, Ocular - drug effects | Neuroglia - drug effects | Retinal Vessels - pathology | Vision, Ocular - physiology | Retinal Diseases - pathology | Angiogenesis Inducing Agents - pharmacology | Receptors, Lysosphingolipid - metabolism | Ischemia - pathology | Disease Models, Animal | Retinal Vessels - metabolism | Nogo Proteins - antagonists & inhibitors | Mice, Inbred C57BL | Ischemia - metabolism | Retinal Vessels - drug effects | Regeneration - physiology | Neovascularization, Physiologic - physiology | Regeneration - drug effects | Animals | Ischemia - drug therapy | Nogo Proteins - metabolism | Neuroglia - metabolism | Retinal Diseases - drug therapy | Eye diseases | Immunotherapy | Retinopathy | Retinal cells | Blocking antibodies | Blood vessels | Retina | Glial cells | Blood | Endothelial cells | Cell adhesion & migration | Nogo protein | Regeneration | Angiogenesis | Ischemia | Restoration | Perfusion | Rodents | Inhibition | Cell migration | Injuries | Index Medicus
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3. Full Text Therapeutic effects of PPARα agonists on diabetic retinopathy in type 1 diabetes models
by Chen, Ying and Hu, Yang and Lin, Mingkai and Jenkins, Alicia J and Keech, Anthony C and Mott, Robert and Lyons, Timothy J and Ma, Jian-Xing
Diabetes, ISSN 0012-1797, 01/2013, Volume 62, Issue 1, pp. 261 - 272
Retinal vascular leakage, inflammation, and neovascularization (NV) are features of diabetic retinopathy (DR). Fenofibrate, a peroxisome proliferator-activated... 
CELLS | METABOLIC SYNDROME | HYPERLIPOPROTEINEMIA | OXYGEN-INDUCED RETINOPATHY | PROCETOFEN | FENOFIBRATE | ENDOCRINOLOGY & METABOLISM | GROWTH-FACTOR | CLOFIBRATE | MODULATION | VASCULAR LEAKAGE | Hypoglycemic Agents - therapeutic use | Retina - drug effects | Diabetic Retinopathy - drug therapy | Retina - metabolism | Mice, Inbred C57BL | Capillary Permeability - drug effects | Rats | Male | Diabetes Mellitus, Type 1 - complications | Leukocytes - physiology | Cell Adhesion - drug effects | Fenofibrate - therapeutic use | Diabetic Retinopathy - physiopathology | Rats, Inbred BN | Fenofibrate - pharmacology | Animals | PPAR alpha - physiology | Leukocytes - drug effects | PPAR alpha - agonists | Mice | Disease Models, Animal | Index Medicus | Abridged Index Medicus | Complications
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4. Full Text Targeting VEGF in canine oxygen-induced retinopathy - A model for human retinopathy of prematurity
by McLeod, D. Scott and Lutty, Gerard A
Eye and Brain, ISSN 1179-2744, 05/2016, Volume 8, pp. 55 - 65
Development of the dog superficial retinal vasculature is similar to the mechanism of human retinal vasculature development; they both develop by... 
Oxygen | Retinopathy | Vascular endothelial cell growth factor | Blood vessels | Angioblasts | Retina | Endothelial cells | Retrolental fibroplasia | Endothelial growth factors | Research | Enzymes | Adenosine | Membranes | Medical imaging | Angiogenesis | Hyperoxia | Dogs | Hypoxia | Ophthalmology | Localization | Laboratory animals | Vascular endothelial growth factor
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5. Full Text NADPH Oxidase, NOX1, Mediates Vascular Injury in Ischemic Retinopathy
by Wilkinson-Berka, Jennifer L and Deliyanti, Devy and Rana, Indrajeetsinh and Miller, Antonia G and Agrotis, Alex and Armani, Roksana and Szyndralewiez, Cédric and Wingler, Kirstin and Touyz, Rhian M and Cooper, Mark E and Jandeleit-Dahm, Karin A and Schmidt, Harald H.H.W
Antioxidants & Redox Signaling, ISSN 1523-0864, 06/2014, Volume 20, Issue 17, pp. 2726 - 2740
Aims: Ischemic retinal diseases such as retinopathy of prematurity are major causes of blindness due to damage to the retinal microvasculature. Despite this... 
Forum Original Research Communications | RETINAL NEOVASCULARIZATION | DIABETIC-RETINOPATHY | NAD(P)H OXIDASE | VITAMIN-E | OXIDATIVE STRESS | VASO-OBLITERATION | OXYGEN-INDUCED RETINOPATHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE MODEL | ENDOCRINOLOGY & METABOLISM | ANIMAL-MODEL | ENDOTHELIAL GROWTH-FACTOR | Pyridines - administration & dosage | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Humans | Membrane Glycoproteins - biosynthesis | Ischemia - metabolism | Vascular System Injuries - enzymology | Ischemia - genetics | NADPH Oxidase 4 | Retinopathy of Prematurity - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Pyrazoles - administration & dosage | Animals | NADPH Oxidases - genetics | Mice | Retinopathy of Prematurity - metabolism | NADPH Oxidases - biosynthesis | Ischemia - pathology | Disease Models, Animal | Retinopathy of Prematurity - pathology | Blood circulation disorders | Reactive oxygen species | Care and treatment | Retrolental fibroplasia | NADP (Coenzyme) | Influence | Research | Risk factors | Index Medicus
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6. Full Text TCF7L2 variation and proliferative diabetic retinopathy
by Luo, Jing and Zhao, Ling and Chen, Aaron Yun and Zhang, Xiaohui and Zhu, Jin and Zhao, Jiagang and Ouyang, Hong and Luo, Hongrong and Song, Yaojun and Lee, Janet and Patel, Sherrina H and Shaw, Peter X and Sadda, Srinivas and Zhuo, Yehong and Rosenfeld, Michael G and Zhang, Kang
Diabetes, ISSN 0012-1797, 07/2013, Volume 62, Issue 7, pp. 2613 - 2617
Proliferative diabetic retinopathy (PDR) is the most severe vision-threatening complication of diabetes. For investigation of genetic association between... 
EYE | OBESITY | POLYMORPHISMS | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | INFLAMMATION | SUSCEPTIBILITY | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | ENDOPLASMIC-RETICULUM STRESS | ENDOTHELIAL GROWTH-FACTOR | Genetic Predisposition to Disease | Transcription Factor 7-Like 2 Protein - genetics | Genetic Association Studies | Diabetes Mellitus, Type 2 - genetics | Gene Frequency | Humans | Genotype | Endoplasmic Reticulum Stress - genetics | Vascular Endothelial Growth Factor A - genetics | Diabetic Retinopathy - genetics | Animals | Alleles | Retinal Neovascularization - genetics | Mice | Polymorphism, Single Nucleotide | Diabetic retinopathy | Transcription factors | Genetic variation | Physiological aspects | Development and progression | Genetic aspects | Research | Index Medicus | Abridged Index Medicus | Original Research
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7. Full Text Induction and Readout of Oxygen-Induced Retinopathy
by Liegl, Raffael and Priglinger, Claudia and Ohlmann, Andreas and Ohlmann, Andreas
Methods in molecular biology (Clifton, N.J.), ISSN 1064-3745, 2019, Volume 1834, pp. 179 - 191
Ischemic retinal diseases can be caused by various pathologies, which often lead to formation of preretinal neovascularization. A very common and... 
Angiogenesis | Retinopathy of prematurity | Hyperoxia | Vaso-obliteration | Vessel regrowth | Oxygen-induced retinopathy | Preretinal neovascularization | Immunohistochemistry | Retinal Diseases - diagnosis | Oxidative Stress | Retinal Vessels - metabolism | Humans | Retinal Neovascularization - metabolism | Oxygen - metabolism | Retinal Vessels - pathology | Animals | Retinal Diseases - etiology | Fluorescent Antibody Technique | Mice | Retinal Neovascularization - pathology | Disease Models, Animal | Index Medicus
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8. Full Text Through the eyes of a child: Understanding retinopathy through ROP - The Friedenwald Lecture
by Smith, Lois E. H
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 2008, Volume 49, Issue 12, pp. 5177 - 5182
RETINAL NEOVASCULARIZATION | MURINE MODEL | PROLIFERATIVE DIABETIC-RETINOPATHY | OXYGEN-INDUCED RETINOPATHY | OPHTHALMOLOGY | POLYUNSATURATED FATTY-ACIDS | VASCULAR-PERMEABILITY FACTOR | FACTOR-I | BINDING-PROTEINS | PREMATURITY | ENDOTHELIAL GROWTH-FACTOR | United States | Humans | Awards and Prizes | Child, Preschool | Erythropoietin - physiology | Retinal Vessels - physiology | Retinopathy of Prematurity - physiopathology | Animals | Societies, Medical | Insulin-Like Growth Factor I - physiology | Retinopathy of Prematurity - metabolism | Ophthalmology | Child | Vascular Endothelial Growth Factor A - physiology | Infant, Newborn | Disease Models, Animal | Index Medicus
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9. Full Text Strain differences in light-induced retinopathy
by Polosa, Anna and Bessaklia, Hyba and Lachapelle, Pierre
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, pp. e0158082 - e0158082
The purpose of this study was to better understand the role of ocular pigmentation and genetics in light-induced retinal damage. Adult pigmented [Long Evans... 
VISUAL CYCLE | OXYGEN-INDUCED RETINOPATHY | ALBINO-RATS | PIGMENT EPITHELIUM | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | RHODOPSIN | INDUCED RETINAL DEGENERATION | DAMAGE | RPE65 | PHOTORECEPTORS | Electroretinography | Retinal Diseases - genetics | Species Specificity | Light - adverse effects | Rats, Long-Evans | Rats, Inbred Lew | Rats | Pigmentation | Rats, Sprague-Dawley | Melanins - biosynthesis | Rats, Inbred BN | Animals | Radiation Injuries - genetics | Retinal Diseases - etiology | Retina - pathology | Care and treatment | Research | Diagnosis | Retinal diseases | Oxidative stress | Neurosciences | Retinopathy | Albinism | Laboratories | Retina | Histology | Exposure | Neurosurgery | Waves | Rodents | Light | Genetics | Diabetes | Damage | Age | Apoptosis | Index Medicus
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10. Propranolol for proliferative diabetic retinopathy
by Hendrick, Andrew M and Lavine, Jeremy and Domalpally, Amitha and Kulkarni, Amol D and Ip, Michael S
Ophthalmic Surgery Lasers and Imaging Retina, ISSN 2325-8160, 01/2018, Volume 49, Issue 1, pp. 35 - 40
BACKGROUND AND OBJECTIVE: To determine the effect of propranolol on retinal neovascularization due to proliferative diabetic retinopathy (PDR). PATIENTS AND... 
SURGERY | OXYGEN-INDUCED RETINOPATHY | HEMANGIOMAS | OPHTHALMOLOGY | ANTAGONISM | RANIBIZUMAB | SEVERITY | Diabetic Retinopathy - drug therapy | Prospective Studies | Follow-Up Studies | Administration, Oral | Fluorescein Angiography - methods | Humans | Middle Aged | Male | Treatment Outcome | Tomography, Optical Coherence - methods | Visual Acuity | Diabetic Retinopathy - physiopathology | Dose-Response Relationship, Drug | Diabetic Retinopathy - diagnosis | Pilot Projects | Time Factors | Propranolol - administration & dosage | Aged, 80 and over | Adult | Female | Aged | Fundus Oculi | Retina - pathology | Adrenergic beta-Antagonists - administration & dosage
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11. Full Text Mechanisms and Management of Retinopathy of Prematurity
by Jasani, B and Nanavati, R and Kabra, N
NEW ENGLAND JOURNAL OF MEDICINE, ISSN 0028-4793, 03/2013, Volume 368, Issue 12, pp. 1161 - 1162
MEDICINE, GENERAL & INTERNAL | OXYGEN-INDUCED RETINOPATHY | PROPRANOLOL | Animals | Retinal Vessels - growth & development | Retinopathy of Prematurity | Humans
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12. Full Text Truncated netrin-1 contributes to pathological vascular permeability in diabetic retinopathy
by Miloudi, Khalil and Binet, François and Wilson, Ariel and Cerani, Agustin and Oubaha, Malika and Menard, Catherine and Henriques, Sullivan and Mawambo, Gaelle and Dejda, Agnieszka and Nguyen, Phuong Trang and Rezende, Flavio A and Bourgault, Steve and Kennedy, Timothy E and Sapieha, Przemyslaw
Journal of Clinical Investigation, ISSN 0021-9738, 08/2016, Volume 126, Issue 8, pp. 3006 - 3022
Diabetic retinopathy (DR) is a major complication of diabetes and a leading cause of blindness in the working-age population. Impaired blood-retinal barrier... 
RETINAL NEOVASCULARIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | MATRIX METALLOPROTEINASES | NERVOUS-SYSTEM | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | GROWTH | SPINAL-CORD | INHIBITORS | GUIDANCE | RECEPTOR UNC5B | Blood-Retinal Barrier | Microglia - metabolism | Retina - metabolism | Capillary Permeability | Netrin-1 | Streptozocin | Humans | Middle Aged | Nerve Growth Factors - metabolism | Case-Control Studies | Matrix Metalloproteinase 9 - metabolism | Diabetes Mellitus, Experimental | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Protein Domains | Disease Models, Animal | Tumor Suppressor Proteins - metabolism | Mice, Inbred C57BL | Macular Edema - metabolism | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Animals | Nerve Growth Factors - genetics | Aged | Mice | Diabetic retinopathy | Complications and side effects | Papilledema | Diabetes | Permeability | Research | Risk factors | Proteins | Studies | Edema | Experiments | Index Medicus | Abridged Index Medicus
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13. Full Text Therapeutic potential of anti-angiogenic multitarget N,O-Sulfated E. Coli K5 Polysaccharide in diabetic retinopathy
by Rezzola, Sara and Dal Monte, Massimo and Belleri, Mirella and Bugatti, Antonella and Chiodelli, Paola and Corsini, Michela and Cammalleri, Maurizio and Cancarini, Anna and Morbidelli, Lucia and Oreste, Pasqua and Bagnoli, Paola and Semeraro, Francesco and Presta, Marco
Diabetes, ISSN 0012-1797, 07/2015, Volume 64, Issue 7, pp. 2581 - 2592
Vascular endothelial growth factor (VEGF) blockers have been developed for the treatment of proliferative diabetic retinopathy (PDR), the leading cause of... 
INTRAVITREAL INJECTION | EYE DISEASE | ISCHAEMIA/REPERFUSION INJURY | OXYGEN-INDUCED RETINOPATHY | MOUSE | ENDOCRINOLOGY & METABOLISM | VEGF AGENTS | RETINAL ANGIOGENESIS | MODEL | HEPARIN | ENDOTHELIAL GROWTH-FACTOR | Diabetic Retinopathy - drug therapy | Heparin - metabolism | Bacterial Capsules | Cricetulus | Humans | Mice, Inbred C57BL | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Chick Embryo | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Animals | Angiogenesis Inhibitors - therapeutic use | Mice | Retinal Neovascularization - drug therapy | CHO Cells | Vitreous Body - drug effects | Diabetic retinopathy | Care and treatment | Polysaccharides | Escherichia coli | Dosage and administration | Research | Vascular endothelial growth factor | Carbohydrates | E coli | Index Medicus | Abridged Index Medicus
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14. Full Text Antibody-Mediated Inhibition of Tspan12 Ameliorates Vasoproliferative Retinopathy Through Suppression of β-Catenin Signaling
by Bucher, Felicitas and Zhang, Ding and Aguilar, Edith and Sakimoto, Susumu and Diaz-Aguilar, Sophia and Rosenfeld, Mauricio and Zha, Zhao and Zhang, Hongkai and Friedlander, Martin and Yea, Kyungmoo
Circulation, ISSN 0009-7322, 07/2017, Volume 136, Issue 2, pp. 180 - 195
BACKGROUND:Anti-angiogenic biologicals represent an important concept for the treatment of vasoproliferative diseases. However, the need for continued... 
β-catenin | antibody | vascular disease | retinopathy | angiogenesis | therapy | VEGF | Tetraspanin12 | beta-catenin | DIABETIC-RETINOPATHY | CARDIAC & CARDIOVASCULAR SYSTEMS | OXYGEN-INDUCED RETINOPATHY | MOUSE | RETINAL VASCULAR DEVELOPMENT | MACULAR DEGENERATION | WNT PATHWAY | ENDOTHELIAL-CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | VESSEL LOSS | Human Umbilical Vein Endothelial Cells | Amino Acid Sequence | Antibodies - therapeutic use | Intravitreal Injections | Humans | Mice, Inbred C57BL | Mice, Transgenic | Retinal Neovascularization - metabolism | Mice, 129 Strain | beta Catenin - metabolism | Mice, Knockout | Antibodies - pharmacology | Tetraspanins - antagonists & inhibitors | Animals | Signal Transduction - drug effects | beta Catenin - antagonists & inhibitors | HEK293 Cells | Antibodies - genetics | Signal Transduction - physiology | Mice | Retinal Neovascularization - drug therapy | Tetraspanins - metabolism | Index Medicus | Abridged Index Medicus
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15. Full Text Reduced nitro-oxidative stress and neural cell death suggests a protective role for microglial cells in TNFα-/- mice in ischemic retinopathy
by Stevenson, Laura and Matesanz, Nuria and Colhoun, Liza and Edgar, Kevin and Devine, Adrian and Gardiner, Tom A and McDonald, Denise M
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 06/2010, Volume 51, Issue 6, pp. 3291 - 3299
PURPOSE. Neovascularization occurs in response to tissue ischemia and growth factor stimulation. In ischemic retinopathies, however, new vessels fail to... 
RETINAL NEOVASCULARIZATION | TUMOR-NECROSIS-FACTOR | OXIDE SYNTHASE | MULLER GLIAL-CELLS | ASTROCYTE DEGENERATION | OXYGEN-INDUCED RETINOPATHY | APOPTOTIC CELLS | OPHTHALMOLOGY | TNF-ALPHA | ENDOTHELIAL GROWTH-FACTOR | TRANSLATIONAL CONTROL
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16. Full Text RhoB antibody alters retinal vascularization in models of murine retinopathy
by Almonte‐Baldonado, Rowena and Bravo‐Nuevo, Arturo and Gerald, Damien and Benjamin, Laura E and Prendergast, George C and Laury‐Kleintop, Lisa D
Journal of Cellular Biochemistry, ISSN 0730-2312, 06/2019, Volume 120, Issue 6, pp. 9381 - 9391
Neovascularization in cancer or retinopathy is driven by pathological changes that foster abnormal sprouting of endothelial cells. Mouse genetic studies... 
retinopathy of prematurity (ROP) | oxygen‐induced retinopathy (OIR) | animal model | diabetic retinopathy | monoclonal antibody | Rho GTPase | macular degeneration | oxygen-induced retinopathy (OIR) | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRAFFICKING | CANCER | CELL BIOLOGY | ADHESION | INHIBITION | ROLES | ENDOTHELIAL-CELLS | BIOLOGY | MOUSE RETINA | NEOVASCULARIZATION | RANIBIZUMAB | Macular degeneration | Medical research | Diabetic retinopathy | Analysis | Physiological aspects | Monoclonal antibodies | Medicine, Experimental | Models | Neovascularization | Vascular endothelial growth factor | Animal models | Retinopathy | Diabetes mellitus | Blood vessels | Retina | Endothelial cells | Vascularization | Angiogenesis | Hemangioma | Ischemia | Physiology | Degeneration | Diabetes | Growth factors | Cancer | Guanosinetriphosphatase
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