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NATURE COMMUNICATIONS, ISSN 2041-1723, 08/2019, Volume 10, Issue 1, pp. 3887 - 20
Oligodendrocyte precursor cells (OPCs) are abundant in the adult central nervous system, and have the capacity to regenerate oligodendrocytes and myelin.... 
PROGENITOR CELLS | PATHOGENESIS | MULTIPLE-SCLEROSIS LESIONS | AUTOIMMUNE | INTERFERON-GAMMA | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | REMYELINATION | CENTRAL-NERVOUS-SYSTEM | CD8(+) T-CELLS | CYTOKINE GM-CSF | Caspase 7 - metabolism | Oligodendroglia - metabolism | Central Nervous System - metabolism | Humans | Caspase 3 - metabolism | Histocompatibility Antigens Class I | Central Nervous System - immunology | Myelin Sheath - metabolism | CD8-Positive T-Lymphocytes | Ovalbumin - metabolism | Remyelination - immunology | Oligodendrocyte Precursor Cells - drug effects | Demyelinating Diseases - immunology | CD4-Positive T-Lymphocytes | Cell Differentiation | Demyelinating Diseases - pathology | Cytokines - genetics | Oligodendrocyte Precursor Cells - metabolism | Disease Models, Animal | Interferon-gamma | Gene Expression | Cytokines - metabolism | T-Lymphocytes | Mice, Inbred C57BL | Oligodendrocyte Precursor Cells - pathology | Mice, Transgenic | Antigen-Presenting Cells - immunology | Animals | Oligodendrocyte Precursor Cells - immunology | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Antigens - immunology | Mice | Astrocytes - metabolism | Brain | Multiple sclerosis | CD8 antigen | Central nervous system | Effector cells | Cytotoxicity | Lymphocytes T | Glial stem cells | Inflammatory diseases | Demyelination | Lymphocytes | Precursors | Oligodendrocytes | Inhibition | Lesions | Immune system | Antigens | Myelin | Astrocytes | Substantia alba | Myelination | Major histocompatibility complex | Cell death | γ-Interferon | In vivo methods and tests
Journal Article
Brain, ISSN 0006-8950, 05/2011, Volume 134, Issue 5, pp. 1315 - 1330
Journal Article
Journal Article
Glia, ISSN 0894-1491, 12/2013, Volume 61, Issue 12, pp. 2078 - 2099
Oligodendrocyte precursor cells (OPCs) originate from restricted regions of the brain and migrate into the developing white matter, where they differentiate... 
Tiam1 | migration | oligodendrocyte precursor cells | NMDA receptors | ERK | Migration | Oligodendrocyte precursor cells | SUBVENTRICULAR ZONE | GLIAL-CELLS | NEUROSCIENCES | GLUTAMATE-RECEPTOR | IN-VITRO | MEDIATED ERK1/2 ACTIVATION | PROGENITOR-CELL | OLFACTORY ENSHEATHING CELLS | CENTRAL-NERVOUS-SYSTEM | CORTICAL NEURONAL MIGRATION | RAT OPTIC-NERVE | Oligodendroglia - metabolism | Calcium Signaling - physiology | Receptors, N-Methyl-D-Aspartate - metabolism | Neurons - cytology | Neoplasm Proteins - metabolism | Neural Stem Cells - cytology | Cell Lineage - drug effects | Cerebral Cortex - cytology | Cell Movement - physiology | Cerebral Cortex - metabolism | N-Methylaspartate - pharmacology | Oligodendroglia - drug effects | Guanine Nucleotide Exchange Factors - metabolism | Neurons - metabolism | Cerebral Cortex - drug effects | Oligodendroglia - cytology | Neurons - drug effects | Cell Differentiation - physiology | T-Lymphoma Invasion and Metastasis-inducing Protein 1 | Neural Stem Cells - drug effects | Rats | Rats, Sprague-Dawley | Cell Lineage - physiology | Cell Movement - drug effects | Animals | Calcium Signaling - drug effects | Cell Differentiation - drug effects | Neural Stem Cells - metabolism | rac1 GTP-Binding Protein - metabolism | Physiological aspects | Methyl aspartate | Purines | Aspartate | Lymphomas | Glutamate | Rodents | Cell adhesion & migration
Journal Article
Journal Article
Glia, ISSN 0894-1491, 12/2017, Volume 65, Issue 12, pp. 2087 - 2098
The regeneration of oligodendrocytes is a crucial step in recovery from demyelination, as surviving oligodendrocytes exhibit limited structural plasticity and... 
oligodendrocyte progenitor cells | demyelination | multiple sclerosis | gray matter | remyelination | PROGENITOR CELLS | MULTIPLE-SCLEROSIS LESIONS | STEM-CELLS | WHITE-MATTER | CNS | NEUROSCIENCES | GLIA GENERATE | AXONAL LOSS | CORPUS-CALLOSUM | GRAY-MATTER | Age Factors | RNA, Untranslated - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Hippocampus - drug effects | Cell Lineage - drug effects | RNA, Untranslated - genetics | Cell Differentiation - genetics | Oligodendrocyte Precursor Cells - drug effects | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Cell Lineage - genetics | Disease Models, Animal | Cuprizone - toxicity | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Mice, Inbred C57BL | Bacterial Proteins - genetics | Corpus Callosum - drug effects | Mice, Transgenic | Hippocampus - pathology | Monoamine Oxidase Inhibitors - toxicity | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Corpus Callosum - pathology | Remyelination - physiology | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Remyelination - drug effects | Luminescent Proteins - metabolism | Medicine, Experimental | Medical research | Multiple sclerosis | Platelet-derived growth factor | Cortex (cingulate) | Myelin | Central nervous system | Substantia alba | Substantia grisea | Plasticity (axonal) | Glial stem cells | Corpus callosum | Atrophy | Regeneration | Myelination | Cuprizone | Sheaths | Demyelination | Oligodendrocytes | Cells (biology) | In vivo methods and tests | Differentiation | Cortex (temporal)
Journal Article
Experimental Neurology, ISSN 0014-4886, 2005, Volume 192, Issue 1, pp. 11 - 24
Chronic demyelination is a pathophysiologic component of compressive spinal cord injury (SCI) and a characteristic finding in demyelinating diseases including... 
NG2 | Radial glia | Demyelination | Remyelination | Nkx2.2 | Oligodendrocyte precursor cell | HOMEODOMAIN TRANSCRIPTION FACTOR | radial glia | TYPE-1 ASTROCYTES | INTERMEDIATE FILAMENT | CNS DEMYELINATION | GLIAL-CELLS | NEUROSCIENCES | oligodendrocyte precursor cell | PROGENITOR-CELL | demyelination | BROMIDE-INDUCED DEMYELINATION | CENTRAL-NERVOUS-SYSTEM | NEURAL STEM-CELL | SCHWANN-CELLS | remyelination | Oligodendroglia - metabolism | Cell Proliferation | Spinal Cord - metabolism | Cell Count | Nerve Regeneration - physiology | Rats, Inbred F344 | Cell Communication - physiology | Demyelinating Diseases - metabolism | Stem Cells - metabolism | Oligodendroglia - ultrastructure | Myelin Sheath - metabolism | Spinal Cord - pathology | Nerve Tissue Proteins - biosynthesis | Female | Schwann Cells - physiology | Demyelinating Diseases - chemically induced | SOXB1 Transcription Factors | Cell Differentiation - physiology | Basic Helix-Loop-Helix Transcription Factors | Disease Models, Animal | Ethidium | Demyelinating Diseases - physiopathology | Neurotoxins | Homeodomain Proteins - biosynthesis | Gene Expression Regulation | Rats | Transcription Factors - biosynthesis | Disease Progression | Stem Cells - ultrastructure | Astrocytes - ultrastructure | Antigens, Differentiation - biosynthesis | Animals | Schwann Cells - ultrastructure | High Mobility Group Proteins - biosynthesis | Myelin Sheath - ultrastructure | Spinal Cord - physiopathology | DNA-Binding Proteins - biosynthesis | Astrocytes - metabolism
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7673, pp. 533 - 537
High-grade gliomas (HGG) are a devastating group of cancers, and represent the leading cause of brain tumour-related death in both children and adults.... 
AUTISM | PEPTIDES | PHOSPHORYLATION | DISINTEGRIN | SUBTYPES | MULTIDISCIPLINARY SCIENCES | OLIGODENDROCYTE PRECURSOR CELLS | MUTATION | HIPPOCAMPAL | SELF-RENEWAL | CLEAVAGE | Neoplasm Transplantation | Neurons - pathology | Oligodendroglia - metabolism | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Nerve Tissue Proteins - deficiency | Brain Neoplasms - pathology | ADAM10 Protein - metabolism | Tumor Microenvironment | Male | Phosphatidylinositol 3-Kinases - metabolism | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Adhesion Molecules, Neuronal - deficiency | Glioma - genetics | Membrane Proteins - deficiency | ADAM10 Protein - antagonists & inhibitors | Heterografts | Glioma - pathology | Adult | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Neurons - metabolism | Oligodendroglia - cytology | Child | Signal Transduction | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Membrane Proteins - genetics | Brain Neoplasms - genetics | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Membrane Proteins - antagonists & inhibitors | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Gliomas | Health aspects | Research | TOR protein | Brain | Phosphorylation | Transcription | Brain tumors | Brain cancer | Genes | Glioblastoma | Genomes | Kinases | Glial stem cells | Proteins | Signal transduction | Cell growth | Proteolysis | Glioma cells | Xenografts | Children | Secretion | Adhesion | 1-Phosphatidylinositol 3-kinase | Oligodendroglioma | Signaling | Proteomics | Focal adhesion kinase
Journal Article
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, ISSN 1422-0067, 09/2019, Volume 20, Issue 18, p. 4486
Aspartate-Glutamate Carrier 1 (AGC1) deficiency is a rare neurological disease caused by mutations in the solute carrier family 25, member 12 (SLC25A12) gene,... 
subventricular zone | AGC1 deficiency | HYPOMYELINATION | PROTEIN | mouse model | growth factors | WHITE-MATTER | MYELINATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KETOGENIC DIET | CHEMISTRY, MULTIDISCIPLINARY | mitochondrial disease | SHUTTLE | N-ACETYLASPARTATE | CALCIUM | GROWTH-FACTOR | Antiporters - deficiency | Cell Line | Cell Proliferation | Reactive Oxygen Species - metabolism | Down-Regulation | Amino Acid Transport Systems, Acidic - metabolism | Gene Silencing | Amino Acid Transport Systems, Acidic - deficiency | Antiporters - metabolism | Lateral Ventricles - metabolism | Mitochondria - metabolism | Animals | Lactates - metabolism | Oligodendrocyte Precursor Cells - cytology | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial | Platelet-Derived Growth Factor | Cell Differentiation | Mice | Neurons - metabolism | Oligodendrocyte Precursor Cells - metabolism | Transforming Growth Factor beta - metabolism | Cell proliferation | Brain | Animal models | Cerebral cortex | Differentiation (biology) | Retina | Glial stem cells | Visual system | Mitochondria | Cell growth | Precursors | Cell cycle | Trophic factors | Dopamine receptors | Recombinant | Dopamine | Axonogenesis | Cloning | Cell differentiation | Calcium (mitochondrial) | Carbachol | Morphology | In vivo methods and tests | Mutation | Hippocampus | Calcium ions
Journal Article
Glia, ISSN 0894-1491, 08/2007, Volume 55, Issue 10, pp. 1001 - 1010
Journal Article