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1. Full Text Glial Membranes at the Node of Ranvier Prevent Neurite Outgrowth
by Jeffrey K. Huang and Greg R. Phillips and Alejandro D. Roth and Liliana Pedraza and Weisong Shan and Wiam Belkaid and Sha Mi and Asa Fex-Svenningsen and Laurence Florens and John R. Yates and David R. Colman and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för neurovetenskap
Science, ISSN 0036-8075, 12/2005, Volume 310, Issue 5755, pp. 1813 - 1817
Nodes of Ranvier are regularly placed, nonmyelinated axon segments along myelinated nerves. Here we show that nodal membranes isolated from the central nervous... 
Axons | Spinal cord | Neurites | Neuroscience | Sprouting | Myelin | Neurons | Neuroglia | Central nervous system | Reports | White matter | LOCALIZATION | PROTEIN | CHONDROITIN SULFATE PROTEOGLYCANS | MULTIDISCIPLINARY SCIENCES | CNS MYELIN | GROWTH | AXONAL REGENERATION | OLIGODENDROCYTE-MYELIN GLYCOPROTEIN | RAT OPTIC-NERVE | CELL | ADULT | Neuroglia - ultrastructure | Neurites - physiology | Humans | GPI-Linked Proteins | Axons - physiology | Oligodendroglia - ultrastructure | Ranvier's Nodes - ultrastructure | Oligodendroglia - physiology | Antigens - analysis | Cattle | Neurites - ultrastructure | Ranvier's Nodes - chemistry | Axons - ultrastructure | Spinal Cord - cytology | Oligodendroglia - chemistry | Neuroglia - chemistry | Cell Surface Extensions - ultrastructure | Cell Surface Extensions - chemistry | Cells, Cultured | Neuroglia - physiology | Ganglia, Spinal - physiology | Myelin-Oligodendrocyte Glycoprotein | Rats | Proteoglycans - analysis | Ganglia, Spinal - ultrastructure | Myelin-Associated Glycoprotein - analysis | Animals | Proteomics | Myelin Proteins | Ranvier's Nodes - physiology | Mice | Myelin Sheath - chemistry | Cell Surface Extensions - physiology | Growth | Analysis | Nerves | Inflammation | Research | Structure | Neurology | Brain | Antigens/analysis | Cells; Cultured | Neurites/physiology/ultrastructure | Myelin Sheath/chemistry | Oligodendroglia/chemistry/physiology/ultrastructure | Ranvier's Nodes/chemistry/physiology/ultrastructure | Myelin-Associated Glycoprotein/analysis | Ganglia; Spinal/physiology/ultrastructure | Spinal Cord/cytology | Neuroglia/chemistry/physiology/ultrastructure | Proteoglycans/analysis | Axons/physiology/ultrastructure | Cell Surface Extensions/chemistry/physiology/ultrastructure
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2. Full Text Elevated phosphatidylinositol 3,4,5-trisphosphate in glia triggers cell-autonomous membrane wrapping and myelination
by Goebbels, Sandra and Oltrogge, Jan H and Kemper, Robert and Heilmann, Ingo and Bormuth, Ingo and Wolfer, Susanne and Wichert, Sven P and Möbius, Wiebke and Liu, Xin and Lappe-Siefke, Corinna and Rossner, Moritz J and Groszer, Matthias and Suter, Ueli and Frahm, Jens and Boretius, Susann and Nave, Klaus-Armin
Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 26, pp. 8953 - 8964
In the developing nervous system, constitutive activation of the AKT/mTOR (mammalian target of rapamycin) pathway in myelinating glial cells is associated with... 
MAMMALIAN TARGET | AXONAL REGULATION | PHOSPHATASE | TUMOR-SUPPRESSOR GENE | TENSIN HOMOLOG | NORMAL CNS MYELINATION | CENTRAL-NERVOUS-SYSTEM | SCHWANN-CELLS | CHROMOSOME-10 PTEN | NEUROSCIENCES | OLIGODENDROCYTES | Sciatic Nerve - physiology | Neuroglia - ultrastructure | Phosphatidylinositol Phosphates - metabolism | Cell Count | Sciatic Nerve - ultrastructure | Axons - physiology | Nerve Fibers, Myelinated - ultrastructure | Brain - physiology | Oligodendroglia - ultrastructure | Oligodendroglia - physiology | Axons - ultrastructure | Brain - ultrastructure | Nerve Fibers, Myelinated - physiology | Aging | Schwann Cells - physiology | PTEN Phosphohydrolase - genetics | Myelin Sheath - physiology | Neuroglia - physiology | PTEN Phosphohydrolase - metabolism | Mice, Transgenic | Collagen - metabolism | Animals | Schwann Cells - ultrastructure | Mice | Myelin Sheath - ultrastructure
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3. Full Text Changes in prefrontal axons may disrupt the network in autism
by Zikopoulos, Basilis and Barbas, Helen
Journal of Neuroscience, ISSN 0270-6474, 11/2010, Volume 30, Issue 44, pp. 14595 - 14609
Neural communication is disrupted in autism by unknown mechanisms. Here, we examined whether in autism there are changes in axons, which are the conduit for... 
DEVELOPING RHESUS-MONKEY | MATTER VOLUME INCREASE | EFFERENT ASSOCIATION PATHWAYS | WHITE-MATTER | SENTENCE COMPREHENSION | ANTERIOR CINGULATE | SPECTRUM DISORDERS | GAP-43 MESSENGER-RNA | ORBITOFRONTAL CORTEX | NEUROSCIENCES | WORKING-MEMORY | Neurons - pathology | Autistic Disorder - pathology | Autistic Disorder - metabolism | Nerve Net - abnormalities | Humans | Male | Nerve Fibers, Myelinated - ultrastructure | GAP-43 Protein - ultrastructure | Neural Pathways - pathology | Prefrontal Cortex - growth & development | Oligodendroglia - ultrastructure | Prefrontal Cortex - pathology | Gyrus Cinguli - abnormalities | Neurons - ultrastructure | Nerve Fibers, Myelinated - pathology | Gyrus Cinguli - pathology | Axons - ultrastructure | Adult | Female | Biomarkers - metabolism | Nerve Net - growth & development | Microscopy, Electron, Transmission | GAP-43 Protein - metabolism | Neural Pathways - growth & development | Prefrontal Cortex - abnormalities | Nerve Net - pathology | Oligodendroglia - pathology | Gyrus Cinguli - growth & development | Axons - pathology | Neural Pathways - ultrastructure
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4. Full Text Ceramide Triggers Budding of Exosome Vesicles into Multivesicular Endosomes
by Katarina Trajkovic and Chieh Hsu and Salvatore Chiantia and Lawrence Rajendran and Dirk Wenzel and Felix Wieland and Petra Schwille and Britta Brügger and Mikael Simons
Science, ISSN 0036-8075, 2/2008, Volume 319, Issue 5867, pp. 1244 - 1247
Intraluminal vesicles of multivesicular endosomes are either sorted for cargo degradation into lysosomes or secreted as exosomes into the extracellular milieu.... 
Ceramides | Lipids | Reports | Cell membranes | Exosomes | Budding | Cholesterols | Freight | Cells | Endosomes | P branes | MICRODOMAINS | TSG101 | BIOGENESIS | PATHWAY | MULTIDISCIPLINARY SCIENCES | HRS | RAFT | ENDOCYTOSIS | Cell Line | Oligodendroglia - metabolism | Ceramides - metabolism | Membrane Microdomains - metabolism | Humans | Ceramides - analysis | Cytoplasmic Vesicles - chemistry | Membrane Microdomains - ultrastructure | Recombinant Fusion Proteins - metabolism | Endosomes - metabolism | Oligodendroglia - ultrastructure | Intracellular Membranes - ultrastructure | Protein Transport | Receptor, Epidermal Growth Factor - metabolism | Animals | Sphingomyelin Phosphodiesterase - antagonists & inhibitors | Cytoplasmic Vesicles - ultrastructure | Sphingomyelin Phosphodiesterase - metabolism | Cytoplasmic Vesicles - metabolism | Endosomes - ultrastructure | Myelin Proteolipid Protein - metabolism | Cell Line, Tumor | Mice | Intracellular Membranes - metabolism | Physiological aspects | Properties | Proteins | Enzymes | Membranes | Cellular biology | Molecular biology
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5. Full Text Ultrastructural alterations of oligodendrocytes in prefrontal white matter in schizophrenia: A post-mortem morphometric study
by Vikhreva, O.V and Rakhmanova, V.I and Orlovskaya, D.D and Uranova, N.A
Schizophrenia Research, ISSN 0920-9964, 2016, Volume 177, Issue 1-3, pp. 28 - 36
Abstract Objective Neuroimaging studies showed abnormalities in frontal white matter (WM) in schizophrenia that were associated with clinical symptoms.... 
Psychiatry | Schizophrenia | Oligodendrocyte | Morphometry | Ultrastructure | White matter | Postmortem | CELLS | PSYCHIATRY | MITOCHONDRIA | PATHOLOGY | DENSITY | MYELIN | CORTEX | BRAIN METABOLISM | GENES | DYSFUNCTION | SPATIAL-DISTRIBUTION | Vacuoles - ultrastructure | Humans | Middle Aged | Male | Schizophrenia - pathology | Microscopy, Electron | White Matter - ultrastructure | Mitochondria - ultrastructure | Oligodendroglia - ultrastructure | Prefrontal Cortex - ultrastructure | Aged, 80 and over | Adult | Female | Aged | Medicine, Experimental | Medical research | Antipsychotic drugs | Analysis
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6. Full Text Axon-glial signaling and the glial support of axon function
by Nave, Klaus-Armin and Trapp, Bruce D
Annual Review of Neuroscience, ISSN 0147-006X, 2008, Volume 31, Issue 1, pp. 535 - 561
Oligodendrocytes and Schwann cells are highly specialized glial cells that wrap axons with a multilayered myelin membrane for rapid impulse conduction.... 
Energy metabolism | Myelination | Neurodegenerative diseases | Oligodendrocytes | Schwann cells | Axonal transport | Growth factors | CELL-ADHESION MOLECULE | MARIE-TOOTH-DISEASE | growth factors | PROTEOLIPID PROTEIN GENE | oligodendrocytes | NEUROSCIENCES | MYELIN-ASSOCIATED GLYCOPROTEIN | PROSTAGLANDIN-D SYNTHASE | neurodegenerative diseases | MICE LACKING GDNF | myelination | axonal transport | PELIZAEUS-MERZBACHER-DISEASE | CENTRAL-NERVOUS-SYSTEM | 2',3'-CYCLIC NUCLEOTIDE 3'-PHOSPHODIESTERASE | CILIARY NEUROTROPHIC FACTOR | energy metabolism | Nerve Fibers, Myelinated - metabolism | Oligodendroglia - metabolism | Demyelinating Diseases - genetics | Humans | Nervous System - metabolism | Nervous System - ultrastructure | Axons - metabolism | Cell Survival - genetics | Cell Communication - physiology | Nerve Fibers, Myelinated - ultrastructure | Schwann Cells - metabolism | Cell Communication - genetics | Cell Survival - immunology | Myelin Proteins - genetics | Oligodendroglia - ultrastructure | Animals | Schwann Cells - ultrastructure | Axons - ultrastructure | Demyelinating Diseases - immunology | Signal Transduction - physiology | Myelin Proteins - metabolism | Axons | Analysis | Neuroglia
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7. Full Text Impaired adult myelination in the prefrontal cortex of socially isolated mice
by Liu, Jia and Dietz, Karen and Deloyht, Jacqueline M and Pedre, Xiomara and Kelkar, Dipti and Kaur, Jasbir and Vialou, Vincent and Lobo, Mary Kay and Dietz, David M and Nestler, Eric J and Dupree, Jeffrey and Casaccia, Patrizia
Nature Neuroscience, ISSN 1097-6256, 12/2012, Volume 15, Issue 12, pp. 1621 - 1623
Protracted social isolation of adult mice induced behavioral, transcriptional and ultrastructural changes in oligodendrocytes of the prefrontal cortex (PFC)... 
DEPRESSION | REMYELINATION | DISORDERS | WHITE-MATTER | OLIGODENDROCYTE PROGENITOR DIFFERENTIATION | NEUROSCIENCES | Age Factors | Myelin Sheath - pathology | Social Isolation - psychology | Mice, Inbred C57BL | Male | Nerve Fibers, Myelinated - ultrastructure | Oligodendroglia - ultrastructure | Prefrontal Cortex - ultrastructure | Prefrontal Cortex - pathology | Oligodendroglia - pathology | Animals | Nerve Fibers, Myelinated - pathology | Mice | Myelin Sheath - ultrastructure | Chromatin | Myelination | Social isolation | Physiological aspects | Diagnosis | Prefrontal cortex | Properties | Health aspects | Myelin proteins
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8. Full Text Global Deprivation of Brain-Derived Neurotrophic Factor in the CNS Reveals an Area-Specific Requirement for Dendritic Growth
by Rauskolb, Stefanie and Zagrebelsky, Marta and Dreznjak, Anita and Deogracias, Ruben and Matsumoto, Tomoya and Wiese, Stefan and Erne, Beat and Sendtner, Michael and Schaeren-Wiemers, Nicole and Korte, Martin and Barde, Yves-Alain
Journal of Neuroscience, ISSN 0270-6474, 02/2010, Volume 30, Issue 5, pp. 1739 - 1749
Although brain-derived neurotrophic factor (BDNF) is linked with an increasing number of conditions causing brain dysfunction, its role in the postnatal CNS... 
BDNF | FACTOR MESSENGER-RNA | RAT HIPPOCAMPUS | PRIMARY SENSORY NEURONS | LONG-TERM POTENTIATION | DOPAMINERGIC-NEURONS | NERVE GROWTH | CONDITIONAL DELETION | SUBSTANTIA-NIGRA | NEUROSCIENCES | IN-SITU | Immunohistochemistry | Dendrites - metabolism | Neostriatum - growth & development | Neostriatum - ultrastructure | Cell Count | Cells, Cultured | tau Proteins - metabolism | Male | Neurons - cytology | Dendrites - ultrastructure | Optic Nerve - ultrastructure | Hippocampus - cytology | Oligodendroglia - ultrastructure | Mice, Knockout | Animals | Neurons - ultrastructure | Female | Brain-Derived Neurotrophic Factor - metabolism | Mice | Oligodendroglia - cytology | Hippocampus - growth & development | Optic Nerve - growth & development
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9. Full Text CNS/PNS boundary transgression by central glia in the absence of schwann cells or Krox20/Egr2 function
by Coulpier, Fanny and Decker, Laurence and Funalot, Benoît and Vallat, Jean-Michel and Garcia-Bragado, Federico and Charnay, Patrick and Topilko, Piotr
Journal of Neuroscience, ISSN 0270-6474, 04/2010, Volume 30, Issue 17, pp. 5958 - 5967
CNS/PNS interfaces constitute cell boundaries, because they delimit territories with different neuronal and glial contents. Despite their potential interest in... 
MIGRATION | MOTOR EXIT POINTS | HINDBRAIN | PERIPHERAL NERVOUS-SYSTEM | SPINAL-CORD | NEUROPATHIES | MECHANISMS | EXPRESSION | DERIVATIVES | NEUROSCIENCES | MYELIN GENE | Peripheral Nervous System - physiology | Central Nervous System - ultrastructure | Neuroglia - ultrastructure | Early Growth Response Protein 2 - genetics | Humans | Infant | Axons - physiology | Cell Movement - physiology | Oligodendroglia - ultrastructure | Central Nervous System - physiology | Oligodendroglia - physiology | Spinal Nerve Roots - physiology | Axons - ultrastructure | Schwann Cells - physiology | Peripheral Nervous System Diseases - pathology | Peripheral Nervous System - ultrastructure | Myelin Sheath - physiology | Neuroglia - physiology | Mice, Transgenic | Spinal Nerve Roots - ultrastructure | Astrocytes - ultrastructure | Peripheral Nervous System Diseases - metabolism | Astrocytes - physiology | Animals | Schwann Cells - ultrastructure | Early Growth Response Protein 2 - metabolism | Mice | Myelin Sheath - ultrastructure | Schwann Cells | Neuroglia | Neurons and Cognition | Genomics | Neurobiology | Peripheral Nervous System | Life Sciences | Early Growth Response Protein 2 | Genetics | Astrocytes | Biochemistry, Molecular Biology | Axons | Central Nervous System | Peripheral Nervous System Diseases | Myelin Sheath | Oligodendroglia | Spinal Nerve Roots | Molecular biology | Human genetics | Cell Movement | Cancer
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10. Full Text Myelin Impairs CNS Remyelination by Inhibiting Oligodendrocyte Precursor Cell Differentiation
by Kotter, Mark R and Li, Wen-Wu and Zhao, Chao and Franklin, Robin J. M
Journal of Neuroscience, ISSN 0270-6474, 01/2006, Volume 26, Issue 1, pp. 328 - 332
Demyelination in the adult CNS can be followed by extensive repair. However, in multiple sclerosis, the differentiation of oligodendrocyte lineage cells... 
Regeneration | Differentiation | Myelin | Remyelination | Macrophage | Oligodendrocyte precursor cell | myelin | PROGENITOR CELLS | RAT SPINAL-CORD | MULTIPLE-SCLEROSIS LESIONS | differentiation | NEUROTROPHIN RECEPTOR | ADULT CNS | regeneration | macrophage | P75(NTR) | NOGO RECEPTOR | NEUROSCIENCES | oligodendrocyte precursor cell | DEMYELINATION | ETHIDIUM-BROMIDE | EXPRESSION | remyelination | Central Nervous System - ultrastructure | Myelin Sheath - physiology | Rats | Nerve Fibers, Myelinated - ultrastructure | Rats, Sprague-Dawley | Oligodendroglia - ultrastructure | Stem Cells - ultrastructure | Central Nervous System - physiology | Neural Inhibition - physiology | Animals | Oligodendroglia - physiology | Nerve Fibers, Myelinated - physiology | Growth Inhibitors - physiology | Female | Stem Cells - physiology | Myelin Sheath - ultrastructure | Cell Differentiation - physiology | Brief Communications
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11. Full Text Early ultrastructural defects of axons and axon–glia junctions in mice lacking expression of Cnp1
by Edgar, Julia M and McLaughlin, Mark and Werner, Hauke B and McCulloch, Mailis C and Barrie, Jennifer A and Brown, Angus and Faichney, Andrew Blyth and Snaidero, Nicolas and Nave, Klaus‐Armin and Griffiths, Ian R
Glia, ISSN 0894-1491, 12/2009, Volume 57, Issue 16, pp. 1815 - 1824
Most axons in the central nervous system (CNS) are surrounded by a multilayered myelin sheath that promotes fast, saltatory conduction of electrical impulses.... 
myelin | inner tongue | proteolipid protein | morphometric analysis | oligodendrocyte | Inner tongue | Oligodendrocyte | Myelin | Morphometric analysis | Proteolipid protein | MOUSE OPTIC-NERVE | WHITE-MATTER | HEREDITARY SPASTIC PARAPLEGIA | MEMBRANE ANCHOR | NEUROSCIENCES | MYELIN-ASSOCIATED GLYCOPROTEIN | MULTIPLE-SCLEROSIS | CNS MYELIN | CENTRAL-NERVOUS-SYSTEM | 2',3'-CYCLIC NUCLEOTIDE 3'-PHOSPHODIESTERASE | Oligodendroglia - metabolism | Spinal Cord - metabolism | Electrophysiology | Nerve Fibers, Myelinated - ultrastructure | Spinal Cord - ultrastructure | Oligodendroglia - ultrastructure | Myelin Sheath - metabolism | Axons - ultrastructure | Myelin Proteolipid Protein - metabolism | Nerve Fibers, Myelinated - metabolism | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Cell Survival | Intercellular Junctions - metabolism | Axons - metabolism | Optic Nerve - ultrastructure | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | Microscopy, Electron | Mice, Knockout | Action Potentials - physiology | Animals | Analysis of Variance | Myelin Proteolipid Protein - genetics | Myelin Sheath - genetics | Mice | Myelin Sheath - ultrastructure | Intercellular Junctions - ultrastructure | Optic Nerve - metabolism
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12. Full Text Oligodendrocyte PTEN is required for myelin and axonal integrity, not remyelination
by Harrington, Emily P and Zhao, Chao and Fancy, Stephen P. J and Kaing, Sovann and Franklin, Robin J. M and Rowitch, David H
Annals of Neurology, ISSN 0364-5134, 11/2010, Volume 68, Issue 5, pp. 703 - 716
Objective Repair of myelin injury in multiple sclerosis may fail, resulting in chronic demyelination, axonal loss, and disease progression. As cellular... 
MAMMALIAN TARGET | NERVOUS-SYSTEM | ADULT CNS | MULTIPLE-SCLEROSIS | CNS REMYELINATION | GENE | PATHWAY | INDUCED DEMYELINATION | DIFFERENTIATION | TRANSCRIPTION FACTOR | NEUROSCIENCES | CLINICAL NEUROLOGY | Axons - enzymology | Age Factors | Humans | PTEN Phosphohydrolase - physiology | Nerve Fibers, Myelinated - ultrastructure | Spinal Cord - ultrastructure | Oligodendroglia - ultrastructure | Myelin Sheath - metabolism | PTEN Phosphohydrolase - antagonists & inhibitors | Oligodendroglia - physiology | Nerve Fibers, Myelinated - pathology | Spinal Cord - pathology | Gene Deletion | Brain - ultrastructure | Nerve Fibers, Myelinated - physiology | Demyelinating Diseases - pathology | Disease Models, Animal | Demyelinating Diseases - physiopathology | Lysophosphatidylcholines | Demyelinating Diseases - enzymology | Myelin Sheath - pathology | Mice, Transgenic | Oligodendroglia - pathology | Animals | Signal Transduction - drug effects | Axons - pathology | Brain - pathology | Oligodendroglia - enzymology | Signal Transduction - physiology | Mice | Myelin Sheath - ultrastructure | Cell Line, Transformed
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13. Full Text The role of oligodendrocyte pathology in schizophrenia
by Uranova, Natalya A and Vostrikov, Victor M and Vikhreva, Olga V and Zimina, Ivetta S and Kolomeets, Natalya S and Orlovskaya, Diana D
The International Journal of Neuropsychopharmacology, ISSN 1461-1457, 8/2007, Volume 10, Issue 4, pp. 537 - 545
Neuroimaging and microarray studies provide evidence for myelin and oligodendrocyte abnormalities in schizophrenia (SZ). Electron microscopy demonstrated... 
Schizophrenia | Post-mortem | Oligodendrocytes | DORSOLATERAL PREFRONTAL CORTEX | ANTERIOR CINGULATE CORTEX | NEUROPATHOLOGY CONSORTIUM | PSYCHIATRY | BIPOLAR-DISORDER | oligodendrocytes | MAJOR-DEPRESSIVE-DISORDER | schizophrenia | NEUROSCIENCES | CLINICAL NEUROLOGY | post-mortem | PYRAMIDAL NEURONS | MOOD DISORDERS | ADULT-RAT | GLIAL-CELL DENSITY | PHARMACOLOGY & PHARMACY | GRAY-MATTER | Caudate Nucleus - ultrastructure | Humans | Middle Aged | Nerve Fibers, Myelinated - ultrastructure | Schizophrenia - pathology | Microscopy, Electron | Case-Control Studies | Oligodendroglia - ultrastructure | Prefrontal Cortex - ultrastructure | Necrosis | Atrophy | Brain - ultrastructure | Hippocampus - ultrastructure | Myelin Sheath - ultrastructure | Cadaver | Apoptosis
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14. Full Text Accumulation of macrophage-like cells expressing NG2 proteoglycan and Iba1 in ischemic core of rat brain after transient middle cerebral artery occlusion
by Matsumoto, Hiroaki and Kumon, Yoshiaki and Watanabe, Hideaki and Ohnishi, Takanori and Shudou, Masachika and Chuai, Miao and Imai, Yoshinori and Takahashi, Hisaaki and Tanaka, Junya
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 01/2008, Volume 28, Issue 1, pp. 149 - 163
Although neurons and glia inevitably undergo degeneration in the core of ischemic lesions, many cells, particularly immune cells, infiltrate the core and... 
Monocytes | Iba1 | Ischemia | Ki67 | Microglia | NG2 glia | monocytes | PROTEIN | MULTIPOTENT | INJURY | SPINAL-CORD | PROLIFERATION | GLIAL-CELLS | ASTROCYTES | NEUROSCIENCES | ischemia | ENDOTHELIAL PROGENITOR CELLS | ENDOCRINOLOGY & METABOLISM | CHONDROITIN SULFATE PROTEOGLYCAN | ACTIVATED MICROGLIA | HEMATOLOGY | microglia | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Male | Monocytes - metabolism | Heterochromatin - ultrastructure | Infarction, Middle Cerebral Artery - metabolism | Stem Cells - metabolism | Oligodendroglia - ultrastructure | Heterochromatin - metabolism | Time Factors | Phagosomes - ultrastructure | Cell Membrane - metabolism | Monocytes - ultrastructure | Macrophages - ultrastructure | Calcium-Binding Proteins - biosynthesis | Phagosomes - metabolism | Gene Expression Regulation | Proteoglycans - biosynthesis | Rats | Cell Membrane - ultrastructure | Infarction, Middle Cerebral Artery - pathology | Stem Cells - ultrastructure | Regeneration | Antigens, Differentiation - biosynthesis | Macrophages - metabolism | Animals | Microfilament Proteins | Microglia - ultrastructure | Antigens - biosynthesis
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