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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2005, Volume 102, Issue 31, pp. 11011 - 11016
Journal Article
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 1, pp. 140 - 150.e7
Journal Article
European Journal of Haematology, ISSN 0902-4441, 05/2011, Volume 86, Issue 5, pp. 361 - 371
Journal Article
Blood, ISSN 0006-4971, 02/2016, Volume 127, Issue 17, pp. 2131 - 2143
Leukemias expressing constitutively activated mutants of ABL1 tyrosine kinase (BCR-ABL1, TEL-ABL1, NUP214-ABL1) usually contain at least 1 normal ABL1 allele.... 
CHRONIC MYELOGENOUS LEUKEMIA | GENOMIC INSTABILITY | APOPTOTIC RESPONSE | CELL-DEATH RESPONSE | C-ABL | TYROSINE KINASE | ACUTE LYMPHOBLASTIC-LEUKEMIA | BCR-ABL | DNA-DAMAGE | HEMATOLOGY | CHRONIC MYELOID-LEUKEMIA | Oxidative Stress | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myeloid, Chronic-Phase - pathology | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Proto-Oncogene Proteins c-abl - physiology | Imatinib Mesylate - therapeutic use | Imidazoles - therapeutic use | Neoplasm Proteins - genetics | Genes, Tumor Suppressor | Proto-Oncogene Proteins c-abl - genetics | Blast Crisis - pathology | Imatinib Mesylate - pharmacology | Leukemia, Experimental - drug therapy | Oncogene Proteins v-abl - genetics | Leukemia, Myeloid, Chronic-Phase - drug therapy | Gene Expression Regulation, Leukemic - drug effects | Imidazoles - pharmacology | Leukemia, Myeloid, Chronic-Phase - enzymology | Cell Division - drug effects | Tumor Suppressor Proteins - physiology | Cell Line, Tumor | Mice, Inbred NOD | Blast Crisis - genetics | Mice | Leukemia, Myeloid, Chronic-Phase - genetics | Oncogene Proteins v-abl - physiology | Genomic Instability | Blast Crisis - drug therapy | Tumor Suppressor Proteins - antagonists & inhibitors | Neoplasm Proteins - physiology | Oncogene Proteins v-abl - antagonists & inhibitors | Leukemia, Experimental - enzymology | Pyridazines - pharmacology | Blast Crisis - enzymology | Oncogene Proteins, Fusion - physiology | Tumor Suppressor Proteins - genetics | Antineoplastic Agents - pharmacology | Cytostatic Agents - pharmacology | Pyridazines - therapeutic use | Leukemia, Experimental - genetics | Leukemia, Experimental - pathology | Mice, SCID | Animals | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Genes, abl | Oncogene Proteins, Fusion - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Neoplastic Stem Cells - enzymology | Myeloid Neoplasia
Journal Article
Nature Cell Biology, ISSN 1465-7392, 12/2004, Volume 6, Issue 12, pp. 1204 - 1211
Journal Article
Journal of Immunological Methods, ISSN 0022-1759, 12/2017, Volume 451, pp. 71 - 77
Antigen receptor gene assembly is accomplished in developing lymphocytes by the V(D)J recombination reaction, which can be separated into two steps: DNA... 
Nonhomologous end joining (NHEJ) | v-Abl transformed pro-B cells | Recombination-activating gene (RAG) endonuclease | CRISPR/Cas9-mediated gene knock-out | V(D)J recombination | STRAND BREAK REPAIR | XRCC4 | BIOCHEMICAL RESEARCH METHODS | IMMUNOLOGY | GENOME | XLF | PAXX | PATHWAY | LIGASE IV | MICE | END RESECTION | DIFFERENTIATION | Apoptosis - drug effects | Homeodomain Proteins - metabolism | Oncogene Proteins v-abl - antagonists & inhibitors | Precursor Cells, B-Lymphoid - metabolism | Precursor Cells, B-Lymphoid - drug effects | DNA Breaks, Double-Stranded | DNA-Binding Proteins - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Precursor Cells, B-Lymphoid - pathology | DNA End-Joining Repair | Clustered Regularly Interspaced Short Palindromic Repeats | Recombinational DNA Repair - drug effects | CRISPR-Associated Proteins - genetics | Imatinib Mesylate - pharmacology | Oncogene Proteins v-abl - genetics | Mice, Inbred C57BL | Genotype | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Oncogene Proteins v-abl - metabolism | Phenotype | Animals | CRISPR-Associated Proteins - metabolism | CRISPR-Cas Systems | Gene Editing - methods | Protein Kinase Inhibitors - pharmacology | G1 Phase Cell Cycle Checkpoints - drug effects | Proto-Oncogene Proteins c-bcl-2 - genetics | Cell Line, Transformed | CRISPR | Cas9-mediated gene knock-out
Journal Article