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Nature Medicine, ISSN 1078-8956, 05/2017, Volume 23, Issue 5, pp. 579 - 589
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2017, Volume 198, Issue 4, pp. 1484 - 1491
OncostatinM(OSM) is a pleiotropic cytokine and a member of the IL-6 family. It has both proinflammatory and anti-inflammatory functions and is involved in the... 
COLLAGEN-INDUCED ARTHRITIS | TREG | LEUKEMIA INHIBITORY FACTOR | MICE | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | TH17 CELLS | PLASTICITY | Interleukin-17 - immunology | Oncostatin M - physiology | Oncostatin M - pharmacology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Interleukins - genetics | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Th17 Cells - drug effects | Interleukin-2 - immunology | Interleukins - immunology | Suppressor of Cytokine Signaling 3 Protein - immunology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Down-Regulation | Gene Expression Regulation | Interleukin-17 - genetics | Suppressor of Cytokine Signaling 3 Protein - genetics | Arthritis, Experimental | Collagen - administration & dosage | T-Lymphocytes, Regulatory - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Th17 Cells - immunology | Mice | CD4-Positive T-Lymphocytes - drug effects | Oncostatin M - genetics | Cell proliferation | Cytokines | Immunoregulation | Helper cells | Stat3 protein | Activation | Lymphocytes T | Inflammation | Arthritis | Stat5 protein | Oncostatin M | Interleukin 17 | CD4 antigen | Interleukin 6 | Signaling | Cell activation | SOCS-3 protein | Interleukin 2 | Lymphocytes | Rheumatoid arthritis | Collagen | Interleukin 21 | Immune Regulation
Journal Article
STEM CELLS, ISSN 1066-5099, 04/2012, Volume 30, Issue 4, pp. 762 - 772
Bone resorption by osteoclasts and bone formation by osteoblasts are tightly coupled processes implicating factors in TNF, bone morphogenetic protein, and Wnt... 
Bone | Monocyte | Oncostatin M | Mesenchymal stem cells | RHEUMATOID-ARTHRITIS | OSTEOBLAST DIFFERENTIATION | LEUKEMIA INHIBITORY FACTOR | CELL & TISSUE ENGINEERING | CELL BIOLOGY | INTERLEUKIN-6 FAMILY | ONCOLOGY | BONE-FORMATION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | FRACTURE REPAIR | IN-VIVO | RECEPTOR ACTIVATOR | STROMAL CELLS | HEMATOLOGY | KAPPA-B LIGAND | Bone Matrix - metabolism | Monocytes - cytology | Humans | Middle Aged | Male | Monocytes - metabolism | Leukemia Inhibitory Factor - metabolism | Mesenchymal Stromal Cells - cytology | Bone Matrix - drug effects | Adenoviridae - genetics | Oncostatin M - metabolism | Adult | Interleukin-6 - metabolism | Gene Transfer Techniques | Mesenchymal Stromal Cells - drug effects | Calcification, Physiologic - drug effects | Mice, Inbred C57BL | Osteogenesis - drug effects | Mesenchymal Stromal Cells - metabolism | Dinoprostone - metabolism | Gene Expression Regulation - drug effects | Monocytes - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Aged | Mice | Macrophage Activation - drug effects | Adenoviridae - drug effects | Adenoviridae | Cyclooxygenase 2 | Dinoprostone | Rhumatology and musculoskeletal system | Mesenchymal Stem Cells | Interleukin-6 | Lipopolysaccharides | Life Sciences | Cell Differentiation | Bone Matrix | Leukemia Inhibitory Factor | Signal Transduction | Gene Expression Regulation | Macrophage Activation | Human health and pathology | Monocytes | Calcification, Physiologic | Osteogenesis
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2015, Volume 19, Issue 6, pp. 1296 - 1307
Oncostatin M (OSM) exhibits many unique biological activities by activating Oβ receptor. However, its role in myocardial I/R injury in diabetic mice remains... 
inositol pyrophosphate 7 | oncostatin M | diabetes | ischaemia/Reperfusion injury | Inositol pyrophosphate 7 | Ischaemia/Reperfusion injury | Diabetes | Oncostatin M | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | MYOCARDIAL-INFARCTION | MYOCYTES | MEDIATOR | Reperfusion injury | CELL BIOLOGY | HEART | SKELETAL-MUSCLE | ischaemia | INHIBITION | REDUCTION | DYSFUNCTION | TANSHINONE IIA PRETREATMENT | Inositol Phosphates - antagonists & inhibitors | Apoptosis - drug effects | Receptors, Oncostatin M - metabolism | Diabetes Mellitus, Experimental - genetics | Insulin Resistance | Apoptosis - genetics | Myocardium - pathology | Mice, 129 Strain | Oncostatin M - pharmacology | Blotting, Western | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Organelle Biogenesis | Animals | Receptors, Oncostatin M - genetics | Mice, Mutant Strains | Protective Agents - pharmacology | Myocardium - metabolism | Glucose - metabolism | Inositol Phosphates - metabolism | Myocardial Reperfusion Injury - genetics | Homeostasis - genetics | Myocardial Reperfusion Injury - prevention & control | Diabetes Mellitus, Experimental - physiopathology | Phosphates | Inositol | Biosynthesis | Glucose | Insulin | Dextrose | Apoptosis | Rodents | Cardiomyocytes | Kinases | Statins | Heart | Bax protein | Diabetes mellitus | Adenine | AKT protein | 1-Phosphatidylinositol 3-kinase | Mitochondria | Sensitivity | Reperfusion | Ischemia | Protein kinase | Insulin resistance | Mice | Inhibition | Injuries | Original
Journal Article
Journal Article
by Guo, L and Chen, C and Shi, M and Wang, F and Chen, X and Diao, D and Hu, M and Yu, M and Qian, L and Guo, N
Oncogene, ISSN 0950-9232, 11/2013, Volume 32, Issue 45, pp. 5272 - 5282
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2013, Volume 288, Issue 30, pp. 21861 - 21875
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2011, Volume 9, Issue 5, pp. 420 - 432
Journal Article
Oncogene, ISSN 0950-9232, 07/2017, Volume 36, Issue 28, pp. 4001 - 4013
Journal Article