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Journal Article
PloS one, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e99299 - e99299
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 08/2009, Volume 106, Issue 32, pp. 13469 - 13474
Journal Article
Journal Article
Nature neuroscience, ISSN 1546-1726, 05/2006, Volume 9, Issue 6, pp. 843 - 852
The optic nerve, like most mature CNS pathways, does not regenerate after injury... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Growth Substances - secretion | Optic Nerve - cytology | Calcium - metabolism | Calcium Signaling - physiology | Nerve Regeneration - physiology | Rats, Inbred F344 | Cell Communication - physiology | Culture Media, Conditioned - pharmacology | Male | Ganglia, Spinal - cytology | Nerve Growth Factors - metabolism | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - cytology | Macrophages - secretion | Calcium-Calmodulin-Dependent Protein Kinase Type 1 | Cyclic AMP - metabolism | Calcium-Binding Proteins - metabolism | Nerve Growth Factors - secretion | Cells, Cultured | Rats | Rats, Sprague-Dawley | Macrophages - metabolism | Animals | Growth Cones - metabolism | Growth Cones - ultrastructure | Signal Transduction - drug effects | Calcium Signaling - drug effects | Calcium-Binding Proteins - secretion | Calcium-Binding Proteins - physiology | Cell Communication - drug effects | Nerve Regeneration - drug effects | Signal Transduction - physiology | Transcriptional Activation - physiology | Growth Cones - drug effects | Ganglia, Spinal - drug effects | Optic Nerve - drug effects | Calcium-Calmodulin-Dependent Protein Kinases - metabolism | Retinal Ganglion Cells - drug effects | Ganglia, Spinal - metabolism | Growth Substances - metabolism | Optic Nerve - metabolism | Regeneration | Nervous system | Optic nerve | Research | Apoptosis | Index Medicus
Journal Article
Developmental cell, ISSN 1534-5807, 03/2017, Volume 40, Issue 6, pp. 566 - 582.e5
A lack of sufficient oligodendrocyte myelination contributes to remyelination failure in demyelinating disorders. miRNAs have been implicated in... 
microRNAs | experimental autoimmune encephalomyelitis | Lingo1 | myelination | miR-338 | gene regulatory network | miR-219 | remyelination | demyelinating injury | Etv5 | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Oligodendroglia - metabolism | Optic Nerve - pathology | Central Nervous System - metabolism | Spinal Cord - drug effects | Spinal Cord - metabolism | Demyelinating Diseases - genetics | Central Nervous System - pathology | Myelin Sheath - drug effects | MicroRNAs - metabolism | Cell Lineage - drug effects | Myelin Sheath - metabolism | Oligodendroglia - drug effects | Spinal Cord - pathology | Gene Deletion | Membrane Proteins - metabolism | Encephalomyelitis, Autoimmune, Experimental - genetics | Wound Healing - genetics | Demyelinating Diseases - pathology | Wound Healing - drug effects | Disease Models, Animal | Lecithins - pharmacology | Nerve Regeneration - genetics | Encephalomyelitis, Autoimmune, Experimental - pathology | Myelin Sheath - pathology | Mice, Inbred C57BL | Multiple Sclerosis - genetics | Optic Nerve - ultrastructure | Multiple Sclerosis - therapy | Disease Progression | Mice, Knockout | Nerve Tissue Proteins - metabolism | Phenotype | Animals | Cell Differentiation - drug effects | Nerve Regeneration - drug effects | Central Nervous System - drug effects | Multiple Sclerosis - pathology | MicroRNAs - genetics | Multiple sclerosis | Analysis | Biotin | Genetic engineering | Birth defects | Cells | Neurophysiology | Index Medicus
Journal Article
Neurotoxicity research, ISSN 1029-8428, 1/2017, Volume 31, Issue 1, pp. 31 - 45
Journal Article