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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2016, Volume 113, Issue 12, pp. E1738 - E1746
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 10, pp. 4039 - 4044
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2010, Volume 107, Issue 13, pp. 6064 - 6069
Journal Article
Cell Death and Disease, ISSN 2041-4889, 2014, Volume 5, Issue 5, pp. e1225 - e1225
The Rho/ROCK/LIMK pathway is central for the mediation of repulsive environmental signals in the central nervous system. Several studies using pharmacological... 
Regeneration | Retinal ganglion cells | ROCK2 | Axonal degeneration | Optic nerve crush | LIMK1 | APOPTOSIS | RHO-KINASE INHIBITION | SIGNALING PATHWAYS | ACTIVATION | regeneration | PHOSPHORYLATION | LIM-KINASE | axonal degeneration | optic nerve crush | CELL BIOLOGY | RETINAL GANGLION-CELLS | NEURITE OUTGROWTH | IN-VIVO | GROWTH | retinal ganglion cells | Dependovirus - genetics | Optic Nerve - pathology | Phosphorylation | Axons - enzymology | Calpain - metabolism | Rats, Wistar | Neurites - enzymology | Caspase 3 - metabolism | Autophagy | Gene Knockdown Techniques | Lim Kinases - metabolism | Retinal Ganglion Cells - pathology | Intercellular Signaling Peptides and Proteins - metabolism | Transfection | RNA Interference | Time Factors | rho-Associated Kinases - metabolism | Cell Death | Female | Optic Nerve Injuries - pathology | Optic Nerve - physiopathology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Gene Transfer Techniques | Optic Nerve - enzymology | Retinal Ganglion Cells - enzymology | Signal Transduction | Optic Nerve Injuries - physiopathology | Cells, Cultured | Nerve Crush | Rats | rho-Associated Kinases - genetics | Nerve Regeneration | Nerve Tissue Proteins - metabolism | Neurites - pathology | Optic Nerve Injuries - enzymology | Optic Nerve Injuries - genetics | Animals | Axons - pathology | Lim Kinases - genetics | Genetic Vectors | Nerve Degeneration | Apoptosis | Original
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 10, pp. 4045 - 4050
Glaucoma, a major cause of blindness worldwide, is a neurodegenerative optic neuropathy in which vision loss is caused by loss of retinal ganglion cells... 
Glaucoma | Axons | Optic nerve | Eyes | Cell death | Neurons | Small interfering RNA | Mice | Physical trauma | Ganglia | Neuroprotection | Drug discovery | MAP3K12 | ELECTROPORATION | INHIBITOR SELECTIVITY | MULTIDISCIPLINARY SCIENCES | AXONAL REGENERATION | INJURY | DEGENERATION | drug discovery | PATHWAY | neuroprotection | IN-VIVO | NEURONS | MOUSE RETINA | GLAUCOMA | Glaucoma - etiology | Up-Regulation | Rats, Wistar | Humans | Cell Survival - genetics | Male | Retinal Ganglion Cells - pathology | Optic Nerve Diseases - pathology | Cell Death - genetics | RNA Interference | Optic Nerve Injuries - pathology | MAP Kinase Kinase Kinases - antagonists & inhibitors | Cell Survival - physiology | Disease Models, Animal | Optic Nerve Injuries - drug therapy | Cell Survival - drug effects | Retinal Ganglion Cells - enzymology | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Down-Regulation | Cells, Cultured | Rats | Glaucoma - drug therapy | Piperazines - pharmacology | Optic Nerve Injuries - enzymology | Animals | Cell Death - physiology | MAP Kinase Kinase Kinases - physiology | Protein Kinase Inhibitors - pharmacology | Optic Nerve Diseases - etiology | Retinal Ganglion Cells - drug effects | Genomics | Physiological aspects | Retina | Genetic aspects | Research | Health aspects | Genetic screening | Methods | Mitogen-activated protein kinases | Biological Sciences
Journal Article